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transfer of tumor
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  “transfer of tumor”译为未确定词的双语例句
     Through the signal transduction of intergrin receptor , FN enhanced the expression of MMP- 2 and accelerated the transfer of tumor.
     FN可通过其整合素受体的信号转导通路启动Hela细胞中MMP蛳2基因表达、降解ECM,从而促进肿瘤的浸润和转移。
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     Conclusion Hela cell can be induced by FN to adhere which causes a series of signal transductions in cell and expresses MMP 2 gene,accelerates the transfer of tumor.
     结论 FN可诱导Hela细胞黏附 ,进而引发一系列细胞内信号转导机制 ,启动MMP 2基因表达 ,促进肿瘤细胞的侵袭与转移
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     CONCLUSION: Desflurane had some effect on heamodynamics and that decreased level of plasma VEGF in patients with chest tumor, which showed some value for decreasing recurrence or transfer of tumor.
     随地氟醚吸入浓度的增加,血浆VEGF水平降低更明显。 结论:低浓度地氟醚吸入对胸部手术病人血液动力学无明显影响,能降低胸部肿瘤病人血浆VEGF水平,这对降低术后肿瘤复发或转移风险可能有一定价值。
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     The invasion and transfer of tumor cells is important biological behavior of tumor and is related with prognosis closely.
     肿瘤细胞的侵袭转移是肿瘤的重要生物行为,与患者的预后密切相关。
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     Objective To investigate the adhesion of Hela cell and the expression of MMP 2 induced by fibronectin(FN),to study the mechanism of the invasion and the transfer of tumor.
     目的 观察纤粘连蛋白 (fibronectin ,FN)诱导的Hela细胞的黏附及基质金属蛋白酶 (matrixmetalloprotein ases,MMPs)基因的表达 ,探讨恶性肿瘤细胞侵袭、转移发生的机制。
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  相似匹配句对
     TECHNOLOGY TRANSFER
     国内外科技成果转让项目
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     EECHNOLOGY TRANSFER
     山东省高新技术转移中心项目推荐
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     Transfer of the Prematures
     早产儿的转运
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     Transfer of Water Rights
     论水权转让制度
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     MicroRNA and tumor
     微小RNA与肿瘤
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  transfer of tumor
These studies indicate that transfer of tumor-reactive lymphocytes with or without the combined administration of IL-2 into syngeneic tumor-bearing animals can lead to the eradication of a disseminated neoplasia when certain conditions are met.
      
Notably, in these animals tumor-targeted IL2 therapy induced tumor regression only after adoptive transfer of tumor-conditioned splenocytes.
      
The adoptive transfer of tumor-draining lymph node (LN) T cells activated ex vivo with anti-CD3 and interleukin 2 (IL-2) mediates the regression of the poorly immunogenic murine melanoma D5.
      
transfer of tumor-sensitized T cells, resulted in rejection of the immunogenic fibrosarcoma, MCA/76-9, by syngeneic C57BL/6J (B6) mice.
      
Passive transfer of tumor immunity with spleen cells from guinea pigs cured of hepatocarcinoma by nonspecific immunotherapy
      
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It was indicated that there was reciprocity interaction between the life and transfer of tumor and the host.The regulation pathway can provide the host some condition to control the tumor.According to the view of TCM,tumor was a local disease in the body,in the treatment of tumor,TCM can control the tumor with regulation of the body status and the balance of body internal environment.It was treated by TCM with multi-pathway and multi-stratification to promote the veversion of the...

It was indicated that there was reciprocity interaction between the life and transfer of tumor and the host.The regulation pathway can provide the host some condition to control the tumor.According to the view of TCM,tumor was a local disease in the body,in the treatment of tumor,TCM can control the tumor with regulation of the body status and the balance of body internal environment.It was treated by TCM with multi-pathway and multi-stratification to promote the veversion of the precancerous lesion of stomach mucosa stomach mucosa reverse.The method of the TCM can be conclude as nourishing qi and spleen nourishing qi and yin to promote the blood circulation and detoxify release the toxity evil,dispersing the depressed liver-qi and regulating the stomach qi to detoxify,clearing the wetness-heat to detoxify.The symptom can be improved with syndrome differentiation and TCM treatment.

有研究资料表明 ,肿瘤的生长转移过程和宿主之间有充分的相互作用。这种完整的调控通路的存在 ,可为宿主对肿瘤控制功能的重新建立提供条件。中医认为 ,肿瘤是全身疾病的局部反应 ,在治疗上最突出的特点是通过调整患者的机体状况 ,全面调节机体内环境的平衡 ,调动内在抗病能力而达到治疗目的。中医药对胃癌前病变的治疗 ,即是通过多途径、多层次的综合调节作用 ,以促进胃粘膜癌前病变的逆转。归纳其治法为 :益气健脾 ,活血解毒法 ;益气养阴 ,活血解毒法 ;疏肝和胃 ,化瘀解毒法 ;清化湿热 ,化瘀解毒法。辨证施治 ,可使临床症状得到改善 ,不同程度地使肠化或异型增生减弱或消退

OBSJECTIVE: To investigate the effect of low concentration of desflurane on heamodynamics and plasma vascular endothelial growth factor (VEGF) in patients with chest tumor. METHODS: Twenty-eight ASA grade Ⅰ-Ⅱ patients with chest tumor, scheduled for elective chest surgery, were studied. Heamodynamics and the level of plasma VEGF were observed before anesthesia, after intubation, inhalation of desflurane at 0.5, I.OMAC, respectively. RESULTS: Compared with those before anesthesia, SBP, DBF, MAP were decreased...

OBSJECTIVE: To investigate the effect of low concentration of desflurane on heamodynamics and plasma vascular endothelial growth factor (VEGF) in patients with chest tumor. METHODS: Twenty-eight ASA grade Ⅰ-Ⅱ patients with chest tumor, scheduled for elective chest surgery, were studied. Heamodynamics and the level of plasma VEGF were observed before anesthesia, after intubation, inhalation of desflurane at 0.5, I.OMAC, respectively. RESULTS: Compared with those before anesthesia, SBP, DBF, MAP were decreased and HR was increased during intubation. Although SBP, DBF, MAP were decreased and HR were increased than those before anesthesia when inhalation of desflurane at 0.5, I.OMAC, blood pressure and HR had not changes compared with intubation. The level of plasma VEGF in patients with chest tumor was higher than that in normal cases. The level of plasma VEGF was decreased after inducing and progressively decreased after inhalation of desflurane. CONCLUSION: Desflurane had some effect on heamodynamics and that decreased level of plasma VEGF in patients with chest tumor, which showed some value for decreasing recurrence or transfer of tumor.

目的:观察低浓度地氟醚吸入麻醉对胸部肿瘤病人手术麻醉时血液动力学和血浆血管内皮细胞生长因子(VEGF)水平的影响。方法:选择胸部肿瘤手术病人28例,ASAⅠ~Ⅱ级。麻醉诱导后气管插管,观察0.5,1.0肺泡气最低有效浓度(MAC)地氟醚对胸部肿瘤手术病人血液动力学和血浆VEGF水平的影响。结果:麻醉诱导后气管插管时,病人SBP,DBP和MAP均呈显著下降,HR显著增快(均P<0.01)。分别吸入0.5,1.0MAC地氟醚时,虽然SBP,DBP,MAP较麻醉前明显下降(P<0.05),但与麻醉诱导后相比,血压和HR无明显变化。肿瘤患者血浆VEGF水平明显高于正常水平;麻醉诱导后,血浆VEGF水平明显降低;吸入地氟醚后,血浆VEGF水平进一步降低;随地氟醚吸入浓度的增加,血浆VEGF水平降低更明显。结论:低浓度地氟醚吸入对胸部手术病人血液动力学无明显影响,能降低胸部肿瘤病人血浆VEGF水平,这对降低术后肿瘤复发或转移风险可能有一定价值。

Objective:To study the role of VEGF during the course of development of astrocytoma and investigate the correlation between them.Methods:Experiment was taken by the method of“S-P”Immunostaining to VEGF of the different malignant degree tumor and the normal brain tissue and FⅧRAg.At the same time,under the electron microscope to find the correlative of VEGF and the malignant degree in human astrocytoma.Results:There are different degree cerebal edema in the tumors of different malignant degree,the more malignant,the...

Objective:To study the role of VEGF during the course of development of astrocytoma and investigate the correlation between them.Methods:Experiment was taken by the method of“S-P”Immunostaining to VEGF of the different malignant degree tumor and the normal brain tissue and FⅧRAg.At the same time,under the electron microscope to find the correlative of VEGF and the malignant degree in human astrocytoma.Results:There are different degree cerebal edema in the tumors of different malignant degree,the more malignant,the more serious edema(F=15.95,P<0.05).There is no expression in the normal brain tissue,blood vessel and the nervous gliocyte,with the development of tumor malignant degree,expression of VEGF is progressively increasing.Grade Ⅲ and grade Ⅳ are the most significant(F=12.08,P<0.05).Microvasculature density has significant difference between the different malignant degree tumors(F=16.65,P<0.05).Observation under electron microscope:There is no significant cell change of structure in astrocytoma of grade Ⅰ and gliocyte neclie of grade Ⅱ,but we can observe obviously change by electron microscopic at the grade Ⅲ,such as vascular endothelial cell,mitochondria,basement membrane defect and vascular wall.Conclusions:VEGF promot the growth and transfer of tumor mainly through stimulating angiogenesis and increasing vescular permeability,and lead to tumor surroundings edema.The malignant degree of brain astrocytoma is positive correlation with VEGF.

目的 :研究血管内皮生长因子 (VEGF)在星形细胞胶质瘤生长过程中起的作用 ,探讨它们之间的相关性。方法 :实验采用免疫组化S -P法对不同恶性程度星形细胞瘤及正常脑组织中VEGF和第八因子相关抗原 (FⅧRAg)进行检测 ,同时行电镜观察 ,找出星形细胞瘤恶性程度与VEGF分泌程度的相关性。结果 :(1 )不同恶性程度肿瘤之间术前均有不同程度的脑水肿 ,肿瘤级别高脑水肿程度重 (F =1 5 95 ,P <0 0 5)。(2 )正常脑组织、血管及神经胶质细胞均无VEGF表达 ,随肿瘤恶性程度增高VEGF表达逐渐增强 ,Ⅲ、Ⅳ级最为显著 (F =1 2 0 8,P <0 0 5)。 (3)不同恶性程度肿瘤微血管密度均有差异 (F =1 6 65 ,P <0 0 5)。 (4)电镜下见星形细胞瘤Ⅰ、Ⅱ级胶质细胞核异型性不大 ,血管内皮细胞均匀 ;Ⅲ级以上肿瘤血管内皮细胞间隙扩张、形状不规则、核大 ,有的变性坏死、线粒体肿胀空泡化、基膜肿胀疏松、部分中断缺损 ,血管壁厚薄不均 ,血管周围间质水肿明显。结论 :VEGF通过刺激肿瘤血管形成、增加血管通透性来促进肿瘤的生长、转移 ,引起肿瘤周围脑水肿 ;人脑...

目的 :研究血管内皮生长因子 (VEGF)在星形细胞胶质瘤生长过程中起的作用 ,探讨它们之间的相关性。方法 :实验采用免疫组化S -P法对不同恶性程度星形细胞瘤及正常脑组织中VEGF和第八因子相关抗原 (FⅧRAg)进行检测 ,同时行电镜观察 ,找出星形细胞瘤恶性程度与VEGF分泌程度的相关性。结果 :(1 )不同恶性程度肿瘤之间术前均有不同程度的脑水肿 ,肿瘤级别高脑水肿程度重 (F =1 5 95 ,P <0 0 5)。(2 )正常脑组织、血管及神经胶质细胞均无VEGF表达 ,随肿瘤恶性程度增高VEGF表达逐渐增强 ,Ⅲ、Ⅳ级最为显著 (F =1 2 0 8,P <0 0 5)。 (3)不同恶性程度肿瘤微血管密度均有差异 (F =1 6 65 ,P <0 0 5)。 (4)电镜下见星形细胞瘤Ⅰ、Ⅱ级胶质细胞核异型性不大 ,血管内皮细胞均匀 ;Ⅲ级以上肿瘤血管内皮细胞间隙扩张、形状不规则、核大 ,有的变性坏死、线粒体肿胀空泡化、基膜肿胀疏松、部分中断缺损 ,血管壁厚薄不均 ,血管周围间质水肿明显。结论 :VEGF通过刺激肿瘤血管形成、增加血管通透性来促进肿瘤的生长、转移 ,引起肿瘤周围脑水肿 ;人脑星形细胞瘤的恶性程度与VEGF呈正相关。

 
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