助手标题  
全文文献 工具书 数字 学术定义 翻译助手 学术趋势 更多
查询帮助
意见反馈
   intensity of hypoxia 的翻译结果: 查询用时:0.01秒
图标索引 在分类学科中查询
所有学科
肿瘤学
更多类别查询

图标索引 历史查询
 

intensity of hypoxia
相关语句
  缺氧程度
     Effects of FGF2 and VEGF on the structural vessels,perfused vessels and intensity of hypoxia in esophageal carcinoma
     FGF2和VEGF对食管鳞癌、腺癌荷瘤裸鼠肿瘤组织结构血管、灌注血管及缺氧程度的影响分析
短句来源
  “intensity of hypoxia”译为未确定词的双语例句
     The results showed that the intensity of hypoxia was positively conincided with the production of polymorphonuclear (PMNs)O - 2 and the LPO,but no significant increase at the same altitude by exposing for different times.
     结果表明,不同程度的缺氧能明显地增加多核粒细胞(PMNs)O-.2的产量,导致机体的脂质过氧化(LPO)反应显著增强,其代谢产物丙二醛(MDA)含量同步增高,但同一高度,缺氧时间长短作用不大。
短句来源
     RESULTS: Fluorescent intensity in hypoxia plus CSDP group was significantly lower (1 217 78±312 07) than that of hypoxia group (1 509±508 48), and the Fluorescent intensity of hypoxia/reoxygenation plus CSDP group was also markedly lower (1 567.91±577 61) than that of hypoxia/reoxygenation group (1 617.60±477.53).
     结果 :缺氧加丹参保护组 ,细胞内钙离子荧光强度 (12 17 78± 312 0 7)明显低于单纯缺氧组 (15 0 9 43± 5 0 8 5 8) ; 缺氧复氧加丹参组 ,细胞内钙离子荧光强度为 (15 6 7 91± 5 77 6 1) ,亦较缺氧再给氧组 (16 17 6± 477 5 3)低。
短句来源
  相似匹配句对
     The intensity of stresses(?)
     并计算了应力强度(?)
短句来源
     (2) respiratory intensity;
     (2 )呼吸强度 ;
短句来源
     FUZZY EARTHQUAKE INTENSITY
     模糊烈度
短句来源
     form and intensity of the change;
     顾客期望价值转变的形式和强度;
短句来源
     ⑤Hypoxia;
     5低氧组 ;
短句来源
查询“intensity of hypoxia”译词为用户自定义的双语例句

    我想查看译文中含有:的双语例句
例句
为了更好的帮助您理解掌握查询词或其译词在地道英语中的实际用法,我们为您准备了出自英文原文的大量英语例句,供您参考。
  intensity of hypoxia
In moderate hypoxia, isolation of the cardiovasomotor center from nonhypoxic afferention provides the compliance of myocardial hyperfunction to the intensity of hypoxia and limits heart participation in different forms of activity of the organism.
      


The influence of acute hypoxia on lipid peroxidation(LPO)of rats at different simulated altitudes (0,4000,6000m)and at the same simulated altitude (6000m) for staying different times(8,12,24h) were studied.The results showed that the intensity of hypoxia was positively conincided with the production of polymorphonuclear (PMNs)O - 2 and the LPO,but no significant increase at the same altitude by exposing for different times.The LPO ...

The influence of acute hypoxia on lipid peroxidation(LPO)of rats at different simulated altitudes (0,4000,6000m)and at the same simulated altitude (6000m) for staying different times(8,12,24h) were studied.The results showed that the intensity of hypoxia was positively conincided with the production of polymorphonuclear (PMNs)O - 2 and the LPO,but no significant increase at the same altitude by exposing for different times.The LPO wat reduced markedly by 30mg/d vitamin E supplementation,but the PMNs O . 2 did not significantly decrease.It is most likely that vitamin E exerts the anti LPO effect by only blocking the free radical reaction.

从4000~6000m的不同模拟高度以及同一高度不同停留时间,观察缺氧对大鼠脂质过氧化的影响及维生素E干预的效应。结果表明,不同程度的缺氧能明显地增加多核粒细胞(PMNs)O-.2的产量,导致机体的脂质过氧化(LPO)反应显著增强,其代谢产物丙二醛(MDA)含量同步增高,但同一高度,缺氧时间长短作用不大。补充维生素E30mg/d,能有效地减轻LPO反应,使动物血清MDA含量显著下降,但补充VE对PMNsO-.2产量无明显影响,说明VE抗LPO很可能是阻断自由基反应,而不是降低自由基产量。

AIM: To examine the effect of compound salvae-dropping-pill (CSDP) on intracellular free calcium in cultured rat myocardial cells subjected to hypoxia and reoxygenation.METHODS: The Fluo- 3/AM was applied to probe intracellular calcium concentration and the fluorescent intensity was detected using laser confocal microscopy technique.RESULTS: Fluorescent intensity in hypoxia plus CSDP group was significantly lower...

AIM: To examine the effect of compound salvae-dropping-pill (CSDP) on intracellular free calcium in cultured rat myocardial cells subjected to hypoxia and reoxygenation.METHODS: The Fluo- 3/AM was applied to probe intracellular calcium concentration and the fluorescent intensity was detected using laser confocal microscopy technique.RESULTS: Fluorescent intensity in hypoxia plus CSDP group was significantly lower (1 217 78±312 07) than that of hypoxia group (1 509±508 48), and the Fluorescent intensity of hypoxia/reoxygenation plus CSDP group was also markedly lower (1 567.91±577 61) than that of hypoxia/reoxygenation group (1 617.60±477.53).CONCLUSION: The cultured rat myocardial cells could be effectively protected by administration of CSDP in case of hypoxia and reoxygenation through decreasing the intracellular calcium concentration. [

目的 :探讨复方中药丹参滴丸对缺氧 /复氧心肌细胞的保护作用。方法 :培养的乳鼠心肌细胞 ,用钙探针Fluo - 3/AM染色 ,利用激光扫描共聚焦显微镜观察心肌细胞缺氧 /复氧及在复方丹参滴丸保护作用下心肌细胞内钙离子荧光强度变化。结果 :缺氧加丹参保护组 ,细胞内钙离子荧光强度 (12 17 78± 312 0 7)明显低于单纯缺氧组 (15 0 9 43± 5 0 8 5 8) ;缺氧复氧加丹参组 ,细胞内钙离子荧光强度为 (15 6 7 91± 5 77 6 1) ,亦较缺氧再给氧组 (16 17 6± 477 5 3)低。结论 :复方丹参滴丸对缺氧心肌细胞有明显保护作用。

Objective To determine the effects of FGF2 and VEGF on density of structural and perfused vessels and to investigate the mechanism of angiogenesis in esophageal carcinoma(EC) . Methods Three cell lines were used in the experiment, TE-1 was esophageal squamous cell carcinoma, SEG-1 and BIC-1 were Barrett s associated adenocarcinoma. FGF2 and VEGF mRNA were determined by multiple-probe RNA protection assay and quantified by densitoraetry; structural vessels, perfused vessels and intensity of hypoxia were...

Objective To determine the effects of FGF2 and VEGF on density of structural and perfused vessels and to investigate the mechanism of angiogenesis in esophageal carcinoma(EC) . Methods Three cell lines were used in the experiment, TE-1 was esophageal squamous cell carcinoma, SEG-1 and BIC-1 were Barrett s associated adenocarcinoma. FGF2 and VEGF mRNA were determined by multiple-probe RNA protection assay and quantified by densitoraetry; structural vessels, perfused vessels and intensity of hypoxia were detected by immunohistochemical staining (CD31) and immunofluorescence (DIOC7,EF5). Results (1)Different concentration of FGF2 (lOng,100ng) didn't affect the proliferation and growth of three EC cell lines in vitro. (2)The basal levels of FGF2 and VEGF mRNA were 4.9, 57.57 in TE-1 ; 49, 53.62 in SEG-1 ; no FGF2 mRNA were observed in BIC-1 ,its VEGF mRNA was 17.04. (3) In three nude mice model, the density of structural vessels (CD31) and perfused vessels (DIOC7) in VEGF-treated group is significantly higher than that in control group (P< 0.05) . The density of structural vessels (CD31) and perfused vessels (DIOC7) in FGF2-trealed group significantly increased only in SEG-1 (P<0.05) .(4)In TE-1 and BIC-1 model, intensity of EF5/Cy3 staining was significantly decreased in VEGF group. In SEG-1 model,no significant difference was found between control and VEGF group, there were no significant difference in FGF2 group comparing with control group in all three models. Conclusion (1)FGF2 didn't affect the proliferation and growth of esophageal carcinoma cell lines in vitro.(2)FGF2 expression were quite different in different histological types, even in same histological types of esophageal carcinoma. (3)The effecl of FGF2 on tumor angiogenesis may be closely related with its endogenous FGF2 mRNA level. (4) In SEG-1 model, FGF2 increased tumor growth mainly by promoting angiogenesis. However, FGF2 didn' t affect both proliferation of tumor cell and angiogenesis in TE-1 and BIC-1 model, which imply FGF2 wasn't the key angiogenic regulator in these two cell lines.(5) VEGF can significantly increase structural vessels and perfused vessels, decrease hypoxia in tumor tissue. VEGF is a key factor to regulate angiogenesis in EC.(6)To detect the structural vessels perfused vessels and intensity of hypoxia in the same area within tumor tissue is a good method to evaluate vascular density and function.

目的 探索FCG2及VEGF对食管癌结构血管和灌注血管的影响,阐明与食管癌快速生长、转移密切相关的血管生成调节机制。方法 本实验采用人食管癌细胞系TE-1(鳞状细胞癌),SEG-1和BIC-1(腺癌)及其荷瘤动物模型。利用体外细胞培养技术观察重组人FGF2、VEGF对三种细胞系生长增殖的影响,采用多探针RNA酶保护性分析方法检测FGF2和VEGFmRNA水平,采用免疫组织化学和免疫荧光方法观察食管癌组织结构血管、灌注血管密度及瘤内缺氧程度的影响。结果 ①体外细胞培养实验中,不同浓度FGF2(10ng,100ng)对人食管癌细胞系TE-1,SEG-1和BIG-1的增殖速度和生长曲线无影响。②FGF2和VEGRmRNA基础水平的定量检测显示食管鳞癌细胞系TE-1的FGF2为4.92,VEGF为57.57。食管腺癌细胞系SEG-1的FGF2、VEGF分别为49和53.62;BIC-1不表达FGF2,VEGF为17.04。③在TE-1、SEG-1、BIC-1三种食管癌荷瘤裸鼠模型中,FGF2、VFGF对食管癌结构血管(CD31)以及灌注血管(DIOC7)平均距离的影响分析显示:VEGF处理组结构血管和灌注血管密度较对...

目的 探索FCG2及VEGF对食管癌结构血管和灌注血管的影响,阐明与食管癌快速生长、转移密切相关的血管生成调节机制。方法 本实验采用人食管癌细胞系TE-1(鳞状细胞癌),SEG-1和BIC-1(腺癌)及其荷瘤动物模型。利用体外细胞培养技术观察重组人FGF2、VEGF对三种细胞系生长增殖的影响,采用多探针RNA酶保护性分析方法检测FGF2和VEGFmRNA水平,采用免疫组织化学和免疫荧光方法观察食管癌组织结构血管、灌注血管密度及瘤内缺氧程度的影响。结果 ①体外细胞培养实验中,不同浓度FGF2(10ng,100ng)对人食管癌细胞系TE-1,SEG-1和BIG-1的增殖速度和生长曲线无影响。②FGF2和VEGRmRNA基础水平的定量检测显示食管鳞癌细胞系TE-1的FGF2为4.92,VEGF为57.57。食管腺癌细胞系SEG-1的FGF2、VEGF分别为49和53.62;BIC-1不表达FGF2,VEGF为17.04。③在TE-1、SEG-1、BIC-1三种食管癌荷瘤裸鼠模型中,FGF2、VFGF对食管癌结构血管(CD31)以及灌注血管(DIOC7)平均距离的影响分析显示:VEGF处理组结构血管和灌注血管密度较对照组显著增加(P<0.05),FGF2处理组结构血管和灌注血管密度在SEG-1模型中显著增加(P<0.05)。④在TE-1和BIC-1模型中,FGF2处理组EF5/Cy3染色的平均强度与对照组无明显差别,VEGF处理组EF5/Cy3染色的平均强度明显降低。在SEG

 
图标索引 相关查询

 


 
CNKI小工具
在英文学术搜索中查有关intensity of hypoxia的内容
在知识搜索中查有关intensity of hypoxia的内容
在数字搜索中查有关intensity of hypoxia的内容
在概念知识元中查有关intensity of hypoxia的内容
在学术趋势中查有关intensity of hypoxia的内容
 
 

CNKI主页设CNKI翻译助手为主页 | 收藏CNKI翻译助手 | 广告服务 | 英文学术搜索
版权图标  2008 CNKI-中国知网
京ICP证040431号 互联网出版许可证 新出网证(京)字008号
北京市公安局海淀分局 备案号:110 1081725
版权图标 2008中国知网(cnki) 中国学术期刊(光盘版)电子杂志社