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noradrenergic nerve
相关语句
  去甲肾上腺素能神经
     The effect of the central noradrenergic nerve on the intracerebral vessels and local cerebral circulation was discussed.
     讨论了中枢去甲肾上腺素能神经对脑实质内血管以及局部脑循环的作用。
短句来源
  “noradrenergic nerve”译为未确定词的双语例句
     The construction pattern and the distribution of noradrenergic nerve to the testis and epididymides of rat Were studied using modified technique of glyc::ylic acid-induced fluorescence for the demonstration of the noradrenergic terminals.
     应用改良的乙醛酸诱发儿茶酚胺(CA)的荧光组织化学方法,证明去甲肾上腺素(NA)能神经终末在大鼠睾丸和附睾内的分布及其构筑形式。
短句来源
     The experimental results showed that the interstitial tissue and the wall of blood vessels of testis and epididymides received noradrenergic nerve innervation.
     实验发现,大鼠皋丸间质结缔组织及血管壁接受NA能神经支配:此种带膨体荧光神经纤维发出分枝,伸进曲细精管壁;
短句来源
     The distribution of central noradrenergic nerve in the intracerebral blood vessels of ten Wistar rats was studied with ABC immunoperoxidase technique, employing Tyrosine-hydroxylase (TH) as label.
     本实验应用ABC免疫过氧化物酶法,以酪氨酸羟化酶(TH)作为标记物,观察了10只Wistar大鼠脑实质内血管的中枢去甲肾上腺素能神经分布。
短句来源
     Specificl lesion of the nucleus locus coeruleus and the noradrenergic nerve terminals by 6-hydroxydopamine affected the doseresponse curve of the xylazine-induced inhibition to rat excitatory activity in a different manner compared with that of xylazine-induced inhibition of rat locomotor activity.
     用6-OH-DA特异性毁损LC或中枢NE能神经末梢,对赛拉嗪抑制大鼠EA作用的影响不同于其影响赛拉嗪抑制LA的作用。
短句来源
     The possible mechanism are regulating of central cholinergic nerve system and noradrenergic nerve system.
     协调中枢胆碱能系统和肾上腺素能系统的功能是其可能的作用机制。
短句来源
  相似匹配句对
     Nes-B in nerve;
     神经为Nes—B;
短句来源
     The possible mechanism are regulating of central cholinergic nerve system and noradrenergic nerve system.
     协调中枢胆碱能系统和肾上腺素能系统的功能是其可能的作用机制。
短句来源
     The effect of the central noradrenergic nerve on the intracerebral vessels and local cerebral circulation was discussed.
     讨论了中枢去甲肾上腺素能神经对脑实质内血管以及局部脑循环的作用。
短句来源
     THE MORPHOLOGY OF THE ACCESSORY NERVE
     国人副神经的形态学研究
短句来源
     THE ORIGIN OF NORADRENERGIC FIBRES IN THE AMYGDALA
     杏仁核内去甲肾上腺素能神经纤维的传入联系
短句来源
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  noradrenergic nerve
DSP-4 is a neurotoxin highly selective for the noradrenergic nerve terminals originating from the locus coeruleus.
      
Trypanosoma cruzi infection in rats induced early lesion of the heart noradrenergic nerve terminals by a complement-independent
      
This investigation tested the hypothesis that oxidative deamination of 5HT in the pineal gland occurs primarily in cellular compartments other than the pinealocyte (i.e., noradrenergic nerve terminals and glia).
      
The data argue against a primary action of lithium on the noradrenergic nerve.
      
It was found that lesioning of the noradrenergic nerve terminals abolished the decrease inβ-adrenoceptors produced by desipramine, mianserin and zimeldine and partially antagonized that of theβ-adrenoceptor agonist clenbuterol.
      
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There are receptors located on the noradrenergic nerve terminals that mcdulate therelease of noradrenaline(NA).Presynaptic inhibitory α-adrenoceptors are involved inthe regulation of the release of NA via a negative feedback mechanism mediated bythe neurone's own transmitter.Presynaptic facilitatcry β-adrenoceptors can be actedupon by circulating adrenaline(ADR)to enhance noradrenergic transmission,ADR canalso be taken up and stored in the noradrenergic nerve terminals and being releasedduring...

There are receptors located on the noradrenergic nerve terminals that mcdulate therelease of noradrenaline(NA).Presynaptic inhibitory α-adrenoceptors are involved inthe regulation of the release of NA via a negative feedback mechanism mediated bythe neurone's own transmitter.Presynaptic facilitatcry β-adrenoceptors can be actedupon by circulating adrenaline(ADR)to enhance noradrenergic transmission,ADR canalso be taken up and stored in the noradrenergic nerve terminals and being releasedduring nerve stimulation thus sustaining a long action.Thereby presynaptic facilitatoryβ-adrenoceptors might be important for the initiation and maintenance of certain formof essential hypertension.The antihypertensive activity of β-adrenoceptor antagonistsmay be due to blockage of presynaptic facilitatory β-adrenoceptors thus preventingADR from activating the facilitatory mechanism in peripheral noradrenergic nerves.The above view has been supported both in vitro and in vivo experiments.

本文系 Rand 教授在本院所作的学术报告,经整理成文。Rand 教授在研究突触传递的基础上提出有关与高血压产生的关系的新见解,具有创见和启发性。

Experiments were performed on 105 anesthetized cats. The drugs were injected into the fourth ventricule. The hypotensive effects of both physostigmine and clonidine were blocked by atropine and yohimbine. The results of peripheral experiments suggest that atropine blocks the hypotensive effect of clonidine not through central α-receptor blockade mechanism. After the central noradrenergic nerve terminal was destroyed by 6-OHDA, the hypotensive effect of clonidine was no longer blocked by atropine. This...

Experiments were performed on 105 anesthetized cats. The drugs were injected into the fourth ventricule. The hypotensive effects of both physostigmine and clonidine were blocked by atropine and yohimbine. The results of peripheral experiments suggest that atropine blocks the hypotensive effect of clonidine not through central α-receptor blockade mechanism. After the central noradrenergic nerve terminal was destroyed by 6-OHDA, the hypotensive effect of clonidine was no longer blocked by atropine. This suggests that an intact noradrenergic nerve in lower brain stem is essential for the blocking effect of atropine. Clonidine could decrease the Ach transmitter release in the overflow of brain ventricular infusion, and the hypotensive effect of clonidine could be blocked by hemicholine.Cocaine did not affect the hypotensive effect of clonidine. Methoxamine did not induce hypotensive effect. It appears that clonidine produces hypotensive effect through activating the central post-synaptic α_2-receptor.It appears that the cholinergic and the noradrenergic nerves synapse on the same cardiovascular neurone in the cat medulla oblongata. When the trigger receptors, Mreceptor and α_-receptor, are activated, both receptors may induce the same inhibitory effect on blood pressure regulation, and clonidine produces hypotensive effect by activating post-synaptic α_2-receptor on this pathway. There seems to be no direct connexion between the hypotensive effect of clonidine and the central cholinergic nerve.

从猫第四脑室给药。毒扁豆碱降压作用可被阿托品及育亨宾阻断;可乐定降压作用可被育亨宾、密胆碱及阿托品阻断,不被可卡因阻断,破坏中枢去甲肾上腺素能末梢后,阿托品的阻断作用消失;可乐定减少脑室灌流液Ach释出量。提示猫延髓胆碱能和去甲肾上腺素能神经在心血管调节功能上相互影响,它们似有可能同在一心血管神经原形成突触联系共同对血压行抑制性调节;可乐定可能通过激动通路上突触后α_2受体产生降压作用。

To define the possible role of central noradrenergic nerve in the regulation of electrical and mechanical activities of stomach, the experiment was performed in rats anesthetized with urethane. 6-hydroxydopamine (6-OHDA) was injected into Ala cinerea. The results obtained are as follows: In the control group, the amplitude of the slow wave and intragastric pressure were decreased to 42.31+9.79% and 34.59±10.20% respectively by stimulation of locus coeruleus. However, in the group treated with 6-OHDA the...

To define the possible role of central noradrenergic nerve in the regulation of electrical and mechanical activities of stomach, the experiment was performed in rats anesthetized with urethane. 6-hydroxydopamine (6-OHDA) was injected into Ala cinerea. The results obtained are as follows: In the control group, the amplitude of the slow wave and intragastric pressure were decreased to 42.31+9.79% and 34.59±10.20% respectively by stimulation of locus coeruleus. However, in the group treated with 6-OHDA the amplitude of slow wave and intragastric pressure were decreased to 84.14+10.13% and 62.86±7.19% respectively. The difference between the two groups was statistically highly significant(p<0.01). The result suggest that the central noradrenergic inhibitory mechanism is involved in the regulation of gastric electrical activity and gastric motility.

电刺激蓝斑核可抑制胃电和胃运动。我们先以6-羟基多巴胺(6-OHDA)损毁大鼠(已用乌拉坦麻醉)的灰部深翼肾上腺素能神经,然后观察了这种损坏对上述抑制效应的影响。结果表明,在对照组,电刺激蓝斑核导致胃电慢波的振幅和胃内压分别下降到对照值的42.31±9.79%和34.59±10.20%。但是,在注射6-OHDA组,刺激蓝斑核后胃电慢波的振幅和胃内压分别下降到对照值的84.14±10.13%和62.86±7.19%,注药组的抑制效应明显弱于对照组,(P<0.01)。本文结果提示:灰翼区肾上腺素能神经参与由刺激蓝斑核区引起的胃电和胃运动的抑制机制。

 
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