Results 176 patients with various liver disease were determined by using blank absorbance A:340nm<0.5, precision (normal level x 4.2U/L) CV:4.68%, line 0-100U/L. Among them, positive rate of acute viral hepatitis was 100%, and liver necrosis due to carbon monoxide poisoning 100%.
Results: Serum IFN-αand IFN-γ level of acute viral hepatitis patients were significantly higher than those in the normal control group ( q=40.96 P<0.01, q= 44.65 P<0.01), and there were statistical difference between the two groups.
Results The level of CHE,ALB and PTA decreased gradually in acute hepatitis,chronic hepatitis and cirrhosis,and was significantly compared with the control group(P<0.001).
To investigate the possible immunopathogenic role of interleukin-6 (IL-6)and tu-mor necrosis factor-α in the liver damage,concentrations of IL-6 and TNF-αin the serum and PBMC were assayed in 15 normal subjects,18 patients with acute hepatitis(AH),37 with chronic liver dis-ease(CLD),and 20 with liver cirrhosis(LC).
[Results] Among the 756 acute virus hepatitis, the rate of HA was 34.66% , HB 37.04% , HC 6.48% , HD 2.25% , HE 25.13% including single infection, mixed infection and overlapping infection.
The occurrences , underreporting and epidemiologic features of aculc viral hepatitis in three field - type units were studied from January to December , 1988. The annual incidence was 1.35 ‰ in the army ,in which hepatitis A covered 0.71 ‰, hepatitis B 0.34 ‰, and hepatitis non - A , non - B 0.07 ‰.
Results:The abnormal rates of ECG in the patients with hepatitis A, B and E were 10/30 , 6/30 and 7/30, respectively. The abnormal rates of myocardial enzymes were 8/30, 3/30 and 3/30, respectively.
R~ The plasma level of TFPI4Ag inGroup I was higher (P<0.05) and itS aCtivation was also increase dramatically (p<0.ol), the level of AT--ill:A was lower in patientS with chronic hepatitis B (P<0.05) and no difference was found in other indicesbetween the tWo groupe.
There were 49 "index cases" of hepatitis-like jaundice: 38 cases of acute viral hepatitis, 5 of chronic agressive hepatitis and 6 of active cirrhosis.
A long-term follow-up of the HBAg carriers showed that three of these subjects progressed to acute viral hepatitis.
The seroepidemiological pattern of acute viral hepatitis
The serum levels of IgA, IgG and IgM were determined by the single radial immunodiffusion method in 86 patients with acute viral hepatitis serologically differentiated by radioimmunoassays.
Acute viral hepatitis A, B and Non-A, Non-B in Stockholm in the 1950s and 1970s: A comparison
Most likely, existing acute hepatitis was of autoimmune origin and emerged from an immune rebound phenomenon after immunosuppressive therapy.
Acute hepatitis in a patient treated with carbamazepine
In each of 23 families in which two or more cases of acute hepatitis-like jaundice (index cases) occurred, all family members were studied to evaluate HBAg clustering and the incidence of asymptomatic liver disease.
Among 466 hospitalized patients with serologically verified acute hepatitis B, 440 individuals (94.4%) could be followed up until normalization of liver function had occured, or for at least one year.
Two hundred and twenty sera from asymptomatic carriers and 129 sera from acute hepatitis patients were subtyped.
They both demonstrate remarkable similarity with acute virus hepatitis in humans.
Endogenous and postheparin monoglyceride hydrolase in plasma of patients with acute virus hepatitis
Endogenous and postheparin monoglyceride hydrolase (MGH) was studied in 9 patients with acute virus hepatitis and in 25 normal subjects with the use of a new spectrophotometric method.
Intravenous glucose tolerance tests were performed in 10 patients with acute virus hepatitis.
The causes leading to coma were acute virus hepatitis A in two cases, hepatitis B in three cases, and Amanita intoxication in one case.
In acute virus hepatitis, HBsAg positive by using reveres passive hemagglutination assay or radioimmunoassay methods was considered as type B; and a 4-fold increase in the Anti-HA titers by complement fixation and immunoadherence hemagglutination assay methods was considered as type A acute hepatitis: Type A tended to occur in younger people with a more acute onset and fever, liver functions usualy recovered more quickly; there was no tendency to be chronic and the prognosis was usually good. In acute hepatitis...
In acute virus hepatitis, HBsAg positive by using reveres passive hemagglutination assay or radioimmunoassay methods was considered as type B; and a 4-fold increase in the Anti-HA titers by complement fixation and immunoadherence hemagglutination assay methods was considered as type A acute hepatitis: Type A tended to occur in younger people with a more acute onset and fever, liver functions usualy recovered more quickly; there was no tendency to be chronic and the prognosis was usually good. In acute hepatitis Type B, the onset was insidious generally there was no fever, liver functions recovered rather slow, and the prognosis was poor.
With follow-up at regular intervals,altogether 622 ECG examinations were donein 250 children with acute viral hepatitis.ECG was made first time within 10 daysof the disease.59.6% of the ECG were found abnormal,manifested by changes of Twave and cardiac rhythm.69.1% of the abnormal ECG were detected in the first 10days and 97% of them reverted to normal within 40 days of the disease.The abnormalECG was not related with the presence or the depth of jaundice,level of SGPT,norwith the types(B or Non-B)of hepatitis,but...
With follow-up at regular intervals,altogether 622 ECG examinations were donein 250 children with acute viral hepatitis.ECG was made first time within 10 daysof the disease.59.6% of the ECG were found abnormal,manifested by changes of Twave and cardiac rhythm.69.1% of the abnormal ECG were detected in the first 10days and 97% of them reverted to normal within 40 days of the disease.The abnormalECG was not related with the presence or the depth of jaundice,level of SGPT,norwith the types(B or Non-B)of hepatitis,but there was significant correlation withage.For patients below 3 years of age,88.9% showed abnormal ECG while abnormalECG was found only in 43.1% in the 6-13 years age group.
本文对250例小儿急性病毒性肝炎进行了622人次的心电图动态观察。发现59.6%的病例有异常心电图,主要为 T 波改变和心律失常。异常心电图的69.1%出现在病程头10天以内其中97%于病程40天内恢复正常。心电图异常与黄疸有无或深浅,SGPT 高低,和乙型与非乙型之间无明显差异,但与年龄大小关系较大,3岁以下者88.9%有心电图异常,6～13岁者只43.1%,差别非常显著。
We have examined the chromosomes of peripheral blood lymphocytes in 26 normal individuals and 53 patients of various viral hepatitis. We found that chromosomal aberrations appeared in both acute and chronic viral hepatitis. These changes often appeare in the acute stage of acute viral hepatitis, and disappeare in the recover stage. At the same time, we have also observed the lymphocyte transformations and found that the chromosomal aberration correlated to low lymphocyte transformation. The cause and the significance...
We have examined the chromosomes of peripheral blood lymphocytes in 26 normal individuals and 53 patients of various viral hepatitis. We found that chromosomal aberrations appeared in both acute and chronic viral hepatitis. These changes often appeare in the acute stage of acute viral hepatitis, and disappeare in the recover stage. At the same time, we have also observed the lymphocyte transformations and found that the chromosomal aberration correlated to low lymphocyte transformation. The cause and the significance of the chromosomal aberrations in viral hepatitis are discussed.
急性病毒性肝炎
acute viral hepatitis
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