Allergic contact dermatitis from natural rubber latex in atopic dermatitis and the risk of later Type I allergy
It was suggested that Type I allergy might be the main pathogenesis.
Conclusion: The nosogenesis of Chronic Urticaria is complex. A part of patients are cased by type I allergy, accompanied by function disorder of T Lymphocyte.
Experimental study on tuo min zu fang against type I hypersensitivity
Effect of hyperbaric oxygen on type I allergic reaction and immune function
There is activation of eosinophils and mast cells in patients with chron ic urticaria. The change of ECP and tryptase is the symbol of the activation of eosinophils and mast cells respectively,which may suggests that t hey are related to the delayed phase in type I allergic reaction.
Type I allergic reaction to the infection may play an important role in pathogenesis of RSV-induced exacerbations of asthma.
结论 :儿童哮喘的发作与RSV感染关系密切 ,其机制与病毒感染损伤呼吸道黏膜、 I型变态反应的发生和IFN -γ水平低下等有关
The IgE antibodies play a vital role in the type I allergic reactions. The binding of IgE with the high-affinity IgE receptor (FcεI ) on the surface of target cells can trigger a series of signal transductions in the cells and then makes mast cells degranulate and release a lot of inflammatory mediators, which are the pathogenesy of allergic diseases, such as asthma.
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More than 25% of the population suffer from type I allergy.
Detection and clinical relevance of a type I allergy with occupational exposure to hexahydrophthalic anhydride and methyltetrahy
Occupational protein contact dermatitis with type I allergy to different kinds of meat and vegetables
Depending on their specificity these antibodies may bind the "fitting" antigens as bridging "allergens" causing the release of various mediators and, thus, the induction of type I allergy reactions.
Plant profilins have recently been shown to be highly crossreactive allergens which bind to IgE antibodies of allergic patients and thus cause symptoms of type I allergy.
Each of these conditions is mediated primarily by type I hypersensitivity reactions.
As in most type I hypersensitivity-based diseases, histamine plays a key role in the pathophysiology of cardiac anaphylaxis.
Many non- type I hypersensitivity responses to sulfonamide antibiotics are attributable to reactive metabolites that cause either direct cytotoxicity or humoral or cellular responses.
Given the importance of immunoglobulin (Ig) E in mediating type I hypersensitivity, inhibiting IgE production would be a general way of controlling allergic disease.
Current data suggest that stabilizing cell-surface CD23 would be a natural means to decrease IgE synthesis and thus control type I hypersensitivity.