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intramembranous osteogenesis
相关语句
  膜性骨发生
     THE HAVERSIAN SYSTEM IN THE INTRAMEMBRANOUS OSTEOGENESIS OF HUMAN FLAT BONE
     人扁骨膜性骨发生中的哈氏系统
短句来源
  “intramembranous osteogenesis”译为未确定词的双语例句
     Conclusion The mechanism of repairing the segmental bony defect by SGM+BMP/HA are that SGM protects defect area from connective tissue invading where it can be repaired by both guiding or inductive bone regeneration achieved by intramembranous osteogenesis throughout the healing.
     结论 SGM管与BMP/HA联合修复骨缺损的机制为SGM的屏障作用使骨缺损区受到引导成骨和诱导成骨的双重作用 ,其中诱导成骨方式为膜内骨发生
短句来源
  相似匹配句对
     THE HAVERSIAN SYSTEM IN THE INTRAMEMBRANOUS OSTEOGENESIS OF HUMAN FLAT BONE
     人扁骨膜性骨发生中的哈氏系统
短句来源
     Cell's adhesion and osteogenesis
     细胞黏附与骨发生
短句来源
     Distraction Osteogenesis of Jaws
     颌骨牵引成骨技术
短句来源
     (2) the new bone formed in the lengthened region might be the result of a combined osteogenesis from endochondral,intramembranous,and fibrous tissue ossification;
     (2)延长区新骨形成方式可能为软骨内成骨、膜内成骨及纤维组织直接转化成骨3种方式并存;
短句来源
     The Treatment of Delayed Nerve Intramembranous Compression Syndromes
     神经膜内迟发性受压综合征的外科处理
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  intramembranous osteogenesis
The effect of hyaluronan on mouse intramembranous osteogenesis in vitro
      


We found that the Haversian system also arised in the intramembranous osteogenesis of human flat bone.The primary Haversir.n system (or the first generation of Haversian system) arised in the developing cranial bones of human fetus as early as the fifth to the sixth months of the embryonic period. At birth, the typical Haversian systems are developed from the primary ones and located both in the compact bone and spongy bone. There are about 1-3 layers ioncentric lamellae, surrounding a central canal of...

We found that the Haversian system also arised in the intramembranous osteogenesis of human flat bone.The primary Haversir.n system (or the first generation of Haversian system) arised in the developing cranial bones of human fetus as early as the fifth to the sixth months of the embryonic period. At birth, the typical Haversian systems are developed from the primary ones and located both in the compact bone and spongy bone. There are about 1-3 layers ioncentric lamellae, surrounding a central canal of Havers. However, there are great different in the numbers, sizes and the layers of Haersian system between Haversian system in the long bone and Haversian system in the flat bone.

人的颅骨(颅顶骨和额骨)和部分扁骨的膜性骨发生中是否存在哈氏系统,以及它的出现时期、形态特征等,无论在教材中或有关文献中至今没有详尽记载,中文作者利用工作便利条件,收集了不同胎龄胎儿及婴幼儿颅骨和都份扁骨等作了系统观察和研究,发现人扁骨膜性骨发生的骨组织中,同样存在哈氏系统,与长骨中的哈氏系统相比,其特点为出现早、数量少、体积小,最早5~6月胎儿颅骨中可见到原始哈氏系统,出生前后哈氏系统已臻成热,完整,2岁后到成人,颅骨的板障中亦可见到哈氏系统。

Osteoblastic cells isolated from newborn rat calvaria were cultured in the DMEM+10% FCS medium supplemented with 50 mg/L ascorbic acid and io mmol/L Na-Beta-glycerophosphate.Clumps and multilayer of cells became evident and the cell nodules were formed.Von Kossa staining showed positive reaction in the nodules indicating presence of calcium phosphate,and alkaline phosphatase staining demonstrated that high activity of the enzyme was associated with formation of the nodules.Ultrastructure analysis of the nodules...

Osteoblastic cells isolated from newborn rat calvaria were cultured in the DMEM+10% FCS medium supplemented with 50 mg/L ascorbic acid and io mmol/L Na-Beta-glycerophosphate.Clumps and multilayer of cells became evident and the cell nodules were formed.Von Kossa staining showed positive reaction in the nodules indicating presence of calcium phosphate,and alkaline phosphatase staining demonstrated that high activity of the enzyme was associated with formation of the nodules.Ultrastructure analysis of the nodules showed mineral crystallites closely associated with collagen fibrils.Osteocyte-like cells were completely surrounded by mineralized matrix.Development of the three dimension structure consisting of the cells,collagen fibrils and mineral deposits resembled that of intramembranous osteogenesis.The results indicate that newborn rat calvaria osteogenic cells elaborate in culture a mineralized matrix,which further support the osteoblastic nature of the cells.

自新生大鼠颅骨分离出的成骨细胞在含50mg/L维生素C和10mmol/Lβ-甘油磷酸钠的DMEM+10%FCS培养液内逐渐出现聚集重叠而形成结节,VonKossa染色证实结节内有钙盐沉积,细胞的碱性磷酸酶染色显示结节的钙化与高活性酶的活动有关。电镜示结节内有大量的钙盐沉积在胶原纤维上,一些类似骨细胞的细胞被包埋在钙化骨质内,整个由细胞、胶原纤维和钙盐组成的三维结构类似于体内膜内成骨。实验结果提示:体外培养的鼠颅骨成骨细胞能形成钙化骨质,进一步证实所分离的细胞具有成骨细胞的性质。

Objective To elucidate the mechanism of silica gel membrane(SGM) with bone morphogenetic protein (BMP) and hydroxyapatite (HA) in repairing segmental bony defect. Methods The 1 2 cm bone defects were designed at radii in rabbits. The defects wrapped by SGM as a tube were filled with BMP/HA? HA as the experimental group, HA only as the control group, and unwrapped as the free group. The defects were observed by X ray, microscope and scanning electronic microscope (SEM) in the 1st,2nd,3rd months after operation...

Objective To elucidate the mechanism of silica gel membrane(SGM) with bone morphogenetic protein (BMP) and hydroxyapatite (HA) in repairing segmental bony defect. Methods The 1 2 cm bone defects were designed at radii in rabbits. The defects wrapped by SGM as a tube were filled with BMP/HA? HA as the experimental group, HA only as the control group, and unwrapped as the free group. The defects were observed by X ray, microscope and scanning electronic microscope (SEM) in the 1st,2nd,3rd months after operation separately. Results The osteoid was found in the defects filled by SGM+BMP/HA in the 1st month. After the 2nd month periosteal cells grew along both the outer and inner surface of SGM and the gap of HA into the defect space where osteogenesis was performed, and much new bone was found in the SGM+BMP/HA group. In the 3rd month the defects were repaired completely in the SGM+BMP/HA group, while little new bone formed in the SGM+HA group and the HA group. All the defects in the free group were filled by fibrous tissue. Conclusion The mechanism of repairing the segmental bony defect by SGM+BMP/HA are that SGM protects defect area from connective tissue invading where it can be repaired by both guiding or inductive bone regeneration achieved by intramembranous osteogenesis throughout the healing.

目的 探讨硅胶膜 (SGM )管与骨形态发生蛋白 (BMP) /羟基磷灰石 (HA)复合物联合修复兔长骨缺损的机制。方法 制备兔桡骨中段 1 2cm缺损模型 ,实验组缺损区外围包绕SGM ,其内分别填充BMP/HA、HA材料 ;对照组仅填充HA ;空白组骨缺损区未填充。通过X线摄片、光学显微镜和扫描电镜观察骨缺损区的影像学、组织学和超微结构变化。结果 术后 1个月 ,SGM +BMP/HA组骨缺损区有大量的类骨质形成。术后 2个月 ,SGM +BMP/HA组大量骨膜来源的分化细胞沿SGM管的内、外面及HA间隙向骨缺损区内生长 ,植入区可见大片的膜内成骨征象。术后 3个月 ,GM +BMP/HA组骨缺损区愈合。此时 ,SGM +HA组与对照组骨缺损区有少量新骨形成 ,空白组缺损区为纤维组织充填。结论 SGM管与BMP/HA联合修复骨缺损的机制为SGM的屏障作用使骨缺损区受到引导成骨和诱导成骨的双重作用 ,其中诱导成骨方式为膜内骨发生

 
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