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retinoblastoma
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  视网膜母细胞瘤
     Expression of TGF-β_1 /Smad4 transduction pathway in retinoblastoma cell line Hxo-Rb44
     TGF-β_1/Smad4信号传导系统在视网膜母细胞瘤株Hxo-Rb44的表达
短句来源
     Expressions of P16,P53 and MDM2 protein in retinoblastoma
     视网膜母细胞瘤中MDM2、P16和P53的表达及其意义
短句来源
     Expression of the p16,p21,p53 genes and PCNA in retinoblastoma
     p16、p21、p53和PCNA在视网膜母细胞瘤中的表达
短句来源
     Significance of expression of PTEN protein and CD_(44)V6 protein in retinoblastoma
     PTEN及CD_(44)V6在视网膜母细胞瘤中的表达及意义
短句来源
     Expressions of P53 and P21 protein in retinoblastoma
     P53蛋白及P21蛋白在视网膜母细胞瘤表达的研究
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  成视网膜细胞瘤
     AIM: To understand the effect of the RB1 gene mutation on the function of pRB (the protein product of the RB1 gene) in the patients with retinoblastoma (RB).
     目的 :了解成视网膜细胞瘤 (RB)患者RB1基因突变至RB发生中RB蛋白 (pRB)的变化。
短句来源
     Materials and methods:CT findings in 150 cases of normal eye,120 cases of traumatic cataract:23 cases of retinoblastoma and 10 cases of Coats disease were reported,which were scanned with a slice thickness and a gap of 2 0 mm,scan time was 2 8 second.
     材料与方法:150 例正常眼、120 例眼外伤性白内障、23 例成视网膜细胞瘤和10 例Coats○病的晶状体CT, 层厚及层距2mm, 扫描时间为2-8s 薄层扫描。
短句来源
     CT manifestations of retinoblastoma and their clinical diagnostic value
     成视网膜细胞瘤的CT表现及其临床诊断价值
短句来源
     D-type cyclin (cyclinD, CycD) regulates the G1/S transition. Under the stimulation of the exogenous signals, CycD accumulates and binds to cyclin dependent kinase (CDK) to form active complexes, which phosphorylate the retinoblastoma (Rb) protein. Rb phosphorylation results in releasing the transcription fac- tor E2F and thereby drives the G1/S transition.
     D型细胞周期蛋白(cyclinD,CycD)调控着细胞周期G1/S的转换,基本过程为CycD在外界环境刺激下积累,并与周期蛋白依赖激酶(cyclin-dependentkinase,CDK)形成有活性的激酶,促进成视网膜细胞瘤蛋白(retinoblastoma,Rb)磷酸化,使E2F因子释放,由此促使G1/S转换,这一调控系统在高等真核生物中具有很高的保守性。
短句来源
     Methods The CT manifestations of 12 patients with retinoblastoma proved by operation and pathology were analyzed.
     方法分析12例经手术及病理证实的成视网膜细胞瘤的CT表现。
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  “retinoblastoma”译为未确定词的双语例句
     BamHI AND RsaI RESTRICTION FRAGMENT LENGTH POLYMORPHISMS IN RETINOBLASTOMA GENE IN A CHINESE POPULATION
     BamHI AND RsaI RESTRICTION FRAGMENT LENGTH POLYMORPHISMS IN RETINOBLASTOMA GENE IN A CHINESE POPULATION
短句来源
     Structural Abnormalities and Polymorphisms of the Retinoblastoma Gene in Nasopharyngeal Carcinoma
     Structural Abnormalities and Polymorphisms of the Retinoblastoma Gene in Nasopharyngeal Carcinoma
短句来源
     The mean MVD was 33.59±1.15 in retinoblastoma and 7.42±0.95 in normal retina (t=17.499, P<0.01).
     Rb组中MVD平均为33.59±1.15,正常对照组为7.42±0.95(t=17.499,P<0.01)。
短句来源
     Results The positive expression rates of the Pl6、P53 and MDM2 in retinoblastoma were 35.7%,69.1%,38.1%. The coexpression rate of P53 and MDM2 was 23.8%.
     结果42例标本中P16、P53和MDM2阳性检出率分别是35.7%、69.1%和38.1%,P53和MDM2的共表达率为23.8%。
短句来源
     · RESULTS: The positive expression rates of the Bcl-2, p16 and PCNA in retinoblastoma were 55.6%, 47.2%,61.1% respectively. There was a significant difference in differentiated and undifferentiated types (P<0.01).
     结果:Bcl-2,p16和PCNA阳性检出率分别是55.6%,47.2%,61.1%,分化型与未分化型之间比较差异有显著性(P<0.01)。
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  retinoblastoma
The principal components of the G1-S checkpoint, such as cyclin-dependent kinases, retinoblastoma and E2F proteins, control the activity of telomerase.
      
Spectrum and Frequencies of RB1 Structural Defects in Retinoblastoma
      
The spectrum and frequencies of RB1 structural defects were studied in tumors and peripheral blood lymphocytes of patients with various forms of retinoblastoma.
      
RB1 and CDKN2A Functional Defects Resulting in Retinoblastoma
      
Methylation of p16 was for the first time observed in retinoblastoma (9 tumors, 17%).
      
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In recent years, with the advancesin research work on chromosome oftumor cells, it was revealed thatthe aberration of chromosome oftumor cells was nonrandom. It hasbeen proved that retinoblastoma(RB)in some patients is associated withdeletion of one chromosome 13q ofsomatic cells and some data concern-ing the chromosomal behavior ofRB tumor cells reported in literatureare found to be similar to those con-cerning somatic cells. The aim ofthe present study was to analyzethe chromosome constitution of tu-mor...

In recent years, with the advancesin research work on chromosome oftumor cells, it was revealed thatthe aberration of chromosome oftumor cells was nonrandom. It hasbeen proved that retinoblastoma(RB)in some patients is associated withdeletion of one chromosome 13q ofsomatic cells and some data concern-ing the chromosomal behavior ofRB tumor cells reported in literatureare found to be similar to those con-cerning somatic cells. The aim ofthe present study was to analyzethe chromosome constitution of tu-mor cells and compare the charac-teristics of chromosome of the malig-nant cells with histological and clini-cal findings of the tumor. Results of chromosome study of so-lid tumor cells from 9 patients withRB are presented in this paper. Noneof the patients had a family historyof RB, and the examination of themetaphase of chromosome of peripher-al blood lymphocytes in 5 patientsrevealed nothing abnormal. Chromo-some preparations of the tumor cellswere obtained by using direct tech-nique and short-term culture (2 and24 h). Chromosome banding wasachieved in 4 cases only. The chromosomes in metaphase oftumor cells in all 9 cases were foundabnormal both in number and struc-ture. 30-206 metaphases of tumorcells were counted in each individualcase. The modal chromosome numberwas 45 in 3 cases, 46 in 2, 47, 66and 92 in 3 cases respectively, andhyper-tetraploidy in 1. Karyotypeanalysis revealed a considerable vari-ability. However, one to five markerchromosomes were found in each case.Six of these cases had aberrations ofD-group chromosome. Six of thesecases lost one E-group chromosome(chromosome 16) and had a metacen-tric chromosome in size similar to C-group chromosome [in some cases, itwas probably i(17q)].Four cases haddouble minutes. These results suggest-ed that the aberration of chromosomeD and E-group were probably relatedto the development and progression ofRB. The investigation of chromoso-mal, histological and clinical findingssuggested that some undifferentiatedtumors tended to have polyploid chro-mosome modes and the chromosomalpolyploidy was probably related toprotracted evolution of tumor cells inconsiderably long duration of malig-nancy too.

本文对9例视网膜母细胞瘤患者实体瘤细胞作染色体研究。瘤细胞染色体数目和结构均有畸变。据检查结果,推测D组和E组染色体的异常可能与视网膜母细胞瘤的发生有一定联系;染色体的多倍化可能与病程较长,瘤细胞长期演化有关。

Retinoblastoma is an intraocularmalignant tumour of early childhoodwhich yields to spontaneous regressionoccasionally. The present paper re-ports four cases of spontaneous regres-sion of retinoblastoma verified histo-logically in 1983 and 1984. At theirfirst clinical visits. one female wassix months old and of the three malestwo were one and a half years old andthe other three years old. However,the onset was observed soon afterbirth, at 23, 30, 90 and 49 days of agerespectively. No family history of re-tinoblastoma...

Retinoblastoma is an intraocularmalignant tumour of early childhoodwhich yields to spontaneous regressionoccasionally. The present paper re-ports four cases of spontaneous regres-sion of retinoblastoma verified histo-logically in 1983 and 1984. At theirfirst clinical visits. one female wassix months old and of the three malestwo were one and a half years old andthe other three years old. However,the onset was observed soon afterbirth, at 23, 30, 90 and 49 days of agerespectively. No family history of re-tinoblastoma was revealed in these pa-tients. Three cases were unilaterallyinvolved while the fourth had one eyewith extraocular extension and the oth-er eye underwent regressive change ofthe tumor.All four cases presented thefeatures of "amaurotic cat's eye" andatrophic changes in the affected eyes.Two of them had histories of high fe-ver. Enucleations were performed in4 eyes and exenteration in the felloweye of the bilateral case. Histopatho-logic examination revealed greyish oryellowish granular tissues and largeareas of necrosis with a number of"corporate" tumor cells and consider-able degree of calcification. The scler-as were thickened especially at theposterior pole and a lot of scar tis-sues were seen in the choroid. In oneunilateral case where a few live tu-mour cells were found in the choroidnear the optic disc, an orbital new-growth occurred two months after enu-cleation and was confirmed to be arecurrent retinoblastoma histopatholo-gically after exenteration.

本文报告4例婴幼儿视网膜母细胞瘤自发退行,其中1例后极脉络膜有存活的瘤细胞,术后2月局部复发;另1例双侧肿瘤,1眼为自发退行,另1眼为眼外期。同时简要讨论了视网膜母细胞瘤自发退行的机理及诊断依据。

The causes of delay in carrying out decisive treatment in 81 patients with proven retinoblastoma are analysed. The most common cause of the delay is the failure of making the diagnosis (51.9%). Delay by refusal or procrastination of accepting prescribed therapy was observed as long as 15.9 months with the overall average of 10.2 months. In 69 cases followed-up for 3 years, the survival rates were 48.4% and 18.2% respectively for those who had been treated 6 months and 1 year after the disease were manifested...

The causes of delay in carrying out decisive treatment in 81 patients with proven retinoblastoma are analysed. The most common cause of the delay is the failure of making the diagnosis (51.9%). Delay by refusal or procrastination of accepting prescribed therapy was observed as long as 15.9 months with the overall average of 10.2 months. In 69 cases followed-up for 3 years, the survival rates were 48.4% and 18.2% respectively for those who had been treated 6 months and 1 year after the disease were manifested clinically.

81例经病理确诊之视网膜母细胞瘤延误治疗的原因以迟诊为最多,占51.9%。延治时间以拒治最为严重,为15.9月。所有病例平均延治时间为10.2月。 69例随访结果:三年以上生存率,延治在6月以内者为48.49%。延治在一年以上者为18.2%。

 
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