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exacerbation
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  加重
     But compared with exacerbation [ 0.13(0.10,0. 18)μg/L],the levels of PCT for patients of group B did not changed [0. 13 (0. 10,0. 15)μg/L, Z = - 1. 614, P = 0. 107 ].
     B组患者稳定期血清PCT含量为0.13(0.10,0.15)μg/L,与急性加重期[0.13 (0.10,0.18)μg/L]比较,差异无统计学意义(Z=-1.614,P=0.107)。
短句来源
     1. The levels of plasma TXB2, Fbgg , D-dimer and blood PaCO2 were significantly increased (P<0.01), and the levels of plasma 6-keto-PGF1α, PT, APTT, AT-Ⅲ:A and blood PaO2 were significantly decreased (P <0.01)in ozagrel group and control group in exacerbation period compared with healthy subjects, but in ozagrel group and control group showed no difference(P > 0.05).
     1. 两组慢性肺心病急性加重期患者治疗前分别与健康组比较,TXB2、Fbgg、D-dimer和PaCO2 显著高于健康组(P <0.01),6-keto-PGF1α、PT、APTT、AT-Ⅲ:A和PaO2显著低于健康组(P <0.01),此两组间上述指标无差别(P > 0.05)。
短句来源
     When they had returned to their stable state, the levels of PCT for patients of group A decreased to 0. 12(0. 10,0. 14)μg/L,which was significantly lower than in exacerbation[0. 24(0. 17,0. 28)μg/L,Z = -3. 298,P =0. 001] ;
     A组患者稳定期血清PCT含量为0.12(0.10,0.14)μg/L,显著低于急性加重期[0.24(0.17,0.28)μg/L,Z= -3.298,P=0.001];
短句来源
     Objective To monitor drug resistance of Pseudomonas aeruginosa in patients with acute exacerbation of chronic obstructive pulmonary disease(AECOPD).
     目的监测慢性阻塞性肺疾病急性加重(acute exacerbation of chronic obstructive pulmonary disease,AECOPD)患者铜绿假单胞菌感染的耐药现状。
短句来源
     (2) The ET-1 level in the patients with CPHD at acute exacerbation stage or stable stage was negatively correlated with PO 2 ( r =-0.72, P <0.05, r =-0.53, P <0.05), while positively correlated with PCO 2 ( r =-0.72, P <0.05, r =-0.53, P <0.05).
     肺心病急性加重期及缓解期血浆ET - 1与PO2 呈负相关(r =- 0 .72 ,r=- 0 .5 3,均P <0 .0 5 ) ,与PCO2 呈正相关 (r=0 .5 5 ,r =0 .5 3,均P <0 .0 5 ) ;
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  加重期
     But compared with exacerbation [ 0.13(0.10,0. 18)μg/L],the levels of PCT for patients of group B did not changed [0. 13 (0. 10,0. 15)μg/L, Z = - 1. 614, P = 0. 107 ].
     B组患者稳定期血清PCT含量为0.13(0.10,0.15)μg/L,与急性加重期[0.13 (0.10,0.18)μg/L]比较,差异无统计学意义(Z=-1.614,P=0.107)。
短句来源
     1. The levels of plasma TXB2, Fbgg , D-dimer and blood PaCO2 were significantly increased (P<0.01), and the levels of plasma 6-keto-PGF1α, PT, APTT, AT-Ⅲ:A and blood PaO2 were significantly decreased (P <0.01)in ozagrel group and control group in exacerbation period compared with healthy subjects, but in ozagrel group and control group showed no difference(P > 0.05).
     1. 两组慢性肺心病急性加重期患者治疗前分别与健康组比较,TXB2、Fbgg、D-dimer和PaCO2 显著高于健康组(P <0.01),6-keto-PGF1α、PT、APTT、AT-Ⅲ:A和PaO2显著低于健康组(P <0.01),此两组间上述指标无差别(P > 0.05)。
短句来源
     When they had returned to their stable state, the levels of PCT for patients of group A decreased to 0. 12(0. 10,0. 14)μg/L,which was significantly lower than in exacerbation[0. 24(0. 17,0. 28)μg/L,Z = -3. 298,P =0. 001] ;
     A组患者稳定期血清PCT含量为0.12(0.10,0.14)μg/L,显著低于急性加重期[0.24(0.17,0.28)μg/L,Z= -3.298,P=0.001];
短句来源
     (2) The ET-1 level in the patients with CPHD at acute exacerbation stage or stable stage was negatively correlated with PO 2 ( r =-0.72, P <0.05, r =-0.53, P <0.05), while positively correlated with PCO 2 ( r =-0.72, P <0.05, r =-0.53, P <0.05).
     肺心病急性加重期及缓解期血浆ET - 1与PO2 呈负相关(r =- 0 .72 ,r=- 0 .5 3,均P <0 .0 5 ) ,与PCO2 呈正相关 (r=0 .5 5 ,r =0 .5 3,均P <0 .0 5 ) ;
短句来源
     Results:The plasma β-EP, ACTH and GC were significantly elevated i n CPHD patients with exacerbation (P<0.01);
     结果 :CPHD急性加重期 β- EP、ACTH、GC均显著升高 (P<0 .0 1) ;
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  病情恶化
     ②exacerbation and death did not appear in any one of 20 CAs with normal ECG,but appeared in 25 of 80 CAs with abnormal ECG(X 2=5 932, P <0 05).
     80例有异常ECG的脑卒中病例 ,2 5例病情恶化和死亡 ,两者差异显著 (X2 =5 932 ;P <0 0 5 ) ;
短句来源
     Of the 11 patients there were 6 cases discharged due to exacerbation, 2 dead in hospital and 3 patients discharged after symptomatic remission by expectant treatment.
     11例患者中6例病情恶化自动出院,2例院内死亡,3例经对症治疗症状缓解出院。
短句来源
     Combination with lamivudine can increase the response rate and reduce the exacerbation rate.
     苦参素单用治疗乙肝部分病例可发生病情恶化,联合拉米夫定治疗可提高应答反应率并降低恶化病例的发生率.
短句来源
     64 cases were cured,one case died,one case was discharged by himself for exacerbation. The death ratio of all was 10.6%,and that of operative group was 3.03%.
     手术治愈 6 4例 ,术后死亡 1例 ,病情恶化自动出院 1例 ,全组死亡率 10 6 0 % (手术组为 3 0 3% )。
短句来源
     According to 21 of major indications and experimental data from bacteriological, serological diagnosis and chest X-ray positive patients, through group coding and probability analysis, the exacerbation of these patients were observed and nursing cares were given.
     根据细菌学、血清学诊断且胸部X线阳性病人的21项主要临床指征、实验数据,通过分组编码和概率分析,及对病人病情恶化进程的观察及护理。
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  “exacerbation”译为未确定词的双语例句
     CNP at acute exacerbation stage in group B1 was higher than that at stable stage in group B2(P<0.05),and group B2 higher than group A(P<0.01).
     血浆CNP水平B1组高于B2组(P<0.05),B2组高于A组(P<0.01);
短句来源
     Result:In cor pulmonale exacerbation, the values of TXB2, 6-K-PGFia, TXB2/6-K-PGFia and PAdT, PAgT and PAF were 204. 76±35. 89pg/ml,49. 08±14. 38pg /ml, 4. 11±1. 72 and 42±11%, 21±8% and 89. 05± 41. 76ng/ml respectivety;
     结果慢性肺心病急性发作期组TXB2、6-K-PGF1a、TXB2/6-K-PGF1a比值和PAdT、PAgT及PAF测定值分别为204.76±3589pg/ml、49.08±1438pg/ml、4.11±1.72和42±11%、21±8%和89.05±41.76ng/ml。
短句来源
     (2) C_ (a-) O_2 in acute exacerbation and stable phases were (5.59±1.6)mL/dL and (3.05±1.07)mL/dL,respectively,with remarkable difference found(P<0.01).
     ②急性期与缓解期C(a-v-)O2分别为(5·59±1·6)mL/dL和(3·05±1·07)mL/dL,有显著性差异(P<0·01)。
短句来源
     ② The levels of interleukin 8 and tumor necrosis factor alpha in sputum were obviously higher in patients at acute exacerbation[(40.52± 7.36),(19.42± 3.25) ng/L] than in those at remission[(16.42± 2.16),(9.30± 2.64)](t=19.06,14.53,P< 0.01).
     ②急性期患者痰液中白介素8和肿瘤坏死因子水平犤(40.52±7.36),(19.42±3.25)ng/L犦明显高于缓解期犤(16.42±2.16),(9.30±2.64)ng/L,t=19.06,14.53,P<0.01犦。
短句来源
     Results The sTNF-R I serum concentrations in exacerbation patients (4.91±0.39 ng/ml) were significantly higher than those with remission asthma (2.35±0.43 ng/ml,P<0.01) and healthy controls (1.26±0.24 ng/ml,P<0.05).
     结果哮喘发作组sTNF-RI浓度为4.91±0.39ng/ml,哮喘缓解组为2.35±0.43ng/ml,明显高于健康对照组(1.26±0.24ng/ml),(P<0.01和P<0.05);
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  exacerbation
Measures of clinical outcome comprised relapse, hospitalization and clinical exacerbation.
      
Acute muscular weakness may be a sign of newly acquired neuromuscular disorders or of an exacerbation of a previously existing disease.
      
Acute exacerbation of COPD is most often caused by pulmonary infections.
      
The term acute exacerbation of chronic obstructive pulmonary disease (COPD) reflects an acute worsening of the stable state with an increase in dyspnea, cough, and count or purulence of sputum.
      
The management of an acute exacerbation is guided mainly clinical severity which has implications on the decision of outpatient management, hospital management or intensive care treatment.
      
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Observations on the histopathology of malignant granuloma in 5 cases were mace One of them was of the generalized type,with widespread pathological changes,confirmed by autopsy.The other 4 were of the localized type and treated by radiating.Two of them died and autopsy examinations were made whereas the clinical symptoms subsided in the other two cases.In one case there was exacerbation of symptoms after local injection of penicillin.One case revealed nodular reticulosis in the spleen.The histopathologic...

Observations on the histopathology of malignant granuloma in 5 cases were mace One of them was of the generalized type,with widespread pathological changes,confirmed by autopsy.The other 4 were of the localized type and treated by radiating.Two of them died and autopsy examinations were made whereas the clinical symptoms subsided in the other two cases.In one case there was exacerbation of symptoms after local injection of penicillin.One case revealed nodular reticulosis in the spleen.The histopathologic features of the disease are quite in accord with these of allergic,necrotizing and granulomatous vasculitis.They may occur locally on the nose,face and lungs and may also become widespread in the whole body.Some focal histological changes after radiotherapy are also discussed in the paper.

本文报告5例恶性肉芽肿,1例经尸检证实为全身性Wegener病,4例为局限性Wegener病,皆用放射治疗,其中2例死亡作了尸检,2例好转。目前多认为本病为变态反应性疾病。作者报告1例局部注射青霉素后病情加重。本病的病理组织学的改变与过敏坏死性血管炎一类疾病相符合。病变可局限在鼻面部或局限于肺部,亦可呈全身播散型。1例脾内网状细胞呈结节状增生,其性质有待进一步研究。

The side-effects in 30 oases of oysticercosis treated with Praziquantel since 1979 were analysed. All oases except one were given Praziquantel orally to a total dose of 120 mg/kg body weight in divided doses for 4-6 days. The remaining case received only three fourths of the total dose.The adverse reactions consisted mainly of headache (23 oases), psychogenia (2 oases), anaphylaotio shock (2 oases). Four oases of cerebral type had their oerebro-spinal fluid raised in pressure, protein level and WBC count during...

The side-effects in 30 oases of oysticercosis treated with Praziquantel since 1979 were analysed. All oases except one were given Praziquantel orally to a total dose of 120 mg/kg body weight in divided doses for 4-6 days. The remaining case received only three fourths of the total dose.The adverse reactions consisted mainly of headache (23 oases), psychogenia (2 oases), anaphylaotio shock (2 oases). Four oases of cerebral type had their oerebro-spinal fluid raised in pressure, protein level and WBC count during the course of the specific treatment and lasted from 1 week to 3 months afterwards. One ophthalmio case had persistent severe local inflammatory reaction after the speoino treatment, so operative removal of the dead oystioercus with surrounding adhesions had to be resorted to. Only 4 cases were found to be free from any adverse reactions.Besides heavy dosage of the specific drug, there seems to have other factors related to the cause of the adverse reactions. Mannitol is recommended to be given concurrently with Praziquantel in order to cope with the increase of intraoranial pressure. In patients with schizophrenia, Praziquantel should be used cautiously to avoid the exacerbation of psychotic symptoms. As to the use of Praziquantel in oysticeroosis with ophthalmio involvement is still an open question awaiting further study.

本文报告30例各型囊虫病患者,除1例外均采用吡喹酮在4~6日口服120mg/kg治疗(体重>60kg者按60kg计算)。服药后全无反应者仅4例(占10.3%)。余26例中有轻重不等的发热、头痛、呕吐、药疹等,2例发生过敏性休克,2例出现精神异常,4例脑型患者治疗后短期内见脑脊液压力、蛋白及白细胞数增高。文中讨论了产生药物不良反应的可能机理。为求减少药物反应,提高疗效,对颅内压增高和痴呆型的脑囊虫病患者必须审慎。眼囊虫病是否适合药物治疗有待进一步探讨。

In 30 cases with SLE, the appearance of LE cells in the blood in relationto the titers of ANA was observed. Dynamic observations were made on 5cases who showed remission and 1 case who showed exacerbation of the dise-ase during treatment with steroids, 10 cases of rheumatic arthritis and other disases were observed as controle. It is preliminarily assumed that the auto-antibodies antinuclear antibodies (ANA), react with the antigens in nucle-us. The degree and extent of the degeneration is directly proportional...

In 30 cases with SLE, the appearance of LE cells in the blood in relationto the titers of ANA was observed. Dynamic observations were made on 5cases who showed remission and 1 case who showed exacerbation of the dise-ase during treatment with steroids, 10 cases of rheumatic arthritis and other disases were observed as controle. It is preliminarily assumed that the auto-antibodies antinuclear antibodies (ANA), react with the antigens in nucle-us. The degree and extent of the degeneration is directly proportional to thetiters of ANA in the patient's blood. If the titers of ANA are low (less than1:100) there may be slight degeneration of the nucleus, part of the normalchromatin structure is still persisted and only the center of it tends to fusetogether or there may be fissures on it. The cleft on it may widen or it maybreak into pieces. According to the characteristics of the degenerated chromat-in described above, we call them "small pattern", and "speckled", LE cells. Ifthe titers of ANA increase more than 1:100, the normal chromatin pattern,under the action of ANA, would be partly dissolved, forming large vacuolesand we call it "reticular", LE cells. All kinds of them are called "atypical LEcells". In the titers of ANA increase up to 1:250 or more, there would befurther of the degenerated chromatin. The chromatin became loose, swelled andfinally lost its normal pattern, giving rise to a large hemogeneous mass-LEbody. When it engulfed by a polymorphonuclear leukocytes, it is called "ty-pical LE cell". With the number of this cells can reach 35 per cent of theneutroleukocytes. In 5 cases of SLE (ANA 1:250), the clinical symptoms subsided aftertreating with steroids with the decreasing of ANA, typical LE cells and LEbodies in the patient's blood disappeared. In one case (ANA 1:1,250), whobecame worse during treatment with steroids, typical LE cells and LE bodiesdid not disappear from the blood, and the titers of ANA did not come down. The presence of typical LE cells in the peripheral blood is valuable forthe diagnosis of SLE. But according to the observation mentioned above, typ-ical LE cells were only present in peripheral blood while the titers of ANA isabove 1:250. Below this level, only different kinds of atypical LE cells couldbe found. Therefore, if the number of these atypical LE cells below 1‰, thediagnosis of SLE would be questionable. If the number of them is above 1‰,SLE and other autoimmune disease could be considered. However, if the numb-er of them is beyond 5‰, it is most likely that the disease is SLE.

本文探讨了典型LE细胞的形成与ANA滴度的关系,认为非典型LE细胞的出现与数量的增多对诊断SLE有参考价值。对典型LE细胞的临床意义本文作了分析。

 
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