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alcoholic hepatitis
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  酒精性肝炎
     The F protein was found in 89.2% patients with acute hepatitis, 85.4% hepatic carcinoma, 79.6% alcoholic hepatitis, and 64.5% liver cirrhosis.
     阳性检出率原发性肝癌85.4%、急性肝炎89.2%、酒精性肝炎79.6%、肝硬化64.5%。
短句来源
     Objective:To observe the expression of cyclooxygenase-2(COX-2)in alcoholic hepatitis(AH)in rats and evaluate the relationship between COX-2and AH.
     目的观察环氧合酶-2(cyclooxygenase-2,COX-2)在大鼠酒精性肝炎(alcoholichepatitis,AH)中的表达,探讨COX-2与AH的关系。
短句来源
     Expression and effect of cyclooxygenase-2 in alcoholic hepatitis in rats
     环氧合酶-2在实验性酒精性肝炎中的表达及作用机制
短句来源
     Results The mAST/AST ratio for patients when they first entered hospital was in the following order: severe hepatitis 0 375±0 073, alcoholic hepatitis 0 311±0 048, chronic hepatitis 0 307±0 039, acute hepatitis 0 302±0 046, drug hepatitis 0 295±0 030, hepatitis cirrhosis 0 276±0 049;
     结果 发病期mAST/AST比值从高到低依次为重型肝炎 0 3 75±0 0 73、酒精性肝炎 0 3 11± 0 0 48、慢性肝炎 0 3 0 7± 0 0 3 9、急性肝炎 0 3 0 2± 0 0 46、药物性肝炎0 2 95± 0 0 3 0、肝炎肝硬化 0 2 76± 0 0 49;
短句来源
     Conclusion Long-term great deal of drinking alcohol is the main reason of alcoholic hepatitis. Drinking everyday 80~120g for 5 years above,90% patients suffered from the fatty liver,10%~35% patients suffered from the alcoholic hepatitis,1.5%~8% suffered from the alcoholic cirrhosis.
     结论长期大量饮酒是引起酒精性肝病的主要原因,每天饮酒80~120g持续5年以上,90%有脂肪肝,10%~35%有酒精性肝炎,1.5%~8%有酒精性肝硬化。
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  “alcoholic hepatitis”译为未确定词的双语例句
     The degree of hepatic steatosis and activity of alcoholic hepatitis were evaluated by semi-quantitation method. Results The score of severity of steatosis were 1.63±0.51(P<0.05), 2.12±0.64 (P<0.01) 和1.50±0.53 (P<0.05) in 3 alcohol groups.
     结果(1)A、B、C组肝脏脂肪变性评分分别为1.63±0.51、2.12±0.64和1.50±0.53,与对照组比较差异均具有统计学意义(P<0.05或0.01); (2)A、B、C组的血清NO水平分别为(76.35±10.56)μmol?
短句来源
     Objective Massive necrosis and apoptosis of liver cells is the common feature of different kinds of liver diseases, such as viral hepatitis, alcoholic hepatitis, autoimmune hepatitis, etc.
     目的:在自身免疫性肝炎和病毒性肝炎引起的肝损伤发病过程中,肝细胞的坏死和凋亡是共同特点。
短句来源
     There are many etiological agents of liver fibrosis, the most common of them are virus hepatitis, alcoholic hepatitis and non-alcoholic fatty liver.
     肝纤维化进展隐匿,最终可发展为肝硬化、肝功能衰竭、甚至肝细胞癌,因此肝纤维化已成为世界上最严重的公共卫生问题之一,阻断肝纤维化是防治肝硬化的重要手段。
短句来源
     Serum leptin was detected by ELISA in 60 patients with ALD (Alcoholic fatty liver AFL n=14, Alcoholic hepatitis AH n=28, Alcoholic cirrhosis ALC n=18). 16 healthy persons serves as normal control(NC). Serum liver fibrosis markers(HA, LN, PCⅢ) and TGF β 1 were determined at the same time.
     用ELISA法检测 6 0例ALD患者血清瘦素水平 ,16例健康人作为对照组 ,同步检测了ALD患者的血清肝纤维化指标HA、LN、PC -Ⅲ及TGFβ1,对血清瘦素与肝纤维化指标、肝组织纤维化分期间的关系进行了分析。
短句来源
     Liver fibrosis is a result of chronic hepatocellular damage due to a variety of liver diseases including viral hepatitis, alcoholic hepatitis, schistosomiasis, primary and secondary biliary cirrhosis, primary sclerosing cholangitis, etc.
     肝纤维化是各种慢性肝脏损害,包括:病毒性肝炎、酒精性肝病、血吸虫病、原发性和继发性胆汁性肝硬化、原发性硬化性胆管炎等的共同的病理基础。
短句来源
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  相似匹配句对
     Alcoholic hepatitis,7cases.
     酒精性肝炎 7例 ;
短句来源
     Advances in treatment of alcoholic hepatitis
     酒精性肝炎的治疗进展
短句来源
     Ischaemia hepatitis
     缺血性肝炎
短句来源
     Hepatitis B vaccination.
     乙型肝炎疫苗免疫
短句来源
     On the Liabilities of Alcoholic Crime
     论醉酒人犯罪的刑事责任
短句来源
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  alcoholic hepatitis
Acute liver diseases such as fulminant hepatic failure or alcoholic hepatitis are considered hypercatabolic diseases and thus require prompt nutritional intervention with a high-calorie enteral or parenteral formula.
      
The TNF-α inhibitor pentoxifylline decreased mortality from alcoholic hepatitis.
      
PPC was beneficial in patients with alcoholic hepatitis, and it opposed fibrosis in heavy drinkers and decreased aminotransferases in patients with hepatitis C.
      
Alcoholic hepatitis is a disease with a wide range of severity.
      
Given the benefit of drug treatment, it is important to identify patients at risk of early mortality from alcoholic hepatitis.
      
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Between January 1984 and December 1991,25 consecutive, histologically diagnosed patients,median age 48 years(range 17 -69)were prospectively studied. The patients were divided into those having alcoholic hepatitis (AH ,n= 10) .alcoholic liver cirrhosis (AC, n = 10)and alcoholic fatty liver(AF,n = 5)on the basis of their histological findings. All the patients were males,those AF being younger than those with AH and AC. The amount of alcohol consumption of AF,AH and AC was not significantlly...

Between January 1984 and December 1991,25 consecutive, histologically diagnosed patients,median age 48 years(range 17 -69)were prospectively studied. The patients were divided into those having alcoholic hepatitis (AH ,n= 10) .alcoholic liver cirrhosis (AC, n = 10)and alcoholic fatty liver(AF,n = 5)on the basis of their histological findings. All the patients were males,those AF being younger than those with AH and AC. The amount of alcohol consumption of AF,AH and AC was not significantlly different. By contrast,the duration of alcohol consumption was significantly different between the AF and AH and AC patients. The clinical features of the patients were quiet similar to those reported from the west except that AH was relatively milder in our patients. Intake of poor of quality country liquor were quite common in the majority. Follow -up period was 2 - 6 years. 6 death of 10 AC patients with continued alcohol consumption in 2-4 years. Among these,5 of 6 AC patients with Child-pugh C grade,portal hypertention and esophageal varices died and 2 died of HCC. 1 of AH died of hepatic failure after continued heavy alcohol consumption .All 5 AF patients recovered after withdrawal of alcohol. Survival is adversely affected by the severity of liver disease at diagnosis,the cont-nued consumption of alcohol and support treatment. These observations indicate that discontinuation of heavy alcohol consumption and treatment of alcohol withdrawal syndrome often results in improvement of liver lesions in patients with alcoholic liver disease.

本文对近年来收治的25例酒精性肝病(ALD)的临床及预后进行了分析,其中酒精性肝炎(AH)10例、酒精性肝硬变(AC)10例.酒精性脂肪肝(AF)5例、并对该病的临床表现、实验室检查、B超等进行了探讨.作者认为戒酒是治疗ALD的紧要环节,戒酒是终生治疗,而治疗乙醇依赖状态和戒酒综合征是起到戒酒的关键.

Serum transferrins were extracted from sera of patients with hepatocellular carcinoma, other liver diseases, and healthy individuals with immobilzed antitransferrin antibody. The extracted transferrins were analyzied by dot-immunobinding assay of Datura Stramonium agglutinin coupled to a peroxidase (HRPDSA) .It was found that sera from the twenty healthy individuals were all negative, and those from the fifteen patients with hepatocellular carcinoma were all positive, including three cases with negative a-fetoprotein...

Serum transferrins were extracted from sera of patients with hepatocellular carcinoma, other liver diseases, and healthy individuals with immobilzed antitransferrin antibody. The extracted transferrins were analyzied by dot-immunobinding assay of Datura Stramonium agglutinin coupled to a peroxidase (HRPDSA) .It was found that sera from the twenty healthy individuals were all negative, and those from the fifteen patients with hepatocellular carcinoma were all positive, including three cases with negative a-fetoprotein (AFP).Two each of ten patients with chronic active hepatitis and ten with pest-hepatitis liver cirrhosis,were found to be positive. Two patients with alcoholic hepatitis and four nonhepetitis patients with a high GPT level all proved negative. These results indicates an effective method for the diagnosis of early stage hepatocellular careinoma, especially those that are AFP negative.

作者用人血清转铁蛋白(Tf)抗体固定化柱快速分离正常人、肝癌及其他肝病患者血清Tf,并直接点在硝酸纤维素膜(NC)上,用辣根过氧化物酶(HRP)标记的欧曼陀罗凝集素(HRP-DSA)进行点印迹分析发现,15例肝癌患者全部显阳性,其中3例AFP阴性的肝癌患者也显阳性,而20例正常人则都显阴性。10例慢性活动性肝炎(慢活肝)患者和10例肝炎后肝硬化患者分别有2例阳性,2例酒精性肝硬化和4例高水平GPT的非肝炎患者则全部为阴性。实验结果为肝癌的早期诊断尤其是AFP阴性的肝癌早期诊断提供了有效的方法。

orphological changes in liver biopsies from 40 alcoholic patients were studied,20 of which being ordinary alcoholics(40-80g ethanol/day)and the other 20 being heavy drinkers(above 80g ethanol/day for over 20 years) All being male who have neither type B nor type C hepatitis.The basic mornhological changes observed being:1.Liver cell degeneration including fatty degeneration & focal ballooning,decrease in liver cell size,occasional giant mitochondrion and Mallory' s body formation. 2. Focal necrosis with neutrophil...

orphological changes in liver biopsies from 40 alcoholic patients were studied,20 of which being ordinary alcoholics(40-80g ethanol/day)and the other 20 being heavy drinkers(above 80g ethanol/day for over 20 years) All being male who have neither type B nor type C hepatitis.The basic mornhological changes observed being:1.Liver cell degeneration including fatty degeneration & focal ballooning,decrease in liver cell size,occasional giant mitochondrion and Mallory' s body formation. 2. Focal necrosis with neutrophil infiltration. 3. Pericellular fibrosis of liver cells,hepatic fibrosis and early cirrhosis.Alcoholic liver disease can be divided into 5 types:Ⅰ.alcoholic fatty liver(AFL),Ⅲ.alcoholic hepatitis(AH),Ⅲ.alcoholic hepatic fibrosis(AHF),Ⅳ. alcoholic liver cirrhosis (ALC),Ⅴ. slight alcohol-ic liver disease (SALD).The degree of liver damage(liver cell necrosis and hepatic fibrosis) is closely re-lated to the amount of daily ethanol intake. The progression of liver damage observed in our study is much milder than reports from Europe,the U. S.and Japan.

研究了临床确定的40例嗜酒者肝穿标本的形态变化,普通嗜酒者与重度嗜酒者各20例。基本病变为:(1)肝细胞变性,包括大泡状肝细胞脂变,灶状肝细胞变小,灶状气球样变等;(2)肝细胞坏死灶形成伴中性粒细胞浸润,肝细胞内巨大线粒体或麦氏小体;(3)窦周纤维化、肝纤维化及细结节性肝硬变等。据此,我们将酒精性肝病分为五个病理类型。研究结果证明,肝脏的病变程度和饮酒量密切相关,饮酒时间超过10年后,肝坏死灶及肝纤维化程度与饮酒量呈正相关关系。40例患者的肝脏病变程度明显轻于欧美和日本病例,这可能与我国的饮酒、饮食习惯有关。

 
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