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dibutyryl cyclic gmp
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  “dibutyryl cyclic gmp”译为未确定词的双语例句
     The excitatory effect of 0.12 mM sodium oleate was partially blocked by intravenous injection of antisecretin serum or dibutyryl cyclic GMP.
     静脉灌流抗促胰液素血清或二丁基cGMP可部分阻断0.12mM油酸钠对胰液分泌的兴奋作用。
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  相似匹配句对
     Regulation of Cell Function by Cyclic GMP.
     cGMP对细胞功能的调控
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     Studies on the Dimer Stacking Conformation of Cyclic GMP
     环化鸟苷酸双聚体堆积构象的研究
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     Influence of Dibutyryl Cyclic Adenosine Monophosphate on Motility of Sperm
     双丁酰环磷酸腺苷对人精子运动功能的影响
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     D—CYCLIC CRAPHS
     D—Cyclic图
短句来源
     The excitatory effect of 0.12 mM sodium oleate was partially blocked by intravenous injection of antisecretin serum or dibutyryl cyclic GMP.
     静脉灌流抗促胰液素血清或二丁基cGMP可部分阻断0.12mM油酸钠对胰液分泌的兴奋作用。
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  dibutyryl cyclic gmp
The effects of CCK-receptor antagonists such as dibutyryl cyclic GMP (dbcGMP), L-364,718, and CR1409 on COOH-terminal octapeptide of CCK (CCK-8) -stimulated chief cell responses were examined using isolated guinea pig gastric chief cells.
      
This secretion was stimulated by dibutyryl cyclic GMP, but was not altered by acetylcholine, carbachol, or 16,16-dimethyl PGE2.
      
A similar change in ODC activity was caused by the addition of oxotremorine (1 mM), isoproterenol (0.1 mM), NE, cyclic AMP (1 mM), or dibutyryl cyclic GMP (1 mM).
      
Calcitonin release from 6.23 rat medullary thyroid carcinoma C-cells was stimulated by dibutyryl cyclic AMP and inhibited by dibutyryl cyclic GMP in concentration dependent fashion.
      
Dibutyryl cyclic GMP by itself, or associated with vinblastine, failed to modify the spontaneous release of enzymes or the secretion induced by dibutyryl cyclic AMP.
      
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The effect of sodium oleate on pancreatic secretion and plasma secretin concentration were tested in anesthetized rats filled with pancreatic cannula. Intraduodenal in- fusion of sodium oleate at doses of 0.03, 0.06 and 0.12 mM/h induced the significant increases in volume, bicarbonate, protein, amylase output and plasma secretin concentration. The excitatory effect of 0.12 mM sodium oleate was partially blocked by intravenous injection of antisecretin serum or dibutyryl cyclic GMP. The duodenal perfu-sate...

The effect of sodium oleate on pancreatic secretion and plasma secretin concentration were tested in anesthetized rats filled with pancreatic cannula. Intraduodenal in- fusion of sodium oleate at doses of 0.03, 0.06 and 0.12 mM/h induced the significant increases in volume, bicarbonate, protein, amylase output and plasma secretin concentration. The excitatory effect of 0.12 mM sodium oleate was partially blocked by intravenous injection of antisecretin serum or dibutyryl cyclic GMP. The duodenal perfu-sate (lumenal factor) also stimulated the pancreatic secretion, indicating that the sodium oleate in the duodenum may trigger the release of an new humoral factor which regulates exocrine pancreatic secretion.

通过麻醉大鼠胰腺插管,观察油酸钠对胰液和促胰液素分泌的影响。实验结果表明,十二指肠灌注0.03、0.06和0.12mM/h油酸钠,可使胰腺分泌量、碳酸氢盐、蛋白质、淀粉酶排出量及血浆促胰液素含量明显增加。静脉灌流抗促胰液素血清或二丁基cGMP可部分阻断0.12mM油酸钠对胰液分泌的兴奋作用。十二指肠流出液(肠腔因子)同样有兴奋胰液分泌的作用。这些结果表明,油酸钠可能刺激肠腔释放一种新的体液因子调节胰腺外分泌。

AIM: To study the effects of dinoprostone (Din) on nitric oxide (NO) production induced by lipopolysaccharides (LPS) and cytokines mixture (CM) in cultured rat hepatocytes, and the role of cAMP signaling pathway in the induction process. METHODS: Rat hepatocytes were incubated with indometacin (Ind), Din, forskolin (For), or dibutyryl cyclic GMP ( db cGMP ) in a medium containing CM (LPS plus TNF α, IL 1β, and IFN γ) for 24 h. NO production in the cultured supernatant was measured with the Griess...

AIM: To study the effects of dinoprostone (Din) on nitric oxide (NO) production induced by lipopolysaccharides (LPS) and cytokines mixture (CM) in cultured rat hepatocytes, and the role of cAMP signaling pathway in the induction process. METHODS: Rat hepatocytes were incubated with indometacin (Ind), Din, forskolin (For), or dibutyryl cyclic GMP ( db cGMP ) in a medium containing CM (LPS plus TNF α, IL 1β, and IFN γ) for 24 h. NO production in the cultured supernatant was measured with the Griess reaction. Intracellular cAMP level was measured with radio ̄immuno ̄assay. RESULTS: NO production stimulated by CM was markedly attenuated by Ind in the cultured supernatant, whereas Din and For increased nitrite concentration in the medium. In the presence of CM, both Din and For increased intracellular cAMP level, and had no effect on cAMP level in absence of the stimuli; db cGMP had no influnence on the cAMP and NO production both in the presence and absence of same stimuli. CONCLUSION: Din potentiates cytokines and LPS to induce NO production in rat hepatocytes, and the induction may be regulated via intracellular second messenger cAMP signaling pathway.

目的:探讨地诺前列酮(Din)对细胞因子(IL1β,TNFα,IFNγ)及脂多糖(LPS)诱导的原代培养大鼠肝细胞一氧化氮(NO)生成的影响,以及环腺苷一磷酸(cAMP)信号通路在诱导过程中的作用.方法:将吲哚美辛(Ind),Din,弗司扣林(For)及双丁酰环鸟苷一磷酸(dbcGMP)分别加入含细胞因子TNFα,IL1β,IFNγ及LPS的培养液中,原代培养肝细胞24h后,采用Gries反应法测定细胞培养上清中NO含量,放射免疫法测定细胞内cAMP水平.结果:Ind明显抑制LPS与细胞因子刺激所致NO生成,反之,Din及For促进NO生成;在同样刺激条件下,Din与For使细胞内cAMP水平增高,而在对照组细胞未观察到上述反应.dbcGMP则无论炎性刺激存在与否均无此作用.结论:Din协同细胞因子及LPS诱导肝细胞NO生成,细胞内第二信使cAMP通路参与此诱导过程.

 
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