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adenosine- -
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     5).
     5)健全社会保障体系。
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     5. G.
     5.连钱草和紫花地丁都有具有校强的耐旱能力。
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     SYNTHESIS ON DERIVATIVES OF ADENOSINE 5-ALDEHYDE
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     Studies on Stereochemistry of Adenosine 3' , 5'-Cycloalkylphosphonate
     腺嘌呤核苷3′,5′-环烷基膦酸类似物的立体化学研究
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     Study on Separation of Adenosine 5'-Triphosphate by Ion Exchange
     离子交换法分离纯化5'-三磷酸腺苷的工艺研究
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  adenosine- -
Mathematical modeling of kinetics of adenosine-5'-triphosphate hydrolysis catalyzed by the Zn2+ ion in the pH range 8.5-9.0
      
The kinetics of adenosine-5'-triphosphate (ATP) hydrolysis catalyzed by Zn2+ at pH 8.5-9.0 is analyzed by numerical simulation.
      
A recent adenosine derivative (Abbott 40557) ethyl-adenosine-5'-carboxylate hydrochloride has marked and prolonged coronary vasodilator activity (2, 3).
      
The goal of this study was to determine whether the protective effects of the A3AR agonist N6-(3-iodobenzyl)adenosine-5'-N-methylcarboxamide (IB-MECA) against myocardial stunning are mediated by the A1AR.
      
The potential of99mTc labeled P1, P4-di (adenosine-5')-tetraphosphate (Ap4A) for imaging experimental atherosclerotic plaques was evaluated in New Zealand white (NZW) rabbits.
      
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As reported by Borst and Slater, dinitrophenol (DNP), the uncoupling agent, can under certain conditions cause an inhibition of the oxidation of succinate by intact mitochondria. We have studied this effect of DNP on succinate oxidation by respirationcontrolled rat-liver mitochondria with a vibrating platinum microelectrode and found that, in absence of added inorganic orthophosphate and adenosine diphosphate (ADP), the action of DNP is closely related to its concentrations. When DNP is added in low concentration...

As reported by Borst and Slater, dinitrophenol (DNP), the uncoupling agent, can under certain conditions cause an inhibition of the oxidation of succinate by intact mitochondria. We have studied this effect of DNP on succinate oxidation by respirationcontrolled rat-liver mitochondria with a vibrating platinum microelectrode and found that, in absence of added inorganic orthophosphate and adenosine diphosphate (ADP), the action of DNP is closely related to its concentrations. When DNP is added in low concentration (5-10μM), succinate oxidation is stimulated, the rate of respiration being increased to greater degrees with increasing concentrations of DNP up to 10μM, with no indication of any inhibition. When the DNP concentration is increased to above 30μM, however, stimulation lasts for only a short time, followed by a decrease in the rate of oxidation. The use of still higher concentrations of DNP results in increasingly lower degrees, and shorter periods, of stimulation, followed sooner by inhibition.In agreement with findings reported by other workers, a preincubation with 2mM amytal has been shown to prevent completely the inhibition of succinate oxidation by high concentrations of DNP. In addition, it has been discovered that even after the appearance of the inhibition, amytal is still effective in alleviating the DNP effect, causing a partial restoration of the respiratory rate.The addition of another uncoupling agent, arsenate, in concentrations of 2-10mM also stimulates succinate oxidation and 4mM arsenate is able to elicit a maximal rate of respiration. In contrast with DNP, no inhibition of respiration can.be observed even with the higher concentrations of arsenate used. Nevertheless, after preincubation with 2 mM amytal, the stimulation of respiration caused by arsenate is found to last for only two minutes, thereafter the rate of oxidation begins to decline and respiration gradually becomes inhibited. Adenosine triphosphate (ATP) is able to prevent the appearance of the inhibited phase of succinate oxidation in both cases, i.e. in either DNP or amytal-arsenate treatment, with however, slightly different efficiency. The addition of ATP enables the respiration in presence of high concentrations of DNP to proceed at a good steady rate, not more, however, than 2/3 of the maximal rate obtainable under optimal conditions, while the same amount of ADP is able to abolish completely the inhibiting effect caused by the amytal-arsenate treatment with the ensuing rate of respiration even higher than that elicited by arsenate alone.The characteristics of the inhibiting actions of these two agents on succinate oxidation and the effect of ATP on them have been discussed. The experimental evidence presented is believed to support the postulation that the oxidation of succinate in liver mitochondria requires an intervention of energy. The possibility that under certain experimental conditions, accumulation of oxaloacetate may play a part in affecting the rate of succinate oxidation is considered likely but of only minor importance.

(1)DNP对于琥珀酸氧化的影响随浓度不同而异,低浓度时,呼吸受激活,其程度随浓度升高而增強,不出現抑制現象。高浓度(30μM以上)DNP只引起短时間氧化激活,随即引起抑制,随浓度升高,抑制出現愈早,氧化激活愈小,預先加入Amytal足以防止上述抑制現象,在抑制出現后加入Amytal亦能使琥珀酸氧化部分恢复。(2)砷酸盐激活的琥珀酸氧化仅在有Amytal的条件下,才出現抑制現象。(3)ATP对上述两种情况引起的琥珀酸氧化抑制都具有一定的解除作用。(4)就实驗結果所做分析支持琥珀酸氧化需要能量激活的看法。

A simple method is suggested for the preparation of relatively large quantity of soluble ribonucleic acid from E. coli by direct extraction of the bacterial cells with phenol, followed by fractionation with ammonium sulphate. The product is found to be homogeneous as judged by its electrophoretic and sedimentation patterns. It contains only adenosine as the terminal nucleoside and from the content of which, the chain is estimated to consist of 78 nucleotides. Its ability to accept S~(35)-methionine is...

A simple method is suggested for the preparation of relatively large quantity of soluble ribonucleic acid from E. coli by direct extraction of the bacterial cells with phenol, followed by fractionation with ammonium sulphate. The product is found to be homogeneous as judged by its electrophoretic and sedimentation patterns. It contains only adenosine as the terminal nucleoside and from the content of which, the chain is estimated to consist of 78 nucleotides. Its ability to accept S~(35)-methionine is rather high as compared with that reported in the literature.The chemical and physico-chemical characteristics of the product, such as protein, polysaccharide, deoxyribonucleic acid and nucleo-base contents, specific rotation, ultraviolet absorption spectrum, and atom phosphorus extinction coefficient, have also been determined.

大腸杆菌經酚直接抽提后以硫酸銨处理,可以得到在电泳与超速离心图譜上均一的, 具有单一末端及接受甲硫氨酸活力較高的sRNA。对sRNA制品的蛋白貭,DNA,多糖含量,比旋光度,紫外光吸收光譜,克原子磷消光系数,核碱組成与链长也进行了测定。

As a direct consequence of the interdigitating filament model of the myofibril and the corresponding theory of muscular contraction put forward by H. E. Huxley and A. F. Huxley, it is evident that the muscle would lose its ability to contract if its sarcomere had been stretched to a length greater than the sum of the lengths of primary and secondary filaments. For the honey bee striated muscle, the sum of the lengths of the two sets of filament would be about 5.4μ, on the basis of the interdigitating filament...

As a direct consequence of the interdigitating filament model of the myofibril and the corresponding theory of muscular contraction put forward by H. E. Huxley and A. F. Huxley, it is evident that the muscle would lose its ability to contract if its sarcomere had been stretched to a length greater than the sum of the lengths of primary and secondary filaments. For the honey bee striated muscle, the sum of the lengths of the two sets of filament would be about 5.4μ, on the basis of the interdigitating filament model. The glycerine extracted myofibril of the honey bee indirect flight muscle, however, still preserved its ability to contract under the action of adenosine triphosphate after the sarcomere length had been stretched to beyond 8μ. Furthermore, the microscopical structure changes of the myofibril caused by stretching the sarcomere to beyond 6μ were still reversible. These results seem to indicate that the fine structure of the myofibril of the honey bee striated muscle was probably different from that expressed by the interdigitating filament model.

本文报告我們观察拉长的蜜蜂間接飞翔肌肌原纤維收縮能力的結果。在肌小节长度超过8微米以后,三磷酸腺苷仍能引起肌原紆維的縮短,而且在肌小节拉长后出現的結构变化也是可逆的。这些結果不能在H.E.Huxley和A.F.Huxley提出的肌原紆維由两組蛋白細絲构成的模型和收縮的細絲滑行理論的基础上来解释。

 
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