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intraventricular
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  脑室
     Results: Mean intraventricular hemorrhage volume was(45.17±23.12)ml in 30 patients,with mean intracerebral hrmorrhage of(8.17±5.52)ml in 13 of them.
     结果:30例患者平均脑室出血量为(45.17±23.12)m l,有13例合并脑内血肿的平均出血量为(8.17±5.52)m l。
短句来源
     of the 3 cases of intraventricular hemorrhage(14%),2 combined with HIE;
     脑室内出血3例(7%),2例合并HIE;
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     Effect of intraventricular administration of insulin on expression of Bcl-2 protein and the apoptosis of neuron in rat hippocampal CA1 region
     脑室内注射胰岛素对大鼠全脑缺血后海马CA1区Bcl-2蛋白表达及神经元凋亡的影响
短句来源
     Results Mean intraventricular hemorrhage volume was 35.17±23.12 cm 3 in 30 patients,with mean intracerebral hemorrhage of 8.17±5.52 cm 3 in 17 of them.
     结果 全组平均脑室出血量为 (35 17± 2 3 12 )cm3,且 17例合并脑内血肿的平均脑内血肿出血量为 (8 17± 5 5 2 )cm3。
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     The effect of intraventricular administration of insulin on expression of Bcl-xL protein and apoptosis of neuron in rat hippocampal CA1 region
     脑室内注射胰岛素对全脑缺血后大鼠海马CA1区Bcl-xL蛋白表达及神经元凋亡的影响
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  “intraventricular”译为未确定词的双语例句
     Effects of Intraventricular Injection of Calmodulin Antagonist(Trifluoperazine)on Pregnancy in Rats
     Effects of Intraventricular Injection of Calmodulin Antagonist (Trifluoperazine)on Pregnancy in Rats
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     Albendazole trial at 15 or 30 mg/kg/day for subarachnoid and intraventricular cysticercosis
     丙硫咪唑15或30mg/(kg·d)治疗蛛网膜下腔和脑室内囊虫病的试验
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     (4) The excitatory response of PGL neurons to intraventricular injection or epicardial application of adenosine was completely inhibited by pretreatment with selective adenosine Al-receptor antagonist 8-cyclopentyl-1, 3-dipropyhanthine (DPCPX, 500μg/kg).
     (4)静脉注射选择性腺苷A1受体拮抗8-cyclopentyl-1,3-dipropylxanthine(DPCPX,500μg/kg)可完全阻断腺苷对PGL神经元的兴奋效应。
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     Dynamic Study on Effect of Intraventricular Administration of pLXSN-bcl-2 cDNA on Apoptosis of Neural Cells, the Volume of Cerebral Infarction and Gene Expression of bcl-2 and Bax Following Focal Cerebral Ischemia in Rats
     侧脑室注射pLXSN-bcl-2 cDNA对局灶性脑缺血大鼠神经细胞凋亡、脑梗死体积及bcl-2/bax蛋白表达影响的动态研究
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     The results showed that there were a significant decrease in left ventricular systolic pressure(LVSP), maximum rising and falling rate of left intraventricular pressure(LV±dp/dtmax) and density of ventrical βARs.
     结果表明,烫伤后大鼠左心室压力(LVSP)、左心室压力最大上升速率和最大下降速率(LV±dp/dtmax)及心室β受体密度明显减低,相关分析表明,β受体密度变化与LVSP、LV±dp/dtmax变化呈高度正相关。
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  相似匹配句对
     Intraventricular hemorrhage
     脑室内出血
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     Traumatic Intraventricular Hemorrhage
     创伤性脑室内出血50例分析
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  intraventricular
She also showed severe atrio-ventricular and intraventricular conduction disorders, so that repeated resuscitation was necessary.
      
The limited number of controlled, randomized studies in spontaneous intracerebral hemorrhage severly limit positive recommendations for any intervention, for blood pressure management or for intraventricular fibrinolysis.
      
g., intravenous intravenous administration of hyperosmolar agents (mannitol) and external intraventricular drainage, are used in addition to antibiotics.
      
Exercise capacity, oxygen uptake and the cardiac index increased, intraventricular gradient, the enddiastolic pressure of the left ventricle and the left atrial size decreased.
      
???None of the pts with normal findings at the EPS showed abnormal p-waves, an AV block, or an intraventricular conduction disturbance during the follow-up.
      
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(1) In mice, intraperitoneal injection of corydalis B (dl-tetrahydropalmatine), 40 mg/kg, potentiated hexobarbital narcosis, and reduced the passive and spontaneous activity in the rotarod, rolling cylinder and photo-electric cage experiments. The hyperactivity induced by small doses of amphetamine and the amphetamine toxicity in aggregated mice were both significantly reduced by this drug. On the motor defense conditioned reflexes in.cat corydalis B had no marked influence in subcutaneous doses of 10—20 mg/kg....

(1) In mice, intraperitoneal injection of corydalis B (dl-tetrahydropalmatine), 40 mg/kg, potentiated hexobarbital narcosis, and reduced the passive and spontaneous activity in the rotarod, rolling cylinder and photo-electric cage experiments. The hyperactivity induced by small doses of amphetamine and the amphetamine toxicity in aggregated mice were both significantly reduced by this drug. On the motor defense conditioned reflexes in.cat corydalis B had no marked influence in subcutaneous doses of 10—20 mg/kg. At the dosage of 30 mg/kg, the cats exhibited at first excitant behavior (running, jumping, growling etc.) and then depression set in with almost complete disappearance of conditioned and unconditioned reflexes. At the intraperitoneal dosage of 10 mg/kg, the conditioned reflex was abolished partially, whilst the unconditioned reflex remained unaffected. In cats, an intraventricular injection of corydalis B, 1—2 mg/kg, caused changes in aggressive behavior, such as hissing. The cats became calm and drowsy, but could be easily awaken by external stimuli. In normal rabbits, spontaneous electric activity of cerebral cortex was recorded. The low voltage fast activity was the main element in EEG, sometimes α-wave and spindles were presented. Within 3—7 minutes following single intravenous injection of 15—20 mg/kg, the high voltage slow waves (1.5—3 c/s, 250—400 μv) were found, and maintained 5—15 minutes.. At the same time the arousal response in EEG evoked by stimulation on skin with direct current was inhibited. Thereafter, in the lapse of 20—40 minutes, EEG recovered gradually. (2) In mice, corydalis B aggravated the strychnine convulsions, but inhibited the metrazol convulsions. It had some synergetic action with phenytoin. (3) A single subcutaneous injection of 40 mg/kg of corydalis B could antagonize emesis induced by 0.1—0.2 mg/kg of apomorphine, but failed to do so when the dosage of the latter was raised to 1 mg/kg. The emesis caused by copper sulfate was not antagonized by corydalis B. (4) In rats, corydalis B produced a slight hypothermia.

(一)延胡索素乙与环己烯巴比妥钠有协同作用,可以减低小白鼠的被动性和自发性活动;它能对抗小量苯丙胺的兴奋现象,又能明显降低大剂量苯丙胺的毒性反应。它们的有效剂量为20—40毫克/公斤。皮下注射乙素10—30毫克/公斤可使猫先兴奋后抑制,较大剂量对防御性条件反射和非条件反射均能抑制,该作用持续2—3小时以上;腹腔给药时作用情况相似。从侧脑室注射乙素1—2毫克/公斤可使猫消除恐惧跳避的行为,出现瞌睡现象,但易为外界刺激所唤醒。正常家兔的皮层脑电波以低电压快波为主,静脉注射乙素15—20毫克/公斤可使其变成高电压慢波,此时对感应电流刺激皮肤的惊醒反应明显消灭,约历40分钟乙素作用消失。 (二)乙素可使士的宁容易产生惊厥,伹能抑制五甲烯四氮唑的惊厥作用。它不能对抗电休克的发生,略能协同苯妥英钠作用。 (三)乙素略有中枢性的镇吐作用,略能降低体温。

Analgesia began 1—4 minutes after intraventricular injections in the rabbit of 20 μg (about or less than 1/500 of i. v. dose) of morphine-HCl and lasted for 1—3 hours, as studied by a modified method of radiant heat focussing on the skin of the nose or of the thigh. 50 μg of morphine topically applied to the somato-sensory cortex of both sides was ineffective, and the same dose injected subarachnoidly into spinal lumbar region could not eliminate the hind leg reaction. When 5 or 10 μg of morphine in 0.002...

Analgesia began 1—4 minutes after intraventricular injections in the rabbit of 20 μg (about or less than 1/500 of i. v. dose) of morphine-HCl and lasted for 1—3 hours, as studied by a modified method of radiant heat focussing on the skin of the nose or of the thigh. 50 μg of morphine topically applied to the somato-sensory cortex of both sides was ineffective, and the same dose injected subarachnoidly into spinal lumbar region could not eliminate the hind leg reaction. When 5 or 10 μg of morphine in 0.002 ml was applied by a microinjector into the periventricular grey matter of both sides of the brain substance in the vicinity of the third ventricle, strong analgesic effect was observed. This action could be antagonised by intravenous nalorphine. When morphine was injected into the other sites including the midbrain reticular formation, nucleus ventralis posteromedialis and nucleus dorsomedialis of the thalamus, caudate nucleus, septum pellucidum, dorso-medial region of the medial geniculate body, etc., no definite analgesic action could be demonstrated. These results indicate that the central grey matter in the medial portion of the brain stem surrounding the third ventricle may be the main, if not the sole, site of the analgesic action of morphine.

(一)本文报告利用家兔脑室內注射和脑內微量注射方法,在保持中枢神經系統完整的条件下,研究嗎啡鎮痛作用部位的結果。 (二)脑室內注射20微克(相当于靜脉剂量的1/500—1/1000左右)可以产生明显而持久的鎮痛作用。注射后1—4分钟即表現作用。根据药液分布的范围等推測作用部位在第四脑室以上水平。 (三)脑室內注射嗎啡对光热刺激鼻部或后肢的痛反应都有效,对电刺激牙髓的痛反应也有效。脑室內注射多种其他药物不产生鎮痛作用。因此鎮痛是全身性的,而且是嗎啡特异作用的表現。 (四)脑內微量注射嗎啡以第三脑室周围灰貭作用最明显,10微克剂量即有鎮痛作用。側脑室壁的尾状核、透明隔以及皮貭下其他部位,注射較大剂量仍无鎮痛作用。 (五)本文提出第三脑室壁灰貭是嗎啡发揮鎮痛的作用部位的新的观点。

In rats and rabbits, the crude venom of A. halys in dosages of 1mg/kg or more given intramuscularly or intravenously caused death from respiratory failure. It was found that when the respiratory movement of the diaphragm had disappeared, the phrenic nerve still showed respiratory discharges. Intraventricular injection of the venom in rather large doses, e. g. 30~50μg/kg, into rabbits did not cause any respiratory distress. On the isolated rat phrenic-diaphragm preparation, the venom at a concentration...

In rats and rabbits, the crude venom of A. halys in dosages of 1mg/kg or more given intramuscularly or intravenously caused death from respiratory failure. It was found that when the respiratory movement of the diaphragm had disappeared, the phrenic nerve still showed respiratory discharges. Intraventricular injection of the venom in rather large doses, e. g. 30~50μg/kg, into rabbits did not cause any respiratory distress. On the isolated rat phrenic-diaphragm preparation, the venom at a concentration of 300μg/ml was found to block the neuromuscular transmission irreversibly.These observations suggested that the respiratory failure caused by the venom of A. halys is due to neuromuscular block.

用记录膈神经和膈肌的呼吸性发放的方法,对蝮蛇毒急性中毒引起的呼吸麻痹,在家兔和大白鼠上进行了实验分析。将稍大于最小致死量(1毫克/公斤体重)的蝮蛇毒,经静脉或肌肉注入动物体内,一般经数小时后出现软瘫性呼吸麻痹,若及时地给予人工呼吸,则在膈肌的呼吸性发放完全停止的情况下,心电和膈神经的呼吸发放都尚可维持相当长的时间。若将蝮蛇毒直接注入侧脑室内,甚至达每公斤体重30~50微克,既不引起动物出现明显的呼吸困难,亦未导致动物死亡。若将脑室注射量加大到每公斤体重100微克以上,则动物经一定时间出现四肢强直、头后仰,抽搐、流涎等症状而死亡。在离体标本上,蝮蛇毒具有不可逆地阻遏接头传递作用,并且这种阻遏的发展过程是缓慢的,和银环蛇毒素乙、肉毒杆菌毒素对接头传递的阻遏过程相似。

 
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