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j point
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  j点
     Compared to Group MI/R, J point of ECG and heartrate decreased smoothly in Group MI/R+GLY (P<0.05, P<0.01).
     MI/R+Gly组在不同时点的J点及心率变化幅度较小,显著小于MI/R组(P<0.05,P<0.01);
短句来源
     Study About the Rates of J Point of ECG Q-X/Q-T and Q-T Tmmediately After Exercise
     运动后即刻ECG的J点Q-X/Q-T及Q-T比值的研究
短句来源
     Method The routine 12 channels and V 3R ~V 7R leads were used. connecting 2 consecutive TP lines of QRS complex as the base line, the amplitudes of ST elevation at 80 ms post J point of each lead of Ⅱ. Ⅲ.
     方法 描记常规 12导联和V3R~V7R,以连续 2个QRS波群的TP连线为基线 ,测量Ⅱ ,Ⅲ ,V3R V7R导联J点后 80ms处ST段抬高的幅度。
短句来源
     When the excursion of ST segment at 0.08 seconds after J point is over 0.1mV, it is judged as positive. The sensitivity,specificity and accuracy of the test were 75.7%,78.7% and 76.6% respectively.
     判定标准以J点后0.08秒ST段偏移0.1mV以上为阳性,诊断冠心病的敏感性,特异性、准确度分别为75.7%,78.7%和76.6%。
短句来源
     The depression of ST segment was measure 80 ms after J point by computer.
     ST段下移测量点为J点后80 ms 处。
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  “j point”译为未确定词的双语例句
     Results The change of ECG J point with ∑△ J of V 3 was most pronounced in ISO-injected rats. Pretreatment with radix astragali reduced V 3(∑△ J)and CPK-MB elevation induced by ISO.
     结果 皮下注射异丙肾上腺素后V3(∑△J)显著增大 ,黄芪注射液组V3(∑△J)及CPK MB含量均较生理盐水组显著降低。
短句来源
     The density of DNFOA was determined to be 1.90 g·cm -3 by suspension method. The values of the standard enthalpy,the detonation velocity and the explosive pressure at C J point were calculated to be 303.6 kJ·mol -1 ,8 559.6 m·s -1 ,33.6 GPa, respectively.
     用悬浮法测定DNFOA密度为1.90 g·cm-3 ,计算其标准生成焓为 3 0 3 .6kJ·mol-1,爆速 85 5 9.6m·s-1,爆压 3 3 .6GPa。
短句来源
  相似匹配句对
     Point of view
     观点
短句来源
     The boiling point (b. p.)
     脂肪醇沸点(b.p.)
短句来源
     Access Point of Subjects
     主题检索点新探
短句来源
     Clinical Application of Jianjing Point
     肩井穴的临床应用
短句来源
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  j point
The criterion for ischemia was ST segment depressions of 1 mm or greater for 40 seconds or more 60 msec after the J point.
      
The algorithm determines the T onset by looking for a point of inflection between the J point and the T peak.
      
These fiducial points are, iso-electric level, the J point, and onsets and offsets of the QRS complex and T wave.
      
The neural networks then address the approximate locations of the J point and the onset of the T-wave (Ton).
      
The early repolarization syndrome, most commonly seen in healthy young individuals, is characterized by a ST-segment elevation of 1 to 4 mm in the mid-precordial leads with a notched and elevated J point in lead V4.
      
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Experiments were performed in the curarized rats, under the artificial respiration. After stimulation of the cantral gray of the midbrain with electrical impulses of frequency 100 Hz, 20 impulses per tran, duration 0.5 msec, and strength 16—32 volts, ventricular tachycardia, junction arrhythmia sino-bradycardia, and J point declined were observed. Ventricular tachycardia could be prevented by administration of propranolol, and transection of the first cervical cord, but it could not be abolished by administration...

Experiments were performed in the curarized rats, under the artificial respiration. After stimulation of the cantral gray of the midbrain with electrical impulses of frequency 100 Hz, 20 impulses per tran, duration 0.5 msec, and strength 16—32 volts, ventricular tachycardia, junction arrhythmia sino-bradycardia, and J point declined were observed. Ventricular tachycardia could be prevented by administration of propranolol, and transection of the first cervical cord, but it could not be abolished by administration of atropine, and section of vagus nervers. It suggested that ventricular tachycardia was possibly due to the sympathetic nerves, Junctional arrhythmia was able to be blocked by admi ninstration of atropine. Sino-bradycardia could be blocked by atropine, and section of vagus nervees. It suggested that both of these types of arrhythmia would be evoked by vagus nerves. J point declined could not be prevented by atropine, propranolol, and was not able to be abolished by secion of vagus nerves. Either, however, it was able to be blocked by transection of the cervical cord. It showed that J point declined was probably elicited by the adrenergic a receptor. The tonic cardiac sympathetic, and vagus discharges were observed. The normal cardiac function, is accomplished, under the interaction of sympathietc and vagus effects. Either of them was hyperfunction, or hypofunction may elicite various arrhythmias.

刺激大白鼠中脑中央灰质区可引起室性心动过速、交界性节律、窦性心动过缓和J点下移等心电图变化。刺激大白鼠中脑中央灰质区出现的室性心动过速是由于交感神经的作用。交界性节律与窦性心动过缓是由于迷走神经的作用。J点下移可能是肾上腺素能α受休的作用。正常的心脏活动是在迷走神经与交感神经相互对抗和相辅相成的作用下维持的,任何一方的减弱或亢进都会导致不同类型的心律失常。

Experiments were performed in the curarized rats, under the artificial respiration. After stimulation of the cantral gray of the midbrain with electrical impulses of frequency 100 Hz, 20 impulses per train, duration 0.5 msec, and strength 16鈥?2 volts, ventricular tachycardia, junction.al arrhythmia sino-bradycardia, and J point declined were observed. Ventricular tachycardia could be prevented by administration of propranolol, and transection of the first cervical cord, but it could not be abolished by...

Experiments were performed in the curarized rats, under the artificial respiration. After stimulation of the cantral gray of the midbrain with electrical impulses of frequency 100 Hz, 20 impulses per train, duration 0.5 msec, and strength 16鈥?2 volts, ventricular tachycardia, junction.al arrhythmia sino-bradycardia, and J point declined were observed. Ventricular tachycardia could be prevented by administration of propranolol, and transection of the first cervical cord, but it could not be abolished by administration of atropine, and section of vagus nervers. It suggested that ventricular tachycardia was possibly due to the sympathetic nerves. Junctional arrhythmia was able to be blocked by admininstration of atropine. Sino-bradycardia could be blocked by atropine, and section of vagus nerves. It suggested that both of these types of arrhythmia would be evoked by vagus nerves. J point declined could not be prevented by atropine, propranolol, and was not able to be abolished by secion of vagus nerves. Either, however, it was able to be blocked by transection of the cervical cord. It showed that J point declined was probably elicited by the adrenergic a receptor. The tonic cardiac sympathetic, and vagus discharges were observed. The normal cardiac function is accomplished, under the interaction of sympathietc and vagus effects. Either of them was hyperfunction, or hypofunction may elicite various arrhythmias.

刺激大白鼠中脑中央灰质区可引起室性心动过速、交界性节律、窦性心动过缓和J点下移等心电图变化。刺激大白鼠中脑中央灰质区出现的室性心动过速是由于交感神经的作用。交界性节律与窦性心动过缓是由于迷走神经的作用。J点下移可能是肾上腺素能α受体的作用。正常的心脏活动是在迷走神经与交感神经相互对抗和相辅相成的作用下维持的,任何一方的减弱或亢进都会导致不同类型的心律失常。

The results of blood pressure, heart rate and ECG changes during maximal bicycle exercise test in 223 healthy school children are reported. During the maximal exercise, 71 children (31.8%) showed ECG changes, including T wave changes, ST depression (<2mm) and J point depression, but none had arrhythemia. Systolic BP increased on the average, 35.8 mmHg in 115 boys and 32.5 mmHg in 108 girls. Diastolic BP was only slightly altered. The slowing of the heart rate was most prominent 1 min after cessation of...

The results of blood pressure, heart rate and ECG changes during maximal bicycle exercise test in 223 healthy school children are reported. During the maximal exercise, 71 children (31.8%) showed ECG changes, including T wave changes, ST depression (<2mm) and J point depression, but none had arrhythemia. Systolic BP increased on the average, 35.8 mmHg in 115 boys and 32.5 mmHg in 108 girls. Diastolic BP was only slightly altered. The slowing of the heart rate was most prominent 1 min after cessation of exercise. Eight min after exercise, the cardiac rate of only 22 children (20%)returned to pre-exercfse level, the mean rate of the remaining 201 children was still faster than that at rest by 18.7%.

本文报道223名健康儿童踏车运动试验,以观察心率、血压及心电图变化的结果。在极量运动过程中,心电图有变化者71名(31.8%),包括T波改变、ST段压低、J点下降等,未出现ST压低≥2mm或出现早搏等心律失常。极量运动时,男孩组收缩压平均升高35.8mmHg;女孩组32.5mmHg,舒张压改变不一,停止运动后1分钟心率减慢最显著,8分钟时全组平均心率仍较运动前增加18.7%。最后作者根据踏车运动试验的方法加以讨论。

 
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