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hypoxic ischemic brain
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  缺氧缺血脑
     Objective To study the effects of 17β estradiol (17β E 2) on extracellular glutamate(Glu) level post hypoxic ischemic brain damage(HIBD) in neonatal rats and to explore whether 17β E 2 has some protective effects on HIBD.
     目的 研究 17β 雌二醇 (17β E2 )对新生大鼠缺氧缺血脑损伤 (HIBD)后谷氨酸 (Glu)释放的影响 ,探讨 17β E2 是否对HIBD有保护作用。
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     Objective This study examined the expression of insulin-like growth factor-1(IGF-1)and its receptor in 3-day-old premature rats with chronic hypoxic ischemic brain damage (HIBD) and investigated the role of IGF-1 in the pathogenesis of this disease.
     目的探讨胰岛素样生长因子-1(IGF-1)在3日龄未成熟大鼠慢性缺氧缺血脑损伤发病机制中的作用。
短句来源
     Conclusions Premature rats with hypoxic ischemic brain damage showed obvious oligodendrocyte damage in brain white matter. It is helpful to understand the pathologic change of premature infants progressively.
     结论3日龄未成熟新生大鼠缺氧缺血脑损伤后,出现以脑白质少突胶质细胞损伤为主的病变,有助于理解早产儿脑损伤发病机制和神经病理变化,并可作为研究早产儿缺氧缺血脑损伤的动物模型。
短句来源
     Parenteral solution of rhEPO (10 U/g) and saline at the same volume were injected into rats through intraperitoneal injection immediately after operation. Mixture of oxygen with volume fraction of 0.08 and nitrogen fume with volume fraction of 0.92 was sucked in rats of the two groups 2 hours after operation to establish neonatal rat models with hypoxic ischemic brain injury and models in the treatment of EPO.
     ②促红细胞生成素组和模型组大鼠结扎右颈动脉,手术后即刻分别腹腔内注射重组人红细胞生成素注射液(10U/g)及同体积生理盐水,手术后2h两组大鼠吸入体积分数为0.08氧气和体积分数为0.92氮气的混合气体2h,建立缺氧缺血脑损伤新生鼠模型及促红细胞生成素治疗模型。
短句来源
     AIM To study the protective effects of monosialotetrahexosylganglioside (GM1) after hypoxic ischemic brain damage (HIBD) in neonatal rats.
     目的 研究外源性单唾液酸四己糖神经节苷脂(GM1)对新生鼠缺氧缺血脑损伤 (HIBD)的保护作用 .
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  “hypoxic ischemic brain”译为未确定词的双语例句
     Expression of NgR mRNA and NgR Protein in Brain Tissues of Newborn Rats with Hypoxic Ischemic Brain Damage
     新生鼠缺氧缺血性脑损伤脑组织NgR mRNA和NgR蛋白表达及意义
短句来源
     The expression of bc1-2 gene in hippocampus area of neonatal rats with hypoxic ischemic brain damage
     新生大鼠脑缺血时海马区bc1-2基因的表达
短句来源
     AIM: To study the expression of HSP70 and pathologic damage in hippocampal CA 1 after hypoxic ischemic brain damage(HIBD)in neonatal rats and the effect of monosialotetrahexosylganglioside(GM 1)on it .
     目的 :研究新生大鼠缺氧缺血性脑损伤 (HIBD)后海马CA1区 70ku热休克蛋白 (HSP70 )表达、病理学损伤变化和外源性单唾液酸四已唐神经节苷脂 (GM1)对HSP70表达及病理学损伤的影响 .
短句来源
     【Methods】 80 seven-day-old Wistar rats were divided into 10 groups(8 for each) randomly as below: normal control group,fake surgery group and hypoxic ischemic brain damage(HIBD) model group-1,3,7,10,12,24,48,72 h.
     【方法】将80只新生7日龄Wistar大鼠随机分为10组(即正常对照组、假手术组和HIBD模型组-1、3、7、10、12、24、48、72 h),每组8只。
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     Protective effects of the MK801 on the hypoxic ischemic brain injury
     MK801对缺氧缺血性脑损伤的保护作用
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  相似匹配句对
     Brain hypoxic and ischemic proconditioning
     脑缺血缺氧预处理
短句来源
     Hypoxic Ischemic Encephalopathy and Apoptosis
     缺氧缺血型脑病与细胞凋亡
短句来源
     Erythropoietinand Hypoxic-ischemic Brain Damage
     促红细胞生成素与缺氧缺血性脑损伤
短句来源
     Hypoxic and ischemic is the most important reason.
     缺氧缺血是HIE发生的本质。
短句来源
     Caspase and hypoxic ischemic brain damage
     半胱天冬酶与缺氧缺血性脑损伤
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  hypoxic ischemic brain
Although autophagy generally prevents cell death, our recent study using conditional Atg7-deficient mice in CNS tissue has demonstrated the presence of autophagic neuron death in the hippocampus after neonatal hypoxic/ischemic brain injury.
      
A neuropathological study of 41 forensic autopsy cases of hypoxic/ischemic brain damage has been undertaken, using immunohistochemical staining to detect the 70-kDa heat shock protein (hsp70) and the status of the glial cells.
      
These neuroimaging findings included acute perinatal focal arterial infarction, hypoxic-ischemic brain injury, and intracranial hemorrhage.
      
It was later determined that plaintiff Mejia had suffered brain damage due to a hypoxic-ischemic brain injury during labor.
      
Excitotoxicity, oxidative stress, and apoptosis comprise the major routes to neuronal death after hypoxic-ischemic brain injury.
      
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in recent years,the results of many experiments show that excitatory amino acids such as glutamate and aspartate mediate hypoxic-ischemic brain damage by its receptors,and excitatory amino acid antagonists can prevent hypoxic-ischemic brain damage.This experiment studied the hypoxia model in murine cortical cell culture,the neuronal injury caused by hypoxia and excitatory amino acid agonist NMDA,and the preventive effect of NMDA antagonists CPP,Ket and non-NMDA antagonist...

in recent years,the results of many experiments show that excitatory amino acids such as glutamate and aspartate mediate hypoxic-ischemic brain damage by its receptors,and excitatory amino acid antagonists can prevent hypoxic-ischemic brain damage.This experiment studied the hypoxia model in murine cortical cell culture,the neuronal injury caused by hypoxia and excitatory amino acid agonist NMDA,and the preventive effect of NMDA antagonists CPP,Ket and non-NMDA antagonist NBQX.The result indicated that (1)the NMDA receptor of excitatory amino acids mediated hypoxia neuronal damage;(2)both NMDA antagonists CPP,Ket and non-NMDA antagonist NBQX prevented neurons from hypoxia injury. Our study suggests that excitatory amino acid antagonists will be the new clinical therapeutic passway of brain ischemia.

近年来,许多研究的结果显示,兴奋性氨基酸谷氨酸、天冬氨酸等通过其受体介导了缺氧、缺血性脑损伤,同时兴奋性氨基酸受体拮抗剂对缺氧、缺血性脑损伤有一定的保护作用。本文研究了离体培养的小鼠皮层神经细胞的缺氧模型,并观察缺氧和兴奋性氨基酸受体激动剂N-甲基-D-天冬氨酸(NMDA)对神经细胞的损伤作用,同时应用兴奋性氨基酸NMDA受体拮抗剂CPP、Ket,及非NMDA受体拮抗剂NBQX观察其对缺氧及NMDA毒性损伤的保护作用。结果表明:(1)兴奋性氨基酸NMDA受体参与介导了缺氧引起的神经元损伤,(2)NMDA受体拮抗剂CPP、Ket和非NMDA拮抗剂NBQX对缺氧引起的神经元损伤均有良好的保护作用。研究结果提示,兴奋性氨基酸受体拮抗剂有望成为临床脑缺血治疗的一个很有价值的途径。

bjective To determine the effect of high risk obstetric factors on neonatal hypoxic-ischemic brain injuries. The obstetric factors were investlgated,including maternal complications during pregnancy and labor,the mcde of delivery. Methods Intensive B-ultrasound brain monitoring in 211 newborns within the first 48 hours of life was carried out.The abnormalities were followed up by B-ultrasound.We assessed the extent of hypo- xic-ischemic brain injuries with two kind of types,mild...

bjective To determine the effect of high risk obstetric factors on neonatal hypoxic-ischemic brain injuries. The obstetric factors were investlgated,including maternal complications during pregnancy and labor,the mcde of delivery. Methods Intensive B-ultrasound brain monitoring in 211 newborns within the first 48 hours of life was carried out.The abnormalities were followed up by B-ultrasound.We assessed the extent of hypo- xic-ischemic brain injuries with two kind of types,mild and severe. The mild type included 1-2 degree intracranial hemorrhage and the local hypoxic-ischemic encephalopathy. The severe type included 3-4 degree intracranial hemorrhage and extensive cerebral edema. Results Thirty-nine cases(18. 5%)were diagnosed intracranial hemorrhage and 22 cases(10.4%)vvere diagnosed hypoxic-ischemic encephalopathy(HIE)or cerebral edema. The total positive rate was 28.9%。Of the positive cases,67.2%were mild brain injuries without clinical symptoms. Those cases need no medical treatment and recovered in a natural course. The rates of brain injuries in groups of pregnancy induced hypertension,fetal distress, neonatal asphyxia and premature newborns,were 46.1%,48.9%,66.7%and 71.4%respectively.It suggested that high risk obstetric factors were closely related to neonatal brain injuries. 15.9%of positive cases were from normal mothers with- out any obstetric complications. The brain injuries in those cases were mild.Conclusions The study showed that as a non-invasive procedure,B ultrasound brain examination is necessary for newborns with perinatal high risk factors of brain injuries. The study also suggested that perinatal care and systematic fetal monitoring were key-points for reduction of neonatal brain injuries.

为从母亲孕期、产时合并症及分娩方式等方面探讨引起新生儿缺氧性脑损伤的高危因素,对211例新生儿于出生后48小时内进行颅脑8超检查。结果:新生儿颅内出血39例(18.5%),脑缺氧缺血改变、脑水肿22例(10.4%),总阳性率28.9%。其中轻型脑损伤67.2%,无临床症状,不需处理即可自行恢复。母亲好高征患儿、胎儿宫内窘迫、新生儿窒息及早产儿的颅脑8超阳性率依次为46.1%、48.9%、66.7%和71.4%。围产期无任何合并症的新生儿颅脑B超阳性的仍有15.9%,但脑损伤程度较轻。提示:好高征、胎儿窘迫及早产系缺氧性脑损伤的主要高危因素,脑损伤程度较重。对有围产期高危因素的新生儿行颅脑B超的筛查是必要的。

To search for the changes of serum neurospecific enolase (NSE) concentration and cranial CT sean in the neonatal hypoxic ischemic encephalopathy (HIE).Methods Twenty neonates of HIE were examined.The concentration of serum NSE was measured by enzyme-linked immunosorbent assay (ELISA) of 3 days and 7 days of life.The cranial CT sean was examined in the first week of life.Results The concentrations of serum NSE at 3 days of life increased in 20 HIE neonates, especilly in moderate and heavy ones, and...

To search for the changes of serum neurospecific enolase (NSE) concentration and cranial CT sean in the neonatal hypoxic ischemic encephalopathy (HIE).Methods Twenty neonates of HIE were examined.The concentration of serum NSE was measured by enzyme-linked immunosorbent assay (ELISA) of 3 days and 7 days of life.The cranial CT sean was examined in the first week of life.Results The concentrations of serum NSE at 3 days of life increased in 20 HIE neonates, especilly in moderate and heavy ones, and they were identical with the clinical manifestation.The cramal CT scan showed no differences with the clinical manifestation in them in the light and moderate ones.Conclusions NSE is a reliable marker for early diagnosis of HIE and estimate of hypoxic-ischemic brain damage. The cranial CT saan together with serum NSf are much help for the diagnosis and cure of HIE.

目的探讨血清神经元特异性烯醇化酶(NSE)和头颅CT在新生儿缺氧缺血性脑病(HIE)诊断中的作用。方法HIR患儿20例,用酶联免疫法测定生后8天、7天血清NSE浓度。生后1周内行头颅CT检查。结果HIE患儿血清NSE在生后3天均升高,尤以中、重度明显,与临床分度一致。重度HIE患儿头颅CT分度与临床一致,轻、中度头颅CT分度与临床不平行。结论血清NSE测定是早期诊断HIE及判断脑损伤的有效指标,头颅CT检查结合血清NSE测定可更为准确地帮助HIE的诊断和治疗。

 
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