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   p 73 genes 的翻译结果: 查询用时:0.01秒
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p genes
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  p73基因
     Hypermethylation of RASSF1A、HIC1 and p73 Genes in Hepatocellular Carcinomas
     肝细胞癌中RASSF1A、HIC1和p73基因启动子异常甲基化
短句来源
     The inactivation of transcription in p73 genes was associated with the methylations of CpG in exon 1 of p73.There was no mutations and deletions in p73 genes.
     p73基因转录失活与p73基因外显子1CpG岛高甲基化有关,不存在基因的突变和缺失;
短句来源
     Hypermethylation of Ras association domain family protein 1A,hypermethylated in cancer 1 and p73 genes in hepatocellular carcinoma
     肝细胞癌组织中RAS相关区域家族蛋白1A、肿瘤高甲基化基因1和P73基因的异常甲基化
短句来源
     The abnormalities of p73 genes may not be important molecular events in the progression of MM.
     p73基因异常可能在MM的发生、发展中不是一个重要的分子事件。
短句来源
     Objective To evaluate the status of promoter hypermethylation of Ras association domain family protein 1A(RASSF1A),hypermethylated in cancer 1(HIC1) and p73 genes in hepatocellular carcinoma(HCC) and to explore the correlation with clinicopathological features.
     目的探讨肝细胞癌中RAS相关区域家族蛋白1A(RASSF1A)、肿瘤高甲基化基因1(H IC1)和P73基因启动子异常甲基化状况及其与患者临床和病理因素之间的关系。
短句来源
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  “p 73 genes”译为未确定词的双语例句
     Alterations of the p53 and p73 Genes in Lympho-plasmacytic Diseases
     淋巴-浆细胞疾病中p53、p73抑癌基因的研究
短句来源
     Conclusion:Both PTEN and P73 genes may play an important role in the development of EHCC.
     尚不能认为PTENmRNA,P73mRNA在肝外胆管癌的表达两者有相关关系。
短句来源
  相似匹配句对
     Jovial (J73) to C Translator
     JOVIAL(J73)到C的转换程序
短句来源
     73 references were quoted.
     引用文献73篇。
短句来源
     Expressionofmorethantwokindsofoncogeneproteinswerepresentin73casesofbladdertransitionalcelcarcinoma.
     73例膀胱移行细胞癌存在两种以上蛋白共同表达。
短句来源
     p73 Gene and Tumor
     p73基因与肿瘤
短句来源
     cytoskeleton genes ;
     细胞骨架相关基因;
短句来源
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  p genes
Promoter hypermethylation of the RB1, p14ARF, p15INK4b, p16INK4a, p21Waf1/Cip1, p27Kip1, and p73 genes was detected in 12 (35%), 2 (6%), 11 (32%), 20 (59%), 1 (3%), 0 (0%), and 4 (12%) of the adenomas, respectively.
      
We examined expressions of the p63 and p73 genes and proteins in normal biliary epithelia, biliary dysplasias, and EBD carcinomas using immunohistochemistry and RT-PCR analysis.
      
The p63 and p73 genes are rarely mutated in human cancer, although p73 loss is observed in neuroblastoma and a subtype of T-cell lymphoma.
      


A survey of the distribution of ABO,P and Rh blood groups was made among960 primary and middle school students of Tujia Minority in Luota,Xiche andMiaoshi Communes in Longshan county.Slide method was used for the identificationof ABO and P blood groups and direct bromelin method,for Rh blood group.Thedistribution of ABO blood group system in The Minority of Tujia is shown inTable 1.The sequence of the frequencies of phenotypes is A>O>B,AB.As for thegene frequencies,the sequence is O>A>B.In view of...

A survey of the distribution of ABO,P and Rh blood groups was made among960 primary and middle school students of Tujia Minority in Luota,Xiche andMiaoshi Communes in Longshan county.Slide method was used for the identificationof ABO and P blood groups and direct bromelin method,for Rh blood group.Thedistribution of ABO blood group system in The Minority of Tujia is shown inTable 1.The sequence of the frequencies of phenotypes is A>O>B,AB.As for thegene frequencies,the sequence is O>A>B.In view of the frequencies of the pheno-types of P blood group system,P_1 is much more frequent than P_2(Table 2),on thecontrary,the gene frequency P(gene of P_2)is higher than P(gene of P_1)(Table 4).As for the Rh blood group,among 930 investigated individuals,we found only8 phenotypes.The sequence of the frequencies of these phenotypes is CCDee>CcDE>ccDE>CcDee>CCDE>ccDee>CCdE and ccdee(Table 3).The rate of Rh posi-tive is 99.78% and Rh negative,0.22%.The sequence of frequencies of Rh genecomplexes is R' R~e>r>R~o>R~z,while r,r'and r~y are zero(Table 4).Detailedcomparisons are made between our results and those reported by the Shanghai Inst-itute of Biological Products(Table 5 to 8).The probable reason why the frequencyof the phenotype P_1 is much higher than P_2 while the gene frequency is quite thecontrary is discussed in thits paper.

共调查了土家族青年学生960人的 ABO、P 及 Rh 等血型系统,结果表明在 ABO 血型系统中,表现型频率的次序为 A>O>B>AB 型,而基因频率的次序为 O 基因>A 基因>B 基因。P 血型的表现型频率,P_1远高于 P_2,但其基因频率却相反,P_2高于 P_1。在我们调查的930例 Rh 血型中各表现型频率的次序为 CCDee>CcDE>ccDE>CeDee>CCDE>ccDee>CCdE 及 ccdee·Rh阳性占99.78%,Rh 阴性仅占0.22%。基因频率的次序为 R~1>R~2>r>R~0>R~z·r′、r″及置 r~y 为O。我们将调查结果与上海生物制品研究所血型组的调查结果作了较详尽的比较,并指出土家族与各民族间某一血型的表现型分布上的差别以及基因频率的差别的显著性基本上一致,但也有个别不尽相同的情况。此外,本文还讨论了土家族 P 血型 P_1远多于 P_2而 P_1基因频率反较 P_2为小的可能原因。

In order to test the promoter function of two HBV DNA fragments, a soluble cell-free system extracted from Hela celis was used. In the in viiro transcriptional system using the 1.4kb DNA fragment as the templa-te, there were two RNA products whose transcriptional initiation sites were supposed to be at nucleotide 276±5% and 821±5% respectively on the HBV map. The first transcriptional initiation site is identical to the one that is directed by the HBV C gene promoter known before.The rela-tionship between...

In order to test the promoter function of two HBV DNA fragments, a soluble cell-free system extracted from Hela celis was used. In the in viiro transcriptional system using the 1.4kb DNA fragment as the templa-te, there were two RNA products whose transcriptional initiation sites were supposed to be at nucleotide 276±5% and 821±5% respectively on the HBV map. The first transcriptional initiation site is identical to the one that is directed by the HBV C gene promoter known before.The rela-tionship between the location of the second initiation site and the gene open reading frame suggests that the promoter may direct the synthesis of P gene mRNA.The 0.8kb DNA fragment starts from the core structure gene, not in-cluding the regulating sequence. Deducing from the 708±5% nt-long RNA product, the transcriptional initiation site is 588 + 5% on the HBV DNA map. Associated with this RNA start site, there is an ATG codon at po-sition 677 downstream, suggsting that the ATG codon may be a start site of a new open reading frame.

自adr亚型乙型肝炎病毒DNA重组质粒中获得两个DNA片段,用体外转录方法研究启动子的位点。其中1.4kb片段有两个转录产物,其转录起始点分别位于乙型肝炎病毒DNA序列的276±5%位和821±5%位,第一个转录起始点与已报道的乙型肝炎病毒核心抗原基因上游启动子位置一致,第二个转录起始点在888位P基因的起始密码子上游。0.8kb片段自校心抗原结构基因起始密码子ATG以下的序列开始,不含有已知的调控序列,其708±5%核苷酸长的RNA产物,根据其长度计算共转录起始点位于乙型肝炎病毒DNA序列588±5%位,与此位置相关的下游ATG密码子位于677位。

The autho rs found that rhizomania resistant charac tor was controlled by two pairs of dominant genes by our observing on the resistance of differ rent original materials, their cross progenies and test cross progenies to the disease in the breeding of rhizomania resistance. The gene types and genetic niodel of sugarbeet (Beta vulgaris) rhizomania resistance were infered acco rding to the result. Let us assume that"P" and "R"represent the two pairs domnant genes, so the RRPP for immune...

The autho rs found that rhizomania resistant charac tor was controlled by two pairs of dominant genes by our observing on the resistance of differ rent original materials, their cross progenies and test cross progenies to the disease in the breeding of rhizomania resistance. The gene types and genetic niodel of sugarbeet (Beta vulgaris) rhizomania resistance were infered acco rding to the result. Let us assume that"P" and "R"represent the two pairs domnant genes, so the RRPP for immune type; RrPP, RRPp for disease resistanc e typel RrPp, RRpp, rrPP for disease tolerant type Ⅰ; Rrpp, rrp for disease tolerant typeⅡ and rrpp for sensitive type.The cultivated sugarbeet has lost a pair of the dominant gene in their cultivating process. If the sugarbeet lost the "p", we obtain only the stock of disease tolerant type, but it is not possible get the stocks of disease resistant and immune types in our selecting works.However, we wi1l get the disease resistant or immune type, after the sugarbeet will pick up the lost"P"gene by crossing with their ancestors.

在从事甜菜抗丛根病育种工作中,通过对各类育种材料的抗病性观察,及对其互交和测交后代的调查,发现甜菜的抗丛根病性是由两对显性基因控制的。经过一步分析,提出了甜菜抗丛根病的基因型和遗传模式。假定以“R”和“P”代表这两对显性基因,免疫型为RPRP;抗病型为RPRp、RPrP耐病Ⅰ型为RPrp、RpRp、rPrP;耐病Ⅱ型为Rprp、rPrP;敏感型为rprp。推测:糖用甜菜在驯化过程中,丢失了其中的一对显性基因,假设丢失的“P”,那么,只有耐病Ⅰ型(RpRp)、耐病Ⅱ型(Rprp)和敏感型(rprp)。这样,采用选择的方法,只能选育出耐病型品种,不可能选育出抗病型和免疫型的品种。只有通过与其祖先杂交,将丢失的“P”基因找回来,才能育成抗病型和免疫型品种。

 
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