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experimental ischemic brain
相关语句
  实验性脑梗死
     The effect of mild hypothermia on oxygen free radicals following the experimental ischemic brain at defined time window
     亚低温在不同时间窗对实验性脑梗死氧自由基的影响
短句来源
     Objective To explore the effect of mild hypothermia on oxygen free radicals(OFR) following the experimental ischemic brain at defined time window.
     目的 探讨亚低温在不同时间窗对实验性脑梗死氧自由基的影响。
短句来源
  “experimental ischemic brain”译为未确定词的双语例句
     Changes of Aqp4 Expression in Experimental Ischemic Brain Edema and the Intervension Study of Aprotinin
     缺血性脑水肿AQP4表达的动态观察及抑肽酶干预研究
短句来源
     Effects of trypsin inhibitors on experimental ischemic brain edema
     胰蛋白酶抑制剂对实验性缺血性脑水肿的作用
短句来源
     Methods: We established the experimental ischemic brain edema model by line-lock using health Wistar rats in the study.
     方法 采用健康的Wistar大鼠,建立线栓法MCAO模型。
短句来源
     Conclusion Experimental ischemic brain edema could be reduced by 66 000KIU/kg trasylol and 50 000U/kg ulinastatin. 
     结论66000KIU/kg特血乐、50000U/kgUTI具有减轻实验性缺血性脑水肿的作用。
短句来源
     Methods: We established the experimental ischemic brain edema model by line-lock using health Wistar rats in the study.
     方法:采用健康的Wistar大鼠,建立线栓法脑梗死(MCAO)模 型。
短句来源
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     EXPERIMENTAL STUDY ON BRAIN ISCHEMIC DAMAGE
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Objective To study effects of trypsin inhibitors on ischemic brain edema of rat.Methods Brain water content was calculated by dryingweighting method and histopathologic changes were observed by light microscope.Results There was no significant change in brain water content in the groups treated with 55mg/kg SBTI and 165mg/kg SBIT(all P>0.05).In the groups which treated with 66 000KIU/kg trasylol or 50 000U/kg ulinastatin,water content of ischemic rat brain reduced significantly(all...

Objective To study effects of trypsin inhibitors on ischemic brain edema of rat.Methods Brain water content was calculated by dryingweighting method and histopathologic changes were observed by light microscope.Results There was no significant change in brain water content in the groups treated with 55mg/kg SBTI and 165mg/kg SBIT(all P>0.05).In the groups which treated with 66 000KIU/kg trasylol or 50 000U/kg ulinastatin,water content of ischemic rat brain reduced significantly(all P<0.05) and histopathologic changes within ischemia area also attenuated.Conclusion Experimental ischemic brain edema could be reduced by 66 000KIU/kg trasylol and 50 000U/kg ulinastatin.

目的观察各种胰蛋白酶抑制剂对大鼠缺血性脑水肿的作用。方法大鼠脑缺血模型采用线栓法,脑含水量测定采用称重法,常规病理学检查。结果55mg/kg和165mg/kg胰蛋白酶抑制剂(SBTI)对LMCAO3h大鼠脑含水量无显著影响(P均>0.05);66000KIU/kg特血乐、50000U/kg尿胰蛋白酶抑制剂(UTI)可显著降低脑水肿大鼠脑含水量(P均<0.05),并可减轻缺血区神经元变性及间质水肿程度。结论66000KIU/kg特血乐、50000U/kgUTI具有减轻实验性缺血性脑水肿的作用。

Objective To explore the effect of mild hypothermia on oxygen free radicals(OFR) following the experimental ischemic brain at defined time window. Methods 110 Sprague-Dawley(SD) rats were randomly divided into 3 groups:normothermia ischemia group,mild hypothermia ischemia group and sham-operated control group. Normothermia and mild hypothermia ischemia group were then divided into 10 subgroups(0h,2h,4h,6h,7h,8h,9h,10h,11h,12h) respectively sham-operated control group was then divided into normothermia...

Objective To explore the effect of mild hypothermia on oxygen free radicals(OFR) following the experimental ischemic brain at defined time window. Methods 110 Sprague-Dawley(SD) rats were randomly divided into 3 groups:normothermia ischemia group,mild hypothermia ischemia group and sham-operated control group. Normothermia and mild hypothermia ischemia group were then divided into 10 subgroups(0h,2h,4h,6h,7h,8h,9h,10h,11h,12h) respectively sham-operated control group was then divided into normothermia and mild hypothermia subgroup,each subgroup contained 5 rats. In ischemia group,after the left middle cerebral artery occluded(LMCAO) using the suture occlusion technique,the rats were exposed to normothermia or mild hypothermia treatment(33+/-1.0℃) at defined time window. The rats in the control group were also exposed to normothermia or mild hypothermia,12 hours after,the rats were killed. The ischemia brain tissues were separated and the nitric oxide(NO),superoxide dismutase(SOD) and malonyldialdehyde(MDA) activities were assayed. Results (1) The levels of SOD in 0h-9h mild hypothermic treatment subgroups(MHTS) were siganificantly higher than those in all normothermia treatment subgroups(NTS) (P<(0.01) or P<(0.05)). (2)The levels of NO and MDA in MHTS (except for the 12h subgroup) were much lower than that in NTS (P<(0.01) or P<(0.05));(3)The SOD activities in some of the MHTS (0h,2h,4h,6h,7h and 8h subgroups) were significantly higher than that in the 12h MHTS(P<(0.01) or P<(0.05));the levels of NO and MDA in the 0h-10h MHTS were much lower than in the 12h MHTS (P<(0.01) or P<(0.05)). Conclusion It is known that oxygen free radicals(OFR) play an important role in the pathophysiological processes of cerebral ischemia. The mild hypothermia treatment in proper time may decrease the OFR and protect neurons against hypoxia,but the threapeutical time windows of mild hypothermia treatment are not the same to different factions in the ischemia brain tissues.

目的 探讨亚低温在不同时间窗对实验性脑梗死氧自由基的影响。方法 将 110只 SD大鼠随机分成手术组和假手术组 ;手术组又分为亚低温组和常温组 ,再各分为手术后 0 h、2 h、4 h、6 h、7h、8h、9h、10 h、11h、12 h亚组 ;假手术组分为常温组和亚低温组 ;每组均 5只。手术组制备成左侧颈动脉近端栓塞的永久性缺血模型 ,假手术组只暴露左颈动脉不栓塞颈动脉。亚低温各亚组将肛温控制在 31.4℃左右 ,维持 12 h;然后处死 ,分离栓塞侧的大脑皮层 ,制备成均浆 ,分别测定一氧化氮 (NO)浓度、超氧化歧酶 (SOD)活性及丙二醛 (MDA)含量。结果  (1)假手术组左、右侧大脑皮质 NO、SOD、MDA含量比较无显著差异 (P>0 .0 5 ) ;(2 )亚低温 0~ 9h亚组 SOD含量较常温组各组增高 ,而 NO、MDA含量除 12 h亚低温亚组均较常温组显著降低 (P<0 .0 1或 P<0 .0 5 ) ;(3)亚低温 0~ 10 h亚组 NO浓度和 MDA含量明显低于 12 h亚组 (P<0 .0 1或 P<0 .0 5 ) ;(4 )亚低温 8h及 8h前各亚组 SOD活性较...

目的 探讨亚低温在不同时间窗对实验性脑梗死氧自由基的影响。方法 将 110只 SD大鼠随机分成手术组和假手术组 ;手术组又分为亚低温组和常温组 ,再各分为手术后 0 h、2 h、4 h、6 h、7h、8h、9h、10 h、11h、12 h亚组 ;假手术组分为常温组和亚低温组 ;每组均 5只。手术组制备成左侧颈动脉近端栓塞的永久性缺血模型 ,假手术组只暴露左颈动脉不栓塞颈动脉。亚低温各亚组将肛温控制在 31.4℃左右 ,维持 12 h;然后处死 ,分离栓塞侧的大脑皮层 ,制备成均浆 ,分别测定一氧化氮 (NO)浓度、超氧化歧酶 (SOD)活性及丙二醛 (MDA)含量。结果  (1)假手术组左、右侧大脑皮质 NO、SOD、MDA含量比较无显著差异 (P>0 .0 5 ) ;(2 )亚低温 0~ 9h亚组 SOD含量较常温组各组增高 ,而 NO、MDA含量除 12 h亚低温亚组均较常温组显著降低 (P<0 .0 1或 P<0 .0 5 ) ;(3)亚低温 0~ 10 h亚组 NO浓度和 MDA含量明显低于 12 h亚组 (P<0 .0 1或 P<0 .0 5 ) ;(4 )亚低温 8h及 8h前各亚组 SOD活性较 12 h亚组显著增高 (P<0 .0 5或 P<0 .0 1)。结论 亚低温在一定的时间内可以通过抑制自由基的产生和促进其清除而发挥脑保护作用 ,但是对不同的因素影响的时间窗并不一致。

Object: The aim of the study is to investigate the relationship between IL-8 and ischemic brain edema through developing observing the change of IL-8 of rats MCAO model. Methods: We established the experimental ischemic brain edema model by line-lock using health Wistar rats in the study. Then estimate the markers brain water content and IL-8 at different time(6h, 12h, 24h, 48h, 72h, 120h)after MCAO. Results:Serum levels of IL-8 showed a significant increased and peaked at 48h after MCAO. The relationship...

Object: The aim of the study is to investigate the relationship between IL-8 and ischemic brain edema through developing observing the change of IL-8 of rats MCAO model. Methods: We established the experimental ischemic brain edema model by line-lock using health Wistar rats in the study. Then estimate the markers brain water content and IL-8 at different time(6h, 12h, 24h, 48h, 72h, 120h)after MCAO. Results:Serum levels of IL-8 showed a significant increased and peaked at 48h after MCAO. The relationship between the change of IL-8 and brain water content is parallel. Conclusion: Serum levels of IL-8 showed a significant increased after ischemia. The parallel relationship between the change of IL-8 and brain water content showed IL-8 involved in the process of ischemic brain edema. The level of elevation of IL-8 reflected the degree of brain edema.

目的:探讨IL-8与缺血性脑水肿的关系;方法:采用健康的Wistar大鼠,建立线栓法脑梗死(MCAO)模 型。测定不同时点的脑组织水含量、IL-8水平的变化。结果:脑梗死后血清中IL-8水平迅速升高,在48小时达到高峰。 IL-8升高趋势与脑组织水含量升高趋势一致。结论:脑梗死后血清中IL-8水平升高,其升高趋势与脑组织水含量升高 趋势一致,说明IL-8参与了缺血性脑水肿的形成,IL-8的测定可以间接反映脑水肿的程度。

 
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