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serious hemorrhagic shock
相关语句
  重度失血性休克
     Two groups of rabbits,control group(n=7) and phentolamine treated one (n = 8), were put into simulated serious hemorrhagic shock. The mean arterial pressure (MAP) was maintained at 5. 33-6. 00kPa for 60min by using reservoir.
     两组家兔——生理盐水对照组(n=7)和酚妥拉明治疗组(n=8),麻醉后放血至血压为5.33~6.00kPa以造成重度失血性休克.
短句来源
     Application of hyperoxia liquid in patients with serious hemorrhagic shock
     高氧液在抢救重度失血性休克中的临床应用
短句来源
     Methods Sixty patients with serious hemorrhagic shock were randomly divided into two groups:the control group(n=30)and the experiment group(n=30).
     方法 选择重度失血性休克患者 6 0例 ,随机分为治疗组和对照组 ,每组 30例。
短句来源
     Methods Sixty patients with serious hemorrhagic shock were randomly divided into two groups: experiment group and control group(n 1,n 2=30).
     方法选择重度失血性休克患者60例,随机分成观察组和对照组,每组30例。
短句来源
     Conclusion Hyperoxia liquid could heighten BP and improve microcirculation in patients with serious hemorrhagic shock.
     结论 高氧液对重度失血性休克有稳定血压和改善微循环的作用。
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  重症失血性休克
     (3) The blood pressure of rat with serious hemorrhagic shock could be increased significantly with a little amount of intestinal lymph infusion, and the rats survived longerthan those of the control group (P<0. 05~0. 01 ).
     (3)重症失血性休克大鼠输入小量肠淋巴液后,血压显著回升,其血压水平及存活时间显著优于对照组(P<0.05~0.01)。
短句来源
     Objective: To study the interference effect of lymph on superior mesenteric artery occlusion(SMAO) shock and serious hemorrhagic shock in rats.
     目的:探讨正常淋浆对肠系膜上动脉闭塞性(SMAO)休克及重症失血性休克的干预作用。
短句来源
     Conclusion: Lymph could maintain the blood pressure and lengthen the survival time on SMAO shock and serious hemorrhagic shock in rats.
     结论:正常淋浆对SMAO休克及重症失血性休克均具有提升血压及延长存活时间的作用。
短句来源
     Objective To observe the effect of mesenteric lymph duct ligation on nitric oxide (NO) and its expression in lung in rats with serious hemorrhagic shock at different periods, and explore the role of intestinal lymphatic pathway in acute lung injury (ALI).
     目的观察结扎肠系膜淋巴管对不同时期重症失血性休克大鼠肺组织一氧化氮(NO)及其表达的影响,探讨肠淋巴途径在休克大鼠急性肺损伤(ALI)中的作用。
短句来源
     Objective To observe the effect of ligation of mesenteric lymph duct on release of oxygen free radical and inflammatory mediator in lung of rat with serious hemorrhagic shock at different periods,and explore the role of intestinal lymphatic pathway on acute lung injury(ALI).
     目的观察结扎肠系膜淋巴管对不同时期重症失血性休克大鼠肺组织自由基、炎症介质的影响,探讨肠淋巴途径在休克大鼠急性肺损伤(ALI)中的作用。
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  “serious hemorrhagic shock”译为未确定词的双语例句
     The changes of mesentery lymph microcirculation in serious hemorrhagic shock rat
     重症休克淋巴微循环的变化
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The purpose of this sdudy is to prove the effects of phentolamine on microcirculatory perfu-sion in hemorrhagic shock. Two groups of rabbits,control group(n=7) and phentolamine treated one (n = 8), were put into simulated serious hemorrhagic shock. The mean arterial pressure (MAP) was maintained at 5. 33-6. 00kPa for 60min by using reservoir. Then the animal was infused with either 5 mg of phentolamine in 25ml equivalent saline and was also transfused with the shed blood via the femoral vein in the period....

The purpose of this sdudy is to prove the effects of phentolamine on microcirculatory perfu-sion in hemorrhagic shock. Two groups of rabbits,control group(n=7) and phentolamine treated one (n = 8), were put into simulated serious hemorrhagic shock. The mean arterial pressure (MAP) was maintained at 5. 33-6. 00kPa for 60min by using reservoir. Then the animal was infused with either 5 mg of phentolamine in 25ml equivalent saline and was also transfused with the shed blood via the femoral vein in the period. The MAP,heart rate (HR) and mesenteric microcirculation before and after infusion were monitored. The blood flow velocity,blood flow rate and capillary calibre were measured by means of the micro-video system frame by frame analyzing method. Following the infusion of phentolamine,HR increased mildly and MAP declined considerably. Blood flow velocity and flow rate also decreased. There was no evidence that the capillary diameter was significantly altered by treatment. The results demonstrated that the phentolamine alone can seriously reduce blood pressure and thus aggravate obstruction of blood flow in mesenteric capillary.

本研究旨在探讨酚妥拉明对失血性休克微循环血液灌流的作用.两组家兔——生理盐水对照组(n=7)和酚妥拉明治疗组(n=8),麻醉后放血至血压为5.33~6.00kPa以造成重度失血性休克.维持60min后将5mg酚妥拉明加入25ml生理盐水中静脉滴注(对照组仅滴注等量生理盐水),同时将放出的全部血液回输.观察休克及用药前后平均动脉血压、心率和肠系膜微循环变化.用显微电视录像静像步进技术测定毛细血管口径、血流速度及血流量.酚妥拉明滴往后,心率略加快,血压明显下降,血流速度和血流量显著降低,毛细血管口径两组间无明显差异.表明休克时酚妥拉明单独应用可使血压严重下降,从而加重肠系膜毛细血管的血流障碍.

The effect of intestinal lymph on blood pressure in rats was observed by the methods of lymph drainage and lymph infusion. The results obtained are as follows: (1) Afte r 150 min of the lymph drained through the cannula of intestinal lymph duct, theblood pressure was significantly lower than that of the sham group (P<0. 05). (2) Equivalent albumin or intralipid infusion was not able to prevent the decrease in bloodpressure when the lymph was lost. But in jugular-intestinal lymph duct shunt group,no significant...

The effect of intestinal lymph on blood pressure in rats was observed by the methods of lymph drainage and lymph infusion. The results obtained are as follows: (1) Afte r 150 min of the lymph drained through the cannula of intestinal lymph duct, theblood pressure was significantly lower than that of the sham group (P<0. 05). (2) Equivalent albumin or intralipid infusion was not able to prevent the decrease in bloodpressure when the lymph was lost. But in jugular-intestinal lymph duct shunt group,no significant blood pressure decrease could be seen during the drainage procedure for 4h.(3) The blood pressure of rat with serious hemorrhagic shock could be increased significantly with a little amount of intestinal lymph infusion, and the rats survived longerthan those of the control group (P<0. 05~0. 01 ). The above results suggest that theintestinal lymph may play an important role in maintenance of blood pressure, in addition to the known function of lymphatic system by returning tissue fluid to blood andmaintaining circulating blood volume.

本文用引流及输入肠淋巴液的方法,研究了畅淋巴液对大鼠血压的影响。结果如下:(1)经肠淋巴管插管引流肠淋巴液的大鼠,自引流后150min起,血压显著低于假手术组(P<0.05)。(2)在丢失肠淋巴液同时,补充等量白蛋白或脂肪乳。仍不能防止血压下降,但肠淋巴管-颈静脉搭桥大鼠,在4h观察过程中,血压不出现见著降低。(3)重症失血性休克大鼠输入小量肠淋巴液后,血压显著回升,其血压水平及存活时间显著优于对照组(P<0.05~0.01)。上述结果表明,淋巴系统除了已知的组织液回流途径及维持血容量等功能外,肠淋巴液可能还具有维持血压的重要作用。

Abstract AIM:To explore the protective mechanisms of ECP (external counterpulsation) on ischemia diseases such as coronary heart disease, ischemia apoplexy and hemorrhagic shock (HS). METHODS:The model of serious hemorrhagic shock was used. After the whole experiment , hearts were excised to detect parameters of energy and free radical metabolism. RESULTS:Blood pressure of ECP and C groups at the end of experiment was significantly higher than that of HS group. ATP level in ventricular tissue of HS group...

Abstract AIM:To explore the protective mechanisms of ECP (external counterpulsation) on ischemia diseases such as coronary heart disease, ischemia apoplexy and hemorrhagic shock (HS). METHODS:The model of serious hemorrhagic shock was used. After the whole experiment , hearts were excised to detect parameters of energy and free radical metabolism. RESULTS:Blood pressure of ECP and C groups at the end of experiment was significantly higher than that of HS group. ATP level in ventricular tissue of HS group was significantly lower than that of control and ECP groups ( P <0 05), and lactic acid concentration in ventricular tissue of HS group was significantly higher than that of control and ECP groups ( P <0 05), SOD activity of HS group was significantly lower than that of control and ECP groups ( P <0 05), and MDA concentration of HS group was significantly higher than that of control and ECP groups ( P <0 05).CONCLUSION: ECP protected the canine heart of HS and its mechanism may be related to improvement of metabolism of energy and free radical. MeSH Counterpulsation; Energy metabolism; Free radicals; Myocardium

目的:体外反搏在治疗许多缺血性疾病中有良好的效果,本文在重症失血性休克模型上探讨其保护机制。方法:采用一侧颈总动脉插管放血制造重症失血性休克模型,全程监测血压,实验结束时取左室心肌进行各项检查。结果:①反搏组反搏后血压显著高于反搏前,且实验结束时反搏组的血压也显著高于失血组。②失血组心肌内ATP水平显著低于对照组及反搏组,而乳酸含量显著高于对照组及反搏组。③失血组心肌MDA含量显著高于对照组及反搏组,而SOD的活性则显著低于反搏组及对照组。结论:体外反搏可改善心肌的能量及自由基代谢

 
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