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central mechanisms
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  中枢机制
     Objective Functional magnetic resonance imaging (fMRI) was used to detect the cerebral cortical somatotopy during maximum voluntary clenching with and without soft splint in patients with hemimasticatory spasms (HMS) and the central mechanisms of HMS and the rule of the splint therapy.
     目的应用功能性磁共振成像(fMRI)技术,探讨单侧咀嚼肌痉挛(HMS)患者紧咬运动及戴软垫紧咬运动诱发痉挛发作时,脑功能活动区域定位,试图探讨HMS发病的中枢机制及软垫的治疗作用机制。
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     Approach to central mechanisms of acupuncture actions by functional brain imaging
     从脑功能成像看针刺作用的中枢机制
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     The present study was undertaken to explore central mechanisms underlying the analgesic effect of melatonin. Push pull perfusion technique and radioimmunoassay were used to observe the changes in the contents of β endorphin (β Ep) and leucine enkephalin (L EK) in the perfusate from the rat periaqueductal gray (PAG) after administration of melatonin.
     本文采用推挽灌流技术、放射免疫测定法 ,观察褪黑素 (melatonin ,MEL)对大鼠中脑导水管周围灰质(PAG)推挽灌流液中 β 内啡肽 (β Ep)、亮氨酸脑啡肽 (L EK)含量的影响 ,以探讨MEL镇痛效应的中枢机制
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     Effect of Daidzein on Laying Performance of Shaoxing Ducks and Its Central Mechanisms Involved
     大豆黄酮对绍兴鸭产蛋性能影响及有关中枢机制初探
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     ObjectiveTo understand advances of studies on func ti onal brain imaging in acupuncture and approach to central mechanisms of acupunct ure treatment.
     目的 :了解针刺负荷脑功能成像的研究进展 ,探讨针刺疗效的中枢机制
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  “central mechanisms”译为未确定词的双语例句
     Advances in the Study on Central Mechanisms of Acupuncture Regulation
     针刺的中枢调节机制研究进展
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     The Study on Behaviors & Peripheral and Central Mechanisms of Masticatory Muscles Mechanical Hyperalgesia Induced by Occlusal Trauma in Rats
     咬合干扰致大鼠咀嚼肌机械性痛觉敏感的动物行为学及中枢外周机制研究
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     Objective: To observe the effect of 2 Hz electroacupuncture (EA) on long term depression (LTD) of synaptic transmission in the spinal dorsal horn in rats with neuropathic pain, so as to explore the central mechanisms of the antinociceptive effects of 2 Hz electroacupuncture on neuropathic pain.
     目的 :观察 2Hz电针对神经病理痛大鼠脊髓背角突触传递长时程抑制 (long termdepression ,LTD)的诱导 ,以阐明电针治疗慢性神经病理痛的神经生物学机制。
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     Studies on the Central Mechanisms Responsible for the Action of Isoflurane
     异氟醚中枢作用机制的初步研究
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     Conclusion: The intestinal barrier function of restrained rats may be protected by Gln supplementation through peripheral and central mechanisms.
     结论: 补充Gln可以保护应激状态下的肠粘膜屏障功能。
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  相似匹配句对
     On Mechanisms
     机制论
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     Advances in the Study on Central Mechanisms of Acupuncture Regulation
     针刺的中枢调节机制研究进展
短句来源
     Studies on the Central Mechanisms Responsible for the Action of Isoflurane
     异氟醚中枢作用机制的初步研究
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     The mechanisms of desulfurizationwith T.
     T.
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     The central Station
     《中央车站》——本片获1998年奥斯卡最佳外语片提名
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  central mechanisms
Central Mechanisms that Control Respiration in the Pond Snail Lymnaea stagnalis: Is the VD4 Neuron Involved in Respiratory Rhyth
      
Central Mechanisms that Control Respiration in the Pulmonate Snail Lymnaea stagnalis: Phasic Coupling of Lung Ventilation to Cyc
      
The hypercapnic drive also increased, which may be determined by readjustment in the central mechanisms of respiratory regulation.
      
These children, like adults with similar impairments, displayed a high degree of strain of the central mechanisms of cardiac regulation.
      
Central mechanisms of fatigue during local static muscular activity
      
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Sustained nociceptive stimuli were applied to the hind limb of 8 monkeys (Macaca mulatta)in three ways:(1)fracturing the fibula;(2)applying a chemical irritant(ether extract of capsicum)to the sciatic nerve topically;(3)injecting hypertonic saline into the gas- trocnemius and its adjacent subcutaneous tissues.All these methods were found to be capable of producing,on the surface of the pinnae,small discrete spots tender to pressure.The topographical distribution of the spots was not regular;but,statistically,they...

Sustained nociceptive stimuli were applied to the hind limb of 8 monkeys (Macaca mulatta)in three ways:(1)fracturing the fibula;(2)applying a chemical irritant(ether extract of capsicum)to the sciatic nerve topically;(3)injecting hypertonic saline into the gas- trocnemius and its adjacent subcutaneous tissues.All these methods were found to be capable of producing,on the surface of the pinnae,small discrete spots tender to pressure.The topographical distribution of the spots was not regular;but,statistically,they often appeared near the tip of the triangular fossa and the antitragus of the pinna.They began to appear from 40 minutes to several hours after the noxious treatment,and persisted for a period from 3 days to a few months(7—10 days on average).Intravenous injection of drugs known to suppress the activity of the reticular forma- tion of the brain stem,such as chlorpromazine or nembutal,augmented the reaction to pressure and increased the number of spots.On the contrary,drugs,such as LSD_(25),amphetamine or adrenaline,which increase the excitability of the reticular formation,suppressed the reaction to pressure and decreased the number of spots.In other experiments,procaine solution was injected into the third ventricle of the brain;1—2 minutes after the injection the referred tender spots disap- peared and reappeared after 13—60 minutes.This fact suggests that the central mechanism con- cerned in the production of the referred tenderness involves neural structure or structures situated in the central portion of the rostral brain stem;and perhaps the mechanism is inhibited by impulses from the reticular formation,as indicated by the experiments of intravenous injection.

用以下三种方法在8只猴人工产生了牵涉性耳郭压痛点:(1)切断腓骨;(2)化学物质(辣椒的乙醚提取物)刺激坐骨神经;(3)下肢肌肉及皮下注射高渗盐水。压痛点可出现于耳郭各处,但比较集中于三角窝尖端及对耳屏附近。压痛点在损伤后40分钟至数小时出现,持续3天至数月(平均7—10天)。静脉注射药理学上认为对脑干网状结构有抑制性影响的药物(氯丙嗪及戊巴比妥)后,压痛点增多,压痛反应增强。注射对网状结构有兴奋性影响的药物(离麦角酸二乙胺、苯异丙胺或肾上腺素),效果相反。这暗示参与耳郭压痛现象的神经结构受来自网状结构的冲动的抑制。第三脑室注射普鲁卡因溶液可在1—2分钟内取消耳郭压痛点,至第13—60分钟又行恢复,表明参与牵涉性耳郭压痛反应的一个中枢环节在脑干前端的中央部分。

Electro-stimulation of periaquaductal central grey in midbrain or administration of angiotensin Ⅱ into the third ventricle causes marked increase in plasma C-Amp and C-Cmp/C-Amp ratio in femoral or renal venous blood of a blood-perfused kidney in vivo in cats. The results suggest that cyclic nucleotides may take part in the release of Angiotensin Ⅱ induced by the central mechanism.

电刺激及注射ANGⅡ均使单只猫的股静脉及灌流猫肾静脉的血中cAMP含量显著升高,cGMP多无明显变化,cAMP/cGMP比值显著增加,提示环核苷酸参与肾内由于中枢效应所引起的 ANGⅡ释放过程。

The central mechanism of gastric acid secretion induced by intracerebro-ven-tricular(icv)injection of caerulein was studied by the effect of similarly applied va-rious receptor antagonists 20 min prior to caerulein.Techniques of continuousgastric perfusion of Ghosh and Schild and cerebral cannula implantation were usedfor acute experiments in rats.Pcrfusates collected every 10 min were titrated toquantify the total acid output.Icv injection of caerulein significantly stimulated gastric acid secretion.Prioricv...

The central mechanism of gastric acid secretion induced by intracerebro-ven-tricular(icv)injection of caerulein was studied by the effect of similarly applied va-rious receptor antagonists 20 min prior to caerulein.Techniques of continuousgastric perfusion of Ghosh and Schild and cerebral cannula implantation were usedfor acute experiments in rats.Pcrfusates collected every 10 min were titrated toquantify the total acid output.Icv injection of caerulein significantly stimulated gastric acid secretion.Prioricv administration of phentolamine or propranolol did not influence the effect ofcaerulein,but the acid output induced by caerulein was completely abolished aftera prior injection of atropine or dibutyryl cGMP(CCK-receptor antagonist).It seems that CCK receptor and cholinergic receptor in brain are involved inthe mechanisms of gastric acid secretion induced by caerulein.

利用特异的受体阻断剂能够拮抗相应的受体激动剂的效应的原理,分析雨蛙肽中枢促胃酸分泌作用的受体机制。向大鼠侧脑室内注射微量雨蛙肽(67ng/鼠),可引起急性灌流大鼠胃酸分泌明显增加。预先向大鼠侧脑室内注射肾上腺素受体阻断剂酚妥拉明或心得安,20min后再向侧脑室内注射雨蛙肽,预处理对雨蛙肽的促胃酸分泌作用影响不大。但事先向侧脑室内注射乙酰胆碱受体阻断剂阿托品或胆囊收缩素(CCK)受体阻断剂二丁酰环化-磷酸鸟苷(Bt_2 cGMP)则可有效地阻断雨蛙肽的作用。以上结果提示,脑内雨蛙肽促胃酸分泌机制中,可能有 CCK 受体和胆碱能受体参与,而与肾上腺素能系统关系不大。

 
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