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chronic bronchitis model
相关语句
  慢性支气管炎模型
     Objective: To study the content change of ET-1 and CGRP in smoke-fumigating rat chronic bronchitis model's blood and plasma and BALF.
     目的 :探讨烟熏致大鼠慢性支气管炎模型血浆及支气管肺泡灌洗液 (BALF)中内皮素(ET -1 )和降钙素基因相关肽 (CGRP)的含量变化及补肾化痰中药对其的影响。
短句来源
     Methods 21 Sprague-Dawley (SD) rats were randomly divided into three groups : ambroxol chloride treatment group ( group A) with intraperitoneal injection of ambroxol chloride basing on chronic bronchitis model, chronic bronchitis model group( group B) induced by lipopolysaccharide instilling into the tracheas of experimental rats combining cigarette smoking, control group(group C).
     方法:21只SD大鼠随机分为AMB干预组(A组)、模型组(B组)、实验对照组(C组); 脂多糖-香烟烟雾诱导法建立慢性支气管炎模型,AMB 30mg/kg腹腔注射进行干预;
短句来源
     (3) Compared with the chronic bronchitis model group, the total T lymphocytes (CD3+) are increased significantly (p<0.01),and the level of CD4+, CD4+/CD8+ is increased significantly in two XQM-treated groups, and XQM&NACH-treated group (p<0.01), and there is the obvious relationship between the dose and the effect.
     3、仙茂合剂各剂量组、仙蔑合剂加核酪口服液组与慢性支气管炎模型组比较,T淋巴细胞CD厂细胞,CD/细胞数量和 CD//CD/细胞比值显著升高,CD/细胞明显减少,两者比较有显著性差异o功刀1),其程度与药量呈明显量效关系。
短句来源
     Results Using passive cigarette smoking and intratracheal instillation of low dose lipopolysacchide may reproduce ideal chronic bronchitis model. The expression of TNF-α,ICAM-1 and IL-8 were weakly obvious in the area of bronchial epithelium in the sham operation group.
     结果熏香烟加气管内低剂量脂多糖注射可复制出较理想的慢性支气管炎模型,假手术组支气管上皮内可见TNF-α,ICAM-1蛋白及IL-8弱阳性表达;
短句来源
     Methods Rat chronic bronchitis model was established by intratracheal instillation of small dose of lipopolysaccharide (LPS, 1 g/L). Experiments were performed in 28 male Sprague Dawley rats, which comprised four groups in random, i.e. chronic bronchitis model group, normal saline treated group, dexamethasone treated group and healthy control group.
     方法 雄性SD大鼠气管内注入浓度为 1g/L的脂多糖 2 0 0 μl制成大鼠慢性支气管炎模型 ,随机分为健康对照组、模型组、生理盐水组及氟美松组 ,每组 7只。
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  慢支模型
     Rats were treated by bilirubin (20mg/kg/d) through feeding 2 hours before the chronic bronchitis model had been estabilished in bilirubin treated group, once a day, 21 days in all.
     胆红素干预组:在制备慢支模型前2小时给予间接胆红素灌胃20mg/kg/日,每日一次,共21天。
短句来源
     In order to survey the influence of smoking on the level of guanylin in lung tissue in rats, a method of passive smoking was used to set up chronic bronchitis model in rats.
     用香烟烟雾使大鼠被动吸烟的方法 ,复制大鼠慢性支气管炎 (慢支 )模型 ,以观察吸烟对大鼠肺组织中鸟苷素含量的影响。 采用被动吸烟方法复制大鼠慢支模型
短句来源
     Rat chronic bronchitis model was estabilished by intratracheal instillation of small dose of Lipopolysaccharide (LPS, lg/L) in chronic bronchitis group.
     慢支组:采用大鼠气管内注入脂多糖(LPS,浓度为1g/L的200μl)的方法制备慢支模型
短句来源
     Methods:Chronic bronchitis model rats and mice infected with inhalational pseudomonas aeruginosa were treated with MZGBG,and examinations were done on:(1)Quantitative determination of bacteria in bronchial tissue culture;
     方法:应用慢支模型鼠,经气道吸入法感染绿脓杆菌,并在设定的条件下,(1)取实验动物气管组织制成匀浆作细菌定量培养;
短句来源
     Method: Sixty male Wistar rats were divided into five groups at random, which consist of blank control group, chronic bronchitis model group, high dose group , low dose group and group. Chronic bronchitis model was induced with refined smoking-fumigating method, and then blood plasma and content of ET-1 and CGRP of BALF were tested in these five groups.
     方法 :雄性Wistar大鼠 60只 ,随机分为正常对照组、慢支模型组、补肾化痰大、小剂量组和气管炎咳嗽痰喘丸组等 5组 ,采用香烟烟雾吸入法建立慢支动物模型 ,测定各组大鼠血浆和BALF中ET -1和CGRP的含量。
短句来源
  “chronic bronchitis model”译为未确定词的双语例句
     Results (1) The levels of TNFα and MIP 2 in BALF and lung tissues, and MPO in lung tissues of chronic bronchitis model group were significantly increased than those of control group ( P <0.05).
     结果 模型组、生理盐水组及氟美松组BALF和肺组织中TNFα和MIP 2的含量显著高于健康对照组 (P <0 .0 5 ) ;
短句来源
     Expression of EGR-1、PDGF-BB in Chronic Bronchitis Model of Rats and the Interference of Drugs
     慢性支气管炎大鼠支气管肺组织EGR-1、PDGF-BB的表达及药物干预的影响
短句来源
     Methods 24 male Sprague-Dawley rats were divided into three groups at random, i.e. control group (8), chronic bronchitis model group (8), bilirubin treated group (8).
     方法 取健康清洁级雄性SD大鼠24只,随机分为对照组(8只)、慢支组(8只)和胆红素干预组(8只)。
短句来源
     Methods: Rat chronic bronchitis model was established by intratracheal instillation of 0.1% lipopolysaccharide 200μl. With image analysis and ELISA,the changes of pathology and mucins in bronchoalveolar lavage fluid(BALF)were observed.
     方法 :Wistar大鼠气管内注入 0 .1%脂多糖2 0 0 μl复制模型 ,采用图象分析、ELISA等方法检测病理学改变、支气管肺泡灌洗液 (BALF)中粘蛋白量的变化。
短句来源
     in chronic bronchitis model group Significant increases in total white blood cell numbers and neutrophils in BALF were found in rat chronic bronchitis group (p<0.001 ).
     BALF白细胞总数及中性粒细胞数较对照组显著增多(P<0.001)。
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This paper has reported the pharmacological effects of SZIQD ou the main pharma-cdynamics. The results were as follow: ( 1 ) Ammonia-induced cough of mice, histamine-acetyl choline chloride-induced asthma of guinea pigs, oil of Croton seed-induced inflammation of mice, and experimental allergy of type f1 of rats were markedly inhibited by SZJQD. ( 2 ) The normal respiratory tract segments and histamine-acetyl choline-induced convulsing respiratory tract segments were markedly relaxed by SZJQD, which couldn't...

This paper has reported the pharmacological effects of SZIQD ou the main pharma-cdynamics. The results were as follow: ( 1 ) Ammonia-induced cough of mice, histamine-acetyl choline chloride-induced asthma of guinea pigs, oil of Croton seed-induced inflammation of mice, and experimental allergy of type f1 of rats were markedly inhibited by SZJQD. ( 2 ) The normal respiratory tract segments and histamine-acetyl choline-induced convulsing respiratory tract segments were markedly relaxed by SZJQD, which couldn't be prevented by propranolol, the bata receptor blocker. (3) Index of sulfur dioxide-induced chronic bronchitis models were significantly decreased by SZJQD.

苏子降气汤对小鼠氨水性咳嗽、豚鼠实验性哮喘、巴豆油引起的小鼠耳部炎症以及实验性I型变态反应均有显著的抑制作用;对正常及由组胺、乙酰胆碱所致痉挛状态的离体气管均有显著的松弛作用,且不被心得安所拮抗;能明显减轻SO_2慢性支气管炎模型的病理改变;对呼吸道酚红排泌的影响不明显。

Objective:Under the guidance of traditional Chinese medical theory of improving the general condition to eliminate pathogens,to study the effects and mechanism of Manzhi Guben Granule(MZGBG),a Chinese tonifying drug in pre- venting respiratory infection.Methods:Chronic bronchitis model rats and mice infected with inhalational pseudomonas aeruginosa were treated with MZGBG,and examinations were done on:(1)Quantitative determination of bacteria in bronchial tissue culture;(2)Pathologic change of epithelial...

Objective:Under the guidance of traditional Chinese medical theory of improving the general condition to eliminate pathogens,to study the effects and mechanism of Manzhi Guben Granule(MZGBG),a Chinese tonifying drug in pre- venting respiratory infection.Methods:Chronic bronchitis model rats and mice infected with inhalational pseudomonas aeruginosa were treated with MZGBG,and examinations were done on:(1)Quantitative determination of bacteria in bronchial tissue culture;(2)Pathologic change of epithelial mucous membrane of trachea and(3)Scanning electron mi- croscopic observation on ultrastructure of tracheal mucous epithelium and its condition of bacteria adhering.And the results were compared with those of non-infected animals.Results:Pseudomonas aeruginosa couldn't adhere to the intact mucous membrane of normal rats and mice,hut could adhere to,settle down and multiply on the infected mucous membrane ep- ithelia,and thus results in a state of long-term bacteria carrier.MZGBG could reduce the degree of pathologic changes of trachea.Conclusion:MZGBG could protect tracheal mucous membrane from bacteria adherence by repairing the pathologic changes.

目的:以中医扶正祛邪理论为指导,进行补益中药抗呼吸道感染的实验研究。方法:应用慢支模型鼠,经气道吸入法感染绿脓杆菌,并在设定的条件下,(1)取实验动物气管组织制成匀浆作细菌定量培养;(2)取气管组织作病理切片,检查粘膜上皮的病理变化;(3)取气管组织作扫描电镜检查,观察粘膜上皮超微结构及细菌粘附于粘膜上皮的情况,据此研究绿脓杆菌感染机理及中药慢支固本冲剂的治疗作用。结果:绿脓杆菌不能粘附于正常鼠完整的气道粘膜,而可粘附于慢支模型鼠病变的粘膜上皮,并定居、繁殖造成长期带菌。结论:给服慢支固本冲剂,能使病变粘膜上皮复愈,从而抵抗绿脓杆菌的粘附。

Objective To study the nature and mechanisms of airway inflammation in chronic bronchitis and observe the effects of inhaled glucocorticoids on inflammatory indices. Methods Rat chronic bronchitis model was established by intratracheal instillation of small dose of lipopolysaccharide (LPS, 1 g/L). Experiments were performed in 28 male Sprague Dawley rats, which comprised four groups in random, i.e. chronic bronchitis model group, normal saline treated group, dexamethasone treated group and healthy...

Objective To study the nature and mechanisms of airway inflammation in chronic bronchitis and observe the effects of inhaled glucocorticoids on inflammatory indices. Methods Rat chronic bronchitis model was established by intratracheal instillation of small dose of lipopolysaccharide (LPS, 1 g/L). Experiments were performed in 28 male Sprague Dawley rats, which comprised four groups in random, i.e. chronic bronchitis model group, normal saline treated group, dexamethasone treated group and healthy control group. The levels of myeloperoxidase (MPO) of blood and lung tissues, and tumor necrosis factor(TNF)α and macrophage inflammatory protein 2 (MIP 2) of plasma, broncho alveolar fluid(BALF) and lung tissues were determined by biochemical and ELISA methods. Total and differential white blood cell counts of BALFwere carried out. Results (1) The levels of TNFα and MIP 2 in BALF and lung tissues, and MPO in lung tissues of chronic bronchitis model group were significantly increased than those of control group ( P <0.05). (2)More significant increase in total white blood cell count and neutrophils in BALF was found in rat chronic bronchitis group than in control group ( P <0.001). (3) Significant positive correlations were observed between the level of MPO and MIP 2 of lung tissues, the level of MPO and TNFα of lung tissue and the total cell counts and the level of MIP 2 of BALF and lung tissue. (4) More significant decrease in total cell counts and neutrophils of BALF and levels of MPO in lung tissue was found in dexamethasone treated group as compared to those of chronic bronchitis group. Conclusion Recruitment and activation of neutrophils seem to be the characteristics of chronic bronchitis. TNFα and MIP 2 may be involved in the process of chemotaxis and activation in airway inflammation in chronic bronchitis. Inhaled steroids might have some effects on chronic bronchitis by limiting the airway inflammation.

目的 探索慢性支气管炎气道炎症的特点。方法 雄性SD大鼠气管内注入浓度为 1g/L的脂多糖 2 0 0 μl制成大鼠慢性支气管炎模型 ,随机分为健康对照组、模型组、生理盐水组及氟美松组 ,每组 7只。应用ELISA法测定血浆、支气管肺泡灌洗液 (BALF)及肺组织中肿瘤坏死因子 (TNF)α、巨噬细胞炎症蛋白 2 (MIP 2 )含量 ,生化法检测血和肺组织中髓过氧化物酶 (MPO)水平 ,并进行BALF中细胞计数和分类。结果 模型组、生理盐水组及氟美松组BALF和肺组织中TNFα和MIP 2的含量显著高于健康对照组 (P <0 .0 5 ) ;肺组织中MPO的含量 ,模型组 [( 15 .0 0 2 +1.85 7)U/g]和生理盐水组 [( 11 888+0 92 6)U/g]显著高于健康对照组 [( 7 812 +1 0 4 3 )U/g](P <0 .0 5 ) ,而氟美松组[( 6 14 1+0 3 64 )U/g]则显著低于模型组 (P <0 .0 5 ) ;模型组肺组织中MPO与TNFα和MIP 2含量之间呈正相关 (r=0 788,r=0 963 ;P <0 .0 5 ,P <0 ....

目的 探索慢性支气管炎气道炎症的特点。方法 雄性SD大鼠气管内注入浓度为 1g/L的脂多糖 2 0 0 μl制成大鼠慢性支气管炎模型 ,随机分为健康对照组、模型组、生理盐水组及氟美松组 ,每组 7只。应用ELISA法测定血浆、支气管肺泡灌洗液 (BALF)及肺组织中肿瘤坏死因子 (TNF)α、巨噬细胞炎症蛋白 2 (MIP 2 )含量 ,生化法检测血和肺组织中髓过氧化物酶 (MPO)水平 ,并进行BALF中细胞计数和分类。结果 模型组、生理盐水组及氟美松组BALF和肺组织中TNFα和MIP 2的含量显著高于健康对照组 (P <0 .0 5 ) ;肺组织中MPO的含量 ,模型组 [( 15 .0 0 2 +1.85 7)U/g]和生理盐水组 [( 11 888+0 92 6)U/g]显著高于健康对照组 [( 7 812 +1 0 4 3 )U/g](P <0 .0 5 ) ,而氟美松组[( 6 14 1+0 3 64 )U/g]则显著低于模型组 (P <0 .0 5 ) ;模型组肺组织中MPO与TNFα和MIP 2含量之间呈正相关 (r=0 788,r=0 963 ;P <0 .0 5 ,P <0 .0 0 1)。结论 气道内中性粒细胞聚集是慢性支气管炎的重要特点 ,TNFα和MIP 2参与了中性粒细胞的激活和趋化过程 ,中性粒细胞产生大量的MPO可造成气道损伤。

 
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