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patients with fulminant hepatitis
相关语句
  重症肝炎患者
     Determination of serum hepatic stimulating activity in patients with fulminant hepatitis
     重症肝炎患者血清肝刺激活性的测定
短句来源
     DETERMINATION OF AMINO ACID CONTENT IN DIFFERENT TISSUES OF THE BRAIN IN PATIENTS WITH FULMINANT HEPATITIS
     重症肝炎患者脑组织氨基酸含量检测
短句来源
  “patients with fulminant hepatitis”译为未确定词的双语例句
     Expression of fgl2 Prothrombinase Gene in Patients with Fulminant Hepatitis
     重型肝炎患者fgl2凝血酶原酶基因的表达
短句来源
     Methods Patients with fulminant hepatitis were investigated for T1762A1764 mutation by direct sequencing of PCRproducts.
     方法PCR产物直接测序,检测重症乙型肝炎患者乙型肝炎病毒(HBV)前C和C基因调节序列的T1762A1764变异;
短句来源
     objective To study prognosis of patients with fulminant hepatitis after plasmaexchange treatment using MELD(model for end-stage liver disease,MELD)scoring system.
     目的应用终末期肝病模型(model for end-stage liver disease,MELD)评分系统预测血浆置换治疗后重型肝炎患者的预后。
     objective To study prognosis of patients with fulminant hepatitis after plasma exchange treatment using MELD(model for end-stage liver disease, MELD) scoring system.
     目的应用终末期肝病模型(model for end-stage liver disease,MELD)评分系统预测血浆置换治疗后重型肝炎患者的预后。
     A Preliminary Study on Gene Mutation of HBV DNA in Patients with Fulminant Hepatitis B and HBV Genotyping in Henan
     重症乙型肝炎HBV DNA CP 区及前S区基因变异与河南地区HBV DNA 基因型的研究
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  相似匹配句对
     As to the patients;
     患者年龄13~76岁。
短句来源
     The patients were
     记录患者平卧10 min(T0)、
短句来源
     Ophthalmoplegia in patients with diabetes
     糖尿病性眼肌麻痹17例临床分析
短句来源
     Psychoanalysis and Psychotherapy of the patients with ED
     心因性勃起功能障碍患者的心理治疗结果分析(附13例报告)
短句来源
     Diagnosis of fulminant duck plague
     暴发性鸭瘟的诊断
短句来源
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  patients with fulminant hepatitis
For the 11 patients with fulminant hepatitis B, the precore/core regions were successfully amplified in 10 patients.
      
To determine the precore/core gene sequence in patients with acute and fulminant hepatitis B, 11 patients with fulminant hepatitis B and seven patients with acute hepatitis B were studied.
      
These data show that patients with fulminant hepatitis commonly had increased circulating levels of CEA, but these elevations were far less than may be found in patients with hepatic metastases.
      
Circulating CEA levels in patients with fulminant hepatitis
      
Survival figures derived by compiling reports on exchange transfusion are somewhat encouraging when the data are compared to the salvage rate in patients with fulminant hepatitis treated conventionally, even after the addition of steroids.
      
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Anti-HAV-IgM, anti-HBc-IgM, and three antibody systems of hepatitis B were tested in 48 patients with fulminant hepatitis to determine which of the hepatitis viruses A and B was the causative agent. HBV was found in 31 cases, HAV in one case, mixed infection of HBV and HAV in 15 cases (31.25%),and uncertain agent in 1 case.Ten cases out of the 15 with mixed infection were subacute, and the other 5 chronic. 13 patients were male and 2 female. The age of the patients ranged from 15 to 49...

Anti-HAV-IgM, anti-HBc-IgM, and three antibody systems of hepatitis B were tested in 48 patients with fulminant hepatitis to determine which of the hepatitis viruses A and B was the causative agent. HBV was found in 31 cases, HAV in one case, mixed infection of HBV and HAV in 15 cases (31.25%),and uncertain agent in 1 case.Ten cases out of the 15 with mixed infection were subacute, and the other 5 chronic. 13 patients were male and 2 female. The age of the patients ranged from 15 to 49 years with an average of 30.39. The clinical conditions of 10 cases became more serious and some of them even died during hospitalization. The clinical and biochemical parameters of the patients with mixed infection showed no statistical difference from those of the patients with single infection of either HBV or HAV of the same time period. 7 patients complained of sudden aggravation of their clinical conditions after they exhibited a group of symptoms and signs of "acute icteric hepatitis". One such case was found during his hospitalization and his anti-HAV-IgM turned positive at the same time. This patient died eventually.It is considered that aggravation of acute or chronic hepatitis can be resulted from superinfection and this problem deserves close attention.

报告经血清免疫学抗HAVIgM、抗-HBcIgM、HBV-DNA、HBsAg/IgM复合物以及乙肝三种抗原抗体系统检测48例重型肝炎中甲、乙型肝炎病毒感染情况。结果:31例为HBV感染,1例为HAV感染,1例未定型,15例(31.25%)为甲、乙型合并感染(混合感染7例、重叠感染8例)。15例中亚急性10例、慢性5例;死亡或恶化10例,与同期“单纯”感染的重肝比较,包括主要临床生化指标均无显著差异。有7例患者病程中有近期出现“急黄肝”症状的历史,随后病情突然加重;1例发生在住院期间,且血清抗-HAVIgM转阳,最后死亡。认为合并感染可使部分急、慢性肝炎病情加重,甚至发展成重型。应注意发现。

Hemorrheology indices in patients with fulminant hepatitis showed apparently

重症肝炎病人血液流变学指标呈明显异常,与急性肝炎病人相应指标比较均有显著差异(P<0.05或P<0.01)。各项指标异常程度随病情的好转或恶化而变化,并与凝血障碍程度及出血呈正相关。当 PT 延长、ELT 缩短以及出血发生时血浆粘度、全血粘度明显降低,亦表明血液流变学严重异常是出血发生的重要原因之一。

Blood sugar, insulin and C-peptide were detected in 11 cases of fulminant heratitis confirmed by liver biopsy before G-I treatment and 15, 30, 90, 120, 180 minutes after instillation.The restults showed that blood sugars were normal or less before instillation, ard elevated slightly then dropped to normal level afler instillation.Insulin C-peptide and glucagon were bigger than normal before instillation and reached peak value 15 minutes after instillation,C-peptide and glucagon recovered to pre-instillation...

Blood sugar, insulin and C-peptide were detected in 11 cases of fulminant heratitis confirmed by liver biopsy before G-I treatment and 15, 30, 90, 120, 180 minutes after instillation.The restults showed that blood sugars were normal or less before instillation, ard elevated slightly then dropped to normal level afler instillation.Insulin C-peptide and glucagon were bigger than normal before instillation and reached peak value 15 minutes after instillation,C-peptide and glucagon recovered to pre-instillation level 3 hours after instilla tion. But insulin persisted in peak value. Therefore in the patients with fulminant hepatitis,function to intake and to degrade insulin decreased, and the function to intake and to degrade glucagon was present.

经肝活检证实的重症肝炎11例,在胰高血糖素-胰岛素(G-I)疗法前及滴完后15、30、60、120、180分钟采血,检测血清糖、胰岛素、C肽及血浆胰升血糖素。结果:血糖在滴注前正常或偏低,滴后轻度上升即降至滴前水平;胰岛素、C肽及胰升血糖素滴前均高于正常,滴后15分钟高峰,滴后3小时C肽及胰升血糖素恢复到滴前水平,而胰岛素持续呈高值。可见重症肝炎对胰岛素的摄取和降解功能降低,而对胰升血糖素有摄取和降解功能。

 
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