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peripheral mechanism
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  外周机制
     Conclusions The peripheral mechanism of EA analgesia is related to raising the activity of cellular factors IL-1 and IL-2.
     结论  EA对炎症痛镇痛的外周机制与其提高细胞因子 IL - 1和 IL - 2的活性有关。
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     Objective To probe into peripheral mechanism of electroacupuncture (EA) analgesia, the effect of EA on acitivity of IL 1 and IL 2 in spleen cell in adjuvant arthritis (AA) rat was observed.
     目的 为了探讨电针 (EA)对炎症痛镇痛的外周机制 ,观察 EA对佐剂性关节炎 (AA)大鼠脾细胞中细胞因子白细胞介素 1(IL- 1)和白细胞介素 2 (IL- 2 )活性的影响。
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     Conclusion EP 1 receptor antagonist relieves hyperalge sia of diabetic rats by blocking PGE 2 to sensitize C units, suggesting tha t PGE 2 plays an important role in the peripheral mechanism of diabetic hyp eralgesia.
     结论 EP1受体阻断剂可阻断PGE2 对C单位的敏化作用而缓解糖尿病大鼠的痛过敏 ,提示PGE2 在糖尿病痛过敏的外周机制中具有重要作用。
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     Advanced Research on Peripheral Mechanism of Insulin Resistance Exercise Induced
     运动干预胰岛素抵抗的外周机制研究进展
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     The previous studies suggested that endogenous opioid peptides (EOP) participate in the peripheral mechanism of electro-acupuncture (EA) analgesia for inflammatory-pain.
     目前,电针(EA)对慢性痛(如炎症痛)的镇痛作用及其外周机制的研究正越来越受到学者们的重视。 以往的研究表明,内源性阿片肽(EOP)参与了EA对炎症痛镇痛的外周机制
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  “peripheral mechanism”译为未确定词的双语例句
     Peripheral Mechanism Underlying the Contribution of VR1 Activation to the Transduction of Noxious Information
     辣椒素受体(VR1)介导外周伤害性信息传递的机制分析
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     Study on the central and peripheral mechanism of analgesia in treatment of rheumatoid arthritis by traditional "heat-enforcing acupuncture"
     传统“热补针法”治疗类风湿关节炎的中枢及外周镇痛机理研究
短句来源
     Whether the Peripheral Mechanism is Involved in the Analgesic Action
     3,15一二乙酰苯甲酰乌头宁的镇痛作用中有无外周成分参与
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     The efficacy was identified on the peripheral mechanism, the central effects of exercise training which could promote the collateral circulation was still in controversy partially because the animal model of chronic coronary stenosis and collateral circulation development had not been satisfactorily established.
     但是其中心机制,即运动可以促进心肌侧支循环的形成目前没有定论,其关键的因素之一是缺乏慢性冠脉狭窄及侧支循环的动物模型。
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     Objective: To better understand the peripheral mechanism of inflammatory pain.
     目的 :研究炎症大鼠背根神经节 (dorsalrootganglion ,DRG)细胞的膜电生理学特征 ,试图解释炎症痛敏的机制。
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  相似匹配句对
     The peripheral analgesic effect of ketamine and its mechanism
     氯胺酮的外周镇痛作用及其机制的初步研究
短句来源
     The analgesic activity involved peripheral and central mechanism.
     镇痛作用机制可能通过外周与中枢两种机制介导;
短句来源
     (2) peripheral;
     (2)周边强化型;
短句来源
     And what is the mechanism?
     机理又是什么?
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     The mechanism was thorougly.
     本文详细讨论了上述症象的形成机制。
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  peripheral mechanism
This paper aims to investigate the peripheral mechanism of taste perception by the use of the grand canonical ensemble in statistical physics.
      
Peripheral mechanism for hydrogenation of aromatic nitro compounds: Reaction of nitrobenzene with copper(II)-alizarin complex
      
Comparisons show that damage to one peripheral mechanism or delay in cognitive development also delays language acquisition.
      
It is postulated that the adrenergic component of the depressor zone of the aortic arch participates in the peripheral mechanism of the regulatory effects of the sympathetic nervous system on the baroreceptor apparatus.
      
Chemosensitivity of dorsal root fibers to potassium ions and a hypothesis concerning the peripheral mechanism of pain
      
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The alkaloids (RAA) of Rauwolfia roots collected from the subtropical zone of Yunnan, identified as Rauwolfia yunnanensis Tsiang var. angustifolia C. Y. Wu., produced a definite hypotensive effect in anesthetized dogs and cats. Tachyphylaxis developed on repeated medications. The fall of blood pressure produced by RAA was neither due to effect on Mcholinergic and N-cholinergic systems, nor to the effect on interoceptors and to the direct dilatation of the blood vessels. Furthermore in unanesthetized rabbits...

The alkaloids (RAA) of Rauwolfia roots collected from the subtropical zone of Yunnan, identified as Rauwolfia yunnanensis Tsiang var. angustifolia C. Y. Wu., produced a definite hypotensive effect in anesthetized dogs and cats. Tachyphylaxis developed on repeated medications. The fall of blood pressure produced by RAA was neither due to effect on Mcholinergic and N-cholinergic systems, nor to the effect on interoceptors and to the direct dilatation of the blood vessels. Furthermore in unanesthetized rabbits intravenous injection of the RAA produced dilatation of ear vessels with mormal innervation. While the denervation ear was not affected. RAA inhibited the pressor responses to carotid occlusion, to electrical stimulation of the afferent vagi and tibial nerves. We suggested that RAA depressed the vasomotor centre. The hypotensive effect was still demonstrable in spinal cats, but to a lesser extent. RAA inhibited the pressor responses to injectious of adrenaline or noradrenaline. Hence the peripheral mechanism of the hypotensive effect of RAA considered as adrenolytic action. The mice survived an oral dose of RAA at 2000 mg per kg, all animals showed sedation.

继中国蘿芙木之后,最近在云南又发現狹叶蘿芙木(Rauwolfia yunnanensis Tsiang var.angustifolia C.Y.Wu.)。其根生物碱对麻醉动物具降压作用,給犬靜脉注射1毫克/公斤后2小时內的降压面积为-32%,給猫靜脉注射3毫克/公斤的降压面积为-30%。同时測得中国蘿芙木根生物碱在犬的降压面积为-45%,猫为-29%。重复給药有快速耐受性。降压的同时呼吸呈兴奋,对心率影响不明显。降压作用与M-胆碱能反应系統、N-胆碱能反应系統以及內感受器都无关系。对血管也无直接作用,单側頸交感神經切除兔耳試驗发現,扩张血管的作用与交感神經有关。狹叶蘿芙木根生物碱可抑制腎上腺素或去甲腎上腺素的加压反应,也可抑制阻断頸总动脉血流和电刺激迷走神經或胫骨神經中枢端所致的加压反射。用脊猫試驗吋其降压作用大为減弱,但并不完全消除,降压面积仅为-14%。因此,其降压机制包括中枢和外周两方面,即对血管运动中枢的抑制作用和抗腎上腺素的作用。急性毒性試驗証明,狹叶蘿芙木根生物碱的毒性很小,小鼠口服2000毫克/公斤无死亡发生,动物呈現安靜状态。

Intravenous injection of l-stepholildine (SPD) 2.5, 10 and 40 mg/kg decreased blood pressure(BP ) by 30, 40 and 55% respectively in anesthetized dogs. SPD also decreased BP in anesthetized rats (0.5mg/kg iv, by 29% and 2-50 mg/kg injected into duodenum, by 17-36%) and rabbits (2.5 mg/kg iv, by 24%). It could inhibit the pressor reflexes induced by occluding carotid, stimulating vagus Or sciatic nerve. Furthermore, SPD 2.5 mg/kg injected into fourth cerebroventricle of dog decreased BP by 20%, and in pithed rats...

Intravenous injection of l-stepholildine (SPD) 2.5, 10 and 40 mg/kg decreased blood pressure(BP ) by 30, 40 and 55% respectively in anesthetized dogs. SPD also decreased BP in anesthetized rats (0.5mg/kg iv, by 29% and 2-50 mg/kg injected into duodenum, by 17-36%) and rabbits (2.5 mg/kg iv, by 24%). It could inhibit the pressor reflexes induced by occluding carotid, stimulating vagus Or sciatic nerve. Furthermore, SPD 2.5 mg/kg injected into fourth cerebroventricle of dog decreased BP by 20%, and in pithed rats SPD 10-40 mg/kg iv depreased BP by 7-40%. These results indicate that both central and peripheral mechanisms are involved in the hypotens-ive action of SPD and the peripheral seems to be more important. As showed in previous papers the effect of SPD on α-adrenoceptors may be one of the chief mechanisms of the hypotensive action induced by SPD.

麻醉犬、大鼠、兔,静脉注射或十二指肠注射SPD均有降压作用。SPD对犬颈总动脉升压反射及刺激迷走神经、坐骨神经升压反射都呈抑制。犬第四脑室及毁脊髓大鼠注射SPD均可降压。这表明SPD降压存在中枢和外周作用部位,外周可能更重要,血管α受体可能是SPD降压的主要机理之一。

The present study investigates the influence of serotonin (5-HT) on the primary humoral immune response to sheep red blood cells (SRBC) in the rat. The results are as follows: Systemic administration of 5-hydroxytryptophaa (S-HTP) resulted in suppression of immune response, parachlorphenylalanine (PCPA) markedly enhanced immune response when injected intraperitoneally. Immune response was significantly affected neither by 5,7-dihydroxytryptamin(5,7-DHT)which was intracerebroventricularly injected into the rat...

The present study investigates the influence of serotonin (5-HT) on the primary humoral immune response to sheep red blood cells (SRBC) in the rat. The results are as follows: Systemic administration of 5-hydroxytryptophaa (S-HTP) resulted in suppression of immune response, parachlorphenylalanine (PCPA) markedly enhanced immune response when injected intraperitoneally. Immune response was significantly affected neither by 5,7-dihydroxytryptamin(5,7-DHT)which was intracerebroventricularly injected into the rat pretreated with desmethylimipramine (DMI)nor by intracerebroven-tricular injection of serotonin. Intraperitoneal administration of serotonin significantly suppressed immune response.Immune function was enhanced following injection of cypro-heptadine, a antagonist of serotoninergic recepeptor. These resulults suggest that sero-ntonin can suppress the primary humoral immune response to SRBC in the rat and im-munomodulation by serotonin is mediated via peripheral mechanism which may be related to the serotoninergic receptor on the immune cells.

本研究用神经药理学方法改变大鼠全身、中枢或外周5-羟色胺(5-HT)的含量,并使用5-HT受体的拮抗剂,观察初次体液免疫应答有无变化,从而探讨5-HT调节体液免疫功能的作用途径和作用机制。结果发现:大鼠腹腔注射5-羟色氨酸(5-HTP)后,体液免疫应答减弱。腹腔注射对氯苯丙氨酸(PCPA)后,免疫应答增强。用去甲丙味嗪(DMI)预处理,然后侧脑室注射5、7-双羟色胺(5、7-DHT);或者单独侧脑室注射5-HT,免疫应答都无显著变化,腹腔注射5-HT后,免疫应答减弱。给予赛庚啶阻断5-HT的受体后,免疫应答增强。本研究结果提示:5-HT对体液免疫应答具有抑制作用;此作用是通过外周途径实现的;其机制可能与免疫细胞上的5-HT受体有关。

 
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