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cerebral ischemic hypoxia
相关语句
  脑缺血缺氧
     STUDY SELECTION: The data were selected firstly to find out those about the pharmacological mechanism of the effect of Ginkgo biloba extract on brain tissue and neural cells, clinical application, the protective effect of its active components on cerebral ischemic hypoxia and hydrops, and the prevention of Parkinson's disease caused by memory lost of the cerebral cortex and pathological changes of brain.
     资料选择:对资料进行初审,纳入银杏叶提取物作用于脑细胞及神经细胞的相关药理机制,临床运用及其有效成分,对脑缺血缺氧,水肿的保护以及大脑皮质层的记忆功能丧失和脑部病变引起的帕金森病的防治相关的文献。
短句来源
     DATA EXTRACTION: A total of 44 articles about protective effect of Ginkgo biloba extract on brain tissue and neural injury were collected, 19 ones about the pharmacological mechanism of the effect of Ginkgo biloba extract on brain tissue and neural cells, clinical application, the protective effect of its active components on cerebral ischemic hypoxia and hydrops, and the prevention of Parkinson's disease caused by memory lost of the cerebral cortex and pathological changes of brain were selected;
     资料提炼:共收集到44篇关于银杏叶提取物对脑组织及神经损伤保护作用的文献,纳入为银杏叶提取物作用于脑细胞及神经细胞的相关药理机制,临床运用及其有效成分,对脑缺血缺氧,水肿的保护以及大脑皮质的记忆功能丧失和脑部病变引起的帕金森病的防治相关的文献19篇。
短句来源
     ②Protective effect of the extract on cerebral ischemic hypoxia and hydrops: The flavonoid glycoside, Terpene lactone and monomer in the extract have many pharmacological activities, which are closely related to the protective effect on cerebral ischemic hypoxia and hydrops and anti platelet activating factor, anti free radical and the improvement of cerebral vessel circulation.
     ②银杏叶提取物对脑缺血缺氧、水肿的保护作用:银杏叶提取物中黄酮苷、萜烯内脂和单体具有多种药理活性,对脑缺血缺氧、水肿保护作用与抗血小板活化因子、抗自由基和改善脑血管循环的作用密切相关。
短句来源
     Conclusion:l-Octacosanol can promote tolerance of mice to isobaric hypoxia, cerebral ischemic hypoxia and myocardial hypoxia.
     结论:二十八烷醇可显著提高小鼠对于常压密闭缺氧、大脑缺血缺氧和心肌缺氧的耐受能力。
短句来源
  相似匹配句对
     Hypoxia-Inducible Factor-1 and Cerebral Ischemic Tolerance
     缺氧诱导因子-1与脑缺血耐受
短句来源
     Pathogenesis of Ischemic Cerebral Apoplexy
     缺血性脑中风的发病机理探讨
短句来源
     Microglia and ischemic cerebral damagse
     小胶质细胞与缺血性脑损伤
短句来源
     Alterations of amino acid zymogram after severe head injury with cerebral ischemic and hypoxia
     重型颅脑损伤合并缺血缺氧后氨基酸谱改变
短句来源
     Protective effect of ischemic preconditioning on cerebral ischemia and hypoxia: historical retrospect and research progress
     缺血预处理防治脑缺血缺氧的历史回顾与研究进展
短句来源
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  cerebral ischemic hypoxia
We describe positron-emission tomography (PET) scintigraphic findings obtained using pyruvate-1-11C in eight patients with cerebral ischemic hypoxia or infarction.
      


BACKGROUND:In the treatment of hypertensive cerebral haemorrhage, many approaches are clinically applied to improve hypoxia at present.Whether the application of oxygenised liquid during surgery had protective effects on neurocyte in the patients with hypertensive cerebral haemorrhage?OBJECTIVE:To investigate the impact and brain protective effect of the application of oxygenised liquid during the surgery of hypertensive cerebral haemorrhage on the myelm basic protein(MBP). DESIGN:A randomised controlled trial.SETTING...

BACKGROUND:In the treatment of hypertensive cerebral haemorrhage, many approaches are clinically applied to improve hypoxia at present.Whether the application of oxygenised liquid during surgery had protective effects on neurocyte in the patients with hypertensive cerebral haemorrhage?OBJECTIVE:To investigate the impact and brain protective effect of the application of oxygenised liquid during the surgery of hypertensive cerebral haemorrhage on the myelm basic protein(MBP). DESIGN:A randomised controlled trial.SETTING and PARTICIPANTS:Study was conducted in the Department of Clinical Anaesthesia of Brain Hospital of Nanjing Medical University.Subjects were surgical patients with hypertensive cerebral haemorrhage admitted by Nanjing Brain Hospital from 1999 to 2001.The inclusive cases were cases with 40 mL to 60 mL of bleeding as shown in cranial CT and grade I-III of American Society of Anaesthesiologist(ASA) classification.INTERVENTIONS:Oxygenised liquid or equilibrium liquid was intravenously injected respectively into the patients of two groups during the surgery.MAIN OUTCOME MEASURES:Serous myelin basic protein(MBP) concentrations before and 12 hours after operation in the patients of two groups were observed respectively.The preoperative and postoperative(on the 7th day) Glasgow coma scale(GCS) as well as the intracranial pressure at each operative stage was observed. RESULTS:There was no difference of intracranial pressure before surgery and after induction between two groups. The intracranial pressures of oxygenised liquid group[(1.87±0.24) and(1.73±0.18) kPa]at haematoma removal and after operation respectively were significantly lower than[(2.10±0.381) and(1.92±0.22) kPa]of control group(t=2.37,P< 0.05;t=3.06,P< 0.01).Serous MBP at postoperative 12 hours in oxygenised liquid group was[(4.1±1.35) μg/L],which was significantly lower than[(2.2±1.42) μg/L] of control group(t=2.58,P< 0.05). The GCS at the 7th postoperative day in oxygenised liquid group was(13.6 ±0.88),which was significantly higher than(12.7±1.22) of control group(t=2.74,P< 0.05).CONCLUSION:The early intervention on haematoma surround tissue by early input oxygenised liquid during hypertensive cerebral haemorrhage surgery that could effectively relieve cerebral oedema and improve cerebral ischemic hypoxia to save reversibly injured neurons surrounding haematoma, is an effective intervention,which is helpful to the cerebral protection in the patients with hypertensive cerebral haemorrhage.

背景:在高血压脑出血的治疗中,目前临床上应用多种方式改善机体缺氧,术中应用携氧液是否对高血压脑出血患者神经细胞有保护作用?目的:研究高血压脑出血术中应用携氧液对患者神经元特异性烯醇化酶的影响及脑保护作用。设计:随机对照的实验研究。地点和对象:研究地点为南京医科大学脑科医院临床麻醉研究室,对象为1999/2001年南京脑科医院收治的高血压脑出血手术患者。选择头颅CT示出血量40~60mL,美国麻醉协会(ASA)分级为I~III级,其中男26例,女14例,平均62岁。随机分为携氧液组和对照组。方法:两组术中分别采用输注携氧液和平衡液。主要观察指标:分别观察两组患者术前和术后12h的血清髓鞘碱性蛋白的浓度,术前和术后第7天的格拉斯格评分以及手术各阶段的颅内压力。结果:两组颅内压在手术前和诱导后无差别,在清除血肿至术毕,携氧液组颅内压犤(1.87±0.24,1.73±0.18)kPa犦明显低于对照组犤(2.10±0.38,1.92±0.22)kPa犦犤(t=2.37,P<0.05;t=3.06,P<0.01)犦;术后12h,携氧液组血清髓鞘碱性蛋白犤(4.1±1.35)μg/L犦明显低于对照组犤(5.2±1.42)μ...

背景:在高血压脑出血的治疗中,目前临床上应用多种方式改善机体缺氧,术中应用携氧液是否对高血压脑出血患者神经细胞有保护作用?目的:研究高血压脑出血术中应用携氧液对患者神经元特异性烯醇化酶的影响及脑保护作用。设计:随机对照的实验研究。地点和对象:研究地点为南京医科大学脑科医院临床麻醉研究室,对象为1999/2001年南京脑科医院收治的高血压脑出血手术患者。选择头颅CT示出血量40~60mL,美国麻醉协会(ASA)分级为I~III级,其中男26例,女14例,平均62岁。随机分为携氧液组和对照组。方法:两组术中分别采用输注携氧液和平衡液。主要观察指标:分别观察两组患者术前和术后12h的血清髓鞘碱性蛋白的浓度,术前和术后第7天的格拉斯格评分以及手术各阶段的颅内压力。结果:两组颅内压在手术前和诱导后无差别,在清除血肿至术毕,携氧液组颅内压犤(1.87±0.24,1.73±0.18)kPa犦明显低于对照组犤(2.10±0.38,1.92±0.22)kPa犦犤(t=2.37,P<0.05;t=3.06,P<0.01)犦;术后12h,携氧液组血清髓鞘碱性蛋白犤(4.1±1.35)μg/L犦明显低于对照组犤(5.2±1.42)μg/L犦(t=2.58,P<0.05)。术后第7天,携氧液组格拉斯哥评分(13.6±0.88)明显高于对照组(12.7±1.22)(t=2.74,P<0.05)。结论:在高血压脑出血术中早期输入携氧液对血肿脑

OBJECTIVE: Ginkgo biloba extract is mainly composed of flavonoid glycoside, Terpene lactone and monomer lactone, its mechanism and curative effect on cardiovascular system, cerebral circulation, blood, nerve and so on have been proved. In this paper, we focus on the studies about the protective effect of Ginkgo biloba extract on brain tissue and neural injury. DATA SOURCES: A computer-based online search of Medline database was undertaken for related articles published in English between January 1994 and December...

OBJECTIVE: Ginkgo biloba extract is mainly composed of flavonoid glycoside, Terpene lactone and monomer lactone, its mechanism and curative effect on cardiovascular system, cerebral circulation, blood, nerve and so on have been proved. In this paper, we focus on the studies about the protective effect of Ginkgo biloba extract on brain tissue and neural injury. DATA SOURCES: A computer-based online search of Medline database was undertaken for related articles published in English between January 1994 and December 2004, with the of "Ginkgo biloba extract". Meanwhile, we searched the Chinese Journal Full-text Database and Wanfang database for related Chinese articles between January 1995 and December 2003 with the same key words in Chinese. STUDY SELECTION: The data were selected firstly to find out those about the pharmacological mechanism of the effect of Ginkgo biloba extract on brain tissue and neural cells, clinical application, the protective effect of its active components on cerebral ischemic hypoxia and hydrops, and the prevention of Parkinson's disease caused by memory lost of the cerebral cortex and pathological changes of brain. Exclusive criteria: review articles and repetitive studies. DATA EXTRACTION: A total of 44 articles about protective effect of Ginkgo biloba extract on brain tissue and neural injury were collected, 19 ones about the pharmacological mechanism of the effect of Ginkgo biloba extract on brain tissue and neural cells, clinical application, the protective effect of its active components on cerebral ischemic hypoxia and hydrops, and the prevention of Parkinson's disease caused by memory lost of the cerebral cortex and pathological changes of brain were selected; 25 review articles and repetitive studies were excluded. DATA SYNTHESIS: ①Protective effect of Ginkgo biloba extract on cerebral thrombosis: The extract can obviously inhibit the increase of tissue plasminogen reactivator inhibitory factor 1, regular the fibrinolytic enzyme process to prevent thrombosis forming. ②Protective effect of the extract on cerebral ischemic hypoxia and hydrops: The flavonoid glycoside, Terpene lactone and monomer in the extract have many pharmacological activities, which are closely related to the protective effect on cerebral ischemic hypoxia and hydrops and anti platelet activating factor, anti free radical and the improvement of cerebral vessel circulation. ③Effect of the extract on memory: The extract can improve the learning and memory ability through increasing the reuptake of hippocampal choline, decreasing the lost of M choline receptor and α2 adrenoceptor and transmitter. ④Preventive effect of the extract on Parkinson's disease: It can protect the survival injured substantia nigra neuron to control the decrease of striatal dopamine and keep it at a certain level; the Ginkgo biloba lactone can increase the dopamine by inhibiting the metabolism of striatal dopamine. ⑤Protective effect of the extract on cerebral nerve: The extract can protect the cerebral nerve by inhibiting the appearance of nitrogen monoxidum in neural cells, the inflammatory reaction mediated by the neutrophil and direct against to glutamic acid receptor. CONCLUSION: Ginkgo biloba extract and monomer, whose research in animal experiment and application in cardiovascular disease has obtained a lot of achievements, can activate brain tissue and neural injured cells, improve cerebral circulation, and effectively treat and prevent some cerebral diseases.

目的:银杏叶提取物主要成分黄酮苷、萜烯内酯和单体内酯等对心血管系统,脑循环,血液及神经等方面作用机制和疗效已基本肯定,现就银杏叶提取物对脑组织及神经损伤保护作用的研究进行综述。资料来源:应用计算机检索Medline数据库1994-01/2004-12期间的相关文章,检索词为“Ginkgobilobaextract”,限定文章语言种类为英文;同时计算机检索中国期刊全文数据库、万方数据库1995-01/2003-12期间的相关文章,检索词为″银杏叶提取物″,限定文章语言种类为中文。资料选择:对资料进行初审,纳入银杏叶提取物作用于脑细胞及神经细胞的相关药理机制,临床运用及其有效成分,对脑缺血缺氧,水肿的保护以及大脑皮质层的记忆功能丧失和脑部病变引起的帕金森病的防治相关的文献。排除标准:综述类文献和重复研究。资料提炼:共收集到44篇关于银杏叶提取物对脑组织及神经损伤保护作用的文献,纳入为银杏叶提取物作用于脑细胞及神经细胞的相关药理机制,临床运用及其有效成分,对脑缺血缺氧,水肿的保护以及大脑皮质的记忆功能丧失和脑部病变引起的帕金森病的防治相关的文献19篇。排除综述类文献和重复研究25篇。资料综合:①银杏叶提取物对脑血栓...

目的:银杏叶提取物主要成分黄酮苷、萜烯内酯和单体内酯等对心血管系统,脑循环,血液及神经等方面作用机制和疗效已基本肯定,现就银杏叶提取物对脑组织及神经损伤保护作用的研究进行综述。资料来源:应用计算机检索Medline数据库1994-01/2004-12期间的相关文章,检索词为“Ginkgobilobaextract”,限定文章语言种类为英文;同时计算机检索中国期刊全文数据库、万方数据库1995-01/2003-12期间的相关文章,检索词为″银杏叶提取物″,限定文章语言种类为中文。资料选择:对资料进行初审,纳入银杏叶提取物作用于脑细胞及神经细胞的相关药理机制,临床运用及其有效成分,对脑缺血缺氧,水肿的保护以及大脑皮质层的记忆功能丧失和脑部病变引起的帕金森病的防治相关的文献。排除标准:综述类文献和重复研究。资料提炼:共收集到44篇关于银杏叶提取物对脑组织及神经损伤保护作用的文献,纳入为银杏叶提取物作用于脑细胞及神经细胞的相关药理机制,临床运用及其有效成分,对脑缺血缺氧,水肿的保护以及大脑皮质的记忆功能丧失和脑部病变引起的帕金森病的防治相关的文献19篇。排除综述类文献和重复研究25篇。资料综合:①银杏叶提取物对脑血栓的保护性作用:银杏叶提取物可明显抑制缺血再灌注损伤所致的组织纤溶酶原激活因子抑制因子Ⅰ的升高,调节纤维蛋白溶酶过程,防止血栓形成。②银杏叶提取物对脑缺血缺氧、水肿的保护作用:银杏叶提取物中黄酮苷、萜烯内脂和单体具有多种药理活性,对脑缺血缺氧、水肿保护作用与抗血小板活化因子、抗自由基和改善脑血管循环的作用密切相关。③银杏叶提取物对大脑记忆力的影响:银杏叶提取物主要是通过促进大脑海马胆碱的再摄取,减少M型胆碱受体和α2肾上腺素受体及递质的丢失,从而发挥改善学习和记忆能力的功能。④银杏叶提取物对帕金森病的防治作用:其机制是通过保护残存的损伤侧黑质神经元,使得纹状体多巴胺的减少得以控制,并保持在一定的水平;银杏内脂抑制纹状体多巴胺的代谢,从而升高多巴胺的水平。⑤银杏叶提取物对脑神经的保护作用:银杏叶提取物可能通过抑制神经细胞一氧化氮的产生、抑制嗜中性粒细胞所介导的炎症反应、直接对抗谷氨酸受体等保护脑神经。结论:银杏叶提取物和单体的研究在动物实验和心血管疾病的运用性研究上取得了广泛的成果,对于激活脑组织及神经损伤的细胞,改善脑血管循环,治疗和预防脑部某些疾病有一定的作用。

Objective:To observe the anti-response to stimulation effect of Aralia elata(Miq) seem.Methods:Poured the medicine at 1,2.5,6.25g.kg or;0.1,0.2g.kg into mice's stomach and reckoned mice's swimming time,time of hypoxia under normal pressure and the surviving time of cerebral ischemic hypoxia.Results:Aralia elata(Miq) seem liquor Could apparently prolong the above hours.Conclusion:Aralia elata(Miq) seem has prominent anti-response to stimulation effect.

目的本课题从实验的角度研究辽东楤木抗应激作用。方法根实验要求将不同剂量的辽东楤木灌入小鼠胃中并计算小鼠的游泳时间、常压耐缺氧时间和脑缺血缺氧存活时间。结果辽东楤木能显著地延长小鼠游泳时间、常压耐缺氧时间和脑缺血缺氧存活时间。结论辽东楤木具有突出的抗应激作用。

 
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