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cerebral ischemic damage
相关语句
  缺血性脑损伤
    We provide new evidence that TNFa is involved in cerebral ischemic damage. We concluded that at early stage of cerebral ischemia, the dominant role of TNFa is induce inflammatory damage to ischemic tissue.
    本研究为TNFa参与缺血性脑损伤提供了新的证据,并且表明在脑缺血的早期(24h内),TNFa的首要作用是促进炎症反应导致组织损伤。
短句来源
    With recent progress in machanism of cerebral ischemic damage, neuroprotective therepy has been an innovative conception in clinical therepy.
    近年来,随着对缺血性脑损伤机制研究的不断深入,神经保护治疗已成为临床神经病学治疗的一个变革性概念。
短句来源
    Objective To study the effects of calcium on cerebral ischemic damage.
    目的探讨Ca2+在缺血性脑损伤中所起的作用。
短句来源
    : In order to study the action and significance of platelet-activating factor (PAF)andLipid peroxidex(LPO) on the cerebral ischemic damage,the levels in rat hipppocampus were observed dynamically.
    目的:运用脑缺血再灌注模型,探索血小板活化因子及自由基在缺血性脑损伤中的地位及意义。
短句来源
  脑缺血损伤
    Objective To study the levels of catecholamines (CA) in plasma of patients with cerebral infarction and in brain tissue of cerebral ischemia reperfusion gerbils and its effect in cerebral ischemic damage as well as the relation with clinical manifestation of infartion patients .
    目的研究脑梗塞患者及脑缺血鼠脑组织儿茶酚胺(CA)浓度变化,和其在脑缺血损伤中的作用以及与脑梗塞患者的临床表现的关系。
短句来源
    Objective:To study the levels of CD 62p in plasma of patients with cerebral infarction and its effect in cerebral ischemic damage.
    目的 :探讨脑梗死患者血浆CD6 2p水平的变化和其在脑缺血损伤中的作用。
短句来源
  “cerebral ischemic damage”译为未确定词的双语例句
    Experimental studies on cerebral ischemic damage following unilateral carotid occlusion and the protective effect of 764-3 in rats
    大鼠单侧颈动脉阻断所致的脑缺血性损害和764-3脑保护作用的实验研究
短句来源
    Progress of study in cerebral ischemic damage
    脑缺血损伤的研究进展
短句来源
    Cerebral ischemic damage and inflammatory reaction
    脑缺血与炎症反应
短句来源
    Effects of Puerarin on the Expression of GAD、VEGF After Experimental Cerebral Ischemic Damage in Rats
    葛根素对实验性脑缺血GAD、VEGF表达变化的影响
短句来源
    Results: The result showed that the content of lipid - bound sialic acid was 2. 67 ± 0. 33 mg/L (x ± s) in normal serum and was 1. 88 ± 0. 24 mg/L in the patient serum with acute cerebral infarct (P < 0. 01). There was a close correlation between the decrease of lipid - bound sialic acid content and the degree and location of cerebral ischemic damage.
    结果:血清脂结合唾液酸含量同龄组健康人为 2.67±0.33 mg/L,脑梗死患者急性期含量显著降低,为1.88±0.24 mg/L(P<0.01);
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  cerebral ischemic damage
These results indicate the feasibility of using mRNA measurement as a parameter to assess cerebral ischemic damage.
      
Thus far, experimental studies using this model have demonstrated a direct interaction of cerebral ischemic damage and AD-type neuropathologies in the primary ischemic area.
      
The authors used a clinically relevant model of focal CNS reperfusion injury to test whether treatment with doxycycline would reduce cerebral ischemic damage and improve functional outcome.
      
Neutrophil elastase inhibition reduces cerebral ischemic damage in the middle cerebral artery occlusion.
      
Involvement of cytokine production in pathogenesis of transient cerebral ischemic damage.
      
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Temporary occlusion of unilateral carotidarteries in the rats was performed.The cere-bral ischemic damage manifested by mi-cropathological changes in neural cells. Nomacro-infarction and neural deficit symptomscould be found.Occlusion of common carotidartery (CCA) internal carotd artery (ICA),and external carotid artery (ECA) in the sametime cerebral ischemic damage were very se-vere.Damage often occlusion of the ICA,showed rather mild and the occlusion of theCCA was very mild.Occlusion of unilateral...

Temporary occlusion of unilateral carotidarteries in the rats was performed.The cere-bral ischemic damage manifested by mi-cropathological changes in neural cells. Nomacro-infarction and neural deficit symptomscould be found.Occlusion of common carotidartery (CCA) internal carotd artery (ICA),and external carotid artery (ECA) in the sametime cerebral ischemic damage were very se-vere.Damage often occlusion of the ICA,showed rather mild and the occlusion of theCCA was very mild.Occlusion of unilateral carotid arteries for more than one hour mayresult irreversible ischemic damage in neuralcells and cerebral edema.764-3 may reducethe level of prostaglandin F_(2α) (PGF_(2α) in cere-bral cortex and the severity of ischemic dam-age of neural cells.

采用三种方式暂时阻断大鼠单侧颈动脉(CA):阻断颈总动脉(CCA),阻断颈内动脉(ICA),阻断 CCA、ICA 和颈外动脉(ECA)。此三种阻断方式可造成脑缺血性损害,但不引起脑梗塞和神经缺失症状。以同时阻断 CCA、ICA 和 ECA 造成的脑缺血性损害最重,阻断 ICA 次之,阻断 CCA最轻。阻断单侧 CA1小时以上,可造成脑皮层神经细胞缺血性损害加重,若缺血时间延长可发生不可逆损害,伴有缺血性脑水肿。764-3作为脑保护剂,可降低由于阻断单侧 CA 引起的脑皮层前列腺素 F_(2α)(PGF_(2α)的增加,减轻神经细胞缺血性损害。

The protective effect of monoclonal antibody to leukocyte CD_18(MoAL CD_18)was observed in an animal model of local cerebral vascular thrombosis induced photochemically.The results demonstrated that the MoAL CD_18 could decrease the size of ceredral infarct areas and improve the neurological functional disabilities significantly.It implied that inhibiting the adherence of Leukocyte could be an alternate way to treat and prevent the cerebral ischemic damage.

本文用光诱导局部血栓形成动物模型,观察了抗白细胞CD18单克隆抗体在脑缺血损伤中的保护作用。研究结果表明,抗白细胞CD18单克隆抗体可显著缩小梗塞面积,减轻脑梗塞后的神经功能缺失状况,提示抑制白细胞粘附介导的缺血损伤是防治脑梗塞的一种新途径。

The cortical cyclic AMP (CAMP) .superoxide (SOD), malondialdehyde (MO A) contents and pathological chandes were obsered during ischemia and reperfusion period in rats either with a single 15 - minute ischemia or with repeated three - 5 minute ischemia. It was found that the cortical cAMP contents in the repeated ischemic group were singnificanty higher,the level of SOD activity decr-dased significantly during ischemia period and the early reperfusion, whereas the contents of MDA showed a delayed and long - lasting...

The cortical cyclic AMP (CAMP) .superoxide (SOD), malondialdehyde (MO A) contents and pathological chandes were obsered during ischemia and reperfusion period in rats either with a single 15 - minute ischemia or with repeated three - 5 minute ischemia. It was found that the cortical cAMP contents in the repeated ischemic group were singnificanty higher,the level of SOD activity decr-dased significantly during ischemia period and the early reperfusion, whereas the contents of MDA showed a delayed and long - lasting increase. Meanwhile, the cortical damage in the group of repeated cerebral ischemia was more severe than single ischemic insult gyoup. The present results swggested that the cAMP and free redical may participate in the pathogenesis of repeated cerebral ischemic damage

本实验动态对比观察单次性15mim和反复性3×5min大鼠全脑缺血/再灌流,大脑皮质cAMP、SOD和MDA含量的变化及相应的病理改变、发现反复性脑缺血及再灌流早期cAMP含量明显增高,SOD活性显著降低,MDA则延迟性持续显著增高;反复性缺血组皮质神经元损害显著重于单次缺血组。结果提示cAMP和自由基均可能参与了反复性脑缺血神经元损害的病生机制。

 
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