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ischemic brain tissues
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  缺血脑组织
     Expressions of TNF-α and IL-1β in human ischemic brain tissues
     人缺血脑组织中TNF-α和IL-1β的表达
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     NO upgrades expression of VEGF protein and mRNA in local ischemic brain tissues of rabbits
     NO增加兔局灶脑缺血后缺血脑组织VEGF表达
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     The Effect of VEGF on the Expression of Fas and FasL in Ischemic Brain Tissues
     EGF对缺血脑组织表达Fas/Fasl的影响
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     In situ hybridization was used to characterize the expression of mRNA for the endostatin. Results Both the protein (at least 50%, P < 0.01) and mRNA (at least 70%, P < 0.05) of endostatin increased significantly in the ischemic brain tissues after MCAO compared with the control group. VEGF increased at least 270% in the brain after cerebral ischemia (P < 0.05).
     结果与对照组相比,MCAO局灶性脑缺血后内皮抑素蛋白和mRNA表达均明显增加,至少分别增加了50%(P<0.01)和70%(P<0.05),同时缺血脑组织VEGF含量也明显增加,至少增加了270%。
短句来源
     RESULTS The erigeron breviscapus increased rCBF and decreased MPO activity, and had no effect on the changes of NO in the focal ischemic brain tissues.
     结果灯盏花素对缺血脑组织NO的产生无明显影响,但可明显改善缺血脑组织的rCBF和显著降低MPO活性。
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  “ischemic brain tissues”译为未确定词的双语例句
     Objective:To study the expression of interleukin-6 (IL-6) and tumor necrosis factor-α(TNF-α) mRNA in ischemic brain tissues and the serum levels of IL-6 and TNF-α in rats with focal brain ischemia.
     目的:研究在大鼠局灶性脑缺血的动物模型中脑组织白细胞介素6(IL-6)和肿瘤坏死因子α(TNF-α)的表达变化及血清中IL-6和TNF-α的含量的变化。
短句来源
     (4) Use the immunohistochemical and the in situ hybridization method to observed the expressions of the TNF-a , E-selectin P-selectin ICAM-1 TGF-P 1 and their mRNA in ischemic brain tissues.
     (4)采用免疫组化和原位杂交技术动态观察缺血侧脑组织TNF-α、E-selectin、P-Selectin、ICAM-1、TGF-β1及其各自mRNA的表达水平。
短句来源
     Results PAF antagonist WEB2086 increased rCBF and decreased MPO activity and NO in the focal ischemic brain tissues.
     结果 PAF受体拮抗剂 WEB2086 对 IR脑组织NO的产生有明显影响,且可明显改善 IR脑组织的 rCBF和显著降低 MPO活性,最终减轻局部 IR脑损害。
短句来源
     Results:Compared with those in sham-operated group,the expression of posityive neuronal cells of TUNEL and positive cells of Fas-L protein increased in model group rats′ ischemic brain tissues.
     结果与假手术组相比,模型组大鼠缺血侧脑组织TUNEL阳性神经细胞及Fas-L阳性细胞表达增多;
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     Conclusion Brain-activating acupuncture can inhibit the synthesis and secretion of IL-1β by ischemic brain tissues so as to reduce IL-1β-caused injury in cerebral ischemia and produce a protective effect on the brain.
     结论 “醒脑开窍”针法能抑制缺血区脑组织 IL - 1β的合成和分泌 ,从而减轻由 IL - 1β引发的一系列脑缺血损害 ,发挥脑保护作用。
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  相似匹配句对
     brain
     脑
短句来源
     Brain hypoxic and ischemic proconditioning
     脑缺血缺氧预处理
短句来源
     The brain of F.
     随发育脑体积增加,髓质占脑体积的比例增加。
短句来源
     Oligodendrocytes and Ischemic Brain Injury
     少突胶质细胞与缺血性脑损伤
短句来源
     Extract from hirudo protects ischemic brain tissues
     水蛭提取物对大鼠脑血栓后脑组织MDA,SOD和NO含量的影响
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  ischemic brain tissues
The Northern blot and Western blot analyses showed that the ischemia-induced expression of CD44 was time dependent in the ischemic brain tissues.
      
Immunoblot analysis of Bax, Bcl-2, and Bcl-X in ischemic brain tissues.
      


Objective It was designed to find out whether the lipid peroxidation caused by free radicals might take part in causing the damage of cerebral ischemic neurons. Method In this study, the focal cerebral ischemia models of rats were made with thread embolism of the middle cerebral artery(MCA). The hydroxyl radical(OH ·) could be trapped by salicylate to form 2,3 and/or 2,5 dihydroxybenzoic acid (DHBA) which were readily measured with high performance liquid chromography (HPLC). Results We found that the...

Objective It was designed to find out whether the lipid peroxidation caused by free radicals might take part in causing the damage of cerebral ischemic neurons. Method In this study, the focal cerebral ischemia models of rats were made with thread embolism of the middle cerebral artery(MCA). The hydroxyl radical(OH ·) could be trapped by salicylate to form 2,3 and/or 2,5 dihydroxybenzoic acid (DHBA) which were readily measured with high performance liquid chromography (HPLC). Results We found that the levels of 2,3 and 2,5 DHBA formation were increased for about 3 4 folds in the striatum and cerebral cortices of the rats after MACO, respectively. Exidently mannitol could attenuate the increased 2,3 and 2,5 DHBA levels in the striatum and cerebral cortices following focal ischemia in the rats and improve the damage of the neuronal function. However, quick dripping injections of mannitol solution could reduce the edematous areas in the ischemic brain tissues. slow dripping injections of mannitol solution showed no significant effect on reducing the edematous areas of the rat brains. Conclusion These results suggested that mannitol injections could reduce free radicals production and this protective activity would probably and incompletely be linked with its dehydration effect.

观察甘露醇对脑缺血鼠脑组织羟自由基的清除作用。方法采用线栓法制成大脑中动脉脑缺血模型,利用高压液相色谱技术测定脑组织中羟自由基的含量。结果脑缺血组鼠脑皮层及纹状体内2,3-二羟基苯甲酸(2,3-DHBA)、2,5-DHBA的水平较对照组增高3~4倍。甘露醇能有效地降低局灶性缺血脑皮层及纹状体2,3-DHBA及2,5-DHBA的水平,改善缺血后神经功能损害,且甘露醇持续慢滴组较快滴组效果明显。甘露醇快速滴注组能减轻缺血脑组织的水肿,而甘露醇慢滴组作用不明显。结论甘露醇的脑保护作用不完全与脱水降颅压有关,可能是通过清除羟自由基而发挥重要的脑保护作用

Objective To study the role of nitric oxide that might have double edged pathophysiological effects during the cerebral ischemic reperfusional injury. Method The rat models of middle cerebral artery (MCA) focal cerebral ischemia reperfusion were established with the suture method in the study. The nonselective NOS inhibitor and selective NOS inhibitor were used for investigating the effects of nitric oxide (NO) changes in the focal ischemic brain tissues and their respective roles...

Objective To study the role of nitric oxide that might have double edged pathophysiological effects during the cerebral ischemic reperfusional injury. Method The rat models of middle cerebral artery (MCA) focal cerebral ischemia reperfusion were established with the suture method in the study. The nonselective NOS inhibitor and selective NOS inhibitor were used for investigating the effects of nitric oxide (NO) changes in the focal ischemic brain tissues and their respective roles during the cerebral ischemia reperfusion. Results This study showed that the nonselective NOS inhibitor L NAME (15 mg/kg) intraperitoneally administrated aggravated the ischemic brain injury,but selective NOS inhibitor aminoguanidine (150mg/kg) intraperitoneally given had a definite, protective effect on the brains. Conclusion The results implied that the NO from different cells and isoforms of NOS had unlike roles in focal cerebral ischemic reperfusional injury.

目的研究一氧化氮在鼠脑局灶性脑缺血再灌注损伤中的作用。方法用线栓法建立大鼠大脑中动脉区缺血再灌注模型,分别用选择性和非选择性诱导型一氧化氮合酶抑制剂对鼠脑局灶性缺血再灌注过程中脑组织一氧化氮的变化规律及可能作用进行探讨。结果非选择性一氧化氮合酶抑制剂(L-NAME)可加重局灶性脑缺血性损害,而选择性诱导型一氧化氮合酶抑制剂(aminoguanidine,AG)具有明确的脑保护作用。结论不同类型的一氧化氮合酶所产生的一氧化氮在脑局灶性缺血性损害中具有不同的作用。

AIM Protein kinase C (PKC) was considered to have an important role in the cerebral ischemic/reperfusional damage, and a regulating role to the nitric oxide synthase. The erigeron breviscapus can inhibite the PKC activity, but its role and machanism in the cerebral ischemia/reperfusion are needed to explore. METHODS The animal models of middle cerebral artery ischemia/reperfusion were established by suture method, the nitric oxide (NO) concentration, regional cerebral blood flow (rCBF), myeloperoxidase of...

AIM Protein kinase C (PKC) was considered to have an important role in the cerebral ischemic/reperfusional damage, and a regulating role to the nitric oxide synthase. The erigeron breviscapus can inhibite the PKC activity, but its role and machanism in the cerebral ischemia/reperfusion are needed to explore. METHODS The animal models of middle cerebral artery ischemia/reperfusion were established by suture method, the nitric oxide (NO) concentration, regional cerebral blood flow (rCBF), myeloperoxidase of leukocytes (MPO) and cerebral infarction volume were measured. RESULTS The erigeron breviscapus increased rCBF and decreased MPO activity, and had no effect on the changes of NO in the focal ischemic brain tissues. CONCLUSION The PKC inhibitor erigeron breviscapus can improve rCBF and prevent the adhesion and infiltration of leukocytes into ischemic brain tissue, which was not concerned with the change of NO concentration, and suggested it may really have a protective effect on ischemic brain tissue.

目的蛋白激酶C(PKC)与脑组织缺血性损伤有密切关系,且证明可调节一氧化氮(NO)合酶的活性。灯盏花素可抑制PKC的活性,其对脑缺血/再灌流损伤的作用和机制需深入研究。方法采用线栓法制成大鼠大脑中动脉缺血/再灌流模型,对缺血脑组织NO含量、局部脑血流量(rCBF)、中性粒细胞髓过氧化物酶(MPO)及脑梗塞体积进行测定。结果灯盏花素对缺血脑组织NO的产生无明显影响,但可明显改善缺血脑组织的rCBF和显著降低MPO活性。结论PKC抑制剂灯盏花素对脑缺血的保护作用与NO的变化无关,其主要是通过抑制PKC、增加rCBF和降低缺血脑组织内中性粒细胞的粘附浸润而实现的。

 
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