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   induced hepatitis 的翻译结果: 查询用时:0.176秒
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induced hepatitis
相关语句
  性肝炎
     Clinical Analysis of 108 Cases of Drug Induced Hepatitis
     药物性肝炎108例临床分析
短句来源
     Results: 1.The incidence rate of male to female was 1:1.51.The on-set peakhappened in the 40—49 year-old in 108 cases of drug induced hepatitis in thisstudy.
     结果:1.本文随机抽取的108 例药物性肝炎,男、女发病率之比为1:1.51,发病高峰年龄段在40—49 岁之间。
短句来源
     A Statistical Analysis of 28 Cases of Drug Induced Hepatitis
     药物性肝炎28例统计分析
短句来源
     Physicians should avoid using drugs withhepatotoxicity. Studies showed that 2-5% patients in abroad hospital withjaundice was drug induced hepatitis and 20-50% fulminant hepatic failure wasrelated with drugs.
     国外报告药物性肝炎可占所有黄疸住院病例的2-5%,在暴发性肝功能衰竭患者中,有相当高的比例(20-50%)与药物有关。
短句来源
     2. Etiology: thecategory of drugs that induced hepatitis and the combined factors of liverdamage.
     2.病因学:引起药物性肝炎的药物种类、同时并发有其它损害肝脏的因素;
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  “induced hepatitis”译为未确定词的双语例句
     there was an significant difference when it was administered at a dose of 800 mg/kg (P<0.01), both dosages lowered the liver MDA content in the CCl-4 induced hepatitis model, both dosages (FGF400 mg/kg), FGF significantly decreased serum ALT and AST elevation at both dosages and increased the SOD and GSH activities while lowered MDA content.
     FGF两个剂量组均使MDA的含量降低。 FGF两个剂量组(400、800 mg/kg)对CCl4 引起的肝损伤模型均使血清ALT、AST水平明显降低,并能提高肝组织SOD活性,降低MDA含量。
短句来源
     AIM To measure the changes of HP450, CytP450 and histamine in the hepatic microsome in CCl 4, acetaminophen and D galactosamine induced hepatitis in rats.
     目的研究四氯化碳、醋氨酚、半乳糖胺诱发大鼠急性肝炎时肝脏微粒体中HP450及CytP450的变化。
短句来源
     4. Laboratory data: the feature inserum of drug induced hepatitis such as routine examination, and AST ALT BIL,and so on.
     4. 实验室检查:血常规、凝血常规及血清AST、ALT、ALP、BIL 等的特点;
短句来源
     In the PAR induced hepatitis model serum ALT and AST levels reduced with a preadministration of FGF at dosages of 800 mg/kg and 1200 mg/kg, and elevation in the SOD activity and a drop in MDA content were observed.
     FGF(800、1 200 mg/kg)对PAR引起的肝损伤模型均使血清ALT、AST水平明显降低,并能提高肝组织SOD活性,降低MDA含量。 在3种模型中FGF均能明显改善肝组织病理损伤程度。
短句来源
     While 8 patients with HBV infection and 6 of other liver diseases including 2 of autoimmune liver disease, 1 of Dubin Johnson syndrome, 1 of fatty liver, 1 of the primary biliary cirrhosis and 1 of drug induced hepatitis were all SEN-V DNA negative.
     另外 8例乙型肝炎及 6例其他肝病 (自身免疫性疾病 2例、DubinJohnson综合征 1例、脂肪肝 1例、原发性胆汁性肝硬化 1例、药物性肝损害 1例 )SEN VDNA检测阴性。
短句来源
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  相似匹配句对
     Induced into E.
     将表达载体转化E.
短句来源
     Hepatitis C virus induced glomerulonephritis
     丙型肝炎病毒感染后肾小球肾炎
短句来源
     HEPATITIS C VIRUS-INDUCED GLOMERULONEPHRITIS
     丙型肝炎病毒感染后肾小球肾炎
短句来源
     Hepatitis C and Detection for it
     丙型肝炎及其检测
短句来源
     Induced Abortion
     人工流产
短句来源
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  induced hepatitis
Depletion of immune cell populations in spleen, however, correlated with severe CMV-induced hepatitis and mortality.
      
Intramuscular injection of amniotic fluid of the 9th and 15th passage into volunteers induced hepatitis without jaundice in 4 out of 6 and 2 out of 5 individuals, respectively, after incubation periods in excess of 60 days.
      
A Comparative Study of the Immunotropic and Hepatotropic Action of Beekeeping Products in Rats with Drug-Induced Hepatitis
      
Hepatoprotector Properties of Eryxin in Animals with Acute Drug-Induced Hepatitis Model
      
The present study was carried out to assess the effect of chloroform insoluble fraction of ethanolic extract of Tridax procumbens (TP) against D-Galactosamine/Lipopolysaccharide (D-GalN/LPS)-induced hepatitis in rats.
      
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The protective effects of total glucosides of paeony (TGP) on eoperimental hepatitis were studied in mice. The elevation of SGPT and the decrese of serum protein and hepatic glycogen contents in D-Galactosamine or CCl4-induced hepatitis were significantly improved by pretreatment with TGP (10, 20mg · kg-1d-1×7d,ip). It also remarkably diminished the hepato-cellular degeneration and necrosis induced by D-Galactosamine or CCI4.

白芍总甙(TGP 10、20mg·kg~(-1),d~(-1)×7d·ip)预防给药可明显对抗D-半乳糖胺或四氯化碳所致小鼠肝损伤后血清谷丙转氨酶升高,血清白蛋的下降及肝糖原含量降低.并使形态学上的肝细胞变性和坏死得到明显地改善和恢复。同时超微结构上肝细胞内线粒体的肿胀、内浆网的空泡变性、溶酶体的脱落也得到明显恢复.上述保护作用的机制有待进一步研究。

Electron microscopy of 36 liver specimens from posttransfussion chronic hep- atitis C was carried out to determine the ultrastructural features in human hepatitis C. All the cases were confirmed by histological examination and serological tests in- cluding anti-HCV antibodies.No identical ultrastructural changes with those de- scribed in experimentally induced hepatitis C was detected,though some ultrastruc- tural alterations,not pathognomomic,did occur in hepatocellular cytoplasm.Promi- nent proliferation...

Electron microscopy of 36 liver specimens from posttransfussion chronic hep- atitis C was carried out to determine the ultrastructural features in human hepatitis C. All the cases were confirmed by histological examination and serological tests in- cluding anti-HCV antibodies.No identical ultrastructural changes with those de- scribed in experimentally induced hepatitis C was detected,though some ultrastruc- tural alterations,not pathognomomic,did occur in hepatocellular cytoplasm.Promi- nent proliferation of smooth endoplasmic reticulum was frequently found,notably in cases with chronic persistent hepatitis,and rough endoplasmic reticulum showed distended cisternae filled with light fluffy material,occasionally forming curved membraned structure in the cisternae.Intercellular and perisinusoidal fibrosis,and invasion of lymphocytes into Disse and intercellular spaces in the absence of con- comitant liver cell injury in the immediate vicinity were ofter noted in the study.

本研究对36例输血后慢性丙型肝炎进行了电镜观察,旨在确定人类丙型肝炎的超微结构特点。所有病例都经各项血清学检查(包括HCV抗体及HCV-RNA的检测)及组织学检查证实。各病例虽均未观察到以往人们在实验性丙型肝炎中报道的那些特殊结构,但在肝细胞及其问质中的确可以出现某些超微结构变化。多数病例(尤其是慢迁肝病例)中可见滑面内质网显著增生;粗面内质网扩张,网池中充满低电子密度絮状物质,偶而形成卷曲的膜性结构;窦周及肝细胞间隙胶原纤维增多;肝窦淋巴细细胞及枯否细胞增多;并常侵入肝细胞间;末见邻近肝细胞明显损伤。

Acetaminophen is a popular analgesic. When its taken in toxic doses, an intermediate meabolite, N-acetyl-para-benzo-quinoneimine is largaly formed. Thus, The hapatic necrosis in going on meanwhile, Some patients medicate several preparations simultaneonsly for increasing therapeutic effects, which may increase acetaminophen poisoning. The main clinical manifestation of the acetaminophen poisoning are drug-induced hepatitis and fulminant hepatic failure moreover. This article reviews some special antidotes...

Acetaminophen is a popular analgesic. When its taken in toxic doses, an intermediate meabolite, N-acetyl-para-benzo-quinoneimine is largaly formed. Thus, The hapatic necrosis in going on meanwhile, Some patients medicate several preparations simultaneonsly for increasing therapeutic effects, which may increase acetaminophen poisoning. The main clinical manifestation of the acetaminophen poisoning are drug-induced hepatitis and fulminant hepatic failure moreover. This article reviews some special antidotes and emergency treatments.

对乙酰氨基酚是常用的解热镇痛药,中毒剂量时形成大量的中间代谢产物N-乙酰-对苯醌亚胺,可导致以急性小叶中央型坏死为特征的肝脏损害。特别是在感冒患者为增强疗效而同时合用多种含对乙酰氨基酚的复方制剂时,可无意中增强其毒性作用,易致中毒事件发生。临床上,对乙酰氨基酚中毒主要表现为“药物性肝炎”或“暴发性肝衰”的改变。同时综述了以N-乙酰半胱氨酸为主的特异解毒药及其它急救措施。

 
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