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brain radiation injury
相关语句
  放射性脑损伤
     The Application Study of Chemical Shift Image of ~1H-MRS on Brain Radiation Injury Following Radiotherapy for Nasopharyngeal Carcinoma
     ~1H-MRS化学位移成像在鼻咽癌放疗后放射性脑损伤中的应用研究
短句来源
     Investigate to MRI and MRS in 15 brain radiation injury cases
     对15例放射性脑损伤MRI及MRS征象的探讨
短句来源
     Objective: To investigate the changes of IL-1, TNF-a and IL-6 in brain and serum at various times after different radiation doses in rats and to explore the role of them in the pathogenesis of brain radiation injury.
     [目的]观察大鼠全脑不同剂量照射后不同时期血清及脑组织中IL-1β、TNF-α和IL-6水平的变化,以探讨它们在放射性脑损伤发病机理中所起的作用。
短句来源
     Objective: Oligodendrocytes develop from oligodendrocyte-type 2 astrocyte progenitor cells. To observe the effects of radiation on differentiation of O-2A, so as to provide basic data on pathogenesis of brain radiation injury.
     [目的] 少突胶质-2型星形胶质前体细胞(oligodendrocyte-type 2astrocyte progenitor cells O-2A)是少突胶质细胞的前体细胞,探索电离辐射对O-2A祖细胞分化的影响,为研究放射性脑损伤发病机理的研究提供基础资料。
短句来源
     Objective: To summarize the MRI and MRS signs of brain radiation injury and improve the diagnostic accuracy.
     目的:总结放射性脑损伤MRI与MRS表现,提高其影像诊断准确率。
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  “brain radiation injury”译为未确定词的双语例句
     Progress in imaging of brain radiation injury
     脑放射损伤的影像学研究进展
短句来源
     2 to study representation of CSI of 'H-MRS of brain radiation injury caused by radiotherapy of NPC (nasopharyngeal carcinoma) and determine its clinical value;
     二、研究NPC放疗后放射性脑损伤的1H-MRS CSI的表现及其诊断价值;
短句来源
     Conclusions The halfbrain irradiation SD rat appears to be an excellent model for study on the brain radiation injury in the early stage.
     结论 该动物模型制作方法简便、可靠,可用于早期放射性脑损伤的实验研究
短句来源
     Results The symptom of brain radiation injury is gotten well mostly (60%) and some patients are recovery partly (20%) or no effective (20%).
     结果  6 0 %的病人症状改善 ,2 0 %部分改善 ,2 0 %无效。
短句来源
     Results The symptom of brain radiation injury is gotten well mostly(60%) and some patients are recovery partly(20%) or no effective(20%).
     结果  6 0 %的病人症状改善 ,2 0 %部分改善 ,2 0 %无效。
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  相似匹配句对
     Radiation Brain Injury
     放射性脑损伤
短句来源
     Diagnosis of radiation brain injury
     放射性脑损伤的诊断
短句来源
     Present investigation of radiation brain injury
     放射性脑损伤研究现状
短句来源
     Progress in imaging of brain radiation injury
     脑放射损伤的影像学研究进展
短句来源
     MRI diagnosis of radiation-induced brain injury
     放射性脑损伤的MRI诊断
短句来源
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Objective To develop an animal model by which study on the brain radiation injuries in early stage. Methods The SD rat half brains were irradiated by 4 MeV electron at a single dose of 10, 20 and 30 Gy, and all the experiment was performed from 1 day to 3 months after radiation. The head skin inside the field, the neurological symptoms, and body weight were examined at the mean time. The histological sections of the rats were scanned as well. Results The peak depth dose of 4 MeV electron was 13mm, and...

Objective To develop an animal model by which study on the brain radiation injuries in early stage. Methods The SD rat half brains were irradiated by 4 MeV electron at a single dose of 10, 20 and 30 Gy, and all the experiment was performed from 1 day to 3 months after radiation. The head skin inside the field, the neurological symptoms, and body weight were examined at the mean time. The histological sections of the rats were scanned as well. Results The peak depth dose of 4 MeV electron was 13mm, and the dosimetry homogeneity of the radiation field was within 7%. Intra-portal alopecia was observed in all the 30 Gy rats and some 20 Gy rats. The abnormal neurological signs were not found in all the rats, but the tend of body weight increase was less pronounced in 1~3 months in the irradiated rats than that in the unirradiated. The necrosis was observed in rats in the 30 Gy 3 months group also. Conclusions The halfbrain irradiation SD rat appears to be an excellent model for study on the brain radiation injury in the early stage.

目的 建立能用于大脑放射性反应与损伤研究的动物模型。方法 在作照射剂量的测量后,对SD大鼠用4 MeV 电子线作10 、20 和30Gy 的单次半脑照射。在照射后3 个月内,分别观察大鼠的照射区皮肤、神经系统体征与体重的变化,并在各观测点作病理组织学检查。结果 该照射条件下的最大剂量点深度约13m m ,各照射点间吸收剂量的差异≤7 % 。所有存活大鼠无异常神经系统体征出现,全部30Gy 和部分20Gy 照射的大鼠有射野内脱毛,照射组大鼠在受照后1 ~3 个月,体重上升趋势低于未照射组大鼠,但无统计学差别。部分30Gy 照射的大鼠大脑中存在放射性坏死灶。结论 该动物模型制作方法简便、可靠,可用于早期放射性脑损伤的实验研究

The mechanisms of brain radiation injury mainly include three hypotheses: vascular injury, glial cells damage and immune response . Most scholars' studies have recently supported the former two ones. Vascular injury plays a major role in the effect of delayed radiation injury. Focal brain injury and diffuse white matter injury can be definitely diagnosed by CT and MRI. T 2 weighted imaging (T 2WI) in MRI shows high sensitivity in water contents, and is not...

The mechanisms of brain radiation injury mainly include three hypotheses: vascular injury, glial cells damage and immune response . Most scholars' studies have recently supported the former two ones. Vascular injury plays a major role in the effect of delayed radiation injury. Focal brain injury and diffuse white matter injury can be definitely diagnosed by CT and MRI. T 2 weighted imaging (T 2WI) in MRI shows high sensitivity in water contents, and is not affected by the beamhardening arifacts from the cranial base. Compared with CT,the sensitivity of MR for detecting white matter lesions is two to threefold higher. When lesions occrs at the site of an irradiated cerebral tumor, tumor recurrence and focal cerebral necrosis cannot be differentiated by CT or MR, PET and MRS now present a certain advantage of differential diagnosis.Tumoror presents high metabolism and necrosis demonstrates low metabolism by utilizing PET scanning, however PET's sensitivity and specificity are far from satisfactory. The amount or ratio of metabolic products in the region of interest measured by MRS contributes to the deferential diagnosis. In addition, PET functmnal imaging and MRS can also predict the early asymptomatic reversible radiation injury so as to allow the early therapy of steroids andpossibly other drugs, prior to the development of irreversible changes.

脑放射损伤发病机制主要有三种学说即血管损伤、胶质损伤和免疫反应机制 ,最近多数学者的研究支持血管和胶质损伤机制 ,血管方面的改变在晚期放射效应中起主要作用。 CT和 MRI对局限性脑损伤和弥漫性脑白质损伤可明确诊断。MRI的 T2 加权成像 (T2 WI)显示水分变化敏感性高 ,又不受颅底线束硬化伪影的影响 ,MRI发现白质病变的敏感性是 CT的 2~ 3倍。如果是脑本身肿瘤放疗后 ,CT和 MRI区别病灶复发或放射性坏死比较困难 ,PET和 MRS(磁共振波谱成像 )在两者鉴别诊断中则初步呈现出一定的优势。行 PET检查时 ,如为肿瘤则代谢活跃 ,坏死则代谢低下 ,但敏感性和特异性欠理想。 MRS测量感兴趣区内代谢产物的量或比率有助于两者的鉴别诊断。另外 ,PET功能成像和 MRS还可预测放疗病人较早期无症状的可逆性放射损伤 ,以便及时应用激素等药物治疗 ,避免其进一步发展为临床症状明显的不可逆性损伤

Objective To investigate the pathogenesis of brain radiation injury by observing the pathological changes of astrocytes in the hippocampus region of rat brain radiation injury model.Methods The brain sections were immunohistochemical stained for glial fibrillary acidic protein (GFAP),which was used to label astrocytes.The morphological characters and the number of GFAP cells in hippocampus were recongnized,and the...

Objective To investigate the pathogenesis of brain radiation injury by observing the pathological changes of astrocytes in the hippocampus region of rat brain radiation injury model.Methods The brain sections were immunohistochemical stained for glial fibrillary acidic protein (GFAP),which was used to label astrocytes.The morphological characters and the number of GFAP cells in hippocampus were recongnized,and the data were analyzed by the Student’s test.Results The astrocytes proliferation was revealed.The relative number of GFAP positive cells increased in the hippocampus.The increase was dose and time course related.Conclusion The pathological changes of astrocytes suggest a possible association in the pathogenesis of brain radiation injury,just as the other tissue components.

目的 通过对脑组织中星形胶质细胞病理学改变的观察 ,研究放射性脑损伤的发病机理。方法 利用已建立的大鼠早期放射性脑损伤的实验模型 ,用胶质纤维酸性蛋白免疫组织化学染色标记星形胶质细胞的方法 ,观察大脑海马区中星形胶质细胞形态与数量的变化 ,并分析照射剂量、照射后时间间隔的影响。结果 受照射侧海马中的阳性细胞表现出胞体肥大、突起增多与延长等改变 ,在剂量增加和观察时间延长的条件下 ,2 0Gy照射后 3个月和 30Gy照射后 1个月与 3个月的三个组别中阳性细胞数有显著增加。结论 随着照射剂量的加大和观测时间的延长 ,在大鼠大脑海马区域内出现了明显的星形胶质细胞增生

 
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