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acute ischemic brain
相关语句
  急性缺血性脑
     1,3,6 ,24 hours after ischemia,rats were killed and c-fos protein(FOS)and HSP70 immune positive cells of cortex tissue in ischemic region were determined by immunohistochemistry assay to observe the influence on anti-apoptosis gene expression at early stages of acute ischemic brain injury.
     分别于缺血后的1、3、6、24h处死 ,进行免疫组织化学染色 ,检测缺血区皮层脑组织c-fos蛋白 (FOS)及HSP70蛋白免疫阳性细胞 ,以观察井穴放血法对急性缺血性脑损伤后早期抑凋亡基因表达的影响。
短句来源
     OBJECTIVE: To discuss the protective effects of nimodipine on acute ischemic brain injury caused by activation of phospholipase A2. DESIGN: A completely randomized controlled trial.
     目的:探讨尼莫地平对磷脂酶A2激活致急性缺血性脑损伤中的保护作用。 设计:完全随机对照实验。
短句来源
     NITRIC OXIDE DERIVED FROM PERIPHERAL BLOOD MONONUCLEAR CELLS AND ACUTE ISCHEMIC BRAIN INJURY
     单个核细胞源性一氧化氮与急性缺血性脑损伤
短句来源
     Cooling for acute ischemic brain damage (COOL AID): A feasibility trial of endovascular cooling
     降温治疗急性缺血性脑损伤:血管内降温的可行性试验
短句来源
     Objective To investigate the change of cognitive function in the elders with first acute ischemic brain stroke.
     目的 探讨老年急性缺血性脑卒中患者首次发病后认知功能的改变。
短句来源
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  “acute ischemic brain”译为未确定词的双语例句
     Methods The perinatal acute ischemic brain damage model was established by ligation of both uterine arteries of full-term pregnant Wistar rats(n=315). They were divided randomly into topiramate one and five times groups and hypoxic-ischemia(HI) group(n=105),and normal collate group consisted of 105 normal Wistar rats.
     方法:夹闭足月妊娠大鼠子宫血管,制成急性脑缺血损伤的新生鼠模型(n=315),随机分为topiramate 1次和5次给药组及缺血组(n=105),另取105只正常Wistar大鼠作为正常对照组。
短句来源
     These suggested that AVP was involved in the pa-thophysiologic process of acute ischemic brain edema, and its mechanism mightbe the effect of AVP on AVP receptor mediated by cAMP, cGMP, and that in turn inhibited the Na+-K+ ATPase activity of brain cell membrane,then exaggerated the formation of ischemic brain edema
     提示AVP可能是通过cAMP、cGMP介导的AVP受体的作用,产生抑制脑细胞膜Na~+-K~+ATP酶活力的效应,从而促进缺血性脑水肿的形成。
短句来源
     Objective To investigate the effects of topiramate on neurons apoptosis and the hydrous capacity in brain tissue in rats with perinatal acute ischemic brain damage.
     目的:观察topiramate对宫内急性脑缺血损伤的Wistar大鼠神经细胞凋亡及脑组织含水量的影响。
短句来源
     Results Plasma levels of vWF and D dimer were significantly higher in pateints with hypertension Ⅲ and acute ischemic brain infarction (P<0 001) and after relief (P<0 05) as compared with those in control subjects and hypertension Ⅱ.
     结果 高血压病 3期血浆 v WF及 D- D水平显著高于高血压 2期及正常对照组 ( P <0 .0 1 ) ,高血压 2期也高于正常对照组 ( P<0 .0 5) ;
短句来源
     PARTICIPANTS:From September 2004 to August 2005,58 patients with acute ischemic brain infarction less than 24 hours after symptom onset were hospitalized in the Department of Neurology of Shandong Provincial Hospital for evaluation and management and enrolled in case group. With the age of 36-86 years and a mean of(68±14)years.
     对象:选择2004-09/2005-08山东省立医院神经内科收治58例缺血性脑梗死患者为病例组,年龄36~86岁,平均(67.7±14.2)岁,男37例,女21例,均于出现症状24h内入组。
短句来源
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  相似匹配句对
     Effect of dipfluzine on acute ischemic brain edema in gerbils
     双苯氟嗪对沙土鼠急性脑缺血性脑水肿的影响
短句来源
     Experimental Observation of the Brain Lactate Changes in Acute Ischemic Rats
     急性缺血性大鼠脑乳酸含量变化的实验观察
短句来源
     The Protective Effect of Tea Pigment on the Acute Brain Ischemic Rats
     茶色素对急性脑缺血小鼠抗脂质过氧化作用的实验研究
短句来源
     The Effect of Arginine Vasopressin ( AVP ) on Acute Ischemic Brain Edema in Gerbil
     精氨酸加压素对沙土鼠急性缺血性脑水肿的作用
短句来源
     Brain hypoxic and ischemic proconditioning
     脑缺血缺氧预处理
短句来源
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The purpose of this experiment was to study the role of arginine vasopressin (AW) in acute cerebral ischemic edema in mongolian gerbils. The results showed that intracerebroventricular injection (ICV) of AVP exacerbated the ischemic brain edema, while ICV of AW antiserum significantly decreased the ischemic brain edema. Nimodipine couldn't block this role of AW in ischemic brain edema. The cortical Na+ -K+ ATPase activity was significantly decreased, the contents of cAMP in the ischemic cortex and hypothalamus...

The purpose of this experiment was to study the role of arginine vasopressin (AW) in acute cerebral ischemic edema in mongolian gerbils. The results showed that intracerebroventricular injection (ICV) of AVP exacerbated the ischemic brain edema, while ICV of AW antiserum significantly decreased the ischemic brain edema. Nimodipine couldn't block this role of AW in ischemic brain edema. The cortical Na+ -K+ ATPase activity was significantly decreased, the contents of cAMP in the ischemic cortex and hypothalamus and the contents of cGMP in the hypothalamus were remarkably increased after ICV of AW. These suggest AW was involved in the pathophysiologic process of acute ischemic brain edema. And its mechanism might be the effect of AW on AW receptor mediated by cAMP, cGMP, and that in turn inhibited the Na+ -K+ ATPase activity of brain cell membrane, then exaggerated the formation of ischemic brain edema.

本实验探讨了精氨酸加压素(AVP)对蒙古种沙土鼠急性缺血性脑水肿的作用及其机理。结果表明,给沙土鼠侧脑室注射AVP后,缺血性皮质水肿显著加重,尼莫地平不能阻断AVP对缺血性脑水肿的影响;侧脑室注射AVP抗血清后,缺血性皮质水肿明显减轻,侧脑室注射AVP后,缺血性皮质Na~+-K~+ATP酶活力显著降低,皮质和下丘脑cAMP以及下丘脑cGMP含量均显著升高。提示AVP参与了急性缺血性脑水肿的病理生理过程,且起着促进或加重缺血性脑水肿形成的作用。其作用机理可能是通过cAMP、cGMP介导的AVP受体的作用,产生抑制脑细胞膜Na~+-K~+ATP酶活力的效应,从而促进缺血性脑水肿的形成。

The purpose of this experiments is to study the role of arginine vasopressin (AVP)iu acute ischemic brain edema and its mechanism. The results showde that intracerebroventricular injection ( ICV ) of AVP exacerbated the ischemic corticalc edema,and the calcium entry Mocker couldn' t block this role of AVP in ischemi brain edema. ICV of AVP antiserum,m V2 and V1/V2 receptor antagonist respectively decreased significantly the ischemic cortical edema. In addition, the Na + -K+ATPase activity...

The purpose of this experiments is to study the role of arginine vasopressin (AVP)iu acute ischemic brain edema and its mechanism. The results showde that intracerebroventricular injection ( ICV ) of AVP exacerbated the ischemic corticalc edema,and the calcium entry Mocker couldn' t block this role of AVP in ischemi brain edema. ICV of AVP antiserum,m V2 and V1/V2 receptor antagonist respectively decreased significantly the ischemic cortical edema. In addition, the Na + -K+ATPase activity of ischemic cortex was significantly decreased, and the contents of cAMP in cortex, hy pothalamus, staiatumand pons-medulla were markedly increased, and the contents of cGMP in hypothalamus and staiatum alsoincreased after ICV of AVP. These suggested that AVP was involved in the pa-thophysiologic process of acute ischemic brain edema, and its mechanism mightbe the effect of AVP on AVP receptor mediated by cAMP, cGMP, and that in turn inhibited the Na+-K+ ATPase activity of brain cell membrane,then exaggerated the formation of ischemic brain edema

本文探讨了精氨酸加压素(AVP)对急性缺血性脑水肿的作用及其作用机理。结果表明,侧脑室注射AVP后,缺血性皮层水肿显著加重,且这一作用不能被钙通道拮抗剂所阻断。侧脑室注射AVP抗血清、V_2及V_1/V_2型AVP受体拮抗剂均显著减轻缺血性皮层水肿。此外,侧脑室注射AVP后,缺血皮屋Na~+-K~+ATP酶活力显著降低;皮层等脑区cAMP含量显著升高,下丘脑和纹状体cGMP含量亦显著升高。提示AVP可能是通过cAMP、cGMP介导的AVP受体的作用,产生抑制脑细胞膜Na~+-K~+ATP酶活力的效应,从而促进缺血性脑水肿的形成。

The purpose of this experiments is to study the role of argi- nine vasopressin (AVP) in acute ischemic brain edema of mongolian gerbils. The results showed that the contents of AVP in ischemic cortex,hypothala- mus and striatum increased remarkably in 15~120 minutes after ischemia, while the contents of AVP had no change in pons-medulla vhich was not affected from ischemia,and there was relationship between the contents of AVP in corte(?) and the ischemic cortical edema.Intracerebroventricular...

The purpose of this experiments is to study the role of argi- nine vasopressin (AVP) in acute ischemic brain edema of mongolian gerbils. The results showed that the contents of AVP in ischemic cortex,hypothala- mus and striatum increased remarkably in 15~120 minutes after ischemia, while the contents of AVP had no change in pons-medulla vhich was not affected from ischemia,and there was relationship between the contents of AVP in corte(?) and the ischemic cortical edema.Intracerebroventricular inje- ction(ICV) of AVP e acerbated the ischemic cortical edema and it showed dose-response correlation.while ICV of AVP antiserum significantly decrea- sed ischemic cortical edema.These suggested that AVP was involved in the pathophysiologic process of acute ischemic brain edema.The inevement of AVP contents in ischemic brain regions could exacerbate the formation of ischemic brain edema.

应用放射免疫分析法和侧脑室微量注射技术,观察了精氨酸加压素(AVP)对急性缺血性脑水肿的作用。结果表明,沙土鼠脑缺血15~120min,皮层、纹状体、下丘脑的 AVP 含量均明显升高,缺血不明显的桥延部,AVP 含量则无明显变化;且缺血不同时间的皮层 AVP 含量与皮层水肿呈非常显著的相关性.侧脑室注射AVP 后,缺血性皮层水肿显著加重,并表现为良好的剂量-效应依赖关系.而侧脑室注射 AVP 抗血清后,缺血性皮层水肿明显减轻。提示 AVP 参与了急性缺血性脑水肿的病理生理过程.脑缺血时,脑内 AVP 含量的升高,可加重或促进缺血性脑水肿的形成.

 
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