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anti-invasive
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  抗侵袭
     Conclusions L-NAME has anti-invasive and anti-metastatic effects on LS-174T cells,and the anti-invasive and anti-metastatic action of L-NAME might be associated with expression of MMP2mRNA,TIMP2mRNA in tumor cells.
     结论L-NAME具有抑制SL-1 7 4T细胞侵袭和转移的作用; L-NAME的抗侵袭活性与肿瘤细胞MMP 2mRNA和TIMP 2mRNA的表达有关。
短句来源
     Anti-invasive effects of curcumin on human prostate carcinoma PC-3M cells and its mechanism
     姜黄素对前列腺癌PC-3M细胞抗侵袭作用的研究
短句来源
     Results Curcumin could down-regulate MMP-2 and up-regulate TIMP-2 expression,also inhibit the the expression of NF-κB.Conclusion The anti-invasive effects of curcumin is probably the result of down-regulation of MMP-2 and up-regulation of TIMP-2 expression,anti-invasive through inhibiting the expression of NF-κB is probably one of its molecular mechanisms.
     结果姜黄素可下调MMP-2和上调TIMP-2的表达,抑制NF-κB的表达。 结论姜黄素通过下调MMP-2和上调TIMP-2的蛋白表达发挥抗侵袭作用,抑制NF-κB的表达可能是抗侵袭作用的机制之一。
短句来源
     The anti- invasive effects of curcumin is probably the result of down-regulation of MMP-2 and up-regulation of TIMP-2 expression.
     姜黄素可能通过下调MMP 2和上调TIMP 2的蛋白表达发挥抗侵袭作用。
短句来源
     Objective To study the mechanism of anti-invasive effects of curcumin on human prostate carcinoma PC-3M cells.
     目的探讨姜黄素对前列腺癌PC-3M细胞抗侵袭作用。
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  “anti-invasive”译为未确定词的双语例句
     PATHOLOGICAL STUDIES ON THE ANTI-INVASIVE CHARACTER BY RECOMBINANT HUMAN INTERLEUKIN-6 GENE-TRANSFECTED MOUSE LEUKEMIA CELLS
     PATHOLOGICAL STUDIES ON THE ANTI-INVASIVE CHARACTER BY RECOMBINANT HUMAN INTERLEUKIN-6 GENE-TRANSFECTED MOUSE LEUKEMIA CELLS
短句来源
     AIM: To study the anti-invasive effect of NS-398 on colon cancer cell line HT-29 in vitro an its regulation by CD44v6 and nm23-H1 genes.
     目的研究NS-398对结肠癌HT-29细胞体外侵袭力的作用及CD44v6、nm23-H1基因的调节。
短句来源
     In the transwell chamber experiment, the anti-invasive effect of 0.001μM, 0.01μM and 0.1μM Taxol on B16BL6 cells were 25%, 55% and 77 % seperately, it showed that Taxol conld inhibit the B16BL6 invasion significantly.
     在小鼠黑色素瘤高转移株B16BL6细胞的侵袭实验中,0.001μM、0.01μM、0.1μMTaxol的侵袭抑制率分别为25%、55%、77%,能够明显抑制B16BL6细胞侵袭; 而0.001μM、0.01μM、0.1μM CPT对B16BL6细胞的侵袭能力没有明显抑制。
短句来源
     ② 0.2, 0.4, 0.8, 1.0 mmol/L L-NAME were used to treate LS174T cell for 72 hours , its inhibitory rate in inhibiting cells migration was 20.76%,24.95%,39.43%,46.85% respectively(P < 0.01=.CONCLUSION: L-NAME has anti-invasive and anti-metastasic effects on LS174T cells.
     ②0.2,0.4,0.8和1.0mmol/LN-硝基精氨酸甲酯处理LS174T细胞72h,对细胞趋化运动抑制率分别为20.76%,24.95%,39.43%,46.85%(P<0.01)。 结论:N-硝基精氨酸甲酯具有抑制LS174T细胞侵袭和转移的作用。
短句来源
     CONCLUSION: NS-398 has an anti-invasive effect on HT-29 cells in vitro. Down-regulation of CD44v6 and up-regulation of nm23-H1 may be its underlying mechanisms.
     结论NS-398具有抑制结肠癌细胞HT-29体外侵袭力的作用,下调CD44v6的表达和上调nm23-H1mRNA的表达可能是其作用机制。
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  相似匹配句对
     Anti-H.
     结果表明,抗H.
短句来源
     Anti-H.
     抗H .
短句来源
     Anti-invasive and Anti-metastatic Effect of Ampelopsin on Melanoma
     蛇葡萄素抗黑色素瘤侵袭和转移的作用
短句来源
     PATHOLOGICAL STUDIES ON THE ANTI-INVASIVE CHARACTER OF IL-6 GENE
     白细胞介素6基因转染的白血病细胞体内抗白血病浸润的病理形态研究
短句来源
     Invasive manipulation.
     接受侵入性操作。
短句来源
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  anti-invasive
SI-27, an anti-MMP agent, has already been shown to possess both in vitro anti-invasive and anti-angiogenic properties against malignant gliomas in non-cytotoxic dose concentrations.
      
HAI-2/PB has anti-invasive effects thought to be mediated primarily by the inhibitory activity against serine proteinase-dependent matrix degradation.
      
We have previously reported on the anti-invasive and angiosuppressive effects of SI-27, an anti-matrix metalloproteinase (MMP) agent.
      
The ability of glioma cells to infiltrate surrounding brain tissue, and ultimately escape current therapeutic modalities, could potentially be minimized using anti-invasive therapies.
      
Representative and valid in vitro experimental systems and animal models of gliomas are necessary for the characterization of the invasive phenotype and further development of anti-invasive therapy.
      
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The anti-invasive ability of cartilage was investigated in human hyaline cartilage co-cultured with OS-732 human osteosarcoma cell line. Invasive osteosarcoma cells were unable to invade normal hyaline cartilage after 7-day co-culture. Observation of TEM showed that the growth and metabolism of tumor cells were inhibited by co-cultured cartilage. However, cartilage extracted with 5M GuHCl was invaded by tumor cells. In this situation, tumor cells indicated a high growth and metabolic status and contained...

The anti-invasive ability of cartilage was investigated in human hyaline cartilage co-cultured with OS-732 human osteosarcoma cell line. Invasive osteosarcoma cells were unable to invade normal hyaline cartilage after 7-day co-culture. Observation of TEM showed that the growth and metabolism of tumor cells were inhibited by co-cultured cartilage. However, cartilage extracted with 5M GuHCl was invaded by tumor cells. In this situation, tumor cells indicated a high growth and metabolic status and contained abundant microtubules, microfilaments, rough endoplasmic reticulum, mitochondria and Golgi bodies. Cartilage matrix collagenous protein(CMCP), from extracted cartilage, was denatured and became more electron dense. In the control group, normal fibroblasts did not invade cartilage or extracted cartilage and its growth and metabolism did not inhibit by cartilage. Our report shows that resistance of hyaline cartilage to tumor invasion could be regulated in part by salt-derived materials which may inhibit the growth, metabolism and locomotion of tumor cells.

本文研究成骨肉瘤OS-732细胞系浸润软骨的机理,指出经盐酸胍处理后的软骨可被瘤细胞所浸润,这些浸润细胞在电镜下见其有生长、代谢旺盛的特点,提示正常软骨内含有抑制肿瘤细胞浸润的因子存在。

In the extracellular plasmin system,the receptor for a urokinase plasminogen activator (uPAR) acts as an anchorage site for uPA on the cell surface where it modulates the activities of the extracellular plasmin system, has a function of internalizing uPA-inhibitors and other complexes, transmits the extracellular signals into cells and represents a new prognostic parameter and a promising approach for anti-invasive therapy in cancer.

尿激酶型纤溶酶原激活物受体作为胞外纤溶酶系统的一员,以糖基磷脂酰肌醇锚的形式固定于细胞膜上,它参与了胞外纤溶酶活性的调节,具有内化受抑制的尿激酶的功能;同时参与了胞外信号的传递;另外它对癌症的临床预后及抗癌转移的研究有重要的意义.

The anti invasive and anti metastatic effects with new retinoid 4 acetamidophenyl retinoate(4 APR) were studied using in vitro and in vivo experiments. 4 APR, at the dose of 43.3 mg·kg -1 ·day -1 po, was shown to reduce the spontaneous lung metastatic foci of Lewis lung carcinoma. 4 APR was also found to inhibit the artificial lung metastasis of B16 F10 cells by 67.9% and 36.6% and suppress the reconstituted basement membrane invasion of B16 F10 cells by 54.2% and 41.9% at the...

The anti invasive and anti metastatic effects with new retinoid 4 acetamidophenyl retinoate(4 APR) were studied using in vitro and in vivo experiments. 4 APR, at the dose of 43.3 mg·kg -1 ·day -1 po, was shown to reduce the spontaneous lung metastatic foci of Lewis lung carcinoma. 4 APR was also found to inhibit the artificial lung metastasis of B16 F10 cells by 67.9% and 36.6% and suppress the reconstituted basement membrane invasion of B16 F10 cells by 54.2% and 41.9% at the concentrations of 10 -5 mol·L -1 and 10 -6 mol·L -1 , respectively.

用体内外实验模型,研究了新维A类化合物4乙酰胺苯基维A酸酯(4APR)对肿瘤侵袭、转移的抑制作用。4APR43.3mg·kg-1po即能减少小鼠Lewis肺癌的自发性肺转移瘤数。半体内实验证明4APR10-5mol·L-1和10-6mol·L-1对B16F10癌细胞的人工肺转移瘤数分别抑制67.9%和36.6%。体外实验显示,4APR对B16F10细胞侵袭重组基底膜的抑制率分别为54.2%和41.9%。

 
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