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hypothermia state
相关语句
  低温状态
     Conclusion:It indicates that the change of concentrations of four type animo acids and free radial system is an important protective mechanism to ischemia neurons under mild hypothermia state.
     结论 :亚低温状态下 4种氨基酸含量及自由基系统的改变是其保护缺血神经元的重要机制
短句来源
     Conclusions Intravenous hypothermic normal saline can induce mild hypothermia state and it can be used as a lowering temperature method in mild hypothermia study.
     结论 静脉滴注低温生理盐水可使家兔达到亚低温状态 ,且对家兔无不良影响 ,它可用来作为家兔亚低温实验研究的一种降温方法。
短句来源
  “hypothermia state”译为未确定词的双语例句
     (2) Aminopyrine induced rat hypothermia state after making model 15 min ( P< 0.05). AC activity rapidly weakened and cAMP content rapidly descended to the lowest value at making model 45 min, subsequently AC activity and cAMP content in hypothalamus gradually increased again to normal range.
     (2 )安痛定腹腔注射后 15min动物即呈现低体温状态 (P <0 0 5 ) ,在造模后45minAC活性及cAMP含量均降至最低点 (P <0 0 5 ) ,其后AC活性及cAMP含量均逐步回升至正常水平。
短句来源
     Aminopyrine induced rats hypothermia state 15 min after peritoneal injection ( P< 0.01). AC activity was higher than normal animal at 15 th minute after the injection of aminopyrine. But subsequently AC activity decreased rapidly with the lowest value at 45 th minute after the injection ( P< 0.05).
     安痛定腹腔注射后15min动物即呈现低体温状态 (P <0 0 1) ,AC活性在 15min时有一短暂上扬后至 4 5min降到最低点 (P <0 0 5 ) ,之后活性逐步回升 ,而PDE活性则在正常水平上下小幅波动。
短句来源
     Aminopyrine induced rats hypothermia state 15 min after peritoneal injection (P<0.01). AC activity was higher than mormal animal at 15 th minute after the injection of aminopyrine. But subsequently AC activity decreased rapidly with the lowest value at 45 th minute after the injection (P<0.05).
     安痛定腹腔注射后 15min动物即呈现低体温状态 (P <0 0 1) ,AC活性在 15min时有一短暂上扬后至 4 5min降到最低点 (P <0 0 5 ) ,之后活性逐步回升 ,而PDE活性则在正常水平上下小幅波动。
短句来源
  相似匹配句对
     State and V. T.
     Stace和V.T.
     state.
     state.
短句来源
     The state of sub-hypothermia can strengthen the protective effect of ischemia pretreatment.
     亚低温状态能增强缺血预处理的保护作用。
短句来源
     Protective approach of sub-hypothermia state on injury of cerebral ischemia and reperfusion in rats
     亚低温状态对大鼠脑缺血再灌注损伤的保护途径
短句来源
     Hypothermia Gluing Technologies
     低温型胶订技术
短句来源
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  hypothermia state
An intranasal BAF injection to white breedless mice previously cooled to 17-19°C under conditions of hypoxia-hypercapnia decelerated the exit of the mice from the hypothermia state.
      


Objective:To evaluate the effect of mild hypothermia to four types amino acids and free radial system during cerebral ischemia and repurfusion.Metheods: Sixtythree Wistat rats were divided randomly into nine groups:control group(n=7),ischemia groupsis3h,is3h/rep1h,is3h/rep3h(each group,n=7),mild hypothermia groups(10mg/kg i.p.)is3h,is3h/rep1h,is3h/rep2h,is3h/rep3h(each group,n=7)with Zea longa's animal middle of cerebral atrery occlusion.Results:Compared with the ischemia groups ,we found the concentrations...

Objective:To evaluate the effect of mild hypothermia to four types amino acids and free radial system during cerebral ischemia and repurfusion.Metheods: Sixtythree Wistat rats were divided randomly into nine groups:control group(n=7),ischemia groupsis3h,is3h/rep1h,is3h/rep3h(each group,n=7),mild hypothermia groups(10mg/kg i.p.)is3h,is3h/rep1h,is3h/rep2h,is3h/rep3h(each group,n=7)with Zea longa's animal middle of cerebral atrery occlusion.Results:Compared with the ischemia groups ,we found the concentrations of glutamate,aspartate,MDA in cerebral cortex decrease markedly and GABA,SOD,GSHPX increased significantly in mild hypothermia groups.Conclusion:It indicates that the change of concentrations of four type animo acids and free radial system is an important protective mechanism to ischemia neurons under mild hypothermia state.

目的 :研究亚低温状态下 ,急性脑缺血时皮质内四种氨基酸和自由基含量的改变 ,探讨亚低温对缺血脑组织的保护机制。方法 :将 63只 Wistar大鼠随机分为 9组 ,假手术组 7只 ,缺血组 (手术后将其颞肌温度控制在 36.5± 0 .5℃ ) -缺血 3h组 ,缺血 3h分别再灌注 1 h、2 h、3h组各 7只 ;亚低温组 (手术后将其颞肌温度控制在 32 .5± 0 .5℃ )一缺血 3h,缺血 3h分别再灌注 1 h、2 h、3h组各 7只。动物模型参照 Zea longa的大脑中动脉线栓模型并加以改进。结果 :比较缺血组和低温组 ,可见低温组缺血皮质内的谷氨酸 (Glu)、天门冬安酸 (Asp)、丙二醛 (MDA)的产生明显减少 ,而超氧化物歧化酶 (SOD)、谷胱甘肽过氧化物酶 (GSH-PX)的生成和释放明显增加 ,γ-氨基丁酸(GABA)的含量亦增加。结论 :亚低温状态下 4种氨基酸含量及自由基系统的改变是其保护缺血神经元的重要机制

AIM: To observe the changes of adengl cyclase(AC) and phosphodiesterase(PDE) activities of at different time point in hypothalamus of rats with fever and hypothermia. METHODS: Radioisotope method was used to measure the activity of AC and PDE. RESULTS:The fresh yeast caused rats fever after subcutaneous injection 4h( P< 0.01). AC activity reached a peak value at 3rd h after making model ( P< 0.05), and PDE activity increased slightly in the full process of experiment action; Aminopyrine induced rats...

AIM: To observe the changes of adengl cyclase(AC) and phosphodiesterase(PDE) activities of at different time point in hypothalamus of rats with fever and hypothermia. METHODS: Radioisotope method was used to measure the activity of AC and PDE. RESULTS:The fresh yeast caused rats fever after subcutaneous injection 4h( P< 0.01). AC activity reached a peak value at 3rd h after making model ( P< 0.05), and PDE activity increased slightly in the full process of experiment action; Aminopyrine induced rats hypothermia state 15 min after peritoneal injection ( P< 0.01). AC activity was higher than normal animal at 15 th minute after the injection of aminopyrine. But subsequently AC activity decreased rapidly with the lowest value at 45 th minute after the injection ( P< 0.05). PDE activity fluctuated slightly within normal range in the full course of experiment .CONCLUSION: In fever model, it might be important cause of febrile response that AC activity in hypothalamus increased markedly, while PDE might be the factor causing long-term febrile response . In hypothermia model, the obvious decrease in AC activity indicating that cAMP content in hypothalamus might be related to hypothermia status.

目的和方法 :采用放射性同位素法观察发热和低体温大鼠下丘脑腺苷酸环化酶 (AC)及磷酸二脂酶(PDE)活性的经时变化。结果 :鲜酵母皮下注射后 4h引起体温明显升高 (P <0 0 1) ,并长时维持在高位水平 ,AC活性在造模后 3h达峰值 (P <0 0 5 ) ,其后活性下降并渐至正常 ,PDE活性在整个过程中略有上升 ;安痛定腹腔注射后15min动物即呈现低体温状态 (P <0 0 1) ,AC活性在 15min时有一短暂上扬后至 4 5min降到最低点 (P <0 0 5 ) ,之后活性逐步回升 ,而PDE活性则在正常水平上下小幅波动。结论 :下丘脑AC活性的增强可能是动物体温升高的重要因素 ,而PDE活性并未相应显著升高则又造成了体温长时的偏高 ;在低体温模型中 ,AC活性的明显下降表明cAMP可能与动物的低温状态有关。

AIM: To observe the changes of AC activity and content of cAMP at different time point in hypothalamus of rats with fever and hypothermia. METHODS: Radioisotope method was used to measure the enzymatic activity of AC and the content of cAMP. RESULTS:(1)The fresh yeast caused fever after making model 4 h( P< 0.01). AC activity reached a peak value 3 h after making model ( P< 0.05), subsequently AC activity rapidly decreased and increased again over normal level ( P< 0.01);The change of cAMP content...

AIM: To observe the changes of AC activity and content of cAMP at different time point in hypothalamus of rats with fever and hypothermia. METHODS: Radioisotope method was used to measure the enzymatic activity of AC and the content of cAMP. RESULTS:(1)The fresh yeast caused fever after making model 4 h( P< 0.01). AC activity reached a peak value 3 h after making model ( P< 0.05), subsequently AC activity rapidly decreased and increased again over normal level ( P< 0.01);The change of cAMP content was positively related to the body temperature. (2) Aminopyrine induced rat hypothermia state after making model 15 min ( P< 0.05). AC activity rapidly weakened and cAMP content rapidly descended to the lowest value at making model 45 min, subsequently AC activity and cAMP content in hypothalamus gradually increased again to normal range. CONCLUSION: (1)In fever model, it might be an important cause of febrile response that AC induced increase in intracellular cAMP content; (2) In hypothermia model, the significant decrease of AC activity and cAMP content indicate that cAMP might play a role in keeping hypothermia.

目的和方法 :采用放射性同位素法观察发热和低体温大鼠下丘脑中腺苷酸环化酶 (AC)活性与cAMP含量的经时变化。结果 :(1)鲜酵母皮下注射后 4h引起体温明显升高 (P <0 0 1) ,并长时间维持在高位水平 ,AC活性在造模后 3h达峰值 (P <0 0 5 ) ,其后活性迅速下降 ,历 3h又再度上扬超出正常水平 (P <0 0 1) ;cAMP含量的变化是由低渐高 ,且与体温的变化趋势相似。 (2 )安痛定腹腔注射后 15min动物即呈现低体温状态 (P <0 0 5 ) ,在造模后45minAC活性及cAMP含量均降至最低点 (P <0 0 5 ) ,其后AC活性及cAMP含量均逐步回升至正常水平。结论 :(1)在发热模型中 ,下丘脑AC活性的增强使细胞内cAMP含量增多是动物体温升高的重要原因。 (2 )在低体温模型中 ,AC活性的明显下降以及cAMP含量的明显减少表明cAMP与动物的低温状态有关 ,但降温的始动环节不依赖于cAMP。

 
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