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delayed hypothermia
相关语句
  延迟性低温
     Counting of neuron in the central CA1 area of hippocampus :more in delayed hypothermia group[(60± 9)%]than that of normothermia group[( 10± 4)%](t=13.20,P< 0.01),but less than that of immediate hypothermia group[( 77± 16)%](t=2.45,P< 0.05).
     海马CA1区中间神经元计数:延迟性低温组(60±9)%较常温缺血组(10±4)%多(t=13.20,P<0.01),不及即刻低温组(77±16)%多(t=2.45,P<0.05)。
短句来源
     Counting of neuron in the lateral CA1 area of hippo campus:numbers in delayed hypothermia group[(71± 13)%]had no difference compa red with that of immediate hypothermia group[(80± 14)%](t=1.25,P >0.05),but m ore than that of normothermia group[(23± 5)%](t=9.14,10.14,P< 0.01).
     海马CA1区外侧成活神经元计数:延迟性低温(71±13)%和即刻低温组(80±14)%之间无差别(t=1.25,P>0.05),均较常温组(23±5)%多(t=9.14,t=10.14,P均<0.01)。
短句来源
     RESULTS:The numbers of grid got over by normothermia group at 10 minutes were 651± 108,which were obviously more than that of in sham-operated group(278± 67),delayed hypothermia group(478± 89) and immediate hypothermia group(368± 4 6)(t=7.76,2.21,6.37,P< 0.01-0.05).
     结果:常温组10min爬过的格子数(651±108)较假手术组(278±67)、延迟性低温组(478±89)、即刻低温组(368±46)有明显增多(t=7.76,2.21,6.37,P<0.0d 1~0.05)。
短句来源
     And the numbers in delayed hypothermia group were distinctly more than that of sham-operated group and immediate hypotherm ia group(t=4.75,2.90,P< 0.01-0.05).
     延迟性低温组较假手术组和即刻低温组增多(t=4.75,2.90,P<0.01~0.05)。
短句来源
     Animals were randomly allocated into 4 groups:sham-operated group, nor mothermia group,delayed hypothermia group and immediate hypothermia group with 7 rat each.
     动物随机分为4组:假手术组、常温组、延迟性低温组和即刻低温组,每组7只。
短句来源
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  “delayed hypothermia”译为未确定词的双语例句
     Objective The effects of delayed hypothermia combined with batroxobin on behavior and histopathology following global cerebral ischemia in gerbils were studied.
     目的 研究短暂全脑缺血后30 分开始的低温复合巴曲酶对沙土鼠行为学和组织病理学的影响。
短句来源
     3.Protective effects of hypothermia at hypoxia onset was better than that of delayed hypothermia, 6 h delayed hypothermia was effective.
     3)缺氧缺血后立即低温干预效果好 ,延迟 6h的低温干预仍有效。
短句来源
     (2) The in-farct area decreased significantly after treatment with hypothermia in all the groups, compared with normothermic group( P < 0.05). The longer delayed hypothermia treated with, the larger infact area got.
     (2)亚低温干预各组大鼠脑梗塞面积与常温恢复组相比显著降低(P<0.05),且随着亚低温干预起始时间的延迟,梗塞面积逐渐增大;
短句来源
     Conclusion The protective effect of hypothermia started immediatedly after hypoxic - ischemic is better than delayed hypothennia, 6 hour delayed hypothermia is effective.
     结论亚低温干预效果与缺氧缺血后低温的起始时间有关,延迟6 h的亚低温干预仍有效。
短句来源
  相似匹配句对
     EFFCTS OF DELAYED HYPOTHERMIA COMBINED WITH BATROXOBIN ON SUPEROXIDE
     延迟性低温复合东菱克栓酶对沙土鼠全脑缺血后氧自由基生成的影响
短句来源
     Delayed Discitis
     迟发性椎间盘炎
短句来源
     DELAYED VISBREAKING
     延迟减粘
短句来源
     Effects of delayed hypothermia on injury of cerebral ischemic repe rfusion in rats
     延迟性低温对沙土鼠全脑缺血再灌注损伤的影响(英文)
短句来源
     Hypothermia Gluing Technologies
     低温型胶订技术
短句来源
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  delayed hypothermia
These data suggest that delayed hypothermia affords protection of integrated mitochondrial function in the neonatal brain following transient hypoxia-ischaemia.
      
Delayed hypothermia preserved NAA and GSH at control levels and enhanced the rate of complex II-III activity.
      
Delayed Hypothermia Prevents Decreases in N-Acetylaspartate and Reduced Glutathione in the Cerebral Cortex of the Neonatal Pig F
      
Barks added that he is currently testing the combination of early drug and delayed hypothermia.
      
Again, CA1 neuronal death occurred throughout this period, because some CA1 loss was postponed by the delayed hypothermia treatment.
      


Objective The effects of delayed hypothermia combined with batroxobin on behavior and histopathology following global cerebral ischemia in gerbils were studied.Methods The ischemia was induced by occlusing the bilateral common carotoid arteries for ten minutes.Thirty five gerbils were divided into five groups:sham operated group;normothermia group;delayed hypothermia group;batroxobin group;delayed hypothermia combined with...

Objective The effects of delayed hypothermia combined with batroxobin on behavior and histopathology following global cerebral ischemia in gerbils were studied.Methods The ischemia was induced by occlusing the bilateral common carotoid arteries for ten minutes.Thirty five gerbils were divided into five groups:sham operated group;normothermia group;delayed hypothermia group;batroxobin group;delayed hypothermia combined with batroxobin group.Open field test were carried out at the fifth day,and histopathology test at the seventh day.Results The exploring activities of gerbils in both batroxobin and delayed hypothermia group were lower than that in normothermia reperfusion group,but higher than in hypothermia combined with batroxobin group.Animals of batroxobin group or delayed hypothermia group showed less neuron loss of CA1 than that of normothermia group,but more neuron loss of CA1 than that of hypothermia combined with batroxobin group.Conclusion Batroxobin or delayed hypothermia provides neuroprotective effects from the behavior deficits as well as neuronal injury following transient global cerebral ischemia,and their efficacy is substantially elevated when the treatments are combined.

目的 研究短暂全脑缺血后30 分开始的低温复合巴曲酶对沙土鼠行为学和组织病理学的影响。方法 采用沙土鼠全脑缺血模型,缺血时间10 分。动物随机分为5组:假手术组、常温组、延迟性低温组、巴曲酶组、延迟性低温复合巴曲酶组,每组7只。低温(32℃)于再灌注30分给予,维持3小时;巴曲酶8BU·kg- 1再灌注30 分经腹腔注入。于动物存活第5 天行开阔法行为学检查,第7 天行海马CA1区组织病理学检查。结果 行为学结果显示,常温组沙土鼠的探索活动明显较假手术组活跃;巴曲酶组或延迟性低温组沙土鼠的探索活动均较常温组明显减弱,较延迟性低温复合巴曲酶组明显活跃。组织病理学结果显示,常温组海马CA1 区内侧、中间和外侧存活神经元计数均较常温组明显增多;延迟性低温组或巴曲酶组海马CA1 区内侧、中间和外侧存活神经元计数均较延迟性低温复合巴曲酶组明显减少。结论 延迟性低温或巴曲酶可以减轻脑缺血后神经功能障碍和海马神经元坏死,两者复合的作用更强。

Objective To study the effects of delayed hypothermia combined with batroxobin on

目的研究沙土鼠全脑缺血后30min开始的亚低温复合东菱克栓酶对脑组织SOD、MDA的影响。方法采用沙土鼠全脑缺血模型,缺血时间10min,再灌注时间3.5h。动物随机分为5组:假手术组、常温组、延迟性低温组、东菱克栓酶组、延迟性低温复合东菱克栓酶组,每组7只动物。低温(32℃)于再灌注30min给予,东菱克栓酶8BU·kg1也在再灌注30min经腹腔注入。再灌注3h断头取脑后分取皮层和海马,测SOD活力和MDA含量。结果延迟性低温组或东菱克栓酶组皮层和海马SOD活力较常温组高(P<0.05),MDA含量较常温组低(P<0.05)。延迟性低温复合东菱克栓酶组皮层和海马SOD活力高于单纯延迟性低温组或东菱克栓酶组(P<0.05或P<0.01),MDA含量低于单纯延迟性低温组或东菱克栓酶组(P<0.05)。结论延迟性低温或东菱克栓酶可以减轻脑缺血后氧自由基造成的脑组织脂质过氧化作用,两者复合的作用强于单纯延迟性低温或东菱克栓酶。

AIM:To investigate the behaviour and histopathological effects of hypothermia after 30-minute of cerebral ischemia in rat and compare with the immediate hy pothermia after cerebral ischemia. METHODS:Cerebral ischemia rat model was introduced and ischemic time was 10 m inutes. Animals were randomly allocated into 4 groups:sham-operated group, nor mothermia group,delayed hypothermia group and immediate hypothermia group with 7 rat each. Open field behavior examine was carried out on the 5th day and...

AIM:To investigate the behaviour and histopathological effects of hypothermia after 30-minute of cerebral ischemia in rat and compare with the immediate hy pothermia after cerebral ischemia. METHODS:Cerebral ischemia rat model was introduced and ischemic time was 10 m inutes. Animals were randomly allocated into 4 groups:sham-operated group, nor mothermia group,delayed hypothermia group and immediate hypothermia group with 7 rat each. Open field behavior examine was carried out on the 5th day and hippoc ampus CA1 area histopathological examine was carried out on the 7th day after ce rebral ischemia. RESULTS:The numbers of grid got over by normothermia group at 10 minutes were 651± 108,which were obviously more than that of in sham-operated group(278± 67),delayed hypothermia group(478± 89) and immediate hypothermia group(368± 4 6)(t=7.76,2.21,6.37,P< 0.01-0.05).And the numbers in delayed hypothermia group were distinctly more than that of sham-operated group and immediate hypotherm ia group(t=4.75,2.90,P< 0.01-0.05).The neuron numbers in hippocampus CA1 area (expressed by percentage of normal hippocampus live neurons,ie.live neurons of e ach group compared with sham-operated group):the numbers of neuron in delayed hypothermia group[(42± 7)%] were more than that of normothermia group[(5± 1) %](t=13.00,P< 0.01),but less than that of immediate hypothermia group[(66± 10 )%](t=5.20,P< 0.01). Counting of neuron in the central CA1 area of hippocampus :more in delayed hypothermia group[(60± 9)%]than that of normothermia group[( 10± 4)%](t=13.20,P< 0.01),but less than that of immediate hypothermia group[( 77± 16)%](t=2.45,P< 0.05).Counting of neuron in the lateral CA1 area of hippo campus:numbers in delayed hypothermia group[(71± 13)%]had no difference compa red with that of immediate hypothermia group[(80± 14)%](t=1.25,P >0.05),but m ore than that of normothermia group[(23± 5)%](t=9.14,10.14,P< 0.01). CONCLUSION:Delayed hypothermia can alleviate neural dysfunction and neuron ne crosis in hippocampus after cerebral ischemia but it function is not as good as immediate hypothermia.

目的:研究全脑缺血后30min开始的低温对沙土鼠行为学和组织病理学的影响,并与脑缺血后即刻低温进行比较。方法:采用沙土鼠全脑缺血模型,缺血时间10min。动物随机分为4组:假手术组、常温组、延迟性低温组和即刻低温组,每组7只。于脑缺血后第5天行开阔法行为学检查,第7天行海马CA1区组织病理学检查。结果:常温组10min爬过的格子数(651±108)较假手术组(278±67)、延迟性低温组(478±89)、即刻低温组(368±46)有明显增多(t=7.76,2.21,6.37,P<0.0d 1~0.05)。延迟性低温组较假手术组和即刻低温组增多(t=4.75,2.90,P<0.01~0.05)。海马CA1区内侧神经元数(以正常海马成活神经元的百分数表示,即各组成活神经元数与假手术组之比):延迟性低温组(42±7)%较常温缺血组(5±1)%多(t=13.00,P<0.01),不及即刻低温组(66±10)%(t=5.20,P<0.01)。海马CA1区中间神经元计数:延迟性低温组(60±9)%较常温缺血组(10±4)%多(t=13.20,P<0.01),不及即刻低温组(77±16)%多(t=2.45,P<0.05)。...

目的:研究全脑缺血后30min开始的低温对沙土鼠行为学和组织病理学的影响,并与脑缺血后即刻低温进行比较。方法:采用沙土鼠全脑缺血模型,缺血时间10min。动物随机分为4组:假手术组、常温组、延迟性低温组和即刻低温组,每组7只。于脑缺血后第5天行开阔法行为学检查,第7天行海马CA1区组织病理学检查。结果:常温组10min爬过的格子数(651±108)较假手术组(278±67)、延迟性低温组(478±89)、即刻低温组(368±46)有明显增多(t=7.76,2.21,6.37,P<0.0d 1~0.05)。延迟性低温组较假手术组和即刻低温组增多(t=4.75,2.90,P<0.01~0.05)。海马CA1区内侧神经元数(以正常海马成活神经元的百分数表示,即各组成活神经元数与假手术组之比):延迟性低温组(42±7)%较常温缺血组(5±1)%多(t=13.00,P<0.01),不及即刻低温组(66±10)%(t=5.20,P<0.01)。海马CA1区中间神经元计数:延迟性低温组(60±9)%较常温缺血组(10±4)%多(t=13.20,P<0.01),不及即刻低温组(77±16)%多(t=2.45,P<0.05)。海马CA1区外侧成活神经元计数:延迟性低温(71±13)%和即刻低温组(80±14)%之间无差别(t=1.25,P>0.05),均较常温组(23±5)%多(t=9.14,t=10.14,P均<0.01)。结论:延迟性低温可以减轻全脑缺血后神经功能障碍和海马神经元坏死,但作用不及即刻低温。

 
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