助手标题  
全文文献 工具书 数字 学术定义 翻译助手 学术趋势 更多
查询帮助
意见反馈
   transient brain ischemic 的翻译结果: 查询用时:0.141秒
图标索引 在分类学科中查询
所有学科
神经病学
更多类别查询

图标索引 历史查询
 

transient brain ischemic
相关语句
  短暂性脑缺血
     Dynamic changes of serum neuron-specific enolase level in patients with transient brain ischemic attack
     短暂性脑缺血发作患者血清神经元特异性烯醇酶水平的动态变化(英文)
短句来源
     ① Comparison of neuron-specific enolase density: It was significantly higher in transient brain ischemic attack group than in control group (23.53±12.35) vs (14.29±6.83) μg/L, t=2.678, P < 0.01.
     ①神经元特异性烯醇酶浓度的比较:短暂性脑缺血发作组明显高于对照组[(23.53±12.35,14.29±6.83)μg/L,t=2.678,P<0.01]。
短句来源
     OBJECTIVE: To observe the changes of serum neuron-specific enolase in patients with transient brain ischemic attack, so as to explore its relationship with the degree of neuronal damage.
     目的:观察短暂性脑缺血发作患者血清神经元特异性烯醇酶的变化,探讨其与疾病的神经损伤程度的关系。
短句来源
     CONCLUSION: Serum neuron-specific enolase increases within a short term after transient brain ischemic attack and reaches the peak level at around 24-36 hours, suggesting that the detection of serum neuron-specific enolase has a guiding value in assessing the severity of transient brain ischemic attack.
     结论:短暂性脑缺血发作患者短期内血清神经元特异性烯醇增高,24~36h达到高峰,提示检测短暂性脑缺血发作患者血清神经元特异性烯醇酶浓度。 对判断病情严重程度有指导意义。
短句来源
  “transient brain ischemic”译为未确定词的双语例句
     Brain ischemic procondition (BIP) or BIT refers to that the brain could produce marked tolerance to lately the more time ischemic brain harm after one or multiple times transient brain ischemic reperfusion.
     脑缺血预适应(brain ischemic procondition,BIP),或称脑缺血耐受(brainischemic tolerance,BIT)是指脑组织一次或多次短暂性缺血再灌注后,大脑对以后较长时间的缺血性损伤产生显著的耐受性,表现为脑神经细胞死亡明显减少,脑梗死体积显著缩小,神经功能缺损明显减轻等。
短句来源
     Study on alleviation of ischemic cerebral injury by transient brain ischemic preconditioning
     短暂缺血预处理减轻缺血性脑损伤的研究
短句来源
  相似匹配句对
     brain
     脑
短句来源
     The brain of F.
     随发育脑体积增加,髓质占脑体积的比例增加。
短句来源
     Health Education for Patients with Transient Ischemic Attack of Brain
     短暂性脑缺血发作患者的健康教育
短句来源
     Transient Dream
     流星梦
短句来源
     Transient ischemic attacks make ischemic tolerance in human brain
     短暂性脑缺血发作的人脑缺血耐受作用
短句来源
查询“transient brain ischemic”译词为用户自定义的双语例句

    我想查看译文中含有:的双语例句
例句
为了更好的帮助您理解掌握查询词或其译词在地道英语中的实际用法,我们为您准备了出自英文原文的大量英语例句,供您参考。
  transient brain ischemic
We report a case of a 61-year-old man who was admitted to our hospital for transient brain ischemic attack.
      


BACKGROUND: Neuron-specific enolase, γ type isoenzyme that is specially present in the cytoplasm of neurons and neuroendocrine cells, is considered as a sensitive predictor for neuronal damage.OBJECTIVE: To observe the changes of serum neuron-specific enolase in patients with transient brain ischemic attack, so as to explore its relationship with the degree of neuronal damage. DESIGN: Case-control observation. SETTING: Department of Neurology, Jinan No. 4 People's Hospital. PARTICIPANTS: A total of 29...

BACKGROUND: Neuron-specific enolase, γ type isoenzyme that is specially present in the cytoplasm of neurons and neuroendocrine cells, is considered as a sensitive predictor for neuronal damage.OBJECTIVE: To observe the changes of serum neuron-specific enolase in patients with transient brain ischemic attack, so as to explore its relationship with the degree of neuronal damage. DESIGN: Case-control observation. SETTING: Department of Neurology, Jinan No. 4 People's Hospital. PARTICIPANTS: A total of 29 patients who were hospitalized in the Department of Neurology, Jinan No. 4 People's Hospital, due to transient brain ischemic attack (all called for emergent medical treatment within the onset of 6 hours) between March 2002 and May 2004 were enrolled in this study. There were 18 males and 11 females with the average age of (60.36±11.67) years. According to the duration of neural functional deficits, all subjects were divided into two groups, namely, transient-symptom group (≤ 6 hours) of 19 cases and lasting-symptom group (> 6 hours) of 10 cases. At the same time, 25 healthy controls, 15 males and 10 females with the average age of (62.34±9.65) years, were selected from those who came for routine health examination.METHODS: Fasting elbow venous blood of 1 mL was collected only once from the subjects in control group; the same amount of blood was collected from the patients in transient ischemic attack group immediately after hospitalization, and at days 2, 3, 4 and 5. Roche Elecsys 2010 automatic analyzer was used to detect serum neuron-specific enolase. Neuronal damage was assessed with neurological deficit scale (defined as practical recovery if scores were reduced by 90%-100%; remarkable improvement if scores were reduced by 46%-89%; improvement if scores were reduced by 18%-45%; ineffective if scores were reduced by less than 17% or even the disease aggravated).MAIN OUTCOME MEASURES: The daily changes of serum neuron-specific enolase.RESULTS: All the 54 subjects remained in the final result analysis. ① Comparison of neuron-specific enolase density: It was significantly higher in transient brain ischemic attack group than in control group (23.53±12.35) vs (14.29±6.83) μg/L, t=2.678, P < 0.01. ② Curve of neuron-specific enolase changes during the acute stage: It began to increase at the early stage, reached the peak level on the next day, and gradually declined to the normal level in 4-5 days. ③ The level of serum neuron-specific enolase in the two groups with various durations of neurological deficit symptoms: It was obviously higher in transient-symptom group than in control group (19.24±8.95) vs (14.29±6.83) μg/L, t=1.893, P < 0.05, and higher in lasting-symptom group than in control group (28.87±13.15) vs (14.29±6.83) μg/L, t=4.367, P < 0.001. ④ The level of neuron-specific enolase was positively correlated with the duration of neuronal damage (r=0.815, P < 0.01). CONCLUSION: Serum neuron-specific enolase increases within a short term after transient brain ischemic attack and reaches the peak level at around 24-36 hours, suggesting that the detection of serum neuron-specific enolase has a guiding value in assessing the severity of transient brain ischemic attack.

背景:神经元特异性烯醇酶是γ型同工酶特异地存在于神经元和神经内分泌细胞的胞浆内,是神经元损伤较敏感的标志物。目的:观察短暂性脑缺血发作患者血清神经元特异性烯醇酶的变化,探讨其与疾病的神经损伤程度的关系。设计:病例-对照观察。单位:济南市第四人民医院神经内科。对象:选择2002-03/2004-05在济南市第四人民医院神经内科入院的短暂性脑缺血发作患者29例(均为起病后6h的急诊观察患者)。男18例,女11例。平均(60.36±11.67)岁。根据神经功能缺失症状持续时间分为两组:短症状组(≤6h)19例,长症状组(>6h)10例。同期选取健康体检者为对照组,共25人。男15人,女10人。平均(62.34±9.65)岁。方法:对照组只取1次空腹肘静脉血1mL,短暂性脑缺血组在入院即时,第2,3,4,5天分别采空腹肘静脉血1mL。采用罗氏Elecsys2010免疫测定分析仪检测血清神经元特异性烯醇酶。用神经功能缺损程度量表(基本痊愈为评分减少90%-100%;显著进步为评分减少46%-89%;进步为评分减少18%-45%;无效为评分减少17%以下或病情恶化)评定患者的神经损伤程度。主要观察指标:观察患者血清神经元...

背景:神经元特异性烯醇酶是γ型同工酶特异地存在于神经元和神经内分泌细胞的胞浆内,是神经元损伤较敏感的标志物。目的:观察短暂性脑缺血发作患者血清神经元特异性烯醇酶的变化,探讨其与疾病的神经损伤程度的关系。设计:病例-对照观察。单位:济南市第四人民医院神经内科。对象:选择2002-03/2004-05在济南市第四人民医院神经内科入院的短暂性脑缺血发作患者29例(均为起病后6h的急诊观察患者)。男18例,女11例。平均(60.36±11.67)岁。根据神经功能缺失症状持续时间分为两组:短症状组(≤6h)19例,长症状组(>6h)10例。同期选取健康体检者为对照组,共25人。男15人,女10人。平均(62.34±9.65)岁。方法:对照组只取1次空腹肘静脉血1mL,短暂性脑缺血组在入院即时,第2,3,4,5天分别采空腹肘静脉血1mL。采用罗氏Elecsys2010免疫测定分析仪检测血清神经元特异性烯醇酶。用神经功能缺损程度量表(基本痊愈为评分减少90%-100%;显著进步为评分减少46%-89%;进步为评分减少18%-45%;无效为评分减少17%以下或病情恶化)评定患者的神经损伤程度。主要观察指标:观察患者血清神经元特异性烯醇酶的每天变化情况。结果:检测者54例均进入结果分析。①神经元特异性烯醇酶浓度的比较:短暂性脑缺血发作组明显高于对照组[(23.53±12.35,14.29±6.83)μg/L,t=2.678,P<0.01]。②急性期内神经元特异性烯醇酶变化曲线:早期增高,次日达高峰,后趋向逐渐下降,四五天基本恢复正常。③神经功能缺失症状持续时间不同的两组血清神经元特异性烯醇酶:短症状组明显高于对照组[(19.24±8.95,14.29±6.83)μg/L,t=1.893,P<0.05],长症状组高于对照组[(28.87±13.15,14.29±6.83)μg/L,t=4.367,P<0.001]。④神经元特异性烯醇酶值的大小与神经功能缺损时间呈正相关(r=0.815,P<0.01)。结论:短暂性脑缺血发作患者短期内血清神经元特异性烯醇增高,24~36h达到高峰,提示检测短暂性脑缺血发作患者血清神经元特异性烯醇酶浓度。对判断病情严重程度有指导意义。

 
图标索引 相关查询

 


 
CNKI小工具
在英文学术搜索中查有关transient brain ischemic的内容
在知识搜索中查有关transient brain ischemic的内容
在数字搜索中查有关transient brain ischemic的内容
在概念知识元中查有关transient brain ischemic的内容
在学术趋势中查有关transient brain ischemic的内容
 
 

CNKI主页设CNKI翻译助手为主页 | 收藏CNKI翻译助手 | 广告服务 | 英文学术搜索
版权图标  2008 CNKI-中国知网
京ICP证040431号 互联网出版许可证 新出网证(京)字008号
北京市公安局海淀分局 备案号:110 1081725
版权图标 2008中国知网(cnki) 中国学术期刊(光盘版)电子杂志社