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common-stent
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  普通支架
     The circumference and area of biliary duct lumen are obviously increased compared to those of the common-stent group (P<0.01);
     胆管腔周长及胆管腔面积均比普通支架组明显增加(P<0.01)。
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     Methods The experimental dogs were randomly divided into common-stent group and 103Pd stent group, each of 6 animals.
     方法 实验犬 12只 (15~ 2 0kg) ,随机分为普通支架组和1 0 3Pd支架组各 6只。
短句来源
     The expression of gene p53 of biliary tissue increased in the 103Pd stent group, and decreased in the common-stent group;
     1 0 3Pd支架组胆管组织p5 3基因表达增高 ,而普通支架组p5 3基因表达降低 ;
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  相似匹配句对
     Objective To study the common clinical manifestations of in-stent restenosis.
     目的 探讨支架内再狭窄的常见表现。
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     On the Common Enterprise
     共同的事业
短句来源
     Common Knowledge
     知识共享——从个人到集体
短句来源
     Acute coronary syndrome is a common clinical manifestation of in-stent restenosis
     急性冠状动脉综合征是支架内再狭窄的常见表现
短句来源
     Feasibility of using endobiliary stent for laparoscopic exploration of common bile duct
     应用胆道内支架行腹腔镜胆总管探查的可行性研究
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Objective To investigate the dose response and optimal radioactivity dose of 103 Pd radioactive stent for preventing restenosis in rabbits, and to prove that it reduces in stent restenosis without edge restenosis. Methods The abdominal aorta of the rabbits randomized to common or radioactive stent groups (including 5,15,25 and 35 Gy subgroups)were balloon injured,followed by stenting. Intravascular ultrasound and angiography on abdominal aortae were performed 8 weeks after stenting. Results Both the...

Objective To investigate the dose response and optimal radioactivity dose of 103 Pd radioactive stent for preventing restenosis in rabbits, and to prove that it reduces in stent restenosis without edge restenosis. Methods The abdominal aorta of the rabbits randomized to common or radioactive stent groups (including 5,15,25 and 35 Gy subgroups)were balloon injured,followed by stenting. Intravascular ultrasound and angiography on abdominal aortae were performed 8 weeks after stenting. Results Both the minimal lumen diameter and in stent lumen area in 103 Pd radioactive stent groups [(1.94±0.21) mm and (5.99±0.43) mm 2, (2.17±0.12) mm and (7.01±0.93) mm 2, (2.43±0.22) mm and (8.09±0.89) mm 2, (2.56±0.27) mm and (8.49±0.54) mm 2 for the 5, 15, 25 and 35 Gy subgroups, respectively] were significantly improved corresponding with the stepwise escalating doses at the end of 8 weeks,revealing a dose dependent effect. At the edge segments, late lumen area loss (magnitude of stenosis), in stent neointimal area and the decrease of external elastic membrane (EEM) area in 25 and 35 Gy 103 Pd radioactive stent subgroups [(1.41±0.84), (0.65±0.60), (0.76±0.36) mm 2 and (1.32±1.02), (0.69±0.60), (0.64±0.53) mm 2, respectively] were lower than that in common stent group [(2.88±1.43), (1.68±1.30) and (1.20±0.41) mm 2, respectively, all P <0.05 versus control]. Conclusions 103 Pd radioactive stent could dose dependently restrain in stent neointimal hyperplasia, reduce the magnitude of in stent stenosis, and the optimal prescribed dose was 25 Gy.The possible mechanism of the preventive effects of 103 Pd radioactive stent on obvious edge restenosis could be both inhibition of in stent neointimal hyperplasia and arterial negative remodeling.

目的 探讨1 0 3Pd支架对血管成形术后再狭窄预防作用的量效关系及是否导致边缘再狭窄。方法  5 0只雄性新西兰兔随机分为普通支架组和核素支架各剂量组 (8只 组 )。支架植入术后 8周行腹主动脉血管内超声和造影检查。结果 核素支架组随剂量增加 ,支架段血管最小内径和支架段血管管腔切面积均增大 ,狭窄程度减小 ;核素支架 2 5及 35Gy组支架边缘段血管管腔内增生内膜切面积及外弹力板切面积变化 (缩小值 )均小于普通支架组 (P <0 .0 5 )。结论 1 0 3Pd支架可抑制支架内内膜增生 ,减轻支架内狭窄程度 ,且不引起边缘再狭窄。

Objective To investigate the dose response of preventive effect of 103 Pd radioactive stent on restenosis in rabbits and its effect on proliferation and apoptosis of VSMC. Methods The abdominal arteries of the rabbits randomized to common or radioactive stents groups (including 5 Gy, 15 Gy, 25 Gy, 35 Gy groups) were balloon injured and stented. Abdominal artery angiography, immunohistochemical study of PCNA, Western blotting for the activity of MAPK and TUNEL assay were performed 8 weeks after...

Objective To investigate the dose response of preventive effect of 103 Pd radioactive stent on restenosis in rabbits and its effect on proliferation and apoptosis of VSMC. Methods The abdominal arteries of the rabbits randomized to common or radioactive stents groups (including 5 Gy, 15 Gy, 25 Gy, 35 Gy groups) were balloon injured and stented. Abdominal artery angiography, immunohistochemical study of PCNA, Western blotting for the activity of MAPK and TUNEL assay were performed 8 weeks after stenting. Results The in stent minimal lumen diameters in 103 Pd radioactive stent groups (1 94±0 21) mm, (2 17±0 12) mm, (2 43±0 22) mm, (2 56±0 27) mm in 5 Gy, 15 Gy, 25 Gy, 35 Gy groups, respectively were significantly improved as increases of the doses at the end of 8 weeks, revealing a dose response effect. At the edge segments, minimal lumen diameters in 25 Gy and 35 Gy 103 Pd stent groups [(2 07±0 14)mm and (2 18±0 17)mm] were larger than that in common stent group (1 86±0 23) mm ( P <0 05). Both the positive ratio of PCNA and the OPTDI of MAPK in each 103 Pd stent groups, both in stent and at edges, were lower than that in commen stent group ( P <0 01), while the ratio of TUNEL positive cells in 15, 25 and 35 Gy of radioactive stent groups were significantly higher than that in commen stent group ( P <0 001). Conclusion 103 Pd radioactive stent dose dependently reduce the magnitude of in stent stenosis, without edge effect. It inhibits proliferation and migration of VSMC and induces the increase of apoptotic cells. The common stent activated the MAPK signal pathway while the 103 Pd stent restrained this activation.

目的 探讨 10 3Pd支架对再狭窄预防作用的量效关系与相应对VSMC增殖和凋亡的影响。方法  40只雄兔随机分为普通支架组和核素支架各剂量组 (8只 /组 )。支架置入术后 8周行腹主动脉造影、免疫组化染色、有丝分裂原激活的蛋白激酶 (MAPK)测定及细胞凋亡检测。结果 核素支架组随剂量增加 ,支架段最小内径增大而狭窄程度减小 ;边缘段最小内径核素支架 2 5Gy和 35Gy组大于普通支架组 (P <0 0 5 )。支架段和边缘段PCNA阳性细胞比率及MAPK积分光密度 ,核素支架各组均小于普通支架组 (P <0 0 1) ,而凋亡指数 ,核素支架除 5Gy组外均大于普通支架组(P <0 0 1)。结论  10 3Pd支架可减少支架内狭窄程度 ,显示出量效关系 ,且不引起边缘再狭窄。10 3Pd支架既抑制VSMC增生、移行也诱导凋亡细胞的数量增加

AIM:To observe the ultra structure changes of vessel wall during the repair of iliac artery injury of rabbits,and explore the preventive effect of magnetic stent on restenosis. METHODS:Twenty New Zealand white rabbits(provided by Fourth Military Medical University of Chinese PLA) were randomly divided into magnetic stent group and common stent group.Ten rabbits were in each group,and magnetic NIR stent implantation and common NIR stent implantation were performed in the left iliac artery of the rabbits respectively.Twenty...

AIM:To observe the ultra structure changes of vessel wall during the repair of iliac artery injury of rabbits,and explore the preventive effect of magnetic stent on restenosis. METHODS:Twenty New Zealand white rabbits(provided by Fourth Military Medical University of Chinese PLA) were randomly divided into magnetic stent group and common stent group.Ten rabbits were in each group,and magnetic NIR stent implantation and common NIR stent implantation were performed in the left iliac artery of the rabbits respectively.Twenty five miligram of aspirine was given to the rabbits in the two groups per day for three days before the operation until the animals were executed.Heparin was administered subcutaneously after the operation 2500 U per day for 7 days.The animals were executed at the 7th and 30th days.The changes of the injured vessel walls were observed by optical microscope,electron microscope and immunohistochemical technique. RESULTS:Thirty days after the operation,the proliferation degree of intima smooth muscle cell(SMC) of iliac artery in the magnetic stent group[(127.59±22.18) μm] was significantly lower than that in the common stent group[(189.45±28.42) μm],and the difference had significant meaning(t=3.43,P=0.014).Observation by electron microscopy showed that more myofibroblasts migrated from adventitia to tunica media and intima in common stent group as compared with the magnetic stent group.Inside the media and intima,there were many synthetic SMCs.Extracellular matrix(ECM) was mainly composed of collegen and elastin.Thirty days after operation,most SMCs under the intima of the magnetic group had transformed to contractile type,and ECMs were less and mainly composed of collegen and elastin.However, SMCs under the intima in common stent group were still synthetic type,with large amount of ECMs around the SMCs,which were mainly composed of proteoglycans. CONCLUTION:Magnetic stent has significant pretective effect on restenosis by inhibiting proliferation and migration of SMCs and the production of ECM.

目的:观察兔髂动脉损伤修复过程中血管壁超微结构的变化,探讨磁化支架对再狭窄的防治作用。方法:新西兰白兔20只(解放军第四军医大学提供),随机分为磁化支架组及普通支架组,每组10只。分别于左髂动脉行磁化NIR支架及普通NIR支架置入术。两组动物术前3d始予阿司匹林25mg/d至处死动物。术后皮下注射肝素2500U/d,连续7d。术后7d和30d处死动物。用光镜、透射电镜及免疫组化染色技术观察损伤部位血管壁变化。结果:术后30d光镜检查发现磁化支架组髂动脉新生内膜平滑肌细胞(smoothmusclecell,SMC)的增生程度犤(127.59±22.18)μm犦明显低于普通支架组犤(189.45±28.42)μm犦,差异有显著性意义(t=3.43,P=0.014)。电镜检查显示,术后7d普通支架组较磁化支架组可见较多从外膜向中膜及内膜迁移的肌纤维母细胞,中膜及内膜大量以合成型为主的SMC,细胞外基质(extracellularmatrix,ECM)以胶原及弹性纤维为主。30d时磁化支架组内膜下SMC已转化为收缩型,ECM较少,以胶原及弹性纤维为主;而普通支架内膜下SMC仍为典型合成型表现,周围大量ECM,以糖氨多...

目的:观察兔髂动脉损伤修复过程中血管壁超微结构的变化,探讨磁化支架对再狭窄的防治作用。方法:新西兰白兔20只(解放军第四军医大学提供),随机分为磁化支架组及普通支架组,每组10只。分别于左髂动脉行磁化NIR支架及普通NIR支架置入术。两组动物术前3d始予阿司匹林25mg/d至处死动物。术后皮下注射肝素2500U/d,连续7d。术后7d和30d处死动物。用光镜、透射电镜及免疫组化染色技术观察损伤部位血管壁变化。结果:术后30d光镜检查发现磁化支架组髂动脉新生内膜平滑肌细胞(smoothmusclecell,SMC)的增生程度犤(127.59±22.18)μm犦明显低于普通支架组犤(189.45±28.42)μm犦,差异有显著性意义(t=3.43,P=0.014)。电镜检查显示,术后7d普通支架组较磁化支架组可见较多从外膜向中膜及内膜迁移的肌纤维母细胞,中膜及内膜大量以合成型为主的SMC,细胞外基质(extracellularmatrix,ECM)以胶原及弹性纤维为主。30d时磁化支架组内膜下SMC已转化为收缩型,ECM较少,以胶原及弹性纤维为主;而普通支架内膜下SMC仍为典型合成型表现,周围大量ECM,以糖氨多糖为主。结论:磁化支架通过抑制SMC增生、迁移及ECM合成对再狭窄形成有显著的防治作用。

 
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