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visceral pathological
相关语句
  内脏病理学
     The visceral pathological and blood bacteriological changes caused by combined projectile-burn wound jointed with seawater soak
     海水浸泡弹烧复合伤血细菌学与内脏病理学的变化
短句来源
  “visceral pathological”译为未确定词的双语例句
     The guinea pigs were challenged with 1130 strain,1219 strain,b858 strain,1290 strain and ATCC 35810 strain to establish the tuberculosis model. The experimental groups were treated with rifampicin. The parameters including macroscopic visceral pathological change index,visceral weight index(spleen,lungs and liver),the colony-forming units(CFU)quantity of visceral Mycobacterium tuberculosis culture(spleen,lungs)and tissue pathology of guinea pigs were observed.
     建立豚鼠1130株、1219株、b858株、1290株和 ATCC 35810株攻毒模型,实验组用利福平治疗,对照组用生理盐水治疗,各组分别于4、7、10周观察豚鼠脏器(脾、肺、肝)致病指数、脏器重量指数(WI)、脏器(脾、肺)结核分枝杆菌培养的荷菌量及组织病理学检查。
短句来源
     Results At the 7th week after challenged with 1130 strain,1219 strain,1290 strain and b858 strain,all animals were sacrificed. The macroscopic visceral pathological change indices of the experimental group were 68.7±13.8,60.0±13.5,70.0±5.8 and 23.8±18.9,whereas all those parameters of the control group were 76.2±18.9,40.0±16.8,63.8±10.3 and 22.5±15.5 respectively,and there was no significance between the experimental group and the control group(t=0.64,1.85,0.35 and 0.10,all P> 0.05).
     结果 1130株、1219株、1290株和 b858株攻毒豚鼠7周时,致病指数值:实验组分别为68.7±13.8、60.0±13.5、70.0±5.8和23.8±18.9,对照组分别为76.2±18.9、40.0±16.8、63.8±10.3和22.5±15.5,差异均无统计学意义(t 值分别为0.64、1.85、0.35和0.10,P 均>0.05);
短句来源
  相似匹配句对
     AND PATHOLOGICAL STUDY
     牙源性粘液瘤的临床、X线和病理学研究
短句来源
     PATHOLOGICAL DIAGNOSIS OF SKIN - AND VISCERAL - TYPE MAREK'S DISEASE OF LAYING HENS FROM SHANXI PROVINCE
     山西省皮肤型和内脏型鸡马立克氏病的病理学诊断
     Conclusion Measurement of visceral organs in the autopsy can assist to an integrative pathological diagnosis.
     结论大体检测器官密度对尸体解剖的综合病理诊断具有一定的辅助意义。
短句来源
     Pathological study of myofibroblastosarcoma
     肌纤维母细胞肉瘤的病理学研究
短句来源
     VISCERAL FUNGAL DISEASE
     内脏霉菌病
短句来源
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Objective To investigate the role of selective decontamination of the digestive tract (SDD) in prevention of bacterial infections secondary to acute necrotizing pancreatitis (ANP). Methods A total of 60 rats were employed to establish the model of ANP with retrograde infusion of artificial bile into the main pancreatic duct. Then animals were randomized into the normal control group (n =6), sham operated group (n = 14) and SDD-treated group (polymycinE, tobramycin and nystatin mixture were used). Visceral...

Objective To investigate the role of selective decontamination of the digestive tract (SDD) in prevention of bacterial infections secondary to acute necrotizing pancreatitis (ANP). Methods A total of 60 rats were employed to establish the model of ANP with retrograde infusion of artificial bile into the main pancreatic duct. Then animals were randomized into the normal control group (n =6), sham operated group (n = 14) and SDD-treated group (polymycinE, tobramycin and nystatin mixture were used). Visceral pathological changes, serum levels of amylase, intestinal flora, incidence of bacterial translocation to organs and mortality were determined 72 h after the operation in all the 3 groups. Results In SDD-treated group, damages in the pancreas were remarkably attenuated and serum level of amylase significantly decreased. At the 72nd h after treatment, the rate of bacterial translocation to other organs and mortality markedly decreased to 33.3% and 14.3% from the original 59.5% and 58.8%, respectively (P<0.05). Conclusions SDD can prevent infections secondary to ANP through controlling bacterial disturbance in the digestive tract and reduce bacterial translocation.

目的 通过急性坏死性胰腺炎 (ANP)动物模型 ,探讨选择性消化道脱污染 (SDD)预防胰腺继发感染的作用。方法 Wistar大鼠 60只 ,向总胆胰管逆行输入人工胆汁复制ANP模型 ,随机设立正常对照组、假手术对照组、ANP组和SDD治疗组 ,观察胰肠组织学、胰淀粉酶、肠道菌群、脏器细菌移位率和死亡率的变化。结果 SDD使ANP动物胰腺和小肠粘膜病理改变减轻 ,胰淀粉酶水平下降 (P <0 0 5 ) ,并使ANP时肠道菌群的紊乱状态得以恢复 ,肠粘膜和肠内容物双歧杆菌 /大肠杆菌比值 (B/E)分别由 - 0 3 7± 0 72升至 1 73± 1 2 3 (P <0 0 1)和由 0 88± 0 77升至无穷大。SDD组 72h脏器细菌移位率由 5 9 5 %降至 3 3 3 % (P <0 0 5 ) ,72h死亡率由 5 8 8%降至 14 3 % (P =0 0 0 5 )。结论 SDD通过减轻肠道菌群紊乱状态 ,减少细菌移位 ,有利于预防ANP继发感染。

Objective To investigate the presence of rifampicin-dependent Mycobacterium tuberculosis strains by use of a guinea pig model of tuberculosis of rifampicin-dependent Mycobacterium tuberculosis.Methods Guinea pigs were randomly divided into groups of infection by rifampicin-dependent Mycobacterium tuberculosis strains(1130 strain,1219 strain,b858 strain),rifampicin-resistant Mycobacterium tuberculosis strain(1290 strain)and ATCC 35810 strain and each group was further divided into an experimental group and a...

Objective To investigate the presence of rifampicin-dependent Mycobacterium tuberculosis strains by use of a guinea pig model of tuberculosis of rifampicin-dependent Mycobacterium tuberculosis.Methods Guinea pigs were randomly divided into groups of infection by rifampicin-dependent Mycobacterium tuberculosis strains(1130 strain,1219 strain,b858 strain),rifampicin-resistant Mycobacterium tuberculosis strain(1290 strain)and ATCC 35810 strain and each group was further divided into an experimental group and a control group.The guinea pigs were challenged with 1130 strain,1219 strain,b858 strain,1290 strain and ATCC 35810 strain to establish the tuberculosis model.The experimental groups were treated with rifampicin.The parameters including macroscopic visceral pathological change index,visceral weight index(spleen,lungs and liver),the colony-forming units(CFU)quantity of visceral Mycobacterium tuberculosis culture(spleen,lungs)and tissue pathology of guinea pigs were observed.Results At the 7th week after challenged with 1130 strain,1219 strain,1290 strain and b858 strain,all animals were sacrificed.The macroscopic visceral pathological change indices of the experimental group were 68.7±13.8,60.0±13.5,70.0±5.8 and 23.8±18.9,whereas all those parameters of the control group were 76.2±18.9,40.0±16.8,63.8±10.3 and 22.5±15.5 respectively,and there was no significance between the experimental group and the control group(t=0.64,1.85,0.35 and 0.10,all P> 0.05).The spleen weight indices of experimental group were 0.229±0.048,0.256±0.067,0.324± 0.054 and 0.199±0.029,whereas all those parameters of control groups were 0.278±0.025,0.216± 0.076,0.368±0.033 and 0.213±0.038 respectively,and there was no significance between the experimental group and the control group(t=1.75,0.79,1.41 and 0.57,all P>0.05).The CFU quantity of spleen Mycobacterium tuberculosis culture of the experimental group were 4.98±0.30,4.68± 1.26,5.07±0.47 and 3.85±0.45,whereas all those parameters of control groups were 4.90±1.03, 4.79±0.45,5.08±0.55 and 4.23±0.95 respectively,and there was no significance between the experimental group and the control group(t=0.11,0.15,0.03 and 0.73,all P>0.05);Moreover,the tissue pathology of both groups was similar.Conclusions The tuberculosis model of rifampicin-dependent Mycobacterium tuberculosis strains was similar to the model of rifampicin-resistant Mycobacterium tuberculosis in guinea pigs.Rifampicin-dependency was not evident in this guinea pig tuberculosis model.

目的通过对依赖利福平结核分枝杆菌的豚鼠结核病模型研究,探讨依赖利福平结核分枝杆菌是否存在。方法豚鼠按随机数字法分成依赖利福平结核分枝杆菌(1130株、1219株、b858株)组、耐利福平结核分枝杆菌(1290株)组和对豚鼠强毒力致病株(ATCC 35810株)组,每株组再设实验组和对照组。建立豚鼠1130株、1219株、b858株、1290株和 ATCC 35810株攻毒模型,实验组用利福平治疗,对照组用生理盐水治疗,各组分别于4、7、10周观察豚鼠脏器(脾、肺、肝)致病指数、脏器重量指数(WI)、脏器(脾、肺)结核分枝杆菌培养的荷菌量及组织病理学检查。结果 1130株、1219株、1290株和 b858株攻毒豚鼠7周时,致病指数值:实验组分别为68.7±13.8、60.0±13.5、70.0±5.8和23.8±18.9,对照组分别为76.2±18.9、40.0±16.8、63.8±10.3和22.5±15.5,差异均无统计学意义(t 值分别为0.64、1.85、0.35和0.10,P 均>0.05);脾 WI 值:实验组分别为0.229±0.048、0.256±0.067、0.324±0.054和0.199...

目的通过对依赖利福平结核分枝杆菌的豚鼠结核病模型研究,探讨依赖利福平结核分枝杆菌是否存在。方法豚鼠按随机数字法分成依赖利福平结核分枝杆菌(1130株、1219株、b858株)组、耐利福平结核分枝杆菌(1290株)组和对豚鼠强毒力致病株(ATCC 35810株)组,每株组再设实验组和对照组。建立豚鼠1130株、1219株、b858株、1290株和 ATCC 35810株攻毒模型,实验组用利福平治疗,对照组用生理盐水治疗,各组分别于4、7、10周观察豚鼠脏器(脾、肺、肝)致病指数、脏器重量指数(WI)、脏器(脾、肺)结核分枝杆菌培养的荷菌量及组织病理学检查。结果 1130株、1219株、1290株和 b858株攻毒豚鼠7周时,致病指数值:实验组分别为68.7±13.8、60.0±13.5、70.0±5.8和23.8±18.9,对照组分别为76.2±18.9、40.0±16.8、63.8±10.3和22.5±15.5,差异均无统计学意义(t 值分别为0.64、1.85、0.35和0.10,P 均>0.05);脾 WI 值:实验组分别为0.229±0.048、0.256±0.067、0.324±0.054和0.199±0.029,对照组分别为0.278±0.025、0.216±0.076、0.368±0.033和0.213±0.038,差异均无统计学意义(t 值分别为1.75、0.79、1.41和0.57,P 均>0.05);脾荷菌量的对数值:实验组分别为(4.98±0.30)、(4.68±1.26)、(5.07±0.47)和(3.85±0.45)CFU,对照组分别为(4.90±1.03)、(4.79±0.45)、(5.08±0.55)和(4.23±0.95)CFU,差异均无统计学意义(t 值分别为0.11、0.15、0.03和0.73,P 均>0.05),实验组和对照组的病理检查结果相似。结论依赖利福平结核分枝杆菌豚鼠结核病模型研究结果与耐利福平结核分枝杆菌株豚鼠结核病模型近似,未见明显依赖利福平现象。

 
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