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acute cerebral ischemic injury
相关语句
  急性缺血性脑损伤
     Mechanism of phospholipase A_2 and protective effect of nimodipine in acute cerebral ischemic injury
     磷脂酶A_2激活在急性缺血性脑损伤中的作用机制及尼莫地平的保护作用
短句来源
     Mechanism of Bajisin's Protective Effect on Acute Cerebral Ischemic Injury
     巴戟素对急性缺血性脑损伤保护作用的机制研究
短句来源
  “acute cerebral ischemic injury”译为未确定词的双语例句
     CONCLUSION DAXXK can relieve damage of cerebral cells and protect brain tissue through clearing oxygen-derived free radidicals inhibiting oxidation decreasing lipid peroxidation and blocking internal flow of Ca2+ during acute cerebral ischemic injury.
     结论:地奥心血康在急性脑缺血损伤过程中,通过清除氧自由基、抗氧化、减少脂质过氧化、阻断Ca2+内流等作用,减轻脑细胞的损伤,对脑组织细胞起保护作用。
短句来源
     Objective:To investigate effects of Buchangnaoxintong (BCNXT,步长脑心通)on the cerebral microcirculation in rats with acute cerebral ischemic injury.
     目的:观察步长脑心通对急性缺血性卒中微小循环障碍的影响。
短句来源
     Study on Effect of Naomai Erhao Injection on Protection of Brain Nerves from Acute Cerebral Ischemic Injury and Its Mechanism
     脑脉Ⅱ号对脑组织缺血性损伤的保护及其机制的研究
短句来源
     It is suggested that the mechanism of Bajisin's protective effect on acute cerebral ischemic injury is related to the counteraction of lipid peroxidation and the improvement of glucose metabolism in brain tissue.
     但对急性脑缺血衰老大鼠脑组织一氧化氮 (NO)含量无明显影响 (P >0 .0 5)。 提示巴戟素对缺血性脑损伤保护作用的机制与抗脑组织的脂质过氧化作用及改善脑组织葡萄糖代谢有关。
短句来源
     Objective To study the effect of ethanol extract from Qufeng tongluo recipe on cerebral blood flow (CBF) in rats with acute cerebral ischemic injury.
     目的通过祛风通络方醇提液经颈内动脉冲洗的方法改善急性缺血性中风脑局部的血液循环,来阻断再建的血流与缺血损伤组织间的相互作用,减少缺血再灌注损伤。
短句来源
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  相似匹配句对
     Pathogenesis of acute focal ischemic cerebral injury
     急性局灶性脑缺血性脑损伤发生机制
短句来源
     The Mechanism of Microirculatory Disturbance in Acute Cerebral Ischemic Injury
     急性脑缺血损伤的微循环障碍机制
短句来源
     Endothelin and Cerebral Ischemic Injury
     内皮素与缺血性脑损伤
短句来源
     Cerebral ischemic injury and Akt
     脑缺血损伤与Akt研究的进展
短句来源
     Mechanism of Bajisin's Protective Effect on Acute Cerebral Ischemic Injury
     巴戟素对急性缺血性脑损伤保护作用的机制研究
短句来源
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  acute cerebral ischemic injury
Acute cerebral ischemic injury can be rapidly detected on diffusion-weighted images.
      
Objective: To investigate muscone's neuroprotective action and its mechanism in rats with acute cerebral ischemic injury.Methods: Rat focal ischemic model of middle cerebral artery occlusion (MCAO) was established by thread inserting method.
      


Purpose: To study the effect of Naomai Erhao Injection—a compound TCM injection for cerebrovascula diseases on protection of brain nerves from acute cerebral ischemic injury and its mechanism. Methods:The structure and functions of the rats' brain were stu died by means of the electronic microscope, radionucliole tracer method and some other me thods. Results: Naomai Erhao Injection reduced the tissue injury of cerebral coxtex of rats with acute cerebral ischemia; increased...

Purpose: To study the effect of Naomai Erhao Injection—a compound TCM injection for cerebrovascula diseases on protection of brain nerves from acute cerebral ischemic injury and its mechanism. Methods:The structure and functions of the rats' brain were stu died by means of the electronic microscope, radionucliole tracer method and some other me thods. Results: Naomai Erhao Injection reduced the tissue injury of cerebral coxtex of rats with acute cerebral ischemia; increased the SOD, GSH-PX activity of ischemic brain ti ssue, reduced the LPO content and raised the 3H-dexoyglucose content in brain tissue. Conclusions: Naomai Erhao Injection can protect the brain nerves by lessening cerebrocellular injury caused by ischemia and its mechanism of action can protect the brain nerves by lesse ning the offensive from oxygen free radicals and lowering the energy consumption.

目的 :探讨中药复方脑脉Ⅱ号对急性脑缺血性损伤的保护作用及其机制。方法 :采用电子显微镜观察及放射性核素示踪法等技术 ,从结构与功能方面进行研究。结果 :脑脉Ⅱ号能减轻急性脑缺血大鼠大脑皮层组织结构的损伤程度 ;提高缺血脑组织SOD、GSH -PX活性 ,并减少LPO含量 ;能增加脑组织3H -脱氧葡萄糖含量。结论 :脑脉Ⅱ号能减轻缺血对脑细胞的损伤 ,从而起保护作用 ,其作用机制可能与对抗自由基攻击和减少能量消耗等有关。

Objective: To evaluate the role of NO、ET and TNF α in the pathogenesis of ischemic stroke. Methods: The levels of TNF α、 NO and ET in peripheral blood were measured by radioimmunorective and chemocolorimetric analysis in 93 patients with acute cerebral infarction (ACI)of different periods and 76 healthy controls. Results: The levels of serum TNF α、NO and plasma ET in acute phase of ACI were significantly higher than those in convalescent and recovery phases of ACI; the above substances in convalescnt and...

Objective: To evaluate the role of NO、ET and TNF α in the pathogenesis of ischemic stroke. Methods: The levels of TNF α、 NO and ET in peripheral blood were measured by radioimmunorective and chemocolorimetric analysis in 93 patients with acute cerebral infarction (ACI)of different periods and 76 healthy controls. Results: The levels of serum TNF α、NO and plasma ET in acute phase of ACI were significantly higher than those in convalescent and recovery phases of ACI; the above substances in convalescnt and recovery phases were significantly higher than those of healthy controls,and related to the stage severity of cerebral infarction; positive correlation between TNF α and ET、NO were found in acute cerebral infarction. Conclusions: The increased cytokines were not merely the response to acute cerebral ischemic injury, but those might play a role in the pathogenesis of ischemic stroke.

目的:探讨肿瘤坏死因子-α(TNF-α)、内皮素(ET)、一氧化氮(NO)在急性脑梗塞发病中的作用。方法:采用放射免疫法和化学比色法测定了93 例急性脑梗塞各期患者和76 例健康人的外周血TNF-α、ET、NO的含量水平。结果:脑梗塞急性期患者血中的TNF-α、ET、NO含量明显高于稳定期和恢复期(P< 0.05~0.001),而稳定期和恢复期TNF-α、ET 、NO均高于正常对照组(P< 0.05~0.001),并与脑梗塞的分期和严重程度相关,且在急性期TNF-α与ET、NO呈正相关(r分别为0.75、0.73)。结论:这些因子的升高不仅在急性期脑缺血性损伤,而且在缺血性脑卒中发病中可能起一定的作用

The protective effect of Xuesaitong Capsule (XC) on cerebral ischemia were studied in rat model established by ligation of bilateral carotid arteries and lowering of blood pressure.The results showed that XC at the dose of 0.1g/kg and 0.2g/kg significantly decreased the water content,permeability of capillary and ischemic injury in brain.In addition,XC significantly increased hypoxie tolerance in mice at the dose of 0.2g/kg .The results indicate that XC exerts a protective effect on acute cerebral...

The protective effect of Xuesaitong Capsule (XC) on cerebral ischemia were studied in rat model established by ligation of bilateral carotid arteries and lowering of blood pressure.The results showed that XC at the dose of 0.1g/kg and 0.2g/kg significantly decreased the water content,permeability of capillary and ischemic injury in brain.In addition,XC significantly increased hypoxie tolerance in mice at the dose of 0.2g/kg .The results indicate that XC exerts a protective effect on acute cerebral ischemic injury.

用结扎颈总动脉合并降压法制备的脑缺血模型研究中药血塞通胶丸对脑缺血的保护作用。结果显示 ,给药后缺血大鼠的脑含水量、脑毛细胞管通透性、脑组织的缺血损伤程度均明显降低。另外 ,该药能明显提高小鼠的耐缺氧能力 ,提示血塞通胶丸有明显的脑缺血保护作用

 
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