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reduce et
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  降低et
     especially SSCM could enhance NO contents in serum,and was reduce ET levels in plasma,evidently cut down TNF-α contents in serum,and was partly superior to Vp.
     同时肾衰合剂可显著降低ET、升高NO的含量,减少TNF-α的产生,其作用部分优于维拉帕米。
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  “reduce et”译为未确定词的双语例句
     ②In type 2 diabetes without hypertension group, Lotensin could significantly decrease ET (P < 0. 05) and obviously increase NO (P < 0. 01) , Whatever under the dosages of 5 mg/d or 10 mg/d, Lotensin could elevate NO level and reduce ET (P < 0. 05) , no difference was seen in the two dosages.
     ②NEH组中,Ⅲ亚组治疗后ET-1水平明显下降(P<0.05),并且NO水平显著升高(P<0.01),而无论是ⅢA亚组还是ⅢB亚组,NO水平均显著升高,ET-1均显著降低水平(P<0.05),两者间的差异无显著性。
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     Conclusions:By elevating the activity of plasma and renal NOS to increase NO levels and by depressing the expression of ET-1mRNA in kidney to reduce ET levels,L-arginine could improve Ccr and RCBF to ameliorate renal dysfunction in obstructive jaundice.
     结论:L鄄Arg通过增强血和肾组织NOS活性来增加体内NO水平、抑制ET1mRNA表达、降低体内ET水平,从而提高Ccr与RCBF,减轻阻塞性黄疸时的肾功能损伤。
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     It could reduce ET content and CRP, TNF-α content.
     并降低寒瘀互结大鼠CRP、TNF -α含量。
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     the results also suggest that underground work probably may reduce ET level of the miners.
     超体重者血浆ET水平较高,而井下作业可能降低了高血压矿工血浆ET水平。
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     The concentration of both plasma and myocardial endothelin (ET) was measured, and the activities of creatine phosphokinase (CK) and lactic dehydrogenase (LDH ) were also measured The results showed that ET levels of both plasma and myocardium in I/R group increased significantly,and myocardium was injured obviously Batroxobin can reduce ET in plasma and myocardium , and can reduce the activities of CK and LDH In addition, it can alleviate the myocardial injury of ischemia /reperfusion
     结果表明 ,I/R组心肌组织及血浆ET水平明显升高 ,心肌组织明显损伤 ,Batroxobin能够显著降低再灌期血浆及心肌ET水平 ,降低CK及LDH活性 ,减轻心肌组织I/R损伤。
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     et C.
     et C.
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     et W.
     et W.
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     reduce F, m and f.
     降低 m,降低 f。
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     ACEI can reduce the plasmatic concentration of ET.
     血管紧张素转换酶抑制剂 (ACEI)卡托普利应用可降低血浆ET浓度。
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     It may reduce the plasma levels of ET and ANP significantly.
     坎地沙坦能明显降低血浆ET、ANP的水平,这可能是血管紧张肽Ⅱ受体拮抗药治疗CHF的机制之一。
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  reduce et
The specific ETA antagonist BQ-123 could not reduce ET-1 pulmonary removal.
      
Therefore, to increase Ek, we must reduce Et and increase Ea.
      
NO to reduce ET-1 expression in lung grafts has not previously been demonstrated.
      


Plasma endothelin (ET) was gradually increased and significantly higher than that of pre-shock period at 6h after hemorrhage/reperfusion (S/R) in rabbit,while sham group had no changes. Furthermore,ET was decreased in 2h after S/R injury by the use of phospholipase A2 inhibitor chloroquine,ME8101 and antioxidant (TFA) during shock period,and in particular,ME810l and TFA markedly reduced ET level within 30min after S/R,which was significantly lower than that of pre-shock period ( P <0. 01 )....

Plasma endothelin (ET) was gradually increased and significantly higher than that of pre-shock period at 6h after hemorrhage/reperfusion (S/R) in rabbit,while sham group had no changes. Furthermore,ET was decreased in 2h after S/R injury by the use of phospholipase A2 inhibitor chloroquine,ME8101 and antioxidant (TFA) during shock period,and in particular,ME810l and TFA markedly reduced ET level within 30min after S/R,which was significantly lower than that of pre-shock period ( P <0. 01 ). Meanwhile,plasma HCO3- and SBC increased at 24h after S/R by the administration of these agents, being significantly higher than that of S/R group ( P <0. 01 ). The results indicated that the increase of ET after S/R appears to be related with the advance of acidosis,and phospholipase A2 inhibitor and antioxidant could be used to block ET release,and to improve acidosis induced by the S/R injuries.

家兔出血性休克再灌注(S/R)后,放免分析发现血浆内皮素逐渐升高,同失血前比较第6h有显著差异(P<0.05),假手术组没有改变。磷脂酶A_2阻断剂氯喹、磷酸萘酚喹和抗氧化剂黄芪酮于失血后再灌注前使用可抑制S/R后2h内血浆内皮素增高,后两者可更明显降低S/R后30min的内皮索水平,与失血前比较差异非常好著(P<0.01)。同时,HCO~-_3和碱贮备(SBC)显著降低,上述三种药物治疗显著增加HCO~-_3和SBC的含量,它们在S/R后24h明显高于S/R组的水平(P<0.01)。结果提示,S/R后血浆内皮素上升可能与代谢性酸中毒加重有一定联系。磷脂酶A_2阻断剂和抗氧化剂可能通过早期抑制内皮素的释放,从而减轻24h后代谢性酸中毒程度。

This paper studied the relations between the middle ear pressure, the opening duration of eustachian tube (ET), and the duration of nasopharyngeal pressure phase during Toynbee maneuver by sonotubometry, tympanometry and nasopharynx manometry. The mucociliary clearance of ET in Toynbee maneuver was also studied by Saccharin method. The results showed that negative middle ear pressure induced by Toynbee maneuver was related to prolonged opening duration of ET and duration of negative pressure...

This paper studied the relations between the middle ear pressure, the opening duration of eustachian tube (ET), and the duration of nasopharyngeal pressure phase during Toynbee maneuver by sonotubometry, tympanometry and nasopharynx manometry. The mucociliary clearance of ET in Toynbee maneuver was also studied by Saccharin method. The results showed that negative middle ear pressure induced by Toynbee maneuver was related to prolonged opening duration of ET and duration of negative pressure in nasopharynx and lower opening pressure of ET. This phenomenon was often shown on the patients with nasal obstruction, and reduced ET mucociliary clearance. The Toynbee phenomenon indicates a closing failure of the ET, clinically, this situation meeds to be prevented and resolved in a timely manner.

采用声管测定法、声阻抗测定法和鼻咽压力测定法,研究在Toynbee(托氏)试验时中耳压力、咽鼓管(ET)开放时程和鼻咽压力时程三者间的关系;同时,测定鼻腔填塞前后鼓室压力的变化,并用糖精法测定鼓膜穿孔者于鼻腔填塞前后的粘膜纤毛运动情况。结果显示:托氏试验引起的鼓室负压与ET开放时程和鼻咽腔负压波时程的延长有关;这种负压可影响ET粘膜纤毛的活动性,使糖精排递时间延长。托氏现象反映出ET的关闭功能不良,它可能是一种病态现象,临床应及时预防和消除这种现象或由其诱发的中耳异常负压。

AIM: To study the protective effect of Gan_wei_ning (GWN) on gastric mucosa. METHODS: Rat experiment: 35 rats were randomly divided into A) saline, B) absolute EtOH, and C) GWN+EtOH. All rats were fasted for 24 h and deprivated of water for 2 h before the experiment. 1 mL of saline was given to Group A and Group B and 1 mL of oral solution of GWN to Group C. After 2 h, 1 mL of 0.9% sodium chloride was given to Group A and 1 mL of EtOH was given to Group B and C. Two hours later, their stomachs were taken out...

AIM: To study the protective effect of Gan_wei_ning (GWN) on gastric mucosa. METHODS: Rat experiment: 35 rats were randomly divided into A) saline, B) absolute EtOH, and C) GWN+EtOH. All rats were fasted for 24 h and deprivated of water for 2 h before the experiment. 1 mL of saline was given to Group A and Group B and 1 mL of oral solution of GWN to Group C. After 2 h, 1 mL of 0.9% sodium chloride was given to Group A and 1 mL of EtOH was given to Group B and C. Two hours later, their stomachs were taken out and the gastric mucosa was observed by macroscopy, optical microscope and electron microscope, and Na +_K +_ATPase of gastric mucosa, homogenate, gastric acid, endothelin (ET) and 6_keto prostaglandin F 1α in serum were determined. Clinical study: Patients with gastritis were treated with GWN 20 g, po, tid×8 wk. RESULTS: GWN enhanced the activity of Na +_K +_ATPase, increased PG level, and reduced ET level in serum, but did not affect the secretion of gastric acid. The gastric mucosal injury in Group C was obviously milder than that in Group A. CONCLUSION: GWN has protective effect on gastric mucosa.

目的 :探讨肝胃宁对胃粘膜的保护作用。方法 :大鼠 35只 ,♀♂不限 ,2 0 0~ 30 0 g ,随机分为正常对照组、无水乙醇对照组及肝胃宁预防组。分别以 0 .9%氯化钠溶液 1mL、0 .9%氯化钠溶液 1mL、肝胃宁 1mL灌胃 ,2h后再分别以 0 .9%氯化钠溶液 1mL、无水乙醇 1mL、无水乙醇 1mL灌胃。再过 2h后麻醉 ,抽血 ,处死取胃 ,进行大体、光镜、电镜、Na+_K+_ATPase活性、胃酸及血内皮素、前列腺素测定。临床治疗慢性胃炎 (糜烂型 ) ,以肝胃宁 2 0g ,tid× 8wk。结果 :肝胃宁能增强胃粘膜Na+_K+_ATPase活力 ,增加胃粘膜前列腺素及降低血内皮素 ,但不影响胃酸分泌。对大鼠胃粘膜损伤有保护作用。肝胃宁治疗慢性胃炎 (糜烂型 )取得较好疗效。结论 :肝胃宁有明显的胃粘膜保护作用

 
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