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reducing et
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  降低et
     Conclusion XNJI could effectively protect the brain from ischemia-reperfusion injury,the mechanism was related to its raising NO level and reducing ET or oxygen free radicals. [
     结论 XNJI对CIRI具有良好的防护作用,其机制与提高机体NO水平、降低ET及氧自由基水平有关。
短句来源
     Conclusion LGTI could effectively protect the brain form ischemia reperfusion injury,the mechanism was related to its raising NO level and reducing ET or oxygen free radicals.
     结论 LGTI对CIRI具有良好的防护作用 ,其机制与提高机体NO水平、降低ET水平及减轻氧自由基损伤等有关
短句来源
     Conclusions:XNJI can effectively protect the brain from ischemia and reperfusion injury by raising NO level and reducing ET level.
     结论 :XNJI可提高机体 NO水平、降低 ET水平而抗 CIRI
短句来源
     CONCLUSION Weixikang Decoction can promote the healing of gastric ulcer, probably by increasing NO, reducing ET content, promoting blood flow of the gastric mucosa and improving the microcirculation of the gastric mucosa.
     结论 胃喜康煎剂可能是通过增加NO、降低ET含量、增加胃黏膜的血流量、改善胃黏膜微循环 ,达到提高胃溃疡愈合质量的目的。
短句来源
     Conclusions:Lipo pro stagladin E_1 can inhibit the progress of atherosclerosis by reducing ET and ra ising NO.
     结论:前列地尔通过升高血清NO、降低ET抑制AS斑块形成。
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  “reducing et”译为未确定词的双语例句
     Results The Sal B could reduce the release of ET and TXB 2 and increase the content of PGI 2.Conclusions Sal B could relieve MIRI of rat probably by reducing ET of plasma, ameliorating the balance of TXA 2/PGI 2 system and protecting the injured myocardial cell.
     结果  Sal B可以减少 ET及 TXB2 的释放 ,提高 PGI2 的含量。 结论  Sal B可以减轻 MIRI,可能通过减少 ET的释放 ,改善 TXA2 / PGI2 系统的平衡状态 ,减轻心肌细胞的损伤。
短句来源
     The serum ET level of L - NAME group was significantly higher than that of contnd and L - Arg groups(P < 0. 01 ), It is suggested that L - Arg plays an important rule in reducing ET level;
     而L-NAME组血浆ET水平明显高于对照组及L-Arg组(P<0.01),与窒息组比较,无统计学意义。 提示:L-Arg能有效地降低窒息新生大鼠血浆ET的水平;
短句来源
     CIPC accomplished protecting function on ischemic cerebral tissue by reducing ET 1 in blood and cerebral tissue and NO in cerebral tissue, as well as rising NO in blood.
     CIPC模型组血、脑组织的ET 1及脑组织的NO含量降低 ,血浆NO含量升高。
短句来源
     Conclusion: ET 1 autocrine by PASMs regulates proloferation of themselves under nomoxia but not hypoxia. Hypoxia attenuates ET 1 synthesis and release by reducing ET 1 mRNA expression.
     结论 :ET 1参与常氧情况下PASM增殖的调节 ,而与缺氧引起的PASM过度增殖无关 ,缺氧可通过抑制PASM的ET 1mRNA的表达而抑制ET 1的合成和分泌。
短句来源
     Conclusion:The locally increased ET may contribute to the cardiovascular hypertrophy and losartan reverse cardiovascular hypertrophy by blocking AT receptor and reducing ET content in plasma and cardiovascular tissues.
     结论 :左室及主动脉组织中ET升高是造成左室及主动脉肥厚的重要因素 ,氯沙坦通过阻断受体和降低血浆及血管组织中的含量产生降压及逆转心血管肥厚的作用
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     et.
     et.
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     et C.
     et C.
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     Monascus could lower hypertension by reducing blood ET value.
     红曲有可能是通过降低L-NNA高血压模型大鼠血中ET水平而发挥降压作用的,但其作用机理有待进一步研究。
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     reducing supervision;
     减少监督程度 ,增加员工自我管理 ;
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     contrarily reducing.
     反之减少。
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Plasma endothelin (ET) was gradually increased and significantly higher than that of pre-shock period at 6h after hemorrhage/reperfusion (S/R) in rabbit,while sham group had no changes. Furthermore,ET was decreased in 2h after S/R injury by the use of phospholipase A2 inhibitor chloroquine,ME8101 and antioxidant (TFA) during shock period,and in particular,ME810l and TFA markedly reduced ET level within 30min after S/R,which was significantly lower than that of pre-shock period ( P <0. 01 )....

Plasma endothelin (ET) was gradually increased and significantly higher than that of pre-shock period at 6h after hemorrhage/reperfusion (S/R) in rabbit,while sham group had no changes. Furthermore,ET was decreased in 2h after S/R injury by the use of phospholipase A2 inhibitor chloroquine,ME8101 and antioxidant (TFA) during shock period,and in particular,ME810l and TFA markedly reduced ET level within 30min after S/R,which was significantly lower than that of pre-shock period ( P <0. 01 ). Meanwhile,plasma HCO3- and SBC increased at 24h after S/R by the administration of these agents, being significantly higher than that of S/R group ( P <0. 01 ). The results indicated that the increase of ET after S/R appears to be related with the advance of acidosis,and phospholipase A2 inhibitor and antioxidant could be used to block ET release,and to improve acidosis induced by the S/R injuries.

家兔出血性休克再灌注(S/R)后,放免分析发现血浆内皮素逐渐升高,同失血前比较第6h有显著差异(P<0.05),假手术组没有改变。磷脂酶A_2阻断剂氯喹、磷酸萘酚喹和抗氧化剂黄芪酮于失血后再灌注前使用可抑制S/R后2h内血浆内皮素增高,后两者可更明显降低S/R后30min的内皮索水平,与失血前比较差异非常好著(P<0.01)。同时,HCO~-_3和碱贮备(SBC)显著降低,上述三种药物治疗显著增加HCO~-_3和SBC的含量,它们在S/R后24h明显高于S/R组的水平(P<0.01)。结果提示,S/R后血浆内皮素上升可能与代谢性酸中毒加重有一定联系。磷脂酶A_2阻断剂和抗氧化剂可能通过早期抑制内皮素的释放,从而减轻24h后代谢性酸中毒程度。

This paper studied the relations between the middle ear pressure, the opening duration of eustachian tube (ET), and the duration of nasopharyngeal pressure phase during Toynbee maneuver by sonotubometry, tympanometry and nasopharynx manometry. The mucociliary clearance of ET in Toynbee maneuver was also studied by Saccharin method. The results showed that negative middle ear pressure induced by Toynbee maneuver was related to prolonged opening duration of ET and duration of negative pressure...

This paper studied the relations between the middle ear pressure, the opening duration of eustachian tube (ET), and the duration of nasopharyngeal pressure phase during Toynbee maneuver by sonotubometry, tympanometry and nasopharynx manometry. The mucociliary clearance of ET in Toynbee maneuver was also studied by Saccharin method. The results showed that negative middle ear pressure induced by Toynbee maneuver was related to prolonged opening duration of ET and duration of negative pressure in nasopharynx and lower opening pressure of ET. This phenomenon was often shown on the patients with nasal obstruction, and reduced ET mucociliary clearance. The Toynbee phenomenon indicates a closing failure of the ET, clinically, this situation meeds to be prevented and resolved in a timely manner.

采用声管测定法、声阻抗测定法和鼻咽压力测定法,研究在Toynbee(托氏)试验时中耳压力、咽鼓管(ET)开放时程和鼻咽压力时程三者间的关系;同时,测定鼻腔填塞前后鼓室压力的变化,并用糖精法测定鼓膜穿孔者于鼻腔填塞前后的粘膜纤毛运动情况。结果显示:托氏试验引起的鼓室负压与ET开放时程和鼻咽腔负压波时程的延长有关;这种负压可影响ET粘膜纤毛的活动性,使糖精排递时间延长。托氏现象反映出ET的关闭功能不良,它可能是一种病态现象,临床应及时预防和消除这种现象或由其诱发的中耳异常负压。

AIM: To study the protective effect of Gan_wei_ning (GWN) on gastric mucosa. METHODS: Rat experiment: 35 rats were randomly divided into A) saline, B) absolute EtOH, and C) GWN+EtOH. All rats were fasted for 24 h and deprivated of water for 2 h before the experiment. 1 mL of saline was given to Group A and Group B and 1 mL of oral solution of GWN to Group C. After 2 h, 1 mL of 0.9% sodium chloride was given to Group A and 1 mL of EtOH was given to Group B and C. Two hours later, their stomachs were taken out...

AIM: To study the protective effect of Gan_wei_ning (GWN) on gastric mucosa. METHODS: Rat experiment: 35 rats were randomly divided into A) saline, B) absolute EtOH, and C) GWN+EtOH. All rats were fasted for 24 h and deprivated of water for 2 h before the experiment. 1 mL of saline was given to Group A and Group B and 1 mL of oral solution of GWN to Group C. After 2 h, 1 mL of 0.9% sodium chloride was given to Group A and 1 mL of EtOH was given to Group B and C. Two hours later, their stomachs were taken out and the gastric mucosa was observed by macroscopy, optical microscope and electron microscope, and Na +_K +_ATPase of gastric mucosa, homogenate, gastric acid, endothelin (ET) and 6_keto prostaglandin F 1α in serum were determined. Clinical study: Patients with gastritis were treated with GWN 20 g, po, tid×8 wk. RESULTS: GWN enhanced the activity of Na +_K +_ATPase, increased PG level, and reduced ET level in serum, but did not affect the secretion of gastric acid. The gastric mucosal injury in Group C was obviously milder than that in Group A. CONCLUSION: GWN has protective effect on gastric mucosa.

目的 :探讨肝胃宁对胃粘膜的保护作用。方法 :大鼠 35只 ,♀♂不限 ,2 0 0~ 30 0 g ,随机分为正常对照组、无水乙醇对照组及肝胃宁预防组。分别以 0 .9%氯化钠溶液 1mL、0 .9%氯化钠溶液 1mL、肝胃宁 1mL灌胃 ,2h后再分别以 0 .9%氯化钠溶液 1mL、无水乙醇 1mL、无水乙醇 1mL灌胃。再过 2h后麻醉 ,抽血 ,处死取胃 ,进行大体、光镜、电镜、Na+_K+_ATPase活性、胃酸及血内皮素、前列腺素测定。临床治疗慢性胃炎 (糜烂型 ) ,以肝胃宁 2 0g ,tid× 8wk。结果 :肝胃宁能增强胃粘膜Na+_K+_ATPase活力 ,增加胃粘膜前列腺素及降低血内皮素 ,但不影响胃酸分泌。对大鼠胃粘膜损伤有保护作用。肝胃宁治疗慢性胃炎 (糜烂型 )取得较好疗效。结论 :肝胃宁有明显的胃粘膜保护作用

 
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