助手标题  
全文文献 工具书 数字 学术定义 翻译助手 学术趋势 更多
查询帮助
意见反馈
   carp retinal 的翻译结果: 查询用时:0.158秒
图标索引 在分类学科中查询
所有学科
生物学
基础医学
更多类别查询

图标索引 历史查询
 

carp retinal
相关语句
  鲫鱼视网膜
     Ca~(2+) Dynamics and Synaptic Plasticity in Carp Retinal Horizontal Cells
     鲫鱼视网膜水平细胞的Ca~(2+)动力学和突触可塑性
短句来源
     It has been shown previously that caffeine sensitive Ca 2+ stores gated by ryanodine receptors (RyRs) and the mechanism of Ca 2+ induced Ca 2+ release (CICR) exist in the soma of carp retinal ON type bipolar cells [1] .
     以前结果表明 ,ryanodine受体 (ryanodinereceptors ,RyRs)门控的咖啡因敏感的钙库和钙引钙释放(Ca2 + inducedCa2 +release ,CICR)机制存在于鲫鱼视网膜ON型双极细胞的胞体中[1] 。
短句来源
     Inhibition of calcium signaling in terminal and soma of carp retinal bipolar cells by GABA
     GABA抑制鲫鱼视网膜双极细胞的轴突末梢和胞体内的钙信号(英文)
短句来源
     GABA and Glycine Receptors on Carp Retinal Ganglion Cells
     鲫鱼视网膜神经节细胞上的γ-氨基丁酸和甘氨酸受体
短句来源
     METHODS: Freshly dissociated carp retinal cells were loaded with fluo-3AM and then the fluorescence measurements were performed on a confocal laser-scanning microscope.
     方法:急性分离的鲫鱼视网膜细胞经fluo-3AM孵育后,用激光共聚焦显微镜检测荧光变化。
短句来源
更多       
  相似匹配句对
     GABA and Glycine Receptors on Carp Retinal Ganglion Cells
     鲫鱼视网膜神经节细胞上的γ-氨基丁酸和甘氨酸受体
短句来源
     CARP PRINCESS
     鲤鱼公主
短句来源
     CHANGES IN RETINAL SENSITIVITY OF SILVER CARP DURING LIGHT ADAPTATION
     鲫鱼(Carassius auratus)视网膜敏感度在明适应过程中的变化
短句来源
     Retinal Vasculitis
     视网膜血管炎
短句来源
     Retinal Detachment
     视网膜脱离
短句来源
查询“carp retinal”译词为用户自定义的双语例句

    我想查看译文中含有:的双语例句
例句
为了更好的帮助您理解掌握查询词或其译词在地道英语中的实际用法,我们为您准备了出自英文原文的大量英语例句,供您参考。
  carp retinal
The synaptic contacts made by carp retinal neurons were studied with electron microscopic techniques.
      
Differential modulation by AMPA of signals from red- and green-sensitive cones in carp retinal luminosity-type horizontal cells
      
However, in the carp retinal slice, other synaptic interactions are possible.
      


To investigate the effect of activation of y-aminobutyric acid (GABA) receptors on high K+-evoked Ca2+ signaling in the terminal and soma of carp retinal ON-type bipolar cells. METHODS: Freshly dissociated carp retinal cells were loaded with fluo-3AM and then the fluorescence measurements were performed on a confocal laser-scanning microscope. RESULTS: Ca2+ signaling evoked by high K+ 35 mmol/L was completely suppressed in both the terminal and soma of bipolar cells by GABA 100μmol/L. However, different...

To investigate the effect of activation of y-aminobutyric acid (GABA) receptors on high K+-evoked Ca2+ signaling in the terminal and soma of carp retinal ON-type bipolar cells. METHODS: Freshly dissociated carp retinal cells were loaded with fluo-3AM and then the fluorescence measurements were performed on a confocal laser-scanning microscope. RESULTS: Ca2+ signaling evoked by high K+ 35 mmol/L was completely suppressed in both the terminal and soma of bipolar cells by GABA 100μmol/L. However, different results were found in the terminal and soma when only one subtype of GABA receptors was activated. While activation of either GABAA or GABAc receptors totally suppressed Ca2+ signaling in the soma, a gradual elevation of [Ca2+]i appeared in the terminal. GABA 10 μmol/L could also completely suppress Ca2+ signaling in the soma, but could only partially reduce Ca2+ signaling in the terminal. CONCLUSION: Activation of both GABAA and GABAc receptors could completely inhibit high K+-evoked Ca2+ signaling in the terminal and soma of carp retinal ON-type bipolar cells. While activation of either GABAA or GABAc receptors alone still totally suppressed Ca2+ signaling in the soma, a gradual elevation of [Ca2+]i appeared in the terminal, which may be due to desensitization of GABA receptors.

目的:研究GABA受体的激活对于鲫鱼视网膜ON-型双极细胞的胞体和轴突末梢内高钾引起的钙信号的影响。方法:急性分离的鲫鱼视网膜细胞经fluo-3AM孵育后,用激光共聚焦显微镜检测荧光变化。结果:GABA 100μmol/L可以完全抑制双极细胞胞体和轴突末梢内高钾35mmol/L引起的钙信号。仅激活GABA受体的一种亚型时,轴突末梢和胞体内出现了不同的结果。当GABA_A受体或GABA_C受体被单独激活时,尽管胞体内的钙信号仍旧被完全抑制,但轴突末梢的[Ca~(2+)]_i逐渐升高。GABA 10μmol/L仍旧可以完全抑制胞体内的钙信号且部分抑制轴突末梢内的钙信号。结论:激活GABA_A和GABA_C受体能够完全抑制鲫鱼视网膜ON-型双极细胞内高钾引起的钙信号。仅激活GABA_A或GABA_C受体时可以完全抑制胞体内的钙信号,而轴突末梢的[Ca~(2+)]_i逐渐升高,这可能与GABA_A或GABA_C受体的脱敏有关。

It has been shown previously that caffeine sensitive Ca 2+ stores gated by ryanodine receptors (RyRs) and the mechanism of Ca 2+ induced Ca 2+ release (CICR) exist in the soma of carp retinal ON type bipolar cells [1] . Using immunocytochemistry of RyRs and intracellular Ca 2+ measurement, we further investigated whether RyR gated Ca 2+ stores are present in the terminal of these cells. Immunocytochemical study of both vertical section and dissociated cells of the retina...

It has been shown previously that caffeine sensitive Ca 2+ stores gated by ryanodine receptors (RyRs) and the mechanism of Ca 2+ induced Ca 2+ release (CICR) exist in the soma of carp retinal ON type bipolar cells [1] . Using immunocytochemistry of RyRs and intracellular Ca 2+ measurement, we further investigated whether RyR gated Ca 2+ stores are present in the terminal of these cells. Immunocytochemical study of both vertical section and dissociated cells of the retina indicates that RyRs were mainly localized in the soma of ON type bipolar cells. Caffeine up to 40 mmol/L could not induce Ca 2+ signal in the terminal. After resting [Ca 2+ ] i was slightly increased by raising external K + to 10 mmol/L, no Ca 2+ signal was induced by caffeine in the terminal. In the presence of forskolin or dopamine, by which intracellular cAMP and in turn cAMP dependent phosphorylation are enhanced, caffeine also failed to bring Ca 2+ signal in the terminal. In addition, 50 μmol/L ryanodine did not show any effect on Ca 2+ signal evoked by 65 mmol/L K + for 1 min or 2 min in the terminal. Taken together, it is indicated that RyR gated caffeine sensitive Ca 2+ stores and CICR are absent in the terminal of carp retinal ON type bipolar cells.

以前结果表明 ,ryanodine受体 (ryanodinereceptors ,RyRs)门控的咖啡因敏感的钙库和钙引钙释放(Ca2 + inducedCa2 +release ,CICR)机制存在于鲫鱼视网膜ON型双极细胞的胞体中[1] 。采用RyRs的免疫细胞化学方法和细胞内钙测量技术 ,我们进一步研究了RyRs门控的钙库是否存在于这些细胞的轴突末梢中。视网膜纵切和分离细胞的免疫细胞化学研究显示 ,RyRs主要位于ON型双极细胞的胞体中。咖啡因浓度升至 4 0mmol/L在轴突末梢不能诱导钙信号。在细胞外K+浓度升至 10mmol/L引起静息 [Ca2 +]i 轻微升高后 ,咖啡因在轴突末梢也不能诱导钙信号。在forskolin或多巴胺引起细胞内cAMP浓度升高 ,进而cAMP依赖的磷酸化增强后 ,咖啡因在轴突末梢仍不能诱导钙信号。此外 ,50 μmol/Lryanodine对 65mmol/LK+作用 1min或 2min诱导的轴突末梢的钙信号没有产生任何效应。这些结果表明 ,在鲫鱼视网膜ON型双极细胞的轴突末梢中不存在RyRs门控的咖啡因敏感的钙库和CICR机制

 
图标索引 相关查询

 


 
CNKI小工具
在英文学术搜索中查有关carp retinal的内容
在知识搜索中查有关carp retinal的内容
在数字搜索中查有关carp retinal的内容
在概念知识元中查有关carp retinal的内容
在学术趋势中查有关carp retinal的内容
 
 

CNKI主页设CNKI翻译助手为主页 | 收藏CNKI翻译助手 | 广告服务 | 英文学术搜索
版权图标  2008 CNKI-中国知网
京ICP证040431号 互联网出版许可证 新出网证(京)字008号
北京市公安局海淀分局 备案号:110 1081725
版权图标 2008中国知网(cnki) 中国学术期刊(光盘版)电子杂志社