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family carcinoma history
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  癌家族史
     There are two factors associated with P16 abnormal expression by using single factor analysis. They are follows:eat fat meat(OR=2.0481),family carcinoma history(OR=13.8092);
     单因素分析结果 ,有两个因素与 P16异常表达有联系 ,分别是好吃肥肉 (OR=2 .0 4 81) ,癌家族史 (OR=13.80 92 ) ;
短句来源
     The stepwise logistic regression analysis shows that the years of smoking(OR=1.060),eat fat meat(OR=3.536),family carcinoma history(OR=37.0815) are the dangerous factors.
     多因素非条件 logistic回归分析 ,有 3个因素进入回归模型 ,分别是吸烟年数 (OR=1.0 6 0 ) ,好吃肥肉 (OR=3.5 36 ) ,癌家族史 (OR=37.815 ) ;
短句来源
     Conclusions:In the study,we can conclude that the years of smoking,eat fat meat and family carcinoma history are the risk factors of P16 abnormal expression in CRC.
     结论 :在暴露于危险因素至癌变过程中 ,P16基因表达缺失可能与吸烟年数、好吃肥肉、癌家族史危险因素有关。
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  “family carcinoma history”译为未确定词的双语例句
     The risk factors of local lung cancer included smoking,air pullution,indoor pollution,pulmonary disease history,family carcinoma history,long term depression. Regular physical exercise was one of the protective factors.
     该地及肺癌的危险因素包括吸烟、大气污染、室内污染、肺部病史、家族肿瘤史及长期精神压抑 ,而体育锻炼是保护因素。
短句来源
  相似匹配句对
     Conclusion There was family assemblage in esophageal carcinoma obviously.
     结论食管癌具有明显的家庭集聚性;
短句来源
     C-1 family.
     1株未扩增出目的条带.
     the family is dispersed.
     家族被分散。
短句来源
     Conclusion:Overexpression and co-overexpression of EGFR family were common in esophageal carcinoma.
     结论 :食管癌中存在EGFR家族的过度表达及联合过度表达。
短句来源
     Objective To evaluate the risk factors of family history of nasopharyngeal carcinoma in south of China.
     目的综合评价鼻咽癌家族史在中国南方地区鼻咽癌发病中的作用。
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Objective:To investigate the prevention strategy of local lung cancer.Methods:The prevention basis for local lung cancer was obtained by comprehensive analysis of the results of two previous surveys about the epidemiological characteristics and risk factors of the local lung cancer.Results:Both the incidence rate and mortality rate of Rongqi town were higher than those of national average levels.Lung cancer accounted for 25.3% local death due to malignance.The risk factors of local lung cancer included smoking,air...

Objective:To investigate the prevention strategy of local lung cancer.Methods:The prevention basis for local lung cancer was obtained by comprehensive analysis of the results of two previous surveys about the epidemiological characteristics and risk factors of the local lung cancer.Results:Both the incidence rate and mortality rate of Rongqi town were higher than those of national average levels.Lung cancer accounted for 25.3% local death due to malignance.The risk factors of local lung cancer included smoking,air pullution,indoor pollution,pulmonary disease history,family carcinoma history,long term depression.Regular physical exercise was one of the protective factors.The mainly chief complains of local lung cancer included chronic cough,chest pain,blood-streaked sputum,short of breath,loss of weight and physical examination.Up to 31.3% young patients(less than 40) were found to suffer from lung cancer by regular physical examination.Conclusion:Comprehensive measures must be taken to better prevent the local lung cancer.

目的 :提出顺德容奇地区肺癌预防策略。方法 :通过对两项调查研究结果进行综合分析 ,找出本地区肺癌防治依据。结果 :容奇地区肺癌发病率及死亡率均高于全国平均水平 ,占当地恶性肿瘤死亡原因的 2 5 .3%。该地及肺癌的危险因素包括吸烟、大气污染、室内污染、肺部病史、家族肿瘤史及长期精神压抑 ,而体育锻炼是保护因素。该地区肺癌患者主要主诉包括慢性咳嗽、胸痛、咳血丝痰或咯血、气促、消瘦和体检。而年轻人因体检而发现肺癌者比例高达 31.3%。结论 :应采取综合措施进行肺癌的三级预防

Objective:To explore the risk factors of P16 protein abnormal expression in colorectal cancer(CRC).Methods:Big four hospitals' CRC patients in Shanxi province were chosen as sample cases.The tumor tissues were detected by s-p immunohistochemistry.The correspond patients wrere divided into P16 abnormal expression positive group and negative group.The case-case study and SAS software were used for factor analysis.Results:The P16 abnormal expression,that is the inactivation of P16 gene expression rate was 51.8%(57/110),it...

Objective:To explore the risk factors of P16 protein abnormal expression in colorectal cancer(CRC).Methods:Big four hospitals' CRC patients in Shanxi province were chosen as sample cases.The tumor tissues were detected by s-p immunohistochemistry.The correspond patients wrere divided into P16 abnormal expression positive group and negative group.The case-case study and SAS software were used for factor analysis.Results:The P16 abnormal expression,that is the inactivation of P16 gene expression rate was 51.8%(57/110),it is positive group.There are two factors associated with P16 abnormal expression by using single factor analysis.They are follows:eat fat meat(OR=2.0481),family carcinoma history(OR=13.8092);The stepwise logistic regression analysis shows that the years of smoking(OR=1.060),eat fat meat(OR=3.536),family carcinoma history(OR=37.0815) are the dangerous factors.Then we analysed the dose-response relatonship of the years of smoking and the number of family carcinoma history person with P16 abnormal expression respectively.The results shows that the number of family carcinoma person and P16 abmormal expression has significance.The years of smoking has no significance,but the OR value of over forty years is far higer than the low years.Conclusions:In the study,we can conclude that the years of smoking,eat fat meat and family carcinoma history are the risk factors of P16 abnormal expression in CRC.

目的 :探讨大肠癌 P16基因异常表达的危险因素。方法 :用免疫组化方法 ,将从山西省 4所综合性医院随机选取 110例原发性大肠癌手术病人 ,病例划分为 P16缺失表达组 (阳性组 )和未缺失表达组 (阴性组 )。应用病例 -病例研究方法及 SAS软件包进行因素分析。结果 :P16基因表达缺失率为 5 1.8% (5 7/ 110 ) ;单因素分析结果 ,有两个因素与 P16异常表达有联系 ,分别是好吃肥肉 (OR=2 .0 4 81) ,癌家族史 (OR=13.80 92 ) ;多因素非条件 logistic回归分析 ,有 3个因素进入回归模型 ,分别是吸烟年数 (OR=1.0 6 0 ) ,好吃肥肉 (OR=3.5 36 ) ,癌家族史 (OR=37.815 ) ;趋势 χ2检验发现吸烟年数与 P16基因表达缺失无明显剂量反应关系 ,但吸烟 4 0年以上组的 OR值是吸烟 2 0年以下组的 2 .9倍 ,家族患肿瘤人数与 P16基因表达缺失有明显剂量反应关系 (χ2 =15 3.38,P<0 .0 0 1)。结论 :在暴露于危险因素至癌变过程中 ,P16基因表达缺失可能与吸烟年数、好吃肥肉、癌家族史危险...

目的 :探讨大肠癌 P16基因异常表达的危险因素。方法 :用免疫组化方法 ,将从山西省 4所综合性医院随机选取 110例原发性大肠癌手术病人 ,病例划分为 P16缺失表达组 (阳性组 )和未缺失表达组 (阴性组 )。应用病例 -病例研究方法及 SAS软件包进行因素分析。结果 :P16基因表达缺失率为 5 1.8% (5 7/ 110 ) ;单因素分析结果 ,有两个因素与 P16异常表达有联系 ,分别是好吃肥肉 (OR=2 .0 4 81) ,癌家族史 (OR=13.80 92 ) ;多因素非条件 logistic回归分析 ,有 3个因素进入回归模型 ,分别是吸烟年数 (OR=1.0 6 0 ) ,好吃肥肉 (OR=3.5 36 ) ,癌家族史 (OR=37.815 ) ;趋势 χ2检验发现吸烟年数与 P16基因表达缺失无明显剂量反应关系 ,但吸烟 4 0年以上组的 OR值是吸烟 2 0年以下组的 2 .9倍 ,家族患肿瘤人数与 P16基因表达缺失有明显剂量反应关系 (χ2 =15 3.38,P<0 .0 0 1)。结论 :在暴露于危险因素至癌变过程中 ,P16基因表达缺失可能与吸烟年数、好吃肥肉、癌家族史危险因素有关。

 
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