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nerve syndrome
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  神经综合征
    Clinical and electromyographical study of patients with posterior interosseous nerve syndrome
    骨间后神经综合征的临床及肌电图研究
短句来源
    RESULTS:After cauda equina nerve syndrome occurred for 1/2 day,there was isch emia and edema in two sides of posterior root ganglion and cellular necrosis of neurons in ganglion.
    结果:临床出现马尾神经综合征1/2d,将导致双侧后根节缺血水肿,节内神经元细胞坏死。
短句来源
    Posterior root g anglion was removed from the model with cauda equina nerve syndrome at different time point,and stained with HE. The number of normal sensory neuron cells were c ounted.
    取不同时间段的马尾神经综合征的模型的神经根的后根节,作HE染色对其内正常感觉神经元细胞记数。
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  nerve syndrome
Eighty percent of the patients had multiple neuraxis syndrome, and the combination of brain plus cranial nerve syndrome was the most common manifestation.
      
Anterior interosseous nerve syndrome secondary to forearm thrombo-phlebitis
      
Anterior interosseous nerve syndrome with atypical nerve course in relation to the pronator teres
      
A total of 1200 cases of nerve compression were operated on in the period of 1974-1981 (72% CCS, 13% Guyon, 11% sulcus of the ulner nerve syndrome, 3.25% supinator, and 0.75% pronator syndrome).
      
The Carpal canal syndrome, the sulcus of the ulnar nerve syndrome, Guyon's syndrome, the supinator syndrome, and the pronator syndrome are documentated by slides.
      
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Objective: To study the possible methods to improve microvascular decompression and to enhance the therapeutic effects in the treatment of cranial nerve syndromes as trigeminal neuralgia and glos-sopharyngeal neuralgia by vascular compression. Methods: A systematic review was made for 1150 cases ( 1120 of trigeminal neuralgia and 30 of glossopharyngeal neuralgia) on whom we applied microvascular decompression. Results: The grand number of cases with satisfactory effects was 1112 with an effective rate...

Objective: To study the possible methods to improve microvascular decompression and to enhance the therapeutic effects in the treatment of cranial nerve syndromes as trigeminal neuralgia and glos-sopharyngeal neuralgia by vascular compression. Methods: A systematic review was made for 1150 cases ( 1120 of trigeminal neuralgia and 30 of glossopharyngeal neuralgia) on whom we applied microvascular decompression. Results: The grand number of cases with satisfactory effects was 1112 with an effective rate of 96. 7% ,and no case died in this group. The complication rate dropped from 5. 6% of 5 years ago to 1. 6% of the recent 5 years. Conclusion: Various factors need to be comsidered in order to enhance the cure rate and to decrease the possible complications of microvascular decompression.

目的:探讨用微血管减压术治疗三叉神经痛、舌咽神经痛等神经血管压迫综合症治疗方法的改进措施和提高治疗效果的临床经验。方法:系统回顾1984年6月至1999年12月我们采用微血管减压术治疗神经血管压迫综合症病例1150例,其中三叉神经痛1120例,舌咽神经痛30例。结果:有效1112例,有效率为96.7%,本组无死亡。并发症发生率由5年前5.6%下降到近5年的1.6%。结论:提高微血管减压术的治愈率和减少并发症,有多方面因素值得考虑。

AIM:To study the pathological mechanism of sensory disturbance caused by cauda equina nerve damage.METHODS:Fifty pure breed healthy male New Zealand rabbits were devided into th ree groups randomly: non-treatment group(10 rabbits);non-compression control g roup(10 rabbits) and trial group(30 rabbits).At the following time points(1/4,1/ 2,3,7 and 15 days).According to the of the compressor screw the sagittal diamete r of vertebral canal,the model group was divided into 1/9,2/9,1/2.The compressio n devices were...

AIM:To study the pathological mechanism of sensory disturbance caused by cauda equina nerve damage.METHODS:Fifty pure breed healthy male New Zealand rabbits were devided into th ree groups randomly: non-treatment group(10 rabbits);non-compression control g roup(10 rabbits) and trial group(30 rabbits).At the following time points(1/4,1/ 2,3,7 and 15 days).According to the of the compressor screw the sagittal diamete r of vertebral canal,the model group was divided into 1/9,2/9,1/2.The compressio n devices were fixed between S2 and S3 to compress cauda equina.Posterior root g anglion was removed from the model with cauda equina nerve syndrome at different time point,and stained with HE.The number of normal sensory neuron cells were c ounted.RESULTS:After cauda equina nerve syndrome occurred for 1/2 day,there was isch emia and edema in two sides of posterior root ganglion and cellular necrosis of neurons in ganglion.The number of normal cells in posterior root ganglion was 3 and 7.The left in unoperation control group at time point of 30d was 32.2±4.2,3 2.2±4.3,32.2±4.3/mm2; The left in uncomprssion operation group was 32.2±2.3,3 2.2±4.3,32.2±3.1/mm2;The left in experiment group was 17.2±3.3,14.2±3.2,14. 1±2.3/ mm2.There was significant difference between groups(P< 0.05).CONCLUSION:Sensory neurons in posterior root ganglion are very sensitive and e asily suffer necrosis,which is one of the important pathological changes of dire ct degeneration in cauda equina nerve damage and also explain why the numbness o f sella turcica region and sensory disturbance are hard to recover.

目的:初步探讨马尾神经损害导致马尾神经综合征,引以感觉障碍的原因。方法:纯种健康雄性新西兰兔40只,按随机数字表法分为3组,非手术对照组10只,手术不加压对照组10只,模型组30只;模型组再根据加压螺丝进入椎管矢状径的深度分为进入1/9,2/9,1/2。模型组加压装置置于S2~3压迫马尾神经组。取不同时间段的马尾神经综合征的模型的神经根的后根节,作HE染色对其内正常感觉神经元细胞记数。结果:临床出现马尾神经综合征1/2d,将导致双侧后根节缺血水肿,节内神经元细胞坏死。后根节的正常细胞记数:3,7,30d时非手术对照组左侧为(32.2±4.2),(32.2±4.3),(32.2±4.3)/mm2;手术不加压对照组左侧为(32.2±2.3),(32.2±4.3),(32.2±3.1)/mm2;实验组左侧为(17.2±3.3),(14.2±3.2),(14.1±2.3)/mm2;差异有显著性意义(P<0.05)。结论:背根结内的感觉神经元极敏感,极易坏死,是马尾神经损害顺行溃变的重要病理变化之一,是导致鞍区麻木、感觉障碍,难以恢复的重要原因。

 
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