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deficit virus
相关语句
  缺陷性病毒
     Expression of LacZ was detected by X gal staining. Necrosis and apoptosis of neural stem cells induced by 6 hydroxy dopamine at dose of 0.02、0.04、0.06 g/L respectively, with or without the protection of recombinant republic deficit virus with NURR1 gene, were detected by flow cytometry.
     【方法】免疫组化检测神经干细胞特异性抗原nestin,X-gal染色检测LacZ基因的表达,流式细胞仪检测剂量为0.02、0.04、0.06g/L6-羟基多巴在添加含有NURR1基因的重组复制缺陷性病毒前后诱发神经干细胞的坏死和凋亡。
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  相似匹配句对
     release of the virus.
     组装病毒病毒颗粒;
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     sendai virus
     仙台病毒
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     (4)with deficit.
     (4)亏损。
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     Refraction of Trade Deficit
     10.27亿美元贸易逆差的折射
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To identify the characteristics of C17.2 neural stem cells (NSC) and to investigate the toxicity of 6 hydroxydopamine and the protective effect of NURR1 gene. Specific nestin expression in nerve stem cell was detected by immunohistochemistry. Expression of LacZ was detected by X gal staining. Necrosis and apoptosis of neural stem cells induced by 6 hydroxy dopamine at dose of 0.02、0.04、0.06 g/L respectively, with or without the protection of recombinant republic deficit virus with NURR1 gene, were...

To identify the characteristics of C17.2 neural stem cells (NSC) and to investigate the toxicity of 6 hydroxydopamine and the protective effect of NURR1 gene. Specific nestin expression in nerve stem cell was detected by immunohistochemistry. Expression of LacZ was detected by X gal staining. Necrosis and apoptosis of neural stem cells induced by 6 hydroxy dopamine at dose of 0.02、0.04、0.06 g/L respectively, with or without the protection of recombinant republic deficit virus with NURR1 gene, were detected by flow cytometry.C17.2 NSC expressed nestin antigen and mostly were stained by X gal. 0.02 g/L 6 hydroxydopamine mainly induced apoptosis of NSC and 0.06g/L dose mostly induced necrosis. The apoptosis and necrosis of C17.2 NSC were decreased by adding NURR1 gene. [Conclusion]Necrosis or apoptosis of NSC induced by 6 hydroxydopamine is closely related to its dose. Overexpression of NURR1 shows protective effect on C17.2 cells.

【目的】初步鉴定C17.2神经干细胞的特性并研究不同浓度6-羟基多巴对C17.2神经干细胞毒性及NURR1基因对C17.2神经干细胞的保护作用。【方法】免疫组化检测神经干细胞特异性抗原nestin,X-gal染色检测LacZ基因的表达,流式细胞仪检测剂量为0.02、0.04、0.06g/L6-羟基多巴在添加含有NURR1基因的重组复制缺陷性病毒前后诱发神经干细胞的坏死和凋亡。【结果】C17.2神经干细胞nestin抗原染色阳性,绝大部分细胞呈X-gal染色阳性,0.02g/L时6-羟基多巴以诱导神经干细胞凋亡为主,随着剂量增加,细胞坏死增多,0.06g/L时则以神经干细胞的坏死为主,NURR1基因过表达能明显减少神经干细胞的坏死和凋亡。【结论】C17.2神经干细胞具有明显的特性,6-羟基多巴诱发的C17.2神经干细胞凋亡或坏死与其剂量有关,NURR1基因对神经干细胞有保护作用。

 
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