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inner retinal
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  内层视网膜
     2. Mean thickness of the inner retinal layers: (l)MTIRL in normal retina was (75.25+3.82) urn.
     2.内层视网膜平均厚度:门)正常组为 OS.25士 3.82户 m。
短句来源
     Ultrastructure observation of cell death during inner retinal ischemia-reperfusion in rat
     大鼠内层视网膜缺血-再灌注过程中细胞死亡的电镜研究
短句来源
     Electron microscopic study of cell death in rat inner retinal ischemia-reperfusion
     大鼠内层视网膜缺血-再灌注过程中细胞死亡的电镜研究(英文)
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  “inner retinal”译为未确定词的双语例句
     ResultsNumerous ED-1-positive macrophages were seen in the vitreous and along the inner retinal surface in experimental group. Retinal ganglion cells(RGCs) showed an intensive upregulation of GAP-43 in their somata in experimental group C(189.26±26.16) versus group B(65.29±21.32) (P﹤0.05),and no GAP-43 expression in the control group.
     结果对照组中未见ED1阳性巨噬细胞,实验组中视网膜内表面见ED1阳性巨噬细胞,C组GAP43较多(189.26±26.16),与B组(65.29±21.32)相比有显著差异,而对照组中无GAP43的表达。
短句来源
     After ischemia for 30min and reperfusion, c-Fos positive neurons increased promptly at 1 h in the inner retinal neurons, peaked at 2h, decreased gradually after 4 h and returned towards normal level 24h thereafter.
     视网膜内层c-Fos阳性神经元数在视网膜缺血再灌注1h明显增多,2h达高峰,4h后逐渐下降,24h恢复正常水平。
短句来源
     The number of retinal ganglion cells and the thickness of the inner retinal layers were measured by HE staining method 1 hour and 1 day,3 days,7 days,10 days,14 days and 28 days after the establishment of HRD and RD,respectively.
     分别在手术后1h、1d、3d、7d、10d、14d、28d对视网膜进行常规HE染色切片,观察细胞丢失情况及测量视网膜神经感觉层厚度。
短句来源
     Results In group A,retinal histological section examination showed that inner retinal layer became significantly thinner after ischemia in right eyes(P<0.05).
     结果缺血后,A组右眼视网膜内层各层的厚度明显变薄(P<0.05)。
短句来源
     Ten microliters (0.1 μg/μl) PEDF was injected into the vitreous of 4 eyes of each group immediately after reperfusion and 4 additional eyes received only normal saline as vehicle controls. The animals were euthanized at 2 or 7 days after reperfusion. The effect of PEDF on retinal degeneration was assessed by measuring the thickness of the inner retinal layers (MTIRL) and counting the retinal ganglion cells (RGC) on plastic embedded retinal sections.
     随后立即向玻璃体注射 10 μ1(0 .1μg/ μl) PEDF,分别于 2 d和 7d摘除眼球 ,测量塑料包埋切片的平均视网膜内层厚度 (mean thicknessof inner retinal layer,MTIRL)和视网膜节细胞 (retinal ganglion cells,RGC)计数。
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  相似匹配句对
     Astrocytes and inner blood-retinal barrier
     星形胶质细胞与血-视网膜屏障
短句来源
     It is inner randomicity.
     这种现象是由系统完全确定的,是一种内随机性。
短句来源
     Retinal Detachment
     视网膜脱离
短句来源
     Retinal Vasculitis
     视网膜血管炎
短句来源
     The experimental study of glutamate neurotoxicity on inner stratum retinal neurons
     谷氨酸对视网膜内层神经细胞影响的研究
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  inner retinal
Small type 2 neurons were located in the inner retinal layer.
      
The necrosis was extensive in the inner retinal layers but focal in the outer retinal layers.
      
Outer and inner retinal pathology (division based on vascular support) do not cause a comparable fall off in resolution in time.
      
We conclude that in young BD patients during extremely prolonged DA there is a significant additional capacity of recovery of rod function and also significant gain of activity in the inner retinal layer.
      
Purpose: Standard ERG a-waves represent contributions from both photoreceptor and inner retinal cells, while the leading edge of the high-intensity a-wave is produced only by photoreceptors.
      
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Photic retinal damage and its medicinal protection: a histochemical quantitative analysis. Chen Wei-heng et al. Department of Ophthalmology, Third Teaching Hospital, Beijing Medical University, Beijing 100083 Eyes of albino rats were exposed to fluorescent illumination of 18 175 Ix for 1 or 2 hours for observation of ultrastructural changes, and a histochemical quantitative analysis was made of the dynamic changes in cytochrome oxidase(CyO) and acetylcholinesterase(AChE) in the retina. It was found that(1) CyO...

Photic retinal damage and its medicinal protection: a histochemical quantitative analysis. Chen Wei-heng et al. Department of Ophthalmology, Third Teaching Hospital, Beijing Medical University, Beijing 100083 Eyes of albino rats were exposed to fluorescent illumination of 18 175 Ix for 1 or 2 hours for observation of ultrastructural changes, and a histochemical quantitative analysis was made of the dynamic changes in cytochrome oxidase(CyO) and acetylcholinesterase(AChE) in the retina. It was found that(1) CyO de- creased significantly in 4 hours to 8 days after 2 hours of light exposure in all rats and AChE in the inner retinal layers decreased mildly and transiently, the results being consistent with the histopathologic damages; (2) the retinas of 1 hour exposure showed less changes than those of 2 hours exposure in cyo, and normal morphology was restored in 8 days; (3) the cyo decrease occurred earlier than the morphologic damages; (4) the photic damage to retinal CyO was ameliorated by vitamine E and betacarotene.

用荧光灯照射大鼠视网膜,应用组织化学微机图像处理方法,定量分析光损伤后视网膜细胞色素氧化酶和乙酰胆碱酯酶的动态改变,观察维生素E和β胡萝卜素对细胞色素氧化酶的防护效果,通过超微结构观察进行比较。结果:光照后细胞色素氧化酶活性下降,并与光照时间有关,提示光损伤可使此酶活性在一定范围内降低或恢复。就恢复时间而言,此酶活性改变与超微结构改变相符。乙酰胆碱脂酶仅呈一过性改变,部位也与超微结构改变吻合。与超微结构的改变相比,组化改变早而敏感。维生素E和β胡萝卜素对细胞色素氧化酶有较好的保护作用。

Purpose [WT5”BZ]To estabalish a quantifying model of retinal neovascularization suitable for the study of pathogenesis and therapeutic intervention for the retinal neovascularization. [WT5”HZ]Methods [WT5”BZ]Sixteen one week old C 57 BL/6 mice were exposed to 75% oxygen for 5 days and then to room air and 16 mice of the same age kept in room air as controls.Ink perfused retinal flatmount was examined to assess the oxygen induced changes of retinal vessels.The proliferated neovascular response was...

Purpose [WT5”BZ]To estabalish a quantifying model of retinal neovascularization suitable for the study of pathogenesis and therapeutic intervention for the retinal neovascularization. [WT5”HZ]Methods [WT5”BZ]Sixteen one week old C 57 BL/6 mice were exposed to 75% oxygen for 5 days and then to room air and 16 mice of the same age kept in room air as controls.Ink perfused retinal flatmount was examined to assess the oxygen induced changes of retinal vessels.The proliferated neovascular response was quantitated by counting the nuclei of endothelial cells of new vessels extending from the retina into the vitreous in 6 μm sagittal cross sections. VEGF and bFGF were determined on the cross sections after immunohistochemcal stain. [WT5”HZ]Results [WT5”BZ]Constriction and closure of the blood vessels were found under the hyperoxia condition,and dilation and proliferation were found under the relatively hypoxia status.There was a mean of 24 neovascular nuclei per cross section in the oxygen treated retina and less than 1 nucleus in the control group ([WT5”BX]P<0.001).VEGF stain was found stronger in the inner retinal layer of oxygen treated mouse than in that of the controls. [WT5”HZ]Conclusion [WT5”BZ]The quantifying model of retinal neovascularization may fascilitate the further researches of medical intervention and pathogenesis of retinal neovacularization. [WT5”HZ]

目的 建立可对视网膜新生血管进行定量研究的小鼠模型 ,用于视网膜新生血管发生机制的探讨及治疗方法的评价。 方法 给氧箱组 16只 1周龄 C5 7BL / 6小鼠在浓度为 75 %的氧箱中生活 5 d,然后回到正常氧环境中 ;16只同龄小鼠生活在正常氧环境中做对照。墨汁灌注视网膜铺片了解氧所导致的视网膜血管改变 ;计数矢状面 6 μm视网膜切片中突破内界膜的血管内皮细胞 ,反映视网膜血管的增生 ;视网膜切片还做了血管内皮细胞生长因子 (vascular endothelial growth factor,VEGF)和碱性成纤维细胞生长因子 (basic fibroblastgrowth factor,b FGF)免疫组织化学染色。 结果 高氧促视网膜血管收缩、闭塞 ,相对低氧致视网膜血管扩张、增生。突破视网膜内界膜的血管内皮细胞数 ,给氧组平均 2 4个 /切面 ,对照组平均<1个 /切面 ,二者相比差异有显著性的意义 (P<0 .0 0 1)。与对照组相比 ,给氧组小鼠视网膜内层 VEGF的表达明显增强。 结论 该小鼠模型中视网膜新生血管的发生与缺氧相关 ,并可对其定量观察 ,是进行视网膜新生血管发生机制及...

目的 建立可对视网膜新生血管进行定量研究的小鼠模型 ,用于视网膜新生血管发生机制的探讨及治疗方法的评价。 方法 给氧箱组 16只 1周龄 C5 7BL / 6小鼠在浓度为 75 %的氧箱中生活 5 d,然后回到正常氧环境中 ;16只同龄小鼠生活在正常氧环境中做对照。墨汁灌注视网膜铺片了解氧所导致的视网膜血管改变 ;计数矢状面 6 μm视网膜切片中突破内界膜的血管内皮细胞 ,反映视网膜血管的增生 ;视网膜切片还做了血管内皮细胞生长因子 (vascular endothelial growth factor,VEGF)和碱性成纤维细胞生长因子 (basic fibroblastgrowth factor,b FGF)免疫组织化学染色。 结果 高氧促视网膜血管收缩、闭塞 ,相对低氧致视网膜血管扩张、增生。突破视网膜内界膜的血管内皮细胞数 ,给氧组平均 2 4个 /切面 ,对照组平均<1个 /切面 ,二者相比差异有显著性的意义 (P<0 .0 0 1)。与对照组相比 ,给氧组小鼠视网膜内层 VEGF的表达明显增强。 结论 该小鼠模型中视网膜新生血管的发生与缺氧相关 ,并可对其定量观察 ,是进行视网膜新生血管发生机制及治疗研究的合适模型。

Objective [WT5”BZ]It has been shown that pigment epithelium derived factor (PEDF) is an effective anti apoptosis agent on several kinds of cells of the central nervous system.This study aimed to evaluate the effect of PEDF on pressure induced retinal ischemia in a rat model. Methods Retinal ischemia was induced by increasing the intraocular pressure to 110 mm Hg for 45 minutes via an intracameral catheter.Ten microliters (0.1 μg/μl) PEDF was injected into the vitreous of 4 eyes of each group immediately after...

Objective [WT5”BZ]It has been shown that pigment epithelium derived factor (PEDF) is an effective anti apoptosis agent on several kinds of cells of the central nervous system.This study aimed to evaluate the effect of PEDF on pressure induced retinal ischemia in a rat model. Methods Retinal ischemia was induced by increasing the intraocular pressure to 110 mm Hg for 45 minutes via an intracameral catheter.Ten microliters (0.1 μg/μl) PEDF was injected into the vitreous of 4 eyes of each group immediately after reperfusion and 4 additional eyes received only normal saline as vehicle controls.The animals were euthanized at 2 or 7 days after reperfusion.The effect of PEDF on retinal degeneration was assessed by measuring the thickness of the inner retinal layers (MTIRL) and counting the retinal ganglion cells (RGC) on plastic embedded retinal sections. [WT5”HZ]Results [WT5”BZ]The MTIRL and the RGC counting in eyes treated with intravitreal PEDF were significantly higher than those in vehicle controls (118.1±5 0) μm vs(94.9±3 0) μm (P<0.05);(6.0±1 0) cells/100 μm vs (4.5±0.5) cells/100 μm (P<0.05) 7 days after reperfusion,respectively. Conclusion Intravitreal administration of PEDF can ameliorate an ischemia reperfusion retinal injury and may be useful to prevent neuronal degeneration in the inner retina.

目的 已证实色素上皮衍生因子 (pigment epithelium- derived factor,PEDF)对中枢神经细胞有抗凋亡作用。本实验评价其对压力诱发的视网膜缺血再灌注的影响。 方法 经前房灌注维持眼压 110mm Hg(1mm Hg=0 .133k Pa) ,45 min,建立大鼠视网膜缺血再灌注模型。随后立即向玻璃体注射 10 μ1(0 .1μg/ μl) PEDF,分别于 2 d和 7d摘除眼球 ,测量塑料包埋切片的平均视网膜内层厚度 (mean thicknessof inner retinal layer,MTIRL)和视网膜节细胞 (retinal ganglion cells,RGC)计数。 结果 PEDF玻璃体注射 7d后治疗组的 MTIRL和 RGC明显高于对照组 [(118.1± 5 .0 )μm对 (94.9± 3.0 )μm,P<0 .0 5 ;(6 .0±1.0 )个 / 10 0μm对 (4.5± 0 .5 )个 / 10 0μm,P<0 .0 5 ]。 结论 玻璃体内注射 PEDF有助于防止视网膜缺血再灌注后神经变性和细胞死亡

 
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