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nephritic rats
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  肾病大鼠
     Clinical and experimental study verified the clinical effect to treat chronic glomerulonephritis and experimental study investigated the renoprotective effect and mechanism of replenish Qi and clear heat extract (益气清热膏) in adriamycin nephritic rats.
     本文通过临床与实验研究进一步验证了益气活血、清热利湿法治疗慢性肾炎的临床疗效,在实验研究中初步探讨了益气清热膏对阿霉素肾病大鼠的肾脏保护作用及其机理。
短句来源
     Effects of the Combination Treatment with Benazepril and Valsartan on Collagen Ⅳand Fibronectin in the Kidney of Nephritic Rats
     苯那普利和缬沙坦联合应用对肾病大鼠肾脏Ⅳ型胶原和纤维连接蛋白的影响
短句来源
     Objective To explore the effects of the combination treatment with benazepril and valsartan on collagen Ⅳand fibronectin(FN)in the kidney of nephritic rats.
     目的探讨苯那普利和缬沙坦联合应用对肾病大鼠细胞外基质Ⅳ型胶原和纤维连接蛋白的影响。
短句来源
     The effect and mechanism of lovastatin on glomerulosclerosis in adriamycin-induced nephritic rats
     洛伐他汀对肾病大鼠肾小球硬化的影响及其机制
短句来源
     Effects of lovastatin therapy on expression of platelet-derived growth factor in nephritic rats
     洛伐他汀降脂治疗对肾病大鼠血小板源生长因子表达的影响(英文)
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  “nephritic rats”译为未确定词的双语例句
     Effects of Tripterium Regelii on serum MMP-2, MMP-9 , and TGF-β_1 in nephritic rats
     东北雷公藤对肾炎大鼠血清中MMP-2、MMP-9和TGF-β_1的影响
短句来源
     Results There were increases in glomerular infiltration of leukocyte, expressions of IL-1β and IL-6 protein and mRNA, and activities of NF-κB in nephritic rats between days 1 and 4 after nephritis induction.
     结果大鼠系膜增殖性肾炎发病后第1~4天,肾小球白细胞计数、IL-1β、IL-6的mRNA与蛋白表达、NF-κB活性升高;
短句来源
     Treatment of nephritic rats with 5-lipoxygenase inhibitorMK886,with thromboxane(TX),synthetase inhibitor,Furegrelate or with 12-lipoxygenaseinhibitor Baicalein inhibited glomerular synthesis of leukotriene(LT)B_4, TXB_2 and also12-hydroxyeicosatetraenoic acid(12-HETE)respectively,and GPA.
     对肾炎鼠给予5-脂氧合酶抑制剂MK886或血栓素(thromboxaneA2,TXA2)合成酶抑制剂Furegrelate或12-脂氧合酶抑制剂Baicalein进行处理,可分别抑制肾小球合成的白三烯(leukotricne,LT)B4 及12-羟二十碳四烯酸(12-hydroxyeicosatetraenoicacid,12-HETE),同时也抑制PCNA(+)细胞及GPA的上升。
短句来源
     Results The level of urine protein (mg/24 h) in nephritic rats treated with Tripterium Regelii was significantly decreased(pre-treatment: 327.38±48.67;post-treatment:170.38±32.62,P<0.01).
     结果 :经东北雷公藤治疗后肾炎大鼠尿蛋白 [(170 .38± 32 .6 2 ) m g/ 2 4 h]明显低于治疗前 [(32 7.38± 4 8.6 7) mg/ 2 4 h,P<0 .0 1];
短句来源
     Objective To investigate the effects of Tripterium Regelii on urine protein and serum MMP-2, MMP9, and TGF-β 1 in nephritic rats.
     目的 :通过观察东北雷公藤对肾炎大鼠尿蛋白及血中 TGF-β1 、 MMP- 2和 MMP- 9的影响 ,研究其在肾炎大鼠中的作用。
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  相似匹配句对
     Renoprotective Effect of Yiqiqingre Extract in Adriamycin Nephritic Rats
     益气清热膏对阿霉素肾病大鼠的肾脏保护作用
短句来源
     All rats
     ②手术对照组:给大鼠施予与实验组大鼠相同
短句来源
     Effects of exercise training and enalapril on nephritic damage rats with adriamycin
     运动训练及依那普利对阿霉素肾病大鼠模型肾损害的影响
短句来源
     METHODS The rats
     方法用075%的腺嘌呤(W/W)长期饲养大鼠。
短句来源
     Dialectical Nursing of Nephritic Syndrome
     肾病综合征的辨证施护
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  nephritic rats
A group of nephritic rats were given daily administration of atRA for 14-16 days.
      
Genistein inhibited the incorporation of [1-14C]acetate into cholesterol and FA in liver slices from nephritic rats when added to an incubation buffer, whereas genistin did not.
      
Effects of genistein and its glycoside genistin were studied in nephritic rats with endogenous hyperlipidemia.
      
While lipoprotein lipase activity was unchanged among the nephritic rats, hepatic triglyceride lipase activity was reduced by either the 8% casein or FO diet.
      
No effect of diet was observed on fatty acid synthesis among the nephritic rats.
      
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In rat mesangioproliferative nephritis induced by anti-Thy 1.1 antibody,therewere enhanced proliferating cell nuclear antigen(PCNA)positive cells per glomerulus andincreased glomerular proliferative activity (GPA)assessed by  ̄3H-thymidine incorporation inshort-term glomerular cultures.Treatment of nephritic rats with 5-lipoxygenase inhibitorMK886,with thromboxane(TX),synthetase inhibitor,Furegrelate or with 12-lipoxygenaseinhibitor Baicalein inhibited glomerular synthesis of leukotriene(LT)B_4, TXB_2 and...

In rat mesangioproliferative nephritis induced by anti-Thy 1.1 antibody,therewere enhanced proliferating cell nuclear antigen(PCNA)positive cells per glomerulus andincreased glomerular proliferative activity (GPA)assessed by  ̄3H-thymidine incorporation inshort-term glomerular cultures.Treatment of nephritic rats with 5-lipoxygenase inhibitorMK886,with thromboxane(TX),synthetase inhibitor,Furegrelate or with 12-lipoxygenaseinhibitor Baicalein inhibited glomerular synthesis of leukotriene(LT)B_4, TXB_2 and also12-hydroxyeicosatetraenoic acid(12-HETE)respectively,and GPA. Leukocyte depletion byX-ray irradiation or platelet depletion by anti-platelet antibody inhibited enhanced PCNApositive cells and GPA. These results indicated that leukocyte and platelet-derived LT,TXA_2and 12-HETE modulate mesangial cell proliferation in rat mesangioproliferative nephritis.

在抗Thy1.1抗体引起的大鼠系膜增殖性肾炎中,肾小球的增殖性细胞核抗原(prolifer-atingcellnuclearantigen,PCNA)阳性细胞增多, ̄3H-胸腺嘧啶核苷( ̄3H-thymidine, ̄3H-TdR)掺入法测定的肾小球增殖活性(glomerularproliferativeactivity,GPA)升高。对肾炎鼠给予5-脂氧合酶抑制剂MK886或血栓素(thromboxaneA2,TXA2)合成酶抑制剂Furegrelate或12-脂氧合酶抑制剂Baicalein进行处理,可分别抑制肾小球合成的白三烯(leukotricne,LT)B4 及12-羟二十碳四烯酸(12-hydroxyeicosatetraenoicacid,12-HETE),同时也抑制PCNA(+)细胞及GPA的上升。用X线照射或抗血小板抗体分别去除肾炎鼠的循环白细胞或血小板,均可抑制PCNA(+)细胞及GPA的上升。结果提示白细胞及血小板衍生的LT、TXA_2及12-HETE参与本模型的系膜细胞增殖。

h after the injection of nephroto xic serum to the accelerated nephrotoxicserum nephritic rat model,the free hydroxyl radical and IL-1 activity produeed byglomerular macrophages(GMφ),own peritoneal Mφ(PMφ),and normal peritoneal Mφ(N-PMφ)were detected by electron spin resonance(ESR)and thymocyte proliferation. Theeffect of IL-1 on production of free hydroxyl radical by rat GMφ and PMφand N-PMφwas also observed.The resuIts showed that IL-1 activity and hydroxyl radical insupernatants of GMφ of the...

h after the injection of nephroto xic serum to the accelerated nephrotoxicserum nephritic rat model,the free hydroxyl radical and IL-1 activity produeed byglomerular macrophages(GMφ),own peritoneal Mφ(PMφ),and normal peritoneal Mφ(N-PMφ)were detected by electron spin resonance(ESR)and thymocyte proliferation. Theeffect of IL-1 on production of free hydroxyl radical by rat GMφ and PMφand N-PMφwas also observed.The resuIts showed that IL-1 activity and hydroxyl radical insupernatants of GMφ of the experimental group was significantly higher than that of ownPMφ and N-PMφ,Free hydroxyl radical production in GMφ stimulated by rIL-1 was in-creased more than that of PMφ and N-PMφ significantly.These results suggested that theoveractivity of infiltrated Mφ in inflammatory glomeruli produeed large quantity of IL-1,furthermore IL-1 stimulated GMφ to produce more reactive oxygen,This vicious circle inlocal glomeruli may play an important role in the pathogenesis of renal injury inaccelerated nephrotoxic nephritis.

本文用大鼠加速肾毒血清肾炎模型,于注射肾毒血清后72h,用电子自旋共振(ESK)和胸腺细胞增殖检测肾炎鼠的肾小球巨噬细胞(GMφ),自身腹腔Mφ(PMφ)及正常鼠腹腔Mφ(N-PMφ)所产生羟自由基(OH)和IL-1活性。同时观察IL-1对GMφ、PMφ和N-PMφ产生OH的影响。结果显示:肾炎鼠GMφIL-1活性和bH明显高于PMφ和N-PMφ;IL-1能刺激Mφ产生OH,且炎症GMφ的OH明显高于PMφ和N-PMφ,揭示:肾小球中浸润的Mφ过度活化并产生大量IL-1及OH,IL-1又能进一步刺激Mφ产生更多的OH,这一肾小球内的局部恶性循环,在加速肾毒血清肾损伤的发病机制中可能起着重要作用。

OBJECTIVE Abnormal lipid metabolism is a frequent complication in renal diseases and plays an important role in the progression of renal injury. Adriamycin(ADR) induced nephritis was used to evaluate the effects of hyperlipidemic chow on the progression of renal damages in this study. METHODOLOGY Forty male Wistar rats were used in this study. ADR(6 0 mg/kg) was used for induction of nephritis in 20 rats. 10 nephritic rats and 10 normal rats were fed with hyperlipidemic chow(1 5% cholesterol,...

OBJECTIVE Abnormal lipid metabolism is a frequent complication in renal diseases and plays an important role in the progression of renal injury. Adriamycin(ADR) induced nephritis was used to evaluate the effects of hyperlipidemic chow on the progression of renal damages in this study. METHODOLOGY Forty male Wistar rats were used in this study. ADR(6 0 mg/kg) was used for induction of nephritis in 20 rats. 10 nephritic rats and 10 normal rats were fed with hyperlipidemic chow(1 5% cholesterol, 0 5% cholic acid and 5% pig tallow added to the basic rodent chow) for 13 weeks, while 10 nephritic rats and 10 normal rats with basic rodent chow. Urinary protein excretion, plasma lipids levels were investigated and renal histologic changes were observed after 13 weeks. RESULTS At the 4, 8 and the 13th week of observation, proteinuria in nephritic rats fed with hyperlipidemic chow increased more significantly than in nephritic rats fed with basic chow(15 91±5 74 vs 11 05±8 97 mg/24h at the 4th week, 20 51±8 53 vs 13 23±3 91 at the 8th week, 29 73±7 94 vs 20 94±6 28 mg/24h at the 13th week, P <0 01); while in the normal rats fed with hyperlipidemic chow, proteinuria also increased significantly in the 8th and 13th week(4 22±2 58 vs 1 99±1 69mg/24h, P <0 05; 12 11±4 69 vs 1 98±1 89 mg/24h, P <0 01). In nephritic rats fed with hyperlipidemic chow, serum total cholesterol, tryglyceride, low density lipoprotein cholesterol and very low density lipoprotein cholesterol increased significantly than those in nephritic rats fed with basic chow at the 13 week of observation( P <0 05 for all); while between normal rats fed with hyperlipidemic chow and basic chow, mild elevations of total cholesterol, low density lipoprotein cholesterol and very low density lipoprotein cholesterol were found. In the histologic study of the renal damages, exacerbated glomerular changes and increased foam cells were found in the nephritic rats fed with hyperlipidemic chow as compared to those in nephritic rats fed with basic chow. CONCLUSION Hyperlipidemic chow increases proteinuria and lipids levels in ADR induced nephritic rats and exacerbates the renal damage through a mechanism in which hyperlipidemia plays an important role.

目的:探讨高脂饲料加剧阿霉素(ADR)肾病大鼠高脂血症及肾组织形态学改变的作用。方法:用ADR静脉注射建立大鼠肾病模型,饲喂含15%胆固醇、5%猪油的高脂饲料。观察大鼠尿蛋白排泄量,测定血清中脂质有关指标以及检查肾组织形态学改变。结果:饲喂高脂饲料的ADR肾病大鼠,血清脂质水平在整个试验过程中均明显高于饲喂基础饲料的ADR肾病大鼠,肾小球病变也显著加重。结论:饲喂高脂饲料所致高脂血症是导致大鼠ADR肾病进行性进展的重要因素之一

 
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