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nephropathy in rats
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  肾病大鼠
     Effect of Jianshenkeli Agent on Renal Function and TGF-β_1mRNA Diabetes Nephropathy in Rats
     健肾颗粒剂对糖尿病肾病大鼠肾功能及肾脏组织TGF-β1_mRNA的影响
短句来源
     Experimental study of immunoloregulation induced by tripterygium glycosides on IgA nephropathy in rats
     雷公藤多甙对IgA肾病大鼠免疫调节的实验研究
短句来源
     ObjectiveTo observe the effect of immunoloregulation induced by tripterygium glycosides on experimental IgA nephropathy in rats.
     目的观察雷公藤多甙对实验性IgA肾病大鼠的免疫调节作用。
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     Establishment of a Radioimmunoassay Method for 8-epi-PGF2 α and Altertation of 8-epi-PGF2 α on Diabetic Nephropathy in Rats and in Patients with Pregnancy-induced Hypertension
     8-表氧-前列腺素F2α放免方法的建立及在糖尿病肾病大鼠和孕高征患者水平变化的研究
短句来源
     Significance of apoptosis during glomerulosclerosis in adriamycin induced nephropathy in rats
     细胞凋亡在阿霉素肾病大鼠肾小球硬化过程中的意义
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  “nephropathy in rats”译为未确定词的双语例句
     AR, Bax/Bcl-2, PPARγ Gene Expressions and Their Relationship to Diabetic Nephropathy in Rats with Diabetes Mellitus
     AR、Bax/Bcl-2、PPARγ基因表达与大鼠糖尿病肾病关系的探讨
短句来源
     The Effect of Matrine on the Expression of TGF-β1 and BMP-7 in Experimental Chronic Cyclosporine Nephropathy in Rats
     苦参碱对大鼠慢性环孢素肾毒性模型TGF-β_1和BMP-7表达的影响
短句来源
     Effects of lovastatin on renal function and expression of phosphorylating -p38 mitogen-activated protein kinase in experimental diabetic nephropathy in rats
     洛伐他汀对糖尿病大鼠肾功能及肾脏组织p38丝裂原激活蛋白激酶表达的影响
短句来源
     A novel gene Dmrs91 related to early stage of diabetic nephropathy in rats and its bioinformatics analysis
     糖尿病大鼠早期肾脏病变相关新基因Dmrs91克隆及其生物信息学分析
短句来源
     Effect of Fluvastatin on Transforming Growth Factor-β1 Expression in Alcoholic Nephropathy in Rats
     氟伐他汀对酒精性肾损伤大鼠肾脏转化生长因子-β1表达的影响
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  相似匹配句对
     Membranous IgA nephropathy
     膜型IgA肾病
短句来源
     PKC and Diabetic nephropathy
     蛋白激酶C和糖尿病肾病
短句来源
     STUDIES OF AUTOANTIBODY IN NEPHROPATHY
     肾脏病自身免疫机理的研究
短句来源
     Prostaglandin and interstitial nephropathy
     前列腺素与肾间质病变
短句来源
     Pathological observation of adriamycin nephropathy in rats
     大鼠阿霉素肾病的病理学观察
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  nephropathy in rats
Beneficial effect of dl-α-lipoic acid on cyclosporine A induced hyperlipidemic nephropathy in rats
      
Stages of modeling of polyetiological electrolyte nephropathy in rats are described.
      
Stages of modeling of polyetiological electrolyte nephropathy in rats are described.
      
Protective Effect of Oral L-arginine Supplementation on Cyclosporine Induced Nephropathy in Rats
      
In this study we examined the effects of PTX on TNF-α, proteinuria, nitrite production, and apoptosis in an experimental model of Adriamycin (ADR) nephropathy in rats.
      
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Objective To explore the effect of antioxidant on nephropathy in rats.Methods A single intravenous injectin of abriamycin(ADR)results in marked proteinuria and morphological changes that aresimilar to minimal change nephrosis in humans.The effect of Vitamin E(VE) on ADR-induced renal injury in rats was examined.The serumlipidperoxide(LPO)level and the superoxide dismutase(SOD)activity of the erythrocyte was examined.Results The full expression of the syndrome occured at the 14th day after using ADR.The...

Objective To explore the effect of antioxidant on nephropathy in rats.Methods A single intravenous injectin of abriamycin(ADR)results in marked proteinuria and morphological changes that aresimilar to minimal change nephrosis in humans.The effect of Vitamin E(VE) on ADR-induced renal injury in rats was examined.The serumlipidperoxide(LPO)level and the superoxide dismutase(SOD)activity of the erythrocyte was examined.Results The full expression of the syndrome occured at the 14th day after using ADR.The serum LPO level wassignificantly higher than that of the nomal cormal control group(P<0.01).Concurrent administration of VE showed amelioration of the normalcontrol group(P<0.01).Concurrent administration of VE showeda melioration of proteinuria and serum biochemical indexes.There were also less severe glmerular morpholgical changes in the VE group versus ADR group.The serum LPO level was singnificantly lower,the SOD activity of erythrocyte was higher than those in the ADR group(P<0.01).Conclusion This study suggests that the protective effect of VE may contribute to its ability to seavenge free radicals and inhibit lipidperoxidation.

目的:探讨抗氧化剂对大鼠肾模型治疗作用。方法:给大鼠1次性静脉注射阿霉素(ADR)制作肾病模型。选用抗氧化剂维生素E(VE)为治疗因素,测定血清中脂质过氧化物(LPO),红细胞超氧化物歧化酶(SOD)水平及相关血尿生化指标。结果:模型物组动物实验第14天出现典型肾病综合征表现,血清中LPO水平明显升高、红细胞SOD活性明显降低。VE治疗组动尿、血生化指标及病理改变明显改善,且血清LPO水平明显降低,红细胞SOD活性明显高于ADR组动物。结论:氧自由基,脂质过氧化损伤与ADR肾病发生密切相关,VE能通过清除自由基、阻断脂质过氧化物对ADR肾病起一定治疗作用

? Shenkangning(SKN) is a compound preparation by composed of Chinese medicines.SKN showed a significantly effects on chronic nephropathy of patients in clinical treatment.The aim of this study is to investigate the effect of SKN on experimental chronic nephropathy.Nonimmunologic damage of chronic nephropathy in rats was induced a single iv administration of ADR(7.5mg/kg body wt) and immunologic damage of chronic nephropathy in rats was induced by repeated sc administration on renal medulla and...

? Shenkangning(SKN) is a compound preparation by composed of Chinese medicines.SKN showed a significantly effects on chronic nephropathy of patients in clinical treatment.The aim of this study is to investigate the effect of SKN on experimental chronic nephropathy.Nonimmunologic damage of chronic nephropathy in rats was induced a single iv administration of ADR(7.5mg/kg body wt) and immunologic damage of chronic nephropathy in rats was induced by repeated sc administration on renal medulla and Freund's complete sdjuvant.The results indicated that proteinuria,urinary creatinine,serum urinary nitrogen and serum urinary creatinine increased singificantly after iv ADR from d10 to d24 and repeated sc administration of renal medulla and Freund's complete sdjuvant from d30.Renal histological examination suggested a tubulointerstitial cellular infiltrate and glomerrular lesions were found in 0.9%NaCl group rats. The proteinuria, urinary creatinine,serum urinary nitrogen and serum urinary creatinine deceased in ADR rats treated by SKN(compare with 0.5% NaCl group rats,P<0.05 or 0.01) and the tubulointerstitial cellular infiltrate and glomerrular lesions were less than 0.9%NaCl group rats. The results suggested SKN was antiproteinuria and protected kidney in exprimental ADR and immunologic nephropathy.

阿霉素7.5mg/kg一次性给大鼠静注和以肾髓质匀浆加完全弗氏佐剂造模形成大鼠慢性肾炎和慢性免疫性肾炎,肾康宁8g/kg、16g/kg治疗组对阿霉素肾炎和免疫性肾炎引起的大鼠尿蛋白排出量增加、尿肌酐、血清肌酐和血清尿素氮升高有明显抑制作用(P<0.05or0.01)。病理切片可见肾康宁对阿霉素肾炎和免疫性肾炎病理变化也有明显的改善作用。

Objective To investigate the effects of high lipid diet and simvastatin on renal histological injuries in adriamycin induced nephropathy in rats Method Urinary protein, blood lipids, BUN, serum creatinine, and histological change were checked in nephropathic rats fed with regular and high lipid chow and treated with simvastatin Results At week 22, the amounts of urinary protein (mg/24h) were (58±21), (120± 41), (92±25), (407±35), (236±54) ( F= 117 65, P< 0 01); the levels...

Objective To investigate the effects of high lipid diet and simvastatin on renal histological injuries in adriamycin induced nephropathy in rats Method Urinary protein, blood lipids, BUN, serum creatinine, and histological change were checked in nephropathic rats fed with regular and high lipid chow and treated with simvastatin Results At week 22, the amounts of urinary protein (mg/24h) were (58±21), (120± 41), (92±25), (407±35), (236±54) ( F= 117 65, P< 0 01); the levels of serum triglyceride (mmol/L) (0 99±0 09), (1 43±0 29), (1 61±0 30), (2 91±0 25), (1 70±0 23) ( F= 68 73, P< 0 01); total cholesterol (mmol/L) (2 53±0 50), (3 28±0 28), (3 02±0 59), (4 52±0 41), (3 42±0 40) ( F= 21 50, P< 0 01); low density lipoprotein cholesterol (LDL ch) (mmol/L) (1 53±0 21), (2 02± 0 09), (1 74±0 36), (2 67±0 22), (1 93±0 22) ( F= 26 57, P< 0 01) in normal control, regular diet control and simvastatin treated, high lipid control and simvastatin treated groups, respectively All these values were significantly higher in adriamycin treated groups than those of control groups; the values of high lipid diet control groups were also higher than those of regular diet control groups ( t= 15 18, P< 0 01; t= 10 93, P< 0 01; t= 7 06, P< 0 01; t= 5 59, P< 0 01, respectively), and in simvastatin treated groups were lower than those of corresponding control groups ( t= 7 49, P< 0 01; t= 10 07, P< 0 01; t= 7 78, P< 0 01; t= 6 73, P< 0 01, respectively) Histological study demonstrated that the percentage of glomeruli in which segmental sclerosis occurred were (3 2±1 4), (55 6±13 5), (13 6±5 0), (86 8±3 4), (37 6±25 6), respectively The differences among control and adriamycin treated groups were significant ( H= 35 1, P< 0 01); mesangial proliferation and matrix expansion significantly increased in the adriamycin treated rats, and were associated with significant tubulointerstitial injuries. The degree of glomerular injuries and extent of tubulointerstitial pathological change were more serious in high lipid diet groups than in regular diet groups; and compared with corresponding control groups, those in simvastatin treated groups were significantly less severe Conclusion In the adriamycin treated rats, high lipid diet may worsen, and simvastatin can ameliorate the renal histological injuries

目的 观察羟甲戊二酰辅酶A还原酶抑制剂辛伐他汀和食物中脂类含量对阿霉素肾病大鼠肾组织损伤程度的影响。方法 对阿霉素肾病大鼠分别饲以标准及高脂鼠食并给予辛伐他汀,观察其尿蛋白排出量、血脂、血尿素氮(BUN) 、肌酐(Scr)和肾脏光镜组织形态学变化。结果 首次给予阿霉素后22 周时,正常对照组、标准鼠食肾病对照组和治疗组、高脂鼠食对照组和治疗组大鼠尿蛋白排出量依次(下同)为:(58±21),(120 ±41) ,(92 ±25) ,(407±35),(236±54) mg/24h(F= 117-65,P< 0-01);血甘油三酯(0-99±0-09),(1-43±0-29),(1-61±0-30),(2-91±0-25),(1-70±0-23) mmol/L(F= 68-73, P<0-01);总胆固醇(2-53 ±0-50) ,(3-28 ±0-28) ,(3-02 ±0-59),(4-52 ±0-41),(3-42 ±0-40) mmol/L(F=21-50, P< 0-01) ;低密度脂蛋白胆固醇(1-53 ±0-21) ,(2-02±0-09),(1-74±0-36) ,(2-67±0-22),(1-9...

目的 观察羟甲戊二酰辅酶A还原酶抑制剂辛伐他汀和食物中脂类含量对阿霉素肾病大鼠肾组织损伤程度的影响。方法 对阿霉素肾病大鼠分别饲以标准及高脂鼠食并给予辛伐他汀,观察其尿蛋白排出量、血脂、血尿素氮(BUN) 、肌酐(Scr)和肾脏光镜组织形态学变化。结果 首次给予阿霉素后22 周时,正常对照组、标准鼠食肾病对照组和治疗组、高脂鼠食对照组和治疗组大鼠尿蛋白排出量依次(下同)为:(58±21),(120 ±41) ,(92 ±25) ,(407±35),(236±54) mg/24h(F= 117-65,P< 0-01);血甘油三酯(0-99±0-09),(1-43±0-29),(1-61±0-30),(2-91±0-25),(1-70±0-23) mmol/L(F= 68-73, P<0-01);总胆固醇(2-53 ±0-50) ,(3-28 ±0-28) ,(3-02 ±0-59),(4-52 ±0-41),(3-42 ±0-40) mmol/L(F=21-50, P< 0-01) ;低密度脂蛋白胆固醇(1-53 ±0-21) ,(2-02±0-09),(1-74±0-36) ,(2-67±0-22),(1-93±0-22) mmol/

 
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