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experimental retinal
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  大鼠视网膜
     Expression of MMP-9 and TIMP-1 in experimental retinal ischemia-reperfusion injury in rats
     MMP-9、TIMP-1在大鼠视网膜缺血再灌注损伤中的表达
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     Aim To investigate the therapeutical effect and mechanisms of basic fibric growth factor (bFGF) on retina ischemia/ reperfusion injury. Methods Experimental retinal ischemia/ reperfusion injury was induced by increasing intraocular pressure in the eyes of rats.
     目的探讨碱性成纤维细胞生长因子(basic fibric growth factor;bFGF)在大鼠视网膜缺血再灌注损伤(retina ischemi—a/reperfusion injury;RIRI)中治疗作用及机制。
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     The Establishment of Rat Model in the Experimental Retinal Photo-injury and Research on its Retinal Pathologic Changes
     实验性大鼠视网膜光损伤模型的建立与研究
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     A Study on the Apoptosis of Photoreceptors in Experimental Retinal Photo-injury Rats
     实验性光损伤大鼠视网膜光感受器细胞凋亡的研究
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     Objective To investigate the dynamic changes and significance of matrix metalloproteinases 9(MMP-9) and tissue inhibitors of metalloproteinases 1(TIMP-1) in an experimental retinal ischemia reperfusion (RIR) injury.
     目的 研究视网膜缺血再灌注(RIR)损伤时大鼠视网膜基质金属蛋白酶-9(MMP-9)和组织金属蛋白酶抑制剂-1(TIMP-1)蛋白表达的动态变化及意义。
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  “experimental retinal”译为未确定词的双语例句
     to illustrate the proliferating effect of IL-1β in experimental retinal detachment and it's cause with the intervention of anti-IL-1β with IL-Ra and IL-1β antibody.
     并应用IL-1β抗体、IL-1Ra进行干预,确定IL-1β在增殖性视网膜病变中所起的作用。
短句来源
     Effects of intravitreal injection of 1 mg dexamethasone on expression of endothelin-1 in the experimental retinal detachment
     地塞米松1mg玻璃体注射对ET-1在实验性视网膜脱离表达的影响
短句来源
     Objective To observe the effect of intravitreal injection of 1 mg dexamethasone(DEX) on the expression of endothelin-1(ET-1) in experimental retinal detachment.
     目的观察1mg地塞米松(dexamethasone,DEX)玻璃体注射对ET1在实验性视网膜脱离中表达的影响。
短句来源
     Objective: To investigate the neuroprotective effect and mechanism of betaxolol on experimental retinal ischemia-reperfusion injury.
     目的:探讨局部滴用倍他洛尔眼药水对实验性视网膜缺血再灌注损伤的治疗作用及其可能的作用机理。
短句来源
     METHODS:The rat model of experimental retinal ischemia-reperfusion injury was established by increasing the intraocular pressure to 110mmHg (1kPa =7.5mmHg) in rat eyes.
     方法:采用前房灌注生理盐水升高大鼠眼压到110mmHg(1kPa=7.5mmHg)持续1h的方法制作实验性视网膜缺血再灌注损伤模型,分别于再灌注不同时间点取材,行免疫组织化学法染色观察HIF-1α在视网膜细胞的阳性表达;
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  experimental retinal
Experimental retinal detachment causes widespread and multilayered degeneration in rabbit retina
      
The ability of retinal Müller glial cells to perform phagocytosis in vivo is studied in a rabbit model of experimental retinal detachment where pigment epithelial cells are occasionally detached together with the neural retina.
      
Retinal pigment epithelium melanin granules are phagocytozed by Müller glial cells in experimental retinal detachment
      
Oscillatory evoked potentials in the rabbit visual system in experimental retinal pathology induced by monoiodoacetate administr
      
Experimental retinal tolerance to very low viscosity silicone oil (100 cs) as a vitreous substitute compared to higher viscosity
      
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OBJECTIVE:To investigate the transporting function of subretinal fluid in retinal pigment epithelium(RPE) of experimental retinal detachment(RD).METHODS:Twenty seven pigmented rabbits with one eye as retinal detachment model and the other eye as control were divided into 4 groups,ie.7 rabbits in each group at random.The Na + K + ATPase activity in RPE choroidal tissue at the detached region in both experimental and control...

OBJECTIVE:To investigate the transporting function of subretinal fluid in retinal pigment epithelium(RPE) of experimental retinal detachment(RD).METHODS:Twenty seven pigmented rabbits with one eye as retinal detachment model and the other eye as control were divided into 4 groups,ie.7 rabbits in each group at random.The Na + K + ATPase activity in RPE choroidal tissue at the detached region in both experimental and control eyes was determined at the lst,2nd,3rd and 4th week after retinal detachment,and also the ultrastructure of the RPE choroidal tissues observed.RESULTS:The activity of Na + K + ATPase of experimental eyes[(2.16±1.26)μmol/(m·h)]was significantly lower than that of the controls[(4.84±1.59)μmol/(m·h)].The degree of decreasing activity was relieving along with increasing of the course of RD.The inward pinocytosis in RPE cells under electron microscope was found from the 2nd week samples afterwards and becoming obvious along with increasing of the disease course.CONCLUSIONS:The outward transporting function of RPE was increased after RD,it became decreased along with increasing of the course of RD and the inward transporting function then developed.

目的:探讨实验性视网膜脱离(retinaldetachment,RD)后视网膜色素上皮(retinalpigmentepithelium,RPE)对视网膜下液(subretinalfluid,SRF)的输导功能。方法:将制作成功的实验性单眼RD28只家兔随机分为4组,每组7只,每只兔的另眼作为对照。分别取RD部位的RPE-脉络膜组织和对照眼相应部位的组织匀浆,再测定ATP水解产物无机磷含量,以判断ATP酶活性;并作实验和对照眼的两种组织相应部位的RPE电镜观察。结果:实验眼Na+-K+-转运ATP酶活性为(2.16±1.26)μmol/(mgh),对照眼为(4.84±1.59)μmol/(mgh),二者差异显著(t=5.52,P<0.01);酶活性降低程度随病程延长而减轻(P<0.05)。电镜观察实验眼RD后第2至4周有自脉络膜向视网膜方向的吞饮泡,且随病程延长而增多。结论:RD发生后RPE的外向输导功能增强,随病程进展,此功能减弱而内向输导功能产生。

Objective Detecting vitreous oxygen pressure(PO2),endothelins(ETs),insulin-like growth factor-1(IGF-1)and retinal blood flow in rabbit's eye before and after hyperbaric oxygen treatment(HBOT),in order to study the mechanism of HBOT in retinal disease.Methods Experimental retinal vein occlusion(RVO) eyes were done by Argon laser.vitreous PO2 value was determined by blood gas analysis;retinal blood flow was measured with radiolabelled microspheres method;vitreous ETs were detected by Radioimmunoassay;vitreous...

Objective Detecting vitreous oxygen pressure(PO2),endothelins(ETs),insulin-like growth factor-1(IGF-1)and retinal blood flow in rabbit's eye before and after hyperbaric oxygen treatment(HBOT),in order to study the mechanism of HBOT in retinal disease.Methods Experimental retinal vein occlusion(RVO) eyes were done by Argon laser.vitreous PO2 value was determined by blood gas analysis;retinal blood flow was measured with radiolabelled microspheres method;vitreous ETs were detected by Radioimmunoassay;vitreous IGF-1 value was acquired with acid-ethanol extraction and immunoradiometric assay.Results Vitreous PO2 value increased from 12.60±1 92kPa before HBOT to 23.34±2.00kPa after HBOT(P<0 01);Retinal blood flow decreased from 3.38±1.51mL ·min-1 ·g-1to 0.25±0.08mL·min-1·g-1(P<0 01);Vitreous ETs increased from 0 to 82.99±25.23ng ·L-1 after 3~5HBOT(P<0 01);Vitreous IGF-1 increased from 7.42±1.18ng·L-1 to 19.81±1.41ng ·L-1 (P<0 01) Conclusion HBO can increase vitreous PO2 and storage of oxygen;HBO decrease retinal blood flow obviously,so we must improve blood flow in HBOT; Vitreous ETs and IGF-1 values are obviously increased after 3~5 HBOT,these factors are activated by HBO and participated in ocularbiologic and pathologic process in HBOT according to their quantities,this is one of factors effecting on HBOT; HBO has direct and indirect treatments.Indirect treatment has two side,it's side effect is one of factors causing complications in HBOT,We must pay attention to it.

目的 检测高压氧治疗前后兔眼玻璃体氧分压、内皮素、胰岛素样生长因子- 1 及视网膜血流量的变化,探讨高压氧治疗视网膜疾病的机理,为高压氧治疗眼病提供理论根据。方法 用氩激光光凝法制作兔眼视网膜静脉阻塞眼模型,血气分析法测定玻璃体氧分压,放射微球标记法检测视网膜血流量;放射免疫分析法测定玻璃体内内皮素含量,酸酒精提取法和免疫放射测定分析法检测玻璃体内胰岛素样生长因子- 1 的含量。经统计学处理分析其相互关系,提出高压氧治疗视网膜疾病的作用机理。结果 高压氧治疗前后视网膜中央静脉阻塞眼玻璃体氧分压由1260±192kPa 升为2334±200kPa(P<001) ;视网膜血流量由338±151mL·min-1 ·g -1降低为025±008mL·min-1 ·g- 1(P< 001); 玻璃体内皮素含量经3~5 次高压氧治疗后, 由0升高为8299±2523ng·L-1(P<001);玻璃体内胰岛素样长生因子-1 含量由742±118ng·L-1 升高为1981±141ng ·L-1(P<001)。结论 高压氧有提高玻璃体氧分压, 增加氧储备作用;高氧可明显降低视网膜血流量,提...

目的 检测高压氧治疗前后兔眼玻璃体氧分压、内皮素、胰岛素样生长因子- 1 及视网膜血流量的变化,探讨高压氧治疗视网膜疾病的机理,为高压氧治疗眼病提供理论根据。方法 用氩激光光凝法制作兔眼视网膜静脉阻塞眼模型,血气分析法测定玻璃体氧分压,放射微球标记法检测视网膜血流量;放射免疫分析法测定玻璃体内内皮素含量,酸酒精提取法和免疫放射测定分析法检测玻璃体内胰岛素样生长因子- 1 的含量。经统计学处理分析其相互关系,提出高压氧治疗视网膜疾病的作用机理。结果 高压氧治疗前后视网膜中央静脉阻塞眼玻璃体氧分压由1260±192kPa 升为2334±200kPa(P<001) ;视网膜血流量由338±151mL·min-1 ·g -1降低为025±008mL·min-1 ·g- 1(P< 001); 玻璃体内皮素含量经3~5 次高压氧治疗后, 由0升高为8299±2523ng·L-1(P<001);玻璃体内胰岛素样长生因子-1 含量由742±118ng·L-1 升高为1981±141ng ·L-1(P<001)。结论 高压氧有提高玻璃体氧分压, 增加氧储备作用;高氧可明显降低视网膜血流量,提示:在高压氧治疗时必?

Objective To further investigate pathologic mechanism of retinal phototrauma. Methods Twenty Wistar rats were divided into control and experimental groups.Their eyes were extracted in 12,24 and 36 hours after light exposure.HE stained retina samples were examined and TDT mediated dUTP nick end labelling(TUNEL)method was employed to distinguish apoptotic cells. Results After 12 hour light exposure,slight vesiculation was observed in the rod outer segment of the retinas.After 24 hour light exposure,the...

Objective To further investigate pathologic mechanism of retinal phototrauma. Methods Twenty Wistar rats were divided into control and experimental groups.Their eyes were extracted in 12,24 and 36 hours after light exposure.HE stained retina samples were examined and TDT mediated dUTP nick end labelling(TUNEL)method was employed to distinguish apoptotic cells. Results After 12 hour light exposure,slight vesiculation was observed in the rod outer segment of the retinas.After 24 hour light exposure,the outer nuclear layer showed predominant fractured and condensed nuclei and fragmented DNA.After 36 hour light exposure,the rod outer and inner segments were lysed and most of the nuclei in the outer nuclear layer were disappeared. Conclusions Apoptosis of photoreceptor cell is one of the important mechanisms which cause experimental retinal photoinjury of rats.

目的 进一步探讨视网膜光损伤的发病机制。 方法 20只Wistar大鼠分为实验组、对照组,分别在光照后12,24,36小时摘除眼球,视网膜组织行HE染色和核苷酸末端转移酶介导的DUTP缺口翻译法(TdTm ediated dUTPnick end labelling m ethod,TUNEL)标记凋亡细胞。 结果 光照后12 小时,视杆细胞外节出现少量空泡变性;24小时后,外核层出现明显的细胞核破碎、浓染和DNA裂解;36 小时后,视杆细胞内、外节溶解,外核层大量细胞核丢失。 结论 视细胞凋亡是大鼠实验性视网膜光损伤的重要机制之一。

 
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