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media necrosis
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  中层坏死
     Conclusions The transplant arteries were characterized by intimal denudation, inflammatory cell infiltration, media necrosis with the smooth muscle cell migration and intimal hyperplasia.
     结论 移植体动脉硬化具有内皮剥脱、炎性细胞浸润、中层坏死、平滑肌细胞迁移及内膜增生 4大特点
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  “media necrosis”译为未确定词的双语例句
     Conclusion ①Three characteristics including intima hyperplasia, media necrosis and cell migration and inflammatory cell infiltation are demonstrat- ed in this model.
     结论①移植动脉硬化具有内膜增生,中层细胞坏死、迁移、炎细胞浸润三大特点;
短句来源
     Conclusion Three characteristics including intima hyperplasia,media necrosis and cell migration and inflammatory cell infiltration are demonstrated in the model.
     结论①大鼠移植动脉变化具有内膜增生,中层细胞坏死、迁移,炎性细胞浸润等特点。
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     Media
     媒体互动
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     Media
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     Interrelation of Recurrent Secretory Otitis Media with Tumor Necrosis Factor-α and Immunoglobulin
     小儿分泌性中耳炎与肿瘤坏死因子及免疫球蛋白的关系
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     Expression of tumor necrosis factor-α in experi-mental otitis media with effusion
     肿瘤坏死因子-α在分泌性中耳炎动物模型中的表达
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  media necrosis
These changes included subendothelial hyperplasia, media necrosis and thrombosis.
      
RDP58 therapy markedly inhibited vascular intimal thickening, media necrosis, and adventitial cellular inflammation.
      
In two patients the diagnosis of Erdheim-Gsell media necrosis was histologically confirmed.
      
This makes hypertension as well as cystic media necrosis factors related to the development of aortic disease.
      


Objective To establish the experimental animal model of chronic rejection and observe the development of arteriosclerosis in the transplant. Methods The pathological changes of transplanted aorta at 3, 7, 14,20, 30 and 60 days after transplantation were observed by electron microscopy and computer image analysis. PCNA and C-myc were detected by immunohistochemical method. Results ①The intime thickening appeared at 14 day after operation, the intima hyperplasia developed persistently until 60 day and the cell...

Objective To establish the experimental animal model of chronic rejection and observe the development of arteriosclerosis in the transplant. Methods The pathological changes of transplanted aorta at 3, 7, 14,20, 30 and 60 days after transplantation were observed by electron microscopy and computer image analysis. PCNA and C-myc were detected by immunohistochemical method. Results ①The intime thickening appeared at 14 day after operation, the intima hyperplasia developed persistently until 60 day and the cell numbers showed a peak at 30th day after operation. A great many synthetic smooth muscle cell in the intima membrane appeared at 20 day after operation. ②The cell necrosis in the media and the cell migrated from the media to initma were observed, the number of α-actin positive cell decreased in the media, while increased in the intima at 14 day after operation. A significant negative correlation existed between them. ③The exprssion of PCNA showed a peak at 20 day after opera- tion. The expression of C-myc and PCNA showed a negative correlation. ④Inflammatory cells infitrated in the adventitia at 3 day after operation. Conclusion ①Three characteristics including intima hyperplasia, media necrosis and cell migration and inflammatory cell infiltation are demonstrat- ed in this model. ②In early phase the intima appears mainly in cell proliferation but extracellular matrix production deposition in later phase and the smooth muscle cell may undergo a phenotypic change during their proliferation process. ③The expression of PCNA shows significant correlation with cell proliferation, the expression of C-myc may be associated with some non-proliferative factors.

目的 建立慢性排斥反应的简化动物模型,观察移植体动脉硬化发展规律。方法 采用病理图像分析及电镜观察移植后3、7、14、20、30、60 d移植动脉硬化情况,结合 PCNA及 C-myc免疫组化了解细胞增殖与原癌基因表达的关系。结果①术后14 d内膜层明显增生,术后 60 d持续增厚,细胞数术后 30 d达高峰,术后 20 d内膜层出现大量合成型平滑肌细胞;②术后 14 d中层细胞变性坏死,向内层迁移,α-actin阳性细胞数下降,而内膜层α-actin阳性细胞数却迅速升高,两者呈显著负相关;③术后3 d外膜大量炎性细胞浸润。结论①移植动脉硬化具有内膜增生,中层细胞坏死、迁移、炎细胞浸润三大特点;②内膜增生在术后早期以细胞增生为主,后期则为细胞外基质增生,且伴随平滑肌形态改变;③PCNA表达反映出细胞增殖进程,而C-myc还可能与非增殖因素有关。

Objective To investigate the pathohistological changes of transplant arteriosclerosis in chronic rejection. Methods Pathological change of transplant aorta was observed by using the computer image analysis system and electron microscopy at 3, 7, 14, 20, 30 and 60 days after arterial transplantation. Results Except two grafts in which the cold ischemia time of donor aorta exceeded 1?h showed minimal intimal thickening at 14 days in the control group post operation, the remaining transplant arteries showed...

Objective To investigate the pathohistological changes of transplant arteriosclerosis in chronic rejection. Methods Pathological change of transplant aorta was observed by using the computer image analysis system and electron microscopy at 3, 7, 14, 20, 30 and 60 days after arterial transplantation. Results Except two grafts in which the cold ischemia time of donor aorta exceeded 1?h showed minimal intimal thickening at 14 days in the control group post operation, the remaining transplant arteries showed no significant difference from the normal arteries of the recipients themselves. In the allografts group, the inflammatory cells infiltrated in the adventitia at the third day post operation, intimal denudation at 7th day, the cell migrated from media to intima and the intimal hyperplasia at the 14th day, and the intimal mid semimonth hyperplasia at the 20th day. The smooth muscle cell was transformed from contractile type into synthetic type and the cell numbers reached peak at the 30th day. At the 60th day, the intimal hyperplasia developed persistently, the stroma fibrocyte was increased and the endothelium recovered. Conclusions The transplant arteries were characterized by intimal denudation, inflammatory cell infiltration, media necrosis with the smooth muscle cell migration and intimal hyperplasia.

目的 了解慢性排斥反应时移植物动脉硬化的病理变化。方法 采用病理图象定量分析及电镜等检测手段 ,观察大鼠动脉移植后 3、7、14、2 0、30及 6 0d的病理变化。结果 同系移植对照组除供者血管冷缺血超过 1h的 2只术后 14d内膜轻度增生外 ,其余移植动脉与受者自身的正常动脉比较 ,差异不显著 ;异系移植实验组术后 3d血管外膜有大量炎性细胞浸润 ,7d时局部内皮剥脱 ,14d内膜中层细胞通过弹力膜裂隙向内膜迁移 ,内膜增生 ,2 0d内膜全层或局部呈半月形增生 ,平滑肌细胞形态由收缩型转变为合成型 ,30d内膜细胞数达峰值 ,6 0d内膜持续增厚 ,基质纤维成分增多 ,内膜修复。结论 移植体动脉硬化具有内皮剥脱、炎性细胞浸润、中层坏死、平滑肌细胞迁移及内膜增生 4大特点

Objective To establish the experimental animal model of transplantation rejection and observe the performance of transplanted of abdominal aorta tissues.Methods The rat model of artery allograft was deve-loped.The grafts were collected to receive immunohistochemical and histopathological examinations and quantitative measurement of TGF-β_1 expression.Results The thickening appeared after operation,strongly expressed TGF-β_1 in intima of artery during the episode of rejection.Conclusion Three characteristics...

Objective To establish the experimental animal model of transplantation rejection and observe the performance of transplanted of abdominal aorta tissues.Methods The rat model of artery allograft was deve-loped.The grafts were collected to receive immunohistochemical and histopathological examinations and quantitative measurement of TGF-β_1 expression.Results The thickening appeared after operation,strongly expressed TGF-β_1 in intima of artery during the episode of rejection.Conclusion Three characteristics including intima hyperplasia,media necrosis and cell migration and inflammatory cell infiltration are demonstrated in the model.The expression level of TGF-β_1 was related to the occurrence and development of rejection.

目的建立大鼠腹主动脉移植慢性排斥反应的模型,观察移植动脉的变化规律。方法应用病理图像分析移植后动脉慢性排斥的变化,并结合免疫组化观察转化生长因子-1β(TGF-β1)在慢性排斥反应中的表达。结果移植术后动脉内膜增生并持续增厚,TGF-β1在移植动脉内膜大量表达并与病理改变相关。结论①大鼠移植动脉变化具有内膜增生,中层细胞坏死、迁移,炎性细胞浸润等特点。②TGF-β1在慢性排斥的发生发展中起着重要作用。

 
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