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ischemic brain
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  缺血性脑
     There were significant negative correlations between stroke indexes and the levels of NE, DA and 5 -HT in ischemic brain (r = -0.485, -0.497 and 0.579 respectively, P<0.01 in all).
     动物的卒中指数与缺血性脑组织中NE、DA及5-HT的含量呈显著负相关(r分别=-0.485,-0.497和-0.579,P均<0.01)。
短句来源
     JAKs-STATs signal transduction and ischemic brain injury
     JAKs-STATs信号转导通路与缺血性脑损伤
短句来源
     Expression of NgR mRNA and NgR Protein in Brain Tissues of Newborn Rats with Hypoxic Ischemic Brain Damage
     新生鼠缺氧缺血性脑损伤脑组织NgR mRNA和NgR蛋白表达及意义
短句来源
     Effect of Dynorphin A 1 ̄13 on Hypoxia Ischemic Brain Injury in Neonatal Rats
     强啡肽A_(1~13)在新生鼠缺氧、缺血性脑损伤中的作用
短句来源
     Progress on the Study of the Relationship of Cyclooxygenase 2 and Plasminogen Activator Inhibitor Type-1 with Ischemic Brain Infarction
     COX-2及PAI-1与缺血性脑梗死关系的研究进展
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  血性脑
     There were significant negative correlations between stroke indexes and the levels of NE, DA and 5 -HT in ischemic brain (r = -0.485, -0.497 and 0.579 respectively, P<0.01 in all).
     动物的卒中指数与缺血性脑组织中NE、DA及5-HT的含量呈显著负相关(r分别=-0.485,-0.497和-0.579,P均<0.01)。
短句来源
     JAKs-STATs signal transduction and ischemic brain injury
     JAKs-STATs信号转导通路与缺血性脑损伤
短句来源
     Expression of NgR mRNA and NgR Protein in Brain Tissues of Newborn Rats with Hypoxic Ischemic Brain Damage
     新生鼠缺氧缺血性脑损伤脑组织NgR mRNA和NgR蛋白表达及意义
短句来源
     Effect of Dynorphin A 1 ̄13 on Hypoxia Ischemic Brain Injury in Neonatal Rats
     强啡肽A_(1~13)在新生鼠缺氧、缺血性脑损伤中的作用
短句来源
     Progress on the Study of the Relationship of Cyclooxygenase 2 and Plasminogen Activator Inhibitor Type-1 with Ischemic Brain Infarction
     COX-2及PAI-1与缺血性脑梗死关系的研究进展
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  缺血脑
     Expressions of TNF-α and IL-1β in human ischemic brain tissues
     人缺血脑组织中TNF-α和IL-1β的表达
短句来源
     Immunohistochemical SABC method was used for detection of ICAM-1 expression in the microvascular endothelial cell of the ischemic brain region, and ELISA method for the soluble ICAM-1 (sICAM-1) content in peripheral blood.
     分别应用免疫组化SABC方法检测脑缺血再灌注大鼠缺血脑区微血管内皮细胞ICAM-1的表达和ELISA法检测外周血中sICAM-1含量及电针对其的影响。
短句来源
     CONCLUSION:VEGF165 gene can transform into the ischemic brain tissue and express VEGF mRNA and VEGF,and VEGF protein protects nerve cells by restraining the expression of Bax and impelling the expression of Bcl 2.
     结论:VEGF165基因可以转化到缺血脑组织中并表达VEGFmRNA和VEGF,后者可能通过抑制Bax和增强Bcl-2的表达而保护神经细胞。
短句来源
     Results After cerebral ischemia-reperfusion, both ICAM-1 expression level in the microvascular endothelium cell of the ischemic brain region and sICAM-1 content in the peripheral blood significantly increased in the model group as compared with the normal group and the sham operation group (P<0.01);
     结果:脑缺血再灌注后,缺血脑区微血管内皮细胞CAM-1表达水平和外周血中sICAM-1含量均增高(模型组与正常组、假手术组比较P<0·01);
短句来源
     Conclusion Cerebral ischemia may induce upregulation of ANG and GFAP expression in the ischemic brain areas.
     结论脑缺血可诱导缺血脑区ANG和GFAP的表达上调。
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  “ischemic brain”译为未确定词的双语例句
     Conclusions: Ginsenoside Rb1,Rb3,Rg1,Rh2,F11 have the protective effects against ischemic brain injury.
     结论:人参皂甙Rb1、Rb3、Rg1、Rh2、F11有明显的抗脑缺血损伤作用。
短句来源
     Results The ANG expression levels(ANG fluorescence intensity)after ischemia-reperfusion 1,3 and 7 d in the ischemic brain areas were 56±21,73±22,and 76±21,respectively.
     结果脑缺血-再灌注1、3、7d脑缺血区ANG表达水平(ANG荧光强度)分别为56±21、73±22和76±21,对照组为35±17;
短句来源
     1.Localization of AQP4 in the Brain 2.Changes of AQP4 Expression in Ischemic Brain Edema
     1.AQP4在大脑中的定位研究 2.AQP4在缺血性脑水肿中表达变化
短句来源
     The area of ischemic brain lesion correlated directy to the number of OX62+ cells in per 100mm2 of brain tissue section(R2 = 0.8914,P < 0.001) .
     脑缺血损伤面积与以每100mm2脑组织片为单位的OX62+数量呈正相关(R2=0.8914 P<0.001)。
     On day 6 after MCAO,the level of DC expressed MHC Ⅱmolecules(OX62+and OX6+) in the ischemic brain tissue also significantly increased as compared with the sham operative group(t=2.975,P< 0.05).
     脑缺血第6天,缺血组与假手术组进行比较,DC表达MHC-Ⅱ类分子(OX62+OX6+)显著升高(t=2.975,P<0.05)。
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  ischemic brain
Thus, choline succinate demonstrated a neuroprotective effect in conditions of ischemic brain injury.
      
EEG and clinical psychophysiological study of functional changes in the chronically ischemic brain upon an increase in cholinerg
      
The most important and highly sensitive and specific findings of CT in stroke patients are intracranial blood and ischemic brain edema.
      
In most cases, post-ischemic brain edema can not be adequately treated by conservative means.
      
Conclusions: The study provides evidence that hemicraniectomy as treatment of severe space occupying ischemic brain edema saves lives and results in good quality of life in a high proportion of patients, especially in the young.
      
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The effect of pentobarbital on cerebral protection and brain monoamine levels during cerebral ischemia were studied in 60 Mongolian gerbils. The animals were randomly divided into three groups. Pentobarbital(n = 20)and control(n = 20) groups which underwent left common carotid artery (CCA) ligation received 60 mg/kg of Pentobarbitalum natricum and equal volume of normal saline respectively at 30 min prior to operation. Sham-operated group (n=20)was treated as same as the control group with exception of not ligating...

The effect of pentobarbital on cerebral protection and brain monoamine levels during cerebral ischemia were studied in 60 Mongolian gerbils. The animals were randomly divided into three groups. Pentobarbital(n = 20)and control(n = 20) groups which underwent left common carotid artery (CCA) ligation received 60 mg/kg of Pentobarbitalum natricum and equal volume of normal saline respectively at 30 min prior to operation. Sham-operated group (n=20)was treated as same as the control group with exception of not ligating CCA. The heurological deficits were evaluated and brain monoariiines measured at the third nour after operation.It was found that the stroke index was reduced, neurological deficits improved and brain norepinephrine(NE), dopamine(DA)and 5-hydroxytrytamine(5-HT) elevated to normal levels in pentobarbital-treated animals. There were significant negative correlations between stroke indexes and the levels of NE, DA and 5 -HT in ischemic brain (r = -0.485, -0.497 and 0.579 respectively, P<0.01 in all).The results suggest that the protective effect of pentobarbital on cerebral ischemia seems to be correlated to its action of improving disorders of NE, DA and 5 -HT in ischemic brain.

本文在60只蒙古沙土鼠研究了戊巴比妥对脑缺血的保护作用及对缺血性脑组织中单胺类神经介质含量的影响。动物随机分成三组:戊巴比妥组,对照组和模拟手术组各20只。戊巴比妥组和对照组分别于术前30分钟给予戊巴比妥钠60mg/kg体重和等容积的生理盐水1P,并作左侧颈总动脉结扎术。模拟手术组除不结扎左颈总动脉外,其它处理均同对照组。术后3小时检查动物的神经系统表现及测定脑组织中单胺类神经介质的含量。 发现戊巴比妥组动物卒中指数下降,神经系统症状明显改善,缺血性脑组织中去甲肾上腺素(NE),多巴胺(DA)和5-羟色胺升高至正常水平;动物的卒中指数与缺血性脑组织中NE、DA及5-HT的含量呈显著负相关(r分别=-0.485,-0.497和-0.579,P均<0.01)。动物的缺血症状越重,单胺类神经介质含量下降越明显。本文结果提示,戊巴比妥对脑缺血的保护作用可能与其改善缺血性脑组织中单胺类神经介质的代谢紊乱有关。

The purpose of this study was to observe the course and mode of collateral circulation between the transplanted omentum and the ischemic cerebral cortex, and to evaluate the effect of intracranial transplantation of omentum.Thirty rabbits were used in this study. At first, different ways to establish experimental models of ischemic brain were studied in rabbits. It revealed that Iectrocoagulation of the main branchs of MCA was a simple and reliable surgical procedure to produce complete ischemia. So we...

The purpose of this study was to observe the course and mode of collateral circulation between the transplanted omentum and the ischemic cerebral cortex, and to evaluate the effect of intracranial transplantation of omentum.Thirty rabbits were used in this study. At first, different ways to establish experimental models of ischemic brain were studied in rabbits. It revealed that Iectrocoagulation of the main branchs of MCA was a simple and reliable surgical procedure to produce complete ischemia. So we consided it a standard method obtain the model of cerebral ischemia.Following experiments were divided into group A and B. In group A, the time needed to form the collateral circulation between omentum and ischemic brain was observed. In group B, the mode of connection between omentum and cerebral cortex was studied. The experiments showed that three days after the transplanaton of the surface of ischemic brain, there existed confirmed vessel anastomosis between them. The omentum keeped connection with the brain through a thick fibrous layer. The omentum itself and the omental vessels also passed through the fibrous layer to connect with the brain directly.

实验目的是了解大网膜移植到缺血脑皮层后,侧支循环建立的时间,方式,从而估价网膜颅内移植的效果。实验采用家兔30只。首先探讨制备兔脑缺血的各种实验模型,发现电凝闭塞大脑中动脉分支操作方便,缺血效果可靠,确认为制备脑缺血的标准实验方法。 实验分甲,乙两组进行。甲组观察带蒂大网膜移植于缺血脑皮层后侧支循环建立的时间;乙组了解网膜和脑皮层之间的联接方式。结果证实,网膜移植到缺血兔脑皮层后3天,发生了肯定的血管吻合,网膜和脑皮层之间有一层增厚的纤维层相隔,网膜和脑组织不仅通过该纤维层发生联系,而且网膜组织本身和网膜内的血管可以穿过该纤维层和脑组织发生直接的连接。

The effect of the restoration of cerebral blood flow (CBF) on ischemic cerebral edema was studied in 73 mongolian gerbils. Following the right common carotid artyer was occluded, occurrence of infarction was confirmed by the visual estimation of neurological symptoms in 37 animals, which were subjected to the following studies. They were divided into three groups: In Group A (N=11), the occlusion was continued for 6 hours, and thereafter the animals were sacrificed. In Group B (N=13), the occlusion was continued...

The effect of the restoration of cerebral blood flow (CBF) on ischemic cerebral edema was studied in 73 mongolian gerbils. Following the right common carotid artyer was occluded, occurrence of infarction was confirmed by the visual estimation of neurological symptoms in 37 animals, which were subjected to the following studies. They were divided into three groups: In Group A (N=11), the occlusion was continued for 6 hours, and thereafter the animals were sacrificed. In Group B (N=13), the occlusion was continued for 9 hours, and the animals were sacrificed. In Group C (N=13), the occlusion was continued for 6 hours, and thereafter the animals were sacrificed 3 hours after the restoration of blood flow. In each group, the brain was removed at the end of experiment, and the cerebral water contetn of the right cerebral hemisphere was measured using the tissue-drying method. In Group C, Evans Blue dye was injected intravenouely 2 hours prior to the extirpation, and the abnormality of blood brain barrier (BBB) permeability was investigated.The normal cerebral water content obtained from 15 control animals was 78.4±0.3%. The cerebral water contents of the right cerebral hemisphere in Groups A and B were 81.4±0.5% and 81.54±0.6% respectively, which were significantly increased compared to the control (p<0.001). The cerebral water content in Group C was 83.4 ±1.3%, which was significantly increased compared to the control (P<0.001). These values were significantly greater than those obtained in Groups A and B (P<0.001). Group C showed extravasation of Evans Blue in the right cerebral hemisphere suggesting that the BBB was disrupted in this territory.The present study suggested that the restoration of CBF in the ischemic brain accelerated the cerebral edema. Probably, the restoration of CBF enhances BBB damage and leads to greater development of cerebral edema.

采用与人类有相似颅底动脉环发育的沙土鼠(Mongolian gerbil)103只,15只作为正常对照组。73只作了右侧颈总动脉结扎术,50.7%(37/73)形成同侧脑梗塞。分成6小时缺血组(N=11),9小时缺血组(N=13),再开通组(6小时缺血后,3小时血流再灌注,N=13)。用干燥重量法测定脑水分含量。另外15只中有脑梗塞的8只用伊凡斯蓝,作了血脑屏障观察。结果各组脑水分含量值以上述排列依次为;78.4±0.30%,81.4±0.50%,81.5±0.59%,83.4±1.30%。提示了两个缺血组和再升通组都产生了脑水肿(P<0.001),而且由于再 开通组已经存在血脑屏障损害,加上血流再灌注使得脑水肿比之两个缺血组的脑水肿更为严重(P<0.001)。

 
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