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pathological staining
相关语句
  病理染色
     Hp was determined by CLOtest combined with pathological staining/14C urea breath test.
     Hp阳性由CLOtest结合病理染色 / 14 C尿素呼吸试验而确定。
短句来源
     H·pylori was determined by CLO test combined with pathological staining/~(14)C urea breath test.
     H·pylori阳性由CLOtest结合病理染色/~(14)C尿素呼吸试验而确定。
短句来源
     Pathological staining and image analysis were used to determine the perimeter and the area of left ventricular cavity,and myocardial nuclei number and collagen content per unit area;
     病理染色图象分析测定左室腔周长和面积、单位面积内心肌细胞核数、单位面积内胶原的含量;
短句来源
     H. pylori was determined by rapid ureasetest combined with pathological staining/14C-urea breath test.
     Hpylori阳性由快速尿素酶试验结合病理染色/14C-尿素呼气试验确定。 结果:40例胃癌患者中,HPylori阳性23例,阴性17例。
短句来源
     Methods:Samples of cholecystolithiasis from 1995.10 to 1996.10 were studied. The routine pathological staining and argyrophilia staining were carried out.
     方法 :收集 1995年 10月至1996年 10月期间的胆囊结石标本做常规病理染色及嗜银蛋白染色分析。
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  “pathological staining”译为未确定词的双语例句
     6.84 106.12 + 23.5x 124.86 + 28.77 mm3 respectively(p<0.05). Pathological staining (TUNEL) and electric microscopy showed that there were significantly more intratumor apoptosis than control group.
     三组荷瘤在治疗25天时的瘤体积分ZlJ为62.17士6.84、一06.12士23.5、x24.s6士28.77mm3,三组相比有统计学差异。
短句来源
     Pathological staining showed that there were significantly more intratumor inflammatory infiltration and CD4~. CD8~ T lymphocytes cell infiltration than that of control group.
     肿瘤组织冻切片免疫组织化学显示Flt3L引起肿瘤局部炎细胞的显著浸润,CD_4~(+)、CD_8~(+)细胞明显多于对照组。
短句来源
     The chracteristies of fibrosis in 66 liver biopsies of aloholic liver disease (ALD) were analyzed by routine pathological staining and immunohistochemistry.
     本文观察了66例酒精性肝病(ALD)的肝纤维化特点。
短句来源
     Two hours after occlusion,infarct index,aspartate aminotransferase(AST),creatine kinase(CK),lactic dehydrogenase(LDH),a-Hy0roxybutyrate Dehydrogenase(HBDH),creatine kinae-MB(CK-MB)and pathological staining were examined to judge the protective effect of Guanxinshu.
     2 h后,通过心肌梗死指数、血清天冬氨酸转氨酶(AST)、肌酸磷酸激酶(CK)、乳酸脱氢酶(LDH)、a-羟丁酸脱氢酸酶(HBDH)、肌酸磷酸激酶同工酶(CK-MB)、病理学染色等指标,观察冠心舒对犬急性心肌缺血的保护作用。
短句来源
     [Methods] 120 patients whose endoscopic diagnoses were gastric and duodenal ulcer and who tested positive for H pylori by rapid unease test and pathological staining were randomized into two groups: Rabeprazole group and Omeprazole group.
     该项研究应用雷贝拉唑和阿莫西林二联疗法治疗Hp相关性溃疡,对溃疡愈合率、Hp清除率及症状缓解程度等进行比较分析,评估其疗效。 方法对120例经胃镜检查确诊为胃和十二指肠溃疡并经快速尿素酶法和病理学特染法测定为Hp阳性的病人,随机分为两组。
  相似匹配句对
     staining.
     染色和大体形态学评分。
     H E staining was used to analyze the pathological changes.
     H- E染色分析转基因小鼠肝组织的病理学变化。
短句来源
     The pathological changes were examined with Masson staining.
     Masson染色观察肾间质病变程度。
短句来源
     Masson staining;
     (2)Masson染色;
短句来源
     AND PATHOLOGICAL STUDY
     牙源性粘液瘤的临床、X线和病理学研究
短句来源
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The chracteristies of fibrosis in 66 liver biopsies of aloholic liver disease (ALD) were analyzed by routine pathological staining and immunohistochemistry. The results showed that aloholie hepatic fibrosis had three morphological types:1.Perisinosoidal fibro-sis (PSF) ;2.Terminal venous fibrosis (TVF) and 3.Portal and Periportal fibrosis. Proliferative collagen was mainly type I col-lagen in serious fibrosis.Close relationship between fibrosis and proliferation of Ito cells was demonstrated by the α-SMA...

The chracteristies of fibrosis in 66 liver biopsies of aloholic liver disease (ALD) were analyzed by routine pathological staining and immunohistochemistry. The results showed that aloholie hepatic fibrosis had three morphological types:1.Perisinosoidal fibro-sis (PSF) ;2.Terminal venous fibrosis (TVF) and 3.Portal and Periportal fibrosis. Proliferative collagen was mainly type I col-lagen in serious fibrosis.Close relationship between fibrosis and proliferation of Ito cells was demonstrated by the α-SMA staining. Image-analysis showed that the percentage of fibrous tissues was higher in ALD than that in chronic viral hepatitis at same stage of fibrosis.

本文观察了66例酒精性肝病(ALD)的肝纤维化特点。酒精性肝纤维化(AHF)主要有三种形成方式:①窦周纤维化,最具特征;②静脉周围纤维化;③汇管区及汇管区周围纤维化,后者包括汇管区胶原增多,扩大,汇管区周围星芒状纤维化及界板侵蚀。重度AHF时以Ⅰ型胶原沉积为主。AHF的形成与Ito细胞增殖密切相关。图像定量分析表明,酒精性肝病肝纤维化程度重于慢性病毒性肝炎者。

Objective:To enhance our understanding of the relation between cerebral ischemic rperfusion injury and heat shock protein70 (HSP70) expression. We evaluated the cellular expression of HSP70 in conjunction with the morphological analysis of rat brain. Methods: Twenty -four Sprague-Dawley (SD) rats were subjected to mild brain ischemia by infusion normal saline 30 minutes from the left common carotid artery. Twelve to them were cerebrel ischemia 30 minutes. The other twelve rats were studied after brain ischemia...

Objective:To enhance our understanding of the relation between cerebral ischemic rperfusion injury and heat shock protein70 (HSP70) expression. We evaluated the cellular expression of HSP70 in conjunction with the morphological analysis of rat brain. Methods: Twenty -four Sprague-Dawley (SD) rats were subjected to mild brain ischemia by infusion normal saline 30 minutes from the left common carotid artery. Twelve to them were cerebrel ischemia 30 minutes. The other twelve rats were studied after brain ischemia 30 minutes and reperfused for 30 minutes by collateral circulation. Brain sections were analyzed by immunohistochemical staining (using a monoclonal antibody to HSP70) and pathological staining (hematoxylin and eosin staining,HE). Results: We found that HSP70 was firstly expressed in parietal cortices when cerebral ischemia 30 minutes. The HSP70 expressed in neuron's membrane, axon and dendrite. Afer brain ischemia 30 minutes and reperfussion 30 minutes by collateral circulation, we can not observe HSP70 in neurons. These results are not the same as reported before. Conclusion: Focal cerebral ischemia could induce the HSP70 in injured cells. If circulation recovery, no HSP70 can be found.

目的:为了加深了解热休克蛋白70(HSP70)与脑缺血/再灌注损伤的关系,探讨HSP70的表达与细胞形态学变化。方法:24只SD大鼠通过左颈总动脉灌注生理盐水形成轻度左侧脑缺血。其中12只大鼠为单纯缺血组,另12只为缺血30分钟后再灌注30分钟组。两组切片分别进行免疫细胞化学和病理组织学观察。结果:我们发现脑缺血30分钟HSP70在顶叶皮层表达,分布在细胞膜,轴突和树突中。再灌注30分钟无HSP70表达。结论:局灶性脑缺血可诱导HSP70表达,恢复血循环无HSP70表达。

Amis: To investigate the expression of tumor suppression gene and regulating gene of apoptosis in gastric mucosal lesions with Helicobacter pylori(H·pylori)infection. Methods: 40 cases of gastric carcinoma patients underwent endoscopy and surgical resection. Samples were taken from tumor site, paracancerous adjacent tissues and paracancerous normal tissues and ABC immunohistochemical staining was used to detect the expression of p53、p16, bcl-2 proteins. H·pylori was determined by CLO test combined with pathological...

Amis: To investigate the expression of tumor suppression gene and regulating gene of apoptosis in gastric mucosal lesions with Helicobacter pylori(H·pylori)infection. Methods: 40 cases of gastric carcinoma patients underwent endoscopy and surgical resection. Samples were taken from tumor site, paracancerous adjacent tissues and paracancerous normal tissues and ABC immunohistochemical staining was used to detect the expression of p53、p16, bcl-2 proteins. H·pylori was determined by CLO test combined with pathological staining/~(14)C urea breath test. Results: No significant difference for the positive rate of p53 expression of gastric carcinoma between H·pylori positive and H·pylori negative(p>0.05). The positive rate and staining intensity of p16 expression of chronic gastritis or intestinal metaplasia with H·pylori positive were significantly lower than those with H·pylori negative (p<0.001, p<0.01), In contrast to p16, bcl-2 expression of intestinal metaplasia with H·pylori positive was significantly higher than that with H·pylori negative. (p<0.05). Conclusion: p16 or bcl-2 gene expression alteration might play a role in the early development/promotion of gastric carcinoma and is associated with H·pylori infection. It seems that H·pylori infection is not related to p53 gene alteration because p53 gene overexpression is relatively later event in stomach tumorigenesis.

目的:研究幽门螺杆菌(H·pylori)感染胃粘膜病变抑癌基因(p53、p16)和关键性凋亡调节基因bcl-2蛋白的表达,进一步探讨H·pylori在胃癌发生发展过程中作用的分子机制。方法:胃镜、外科手术中取40例胃癌患者的癌组织、癌旁组织(靠近癌的正常组织)各两块,石蜡包埋。切片HE染色作病理及免疫组织化学检查p53、p16、bcl-2蛋白表达。H·pylori阳性由CLOtest结合病理染色/~(14)C尿素呼吸试验而确定。结果:p53阳性表达率在H·pylori阳性及阴性胃癌组之间无显著性差别(p>0.05)。H·pylori阳性组慢性胃炎或肠化中p16阳性表达率及阳性表达强度均显著低于H·pylori阴性组(p<0.05,p<0.01)。而H·pylori阳性组肠化中bcl-2阳性表达率及阳性表达强度均显著高于H·pylori阴性组(pall<0.05)。结论:在胃癌发生的早期即存在较明显的p16基因表达低下与bcl-2基因过度表达,并与H·pylori感染有一定的关系。p53基因过度表达是胃癌发展过程中较晚期事件,与H·pylori感染无明显相关性。

 
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