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-triphosphate
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  5’-三磷酸
     The Process Research for Cytidine 5'-Triphosphate Separation and Purification by Ion Exchange Resin
     离子交换法分离纯化5’-三磷酸胞苷的研究
短句来源
  “5 '-triphosphate”译为未确定词的双语例句
     1 ̄HNMR STUDY OF INTRAMOLECULAR STACKING IN TERNARY PALLADIUM(Ⅱ) COMPLEXES INVOLVING ADENOSLNE 5'-TRIPHOSPHATE AND LIGANDS CONTAINING OXYGEN AND/OR NITROGEN AS DONOR ATOMS
     1~HNMR STUDY OF INTRAMOLECULAR STACKING IN TERNARY PALLADIUM(Ⅱ) COMPLEXES INVOLVING ADENOSLNE 5'-TRIPHOSPHATE AND LIGANDS CONTAINING OXYGEN AND/OR NITROGEN AS DONOR ATOMS
短句来源
     the stability constants of the ternary mixed-ligand complexes Pt (A)(ATP) n- (A=Phen,Bpy,Trp;ATP=adenosine5'-triphosphate;n=2or 3)have been determined by potentiometric pH titration in dilute aqueous solution (I=0.1mol/L,KNO 3 ;25℃).
     用pH电位法测定了Pt(Ⅱ)与腺苷5'-三磷酸(ATP)的三元混配配合物Pt(A)(ATP)n-(杂环碱A=PhenBpyTrpn=2或3)在水溶液中的稳定常数(I=0.1mol/L,KNO3;25℃)。
短句来源
     A STUDY ON THE COMPLEX BETWEEN AI (Ⅲ) AND ADENOSINE 5'-TRIPHOSPHATE BY ~(27)l1, ~1H AND ~(31)P NMR
     Al(Ⅲ)-ATP配合物的~(27)Al、~1H和~(31)P NMR研究
短句来源
     A repeat angiogram in the same projection was recorded 15 seconds after an intracoronary bolus of adenosine 5'-triphosphate (ATP) (the left coronary artery 50ug, the right coronary artery 20jig), then the number of cineframes was measured (hyperemic TFC, H-TFC).
     冠状动脉内注射三磷酸腺苷(ATP)(左冠状动脉50μg,右冠状动脉20μg)15秒后在相同角度重复造影,取得充血相TFC(H-TFC)。
短句来源
     Biosynthesis of Adenosine 5'-Triphosphate by Using Immobilized Beer Yeast Cells
     固定化啤酒酵母细胞合成5’-三磷酸腺苷
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  相似匹配句对
     (5) C
     5、儿茶酚胺作用于心肌细胞后,心肌肌球蛋白重链(MHC)基
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     5).
     5、行政案件不能独任审判。
短句来源
     Enzymatic synthesis of ~3H-thymidine-5-triphosphate
     ~3H-甲基-胸腺嘧啶核苷-5′-三磷酸的酶促合成
短句来源
     Study on Separation of Adenosine 5'-Triphosphate by Ion Exchange
     离子交换法分离纯化5'-三磷酸腺苷的工艺研究
短句来源
     Comparison of the determination methods of adenosine 5-triphosphate disodium
     三磷酸腺苷二钠含量测定方法的比较
短句来源
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  -triphosphate
Using several lipids immobilized on nitrocellulose membranes, it was shown that phosphatidylinositol-3,4,5-triphosphate is the specific ligand for the studied protein.
      
Divergence of de novo biosynthesis of inosine-5'-triphosphate
      
Mathematical modeling of kinetics of adenosine-5'-triphosphate hydrolysis catalyzed by the Zn2+ ion in the pH range 8.5-9.0
      
The kinetics of adenosine-5'-triphosphate (ATP) hydrolysis catalyzed by Zn2+ at pH 8.5-9.0 is analyzed by numerical simulation.
      
Mathematical Modeling of Kinetics of Adenosine 5"-Triphosphate Hydrolysis Catalyzed by the Zn2+ Ion in the pH Range 7.4-8.3
      
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As reported by Borst and Slater, dinitrophenol (DNP), the uncoupling agent, can under certain conditions cause an inhibition of the oxidation of succinate by intact mitochondria. We have studied this effect of DNP on succinate oxidation by respirationcontrolled rat-liver mitochondria with a vibrating platinum microelectrode and found that, in absence of added inorganic orthophosphate and adenosine diphosphate (ADP), the action of DNP is closely related to its concentrations. When DNP is added in low concentration...

As reported by Borst and Slater, dinitrophenol (DNP), the uncoupling agent, can under certain conditions cause an inhibition of the oxidation of succinate by intact mitochondria. We have studied this effect of DNP on succinate oxidation by respirationcontrolled rat-liver mitochondria with a vibrating platinum microelectrode and found that, in absence of added inorganic orthophosphate and adenosine diphosphate (ADP), the action of DNP is closely related to its concentrations. When DNP is added in low concentration (5-10μM), succinate oxidation is stimulated, the rate of respiration being increased to greater degrees with increasing concentrations of DNP up to 10μM, with no indication of any inhibition. When the DNP concentration is increased to above 30μM, however, stimulation lasts for only a short time, followed by a decrease in the rate of oxidation. The use of still higher concentrations of DNP results in increasingly lower degrees, and shorter periods, of stimulation, followed sooner by inhibition.In agreement with findings reported by other workers, a preincubation with 2mM amytal has been shown to prevent completely the inhibition of succinate oxidation by high concentrations of DNP. In addition, it has been discovered that even after the appearance of the inhibition, amytal is still effective in alleviating the DNP effect, causing a partial restoration of the respiratory rate.The addition of another uncoupling agent, arsenate, in concentrations of 2-10mM also stimulates succinate oxidation and 4mM arsenate is able to elicit a maximal rate of respiration. In contrast with DNP, no inhibition of respiration can.be observed even with the higher concentrations of arsenate used. Nevertheless, after preincubation with 2 mM amytal, the stimulation of respiration caused by arsenate is found to last for only two minutes, thereafter the rate of oxidation begins to decline and respiration gradually becomes inhibited. Adenosine triphosphate (ATP) is able to prevent the appearance of the inhibited phase of succinate oxidation in both cases, i.e. in either DNP or amytal-arsenate treatment, with however, slightly different efficiency. The addition of ATP enables the respiration in presence of high concentrations of DNP to proceed at a good steady rate, not more, however, than 2/3 of the maximal rate obtainable under optimal conditions, while the same amount of ADP is able to abolish completely the inhibiting effect caused by the amytal-arsenate treatment with the ensuing rate of respiration even higher than that elicited by arsenate alone.The characteristics of the inhibiting actions of these two agents on succinate oxidation and the effect of ATP on them have been discussed. The experimental evidence presented is believed to support the postulation that the oxidation of succinate in liver mitochondria requires an intervention of energy. The possibility that under certain experimental conditions, accumulation of oxaloacetate may play a part in affecting the rate of succinate oxidation is considered likely but of only minor importance.

(1)DNP对于琥珀酸氧化的影响随浓度不同而异,低浓度时,呼吸受激活,其程度随浓度升高而增強,不出現抑制現象。高浓度(30μM以上)DNP只引起短时間氧化激活,随即引起抑制,随浓度升高,抑制出現愈早,氧化激活愈小,預先加入Amytal足以防止上述抑制現象,在抑制出現后加入Amytal亦能使琥珀酸氧化部分恢复。(2)砷酸盐激活的琥珀酸氧化仅在有Amytal的条件下,才出現抑制現象。(3)ATP对上述两种情况引起的琥珀酸氧化抑制都具有一定的解除作用。(4)就实驗結果所做分析支持琥珀酸氧化需要能量激活的看法。

As a direct consequence of the interdigitating filament model of the myofibril and the corresponding theory of muscular contraction put forward by H. E. Huxley and A. F. Huxley, it is evident that the muscle would lose its ability to contract if its sarcomere had been stretched to a length greater than the sum of the lengths of primary and secondary filaments. For the honey bee striated muscle, the sum of the lengths of the two sets of filament would be about 5.4μ, on the basis of the interdigitating filament...

As a direct consequence of the interdigitating filament model of the myofibril and the corresponding theory of muscular contraction put forward by H. E. Huxley and A. F. Huxley, it is evident that the muscle would lose its ability to contract if its sarcomere had been stretched to a length greater than the sum of the lengths of primary and secondary filaments. For the honey bee striated muscle, the sum of the lengths of the two sets of filament would be about 5.4μ, on the basis of the interdigitating filament model. The glycerine extracted myofibril of the honey bee indirect flight muscle, however, still preserved its ability to contract under the action of adenosine triphosphate after the sarcomere length had been stretched to beyond 8μ. Furthermore, the microscopical structure changes of the myofibril caused by stretching the sarcomere to beyond 6μ were still reversible. These results seem to indicate that the fine structure of the myofibril of the honey bee striated muscle was probably different from that expressed by the interdigitating filament model.

本文报告我們观察拉长的蜜蜂間接飞翔肌肌原纤維收縮能力的結果。在肌小节长度超过8微米以后,三磷酸腺苷仍能引起肌原紆維的縮短,而且在肌小节拉长后出現的結构变化也是可逆的。这些結果不能在H.E.Huxley和A.F.Huxley提出的肌原紆維由两組蛋白細絲构成的模型和收縮的細絲滑行理論的基础上来解释。

This paper presents the results of experiments concerning the influence of salt concentration, adenosine triphosphate, pH value, alcohol and urea on the binding of Congo Red with the glycerinated rabbit psoas muscle and toad sartorious muscle fibers. Within a certain range of concentrations, salt promotes the binding of Congo Red with the muscle fibers and impedes the washing out of Congo Red from stained muscle fibers in solutions of similar electrolytic composition without Congo Red. Adenosine triphosphate...

This paper presents the results of experiments concerning the influence of salt concentration, adenosine triphosphate, pH value, alcohol and urea on the binding of Congo Red with the glycerinated rabbit psoas muscle and toad sartorious muscle fibers. Within a certain range of concentrations, salt promotes the binding of Congo Red with the muscle fibers and impedes the washing out of Congo Red from stained muscle fibers in solutions of similar electrolytic composition without Congo Red. Adenosine triphosphate has two distinct effects. When the salt concentration of the washing solution is low, its effect is similar to that of the other salts. If the salt concentration is already rather high, it exerts a promoting effect on the washing out process. An alkaline pH is unfavourable to the binding. Furthermore, the muscle fiber stained with alkaline Congo Red solution (pH 9—10) fails to show dichroic properties. Alcohol has a strong inhibitory effect on the binding. Urea can increase the speed of the washing out process. The nature of the binding between Congo Red and the contractile protein molecules is discussed and it is argued that both salt linkage and hydrogen bond formation may play a role.

本文报告一些外界条件对剛果紅与甘油抽提的骨骼肌肌纤維結合的影响。所用方法大体上可以分为两类:(1)将肌纤維束浸于不同的剛果紅溶液中,观察染色的速率和深度有何差別;(2)将已經染色的肌纤維束浸于其他成分和用来染色的剛果紅溶液相同,但是不含剛果紅的洗脫液中,过一定时間后改变洗脫液的成分,观察由此所引起的洗脫速率的变化。得到的結果主要如下: (一)在一定范圍內增加盐濃度能促进剛果紅和肌纤維的結合,也能减低剛果紅从已經染色的肌纤維洗脫的速率,甚至使已被洗脫的剛果紅又重新和肌纤維結合。 (二)在洗脫液不含盐或含盐較少的情况下,三磷酸腺苷的作用和其它盐类的相仿,可是在洗脫液盐濃度較高的情况下,它却能增加洗脫的速率。 (三)两类实驗結果都表示硷性溶液(pH 9—10)不利于剛果紅和肌纤維結合,而且在硷性剛果紅溶液中染色的肌纤維或已經染色的肌纤維用硷性溶液处理后都基本上不出現二向色性。 (四)甲醇和乙醇能可逆地阻止剛果紅和肌纤維結合。 (五)脲能增加剛果紅从已經染色的肌纤維中洗脫的速率。文中还討論了剛果紅分子和收縮蛋白分子結合方式的問題,认为盐鍵和氫鍵大概都在起作用,而且在肌纤維中排列有規則的一部分剛果紅分子可能是通过氫鍵和...

本文报告一些外界条件对剛果紅与甘油抽提的骨骼肌肌纤維結合的影响。所用方法大体上可以分为两类:(1)将肌纤維束浸于不同的剛果紅溶液中,观察染色的速率和深度有何差別;(2)将已經染色的肌纤維束浸于其他成分和用来染色的剛果紅溶液相同,但是不含剛果紅的洗脫液中,过一定时間后改变洗脫液的成分,观察由此所引起的洗脫速率的变化。得到的結果主要如下: (一)在一定范圍內增加盐濃度能促进剛果紅和肌纤維的結合,也能减低剛果紅从已經染色的肌纤維洗脫的速率,甚至使已被洗脫的剛果紅又重新和肌纤維結合。 (二)在洗脫液不含盐或含盐較少的情况下,三磷酸腺苷的作用和其它盐类的相仿,可是在洗脫液盐濃度較高的情况下,它却能增加洗脫的速率。 (三)两类实驗結果都表示硷性溶液(pH 9—10)不利于剛果紅和肌纤維結合,而且在硷性剛果紅溶液中染色的肌纤維或已經染色的肌纤維用硷性溶液处理后都基本上不出現二向色性。 (四)甲醇和乙醇能可逆地阻止剛果紅和肌纤維結合。 (五)脲能增加剛果紅从已經染色的肌纤維中洗脫的速率。文中还討論了剛果紅分子和收縮蛋白分子結合方式的問題,认为盐鍵和氫鍵大概都在起作用,而且在肌纤維中排列有規則的一部分剛果紅分子可能是通过氫鍵和收縮蛋白分子結合的。

 
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