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metabolites release
相关语句
  产物的释放
     This paper mainly introduces characteristics of immobilization culture, immobilization method, immobilization reactor and metabolites release.
     文章重点介绍了固定化培养的特点、固定化方法、固定化反应器和产物的释放四个方面的内容.
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  “metabolites release”译为未确定词的双语例句
     Conclusions:PLA 2 activation and its metabolites release appear to be strongly involved in the acute lung injury following in testinal IR injury.
     结论:PLA2激活及其代谢产物的释放在肠IR急性肺损伤中发挥重要作用。
短句来源
     The observations suggest that intestinal I/R injury may cause increase of plasma CGRP, PLA 2 activation and its metabolites release seem to have some influences on the plasma and pulmonary CGRP levels.
     结果表明,肠缺血/再灌注(I/R)诱导血浆中CGRP增加,磷脂酶A2激活及其产物可能对肺和循环中CGRP的水平有明显的影响。
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  相似匹配句对
     INDUCING RELEASE OF METABOLITES IN PLANT CELL
     植物细胞代谢产物的诱导释放
短句来源
     Pressure pulse can enhance the release of intracelluler metabolites.
     压力脉冲是促进胞内生物碱释放的有效手段;
短句来源
     The release of Windows.
     Windows.
短句来源
     Intelligent Release
     智能脱扣器
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     Pharmacokinetics of amitriptyJine and its metabolites
     阿米替林及其代谢产物的药物动力学
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[3H]arachidonic acid (AA)prelabeled mouse peritoneal macrophageswere stimulated by zymosan to release [3H]AA metabolites.The major labeled products which were co-chromatographed with the authentic PG and LT standareds by TLC and determined by liquid scintillation were LTC4,LTB4,TXB2,6-keto-PGF1α and PGE2 Aniso-daminc (Ani)significantly inhibited the zymosan-induced release of PG and LT from mouse,macrophages in a dose-dependent manner.Both the total 3H and each of AA metabolites released...

[3H]arachidonic acid (AA)prelabeled mouse peritoneal macrophageswere stimulated by zymosan to release [3H]AA metabolites.The major labeled products which were co-chromatographed with the authentic PG and LT standareds by TLC and determined by liquid scintillation were LTC4,LTB4,TXB2,6-keto-PGF1α and PGE2 Aniso-daminc (Ani)significantly inhibited the zymosan-induced release of PG and LT from mouse,macrophages in a dose-dependent manner.Both the total 3H and each of AA metabolites released in media reduced in the presence of Ani.At 0.5 mmol / L of Ani,the zymosan-induced release of total 3H was reduced by 46%(2 h),45%(5 h),and 34%(15 h),respectively.The results of thepresent study further confirmed the hypothesis that Ani decreased the formation of PG and LT via the inhibiting release of AA from the cell membrane.

酵母多糖刺激[~3H]AA预标的小鼠腹腔巨噬细胞可释放[~3H]AA代谢物LTC_4,LTB_4,TXB_2,6-keto-PGF_(1α)及PGE_2.山莨菪碱可显著抑制上述刺激释放作用,使总~3H释放量及各[~3H]AA代谢物释放量均显著减少.这种抑制作用随山莨菪碱剂量增加而增强.当其浓度为0.5 mmol/L时.总~3H释放量下降46%(2 h),45%(5 h)及34%(15 h).本实验结果进一步证实山莨菪碱是通过抑制AA释放而减少PG及LT的形成.

In April 1995, a study on the effect of metabolites released from Pearloyster Pinctada martensi (collected from the experimental site) on the growth of redalga Kopaphycus alvarezii (collected from Lingshui County, Hainan lsland) wasconducted at the Hainan Tropic Marine Biology Experimental Station, ChineseAcademy of Sciences. Laboratory study and in situ experiments were carried out. Inthe laboratory experiment pearl oysters were placed for 6 hours in sterilized seawaterin a glass container and the concentrations...

In April 1995, a study on the effect of metabolites released from Pearloyster Pinctada martensi (collected from the experimental site) on the growth of redalga Kopaphycus alvarezii (collected from Lingshui County, Hainan lsland) wasconducted at the Hainan Tropic Marine Biology Experimental Station, ChineseAcademy of Sciences. Laboratory study and in situ experiments were carried out. Inthe laboratory experiment pearl oysters were placed for 6 hours in sterilized seawaterin a glass container and the concentrations of NH4-N, NO3 -N and NO2-N wereanalysed after the removal of the Pearl oysters. The alga was then placed for 1h inthe Pearl oyster treated seawater. After removal of the alga the concentration of thethree nitrides were analyzed again. The alga was placed in cages suspended under afloating raft in the open sea and increase in fresh weight was measured. Theexperiments were repeated on day 1, 4, 7, 10 and 13. The experimental resultsshowed that after one day in the Pearl oyster treated seawater, NH4-N was the maincomponent (0.244mmol / L), NO3-N the secondary (0.188 mm0l / L) and NO2-N thesmallest (8.8 x 10 ) mmol / L), and that their absorption by alga were in the order26; 15; 1. The growth rate of test alga was much higher than that of the control. Inthe simulation expenment. because the added amount of the three nitrides were equalto the amounts naturally released by pearI oysters, their growth rate approached that ofalga in the experimental group. These resultS showed that the three nitrides releasedby Pearl oyster were the main cause of the high growth rate of the alga. K.alvareziican therefore be used as a nitrogenous waste remover in peari oyster farm to improvethe seawatCr quality and pearl oyster production. Since the number of sites favorableto aquacultUre are limited, it is advantageous to find ways of increasing producivitywithin existing sites. The solution to conflicting demands between development ofhighdensity mariculture and protection of a marine environment may rely on cultureand the interaction between primary producers and consumers.

于1995年4月间在海南省三亚市、陵水县分别采集合浦珠母贝和异枝麒麟菜并进行混养实验,测定贝释放的三氮(NH4-N,NO3-N,NO2-N)以及藻类对贝释放的三氮的吸收及其生长,以期探讨合浦珠母贝代谢产物促进异枝麒麟菜生长的原因。结果表明,贝释放物中以NH4-N为主,NO3-N次之,NO2-N含量较少;藻对三氮的吸收亦依此顺序递减。实验组藻体的生长速率明显高于对照组的,在模拟实验组中,通过加入无机氮使水体中NH4—N,NO3-N,NO2-N浓度与实验组贝释放后的浓度相同,藻体生点速率接近实验组的。本结果直接证明了贝释放的三氮是促进藻体快速生长的主要原因。另一方面,藻类吸收了贝类代谢产物,也优化了养殖区的生态环境。

By radioimmunoassay, it was discovered that intestinal ischemia reperfusion caused a 3.5 fold increase of the CGRP in rat plasma, and a 2.2 fold elevation compared with sham group. In pulmonary lavage fluid, the level of CGRP was 39% heigher than that of sham. Intra peritoneal use of phospholipase A 2 or cyclooxgenase inhibitor (TRIF or INDO) caused an increase of CGRP in plasma but decrease in pulmonary lavage fluid. However, CGRP release from normal tissue was promoted by both above drugs in vitro experiment....

By radioimmunoassay, it was discovered that intestinal ischemia reperfusion caused a 3.5 fold increase of the CGRP in rat plasma, and a 2.2 fold elevation compared with sham group. In pulmonary lavage fluid, the level of CGRP was 39% heigher than that of sham. Intra peritoneal use of phospholipase A 2 or cyclooxgenase inhibitor (TRIF or INDO) caused an increase of CGRP in plasma but decrease in pulmonary lavage fluid. However, CGRP release from normal tissue was promoted by both above drugs in vitro experiment. The observations suggest that intestinal I/R injury may cause increase of plasma CGRP, PLA 2 activation and its metabolites release seem to have some influences on the plasma and pulmonary CGRP levels.

经RIA分析发现在大鼠肠缺血再灌注后血浆中降钙素基因相关肽(CGPR)较损伤前平均增加3.5倍,比假手术相应水平增加2.2倍,肺灌洗液中损伤组比假手术组又增加39%。磷脂酶A2阻断剂三氟拉嗪和环氧化酶抑制剂消炎痛预先腹腔治疗可增加CGRP在血浆中的浓度,而同时肺灌洗液中CGRP的含量减少,但体外局部用药二者可明显促进正常肺组织释放CGRP。结果表明,肠缺血/再灌注(I/R)诱导血浆中CGRP增加,磷脂酶A2激活及其产物可能对肺和循环中CGRP的水平有明显的影响。

 
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