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lower map
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  map较低
     2. The patients in the high effect group had a lower MAP and a better renal function compared to the other patients;
     2 .与其他组相比 ,显效组患者治疗前肾功能良好、MAP较低 ;
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  “lower map”译为未确定词的双语例句
     Hypotension was induced with 0.02% sodium nitroprusside to lower MAP to 50mmHg.
     控制性降压期间,0.02%硝 普钠溶液泵入,使MAP降至50mmHg维持30min。
短句来源
     Results The rats in HL-Group displayed significantly higher Tr (43.04±0.11 ℃), HR (660±42 beats/min), and RR (150±11/min) but lower MAP (49.0±3.5 mmHg) as compared with the C Group.
     结果(1)HL组动物肛温上升到(43.04±0.11)℃,心率加快到(660±42)次/min,呼吸频率加快到(150± 11)次/min,平均动脉压下降到(49.0±3.5)mmHg;
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     RESULTS:Untreated cirrhotic rats had significantly lower MAP, SVR and higher PP,CO,CI,SBF and nitrite concentration than those of the controls (all, P<0.01).
     结果 :未治疗组MAP、SVR显著低于正常对照组 ,而PP、CO、CI、SBF及亚硝酸盐含量则显著高于正常对照组 (P值均小于0 0 1)。
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     Results Patients' HR in the two groups changed slightly. Patients in group A had lower MAP,with a significant difference as compared with group B(P<0.05).
     结果两组心率变化不大,A组病人平均动脉压比B组低(P<0.05);
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     Furthermore, the histologic damages of lung were compared with those of sham operation group after stained by hematoxylin and eosin. Particularly, the correlation between the activity of PLA2 and lung coefficient, LPI, protein content in BALF, activity of NOS in lung tissue were analysed, respectively. Results Endotoxin can lower MAP and increase the levels of lung-body coefficient, LPI, protein content in BALF, activity of NOS and PLA2 in lung tissue, and mRNA expression of sPLA2-ⅡA.
     结果 内毒素可导致MAP下降,同时可引起肺体指数、BALF中蛋白含量、LPI、肺组织中NOS活力、PLA2的活性及sPLA2-ⅡA的基因表达水平等指标明显增高,从而导致肺组织病理改变,并且PLA2的活性与肺体指数、BALF中蛋白含量、LPI、肺组织中NOS活力呈显著的正相关。
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     A and MAP.
     MAP.
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     MAP was significantly lower in Group C than that in Group B( P <0.05).
     C组MAP(注药后 1~ 3min)虽有下降趋势 ,但与B组比较无显著性差异 (P >0 .0 5 )。
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     VENUE MAP
     会议地图
     The biomass was lower.
     生物量低。
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     ( b ) lower cost;
     (b)成本低;
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  lower map
The ratio of radial to femoral pressure correlated with MAP (i.e., lower MAP produced greater differences), and the ratio of systolic radial to systolic femoral pressure inversely correlated with systemic vascular resistance index.
      
Conclusion: We concluded that the induction of general anesthesia caused more hemodilution, volume expansion and volume expansion efficiency than epidural anesthesia, which was triggered only by the lower MAP.
      
The lower map shows the positions of the genetic markers used.
      
The lower map shows the suitability of each cell in 2D geographic space for one of the species as a function of the three factors.
      
The lower map is an expanded view of the sequenced region, showing the boundaries of the plasmid subclones derived from T>amp;y51 and pA36.
      
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The effect of high-frequency oscillation ( HFO ) and conventional mechanical ventilation on oxygenation and hemodynamics at the same mean airway pressure ( MAP )was comparatively observed in normal lung and the" injured lung induced by aleic acid in dogs. The results showed that the oxygenation effect, heart rate, mean femoral arterial pressure, mean pulmonary arterial pressure( P pa), and cardiac output(CO) dnrjng HFO were similar to those during the intermittent positive pressure ventilation. When MAP...

The effect of high-frequency oscillation ( HFO ) and conventional mechanical ventilation on oxygenation and hemodynamics at the same mean airway pressure ( MAP )was comparatively observed in normal lung and the" injured lung induced by aleic acid in dogs. The results showed that the oxygenation effect, heart rate, mean femoral arterial pressure, mean pulmonary arterial pressure( P pa), and cardiac output(CO) dnrjng HFO were similar to those during the intermittent positive pressure ventilation. When MAP was at 1.6 - 2.0 kPa in injured lung, there was no significant difference between HFO and positive end-expiratory pressure ventilaton, At lower MAP (0,8-1.2 kPa), however, the difference of other hemodynamic indices had no significance in statistics, except that the HFO in P pa was obviously higher than that of PEEP, we conclude that HFO in normal lung and injured lung all has good oxygenation effect, but no obvious deteriorated phenomenon in hemodynamics. In addition, oxygenation, P pa and CO during HFO are directly influenced by MAP as much as during PEEP.

在相同的平均气道压(MAP)下,对比观察了高频振荡通气(HFO)和常规通气对正常及油酸型肺损伤狗的氧合与血液动力学的影响。结果表明:正常肺在HFO时的氧合效果及心率、股动脉平均压、肺动脉平均压(Ppa)、心输出量(CO)均与间歇正压通气时相同。损伤肺在MAP为1.6~2.0kPa时,HFO与呼气末正压通气(PEEP)间差异无显著意义;MAP较低(0.8~1.2kPa)时,除HFO的Ppa较PEEP明显升高外,其它血液动力学指标差异无统计学意义。认为HFO对正常肺和损伤肺均有良好的氧合效果,而无明显的血液动力学恶化现象。另外,HFO与PEEP一样,氧合、Ppa与CO直接受MAP的影响。

The changes of heart rate(HR)and mean arterial pressure(MAP)during acute hypoxia Were studied in rabbits under the anesthetization and artificial respiration.The animals were divided into two groups:the control group with the intact vagus and the experimental group without the intact vagus.The HR in the con- trol group decteased remarkably at5,10,20and 30min of hypoxia(p<0.01),then reached to the control level at 20,30min after normoxia,the MAP reduced in hypoxia(p<0.01),but with a short recovering...

The changes of heart rate(HR)and mean arterial pressure(MAP)during acute hypoxia Were studied in rabbits under the anesthetization and artificial respiration.The animals were divided into two groups:the control group with the intact vagus and the experimental group without the intact vagus.The HR in the con- trol group decteased remarkably at5,10,20and 30min of hypoxia(p<0.01),then reached to the control level at 20,30min after normoxia,the MAP reduced in hypoxia(p<0.01),but with a short recovering pe- riod after normoxia.In addition,a significant position correlation between the slow HR and the lower MAP (r=0.8991,p<0.001)was found,In the experimental group,the HR changes were similar to the control group during and after hypoxia,however,the MAP was significant higher than the ontrol level at 5,10min and equal to the control level at 20,30min of hypoxia and stayed at the control valueafter normoxia.The mechahism of the changes were discussedin the paper.

本文在麻醉制动家兔上观察了吸入11%低氧混合气30min及恢复过程中平均动脉血压(MAP)、心率(HR)的变化。迷走神经完整组(N=11)在低氧期间HR明显减慢,停止低氧后20min才能恢复。MAP在低氧5min时已下降,10、20、30min均明显低于对照值,停低氧5min既可恢复。HR减慢与MAP下降呈高度正相关。切断迷走神经组(N=7)HR的变化同完整组,MAP在低氧5—10min时明显高于对照水平,20—30min恢复到对照水平,停止低氧后维持不变。文中对有关机制进行了讨论。

The hemodynamics of 49 scheduled patients during anesthesia and opration for mitral and aortic valve replacement were studied with the insertion of SwanGanz catheter and by the method of thermodilution technics.Pre-anesthesia CI and LVWI were in the nor mal range with abnormal MPAP, PCWP, PTRI and RVWI.Increase of RAP and decrease in MPA,PCWP, MAP, CI, LVWI and RVWI were observed during induction of anesthesia. The hemodynamic parameters returned to or exceded the pre-anesthesia values after trachcal...

The hemodynamics of 49 scheduled patients during anesthesia and opration for mitral and aortic valve replacement were studied with the insertion of SwanGanz catheter and by the method of thermodilution technics.Pre-anesthesia CI and LVWI were in the nor mal range with abnormal MPAP, PCWP, PTRI and RVWI.Increase of RAP and decrease in MPA,PCWP, MAP, CI, LVWI and RVWI were observed during induction of anesthesia. The hemodynamic parameters returned to or exceded the pre-anesthesia values after trachcal intubation and sternotomy. The CI, MAP, MPAP, PCWP, LVWI and RVWI decreased signifi- cantly after insertion of aorta and vena cava catheter. The reduction of MPAP, P(IWP,PTRI and RVWI post-cardiopulmonary bypass (CPB) were significant and persistent with no change of PVRI.The lower MAP,SVRI and deterioration of cardiac performance post-CPB indicate that the administeration of positive inotropir agents and restoration of hematocrit are nec essary as early as possible.

采用Swan-Ganz导管及热稀释原理测定心排血量方法,对49例择期行二尖瓣及主动脉瓣双瓣置换术麻醉期间的血流动力学进行了测定。结果表明:麻醉前心脏指数(CI)、左心功指数(LVWI)属正常范围,但平均肺动脉压(MPAP)、肺毛细血管嵌压(PCWP)、肺总阻力指数(PTRI)、右心功指数(RVWI)明显异常。诱导后右心前负荷(RAP)升高,右心后负荷(MPAP、PCWP)、平均动脉压(MAP)、(CI及左、右心作功降低,显示有心功能可能受抑;气管插管及锯胸骨后血流动力学各项指标均不同程度地回升并超过诱导前,提示心血管功能仍保持对应激的反应能力;主动脉及腔静脉插管时CI、MAP、MPAP、PCWP、LVWI及RVWI均显著降低,显然与此期间近心性、出血及搬动、挤压心脏、严重心律失常等干扰左、右心前后负荷的平衡有关;体外循环后,由于手术纠正了瓣膜病变所致的血流障碍,MPAP、PCPP、PTRI、及 RVWI显著持续下降,而反映肺血管阻力的PVRI无变化;心肺转流(CPB)结束后MAP、SVRI明显减低,心脏功能也受损,提示此期间应尽早应用心血管活性药并提高患者的血细胞比容。

 
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